ライフサイエンスコーパス: nasal mucosa

1 -mediated ion transport were measured in the nasal mucosa.

2 mRNA) and function was investigated in human nasal mucosa.

3 by ex vivo permeation studies across porcine nasal mucosa.

4 RPV1-SP nociceptive signaling pathway in the nasal mucosa.

5 and Gammapapillomavirus have tropism for the nasal mucosa.

6 derived from cytotoxic NK or T cells of the nasal mucosa.

7 that Solutol HS15 had no toxic effect on the nasal mucosa.

8 on, which contained PBS-diluted normal human nasal mucosa.

9 e in promoting S. aureus colonization of the nasal mucosa.

10 plasmacytoid dendritic cells (pDCs), to the nasal mucosa.

11 d may also involve chromatography across the nasal mucosa.

12 as well as eosinophilic inflammation in the nasal mucosa.

13 -1beta or tumor necrosis factor alpha in the nasal mucosa.

14 ls (DCs) and plasmacytoid DCs (pDCs), to the nasal mucosa.

15 d to delayed clearance of influenza from the nasal mucosa.

16 cific immune responses by vaccination at the nasal mucosa.

17 t infections, its main entry route being the nasal mucosa.

18 within the nasal-associated lymphoid tissue/nasal mucosa.

19 microenvironments have been described in the nasal mucosa, a site where the complex functions of olfa

20 )NP(366)-specific CD8 T cells persist in the nasal mucosa after primary influenza infection and predo

21 livers standardized allergens locally to the nasal mucosa allowing clinical symptoms and biospecimens

22 creases in pDC and mDC numbers at 8 h in the nasal mucosa and at 8-48 h in the skin.

23 fects of stimulation of the receptors in the nasal mucosa and carotid body chemoreceptors on vascular

24 ffects or evidence of absorption through the nasal mucosa and did not interfere with development of n

25 ninfected brain homogenate rapidly cross the nasal mucosa and enter the lumen of lymphatic vessels fo

26 mily V, receptor 1 (TRPV1) expression in the nasal mucosa and higher concentrations of substance P (S

27 R features an overexpression of TRPV1 in the nasal mucosa and increased SP levels in nasal secretions

28 The distribution of SP and TLRs in the nasal mucosa and local airway neurons was assessed with

29 The rate of viral clearance from nasal mucosa and lungs was not altered by removal of NAL

30 he severity of patients' symptoms, aspect of nasal mucosa and medical intake as parameters of CRS con

31 infection resulted in the degradation of the nasal mucosa and nasal-associated lymphoid tissue (NALT)

32 d with the induction of ICOS in cells of the nasal mucosa and on both CD4+Foxp3+ and CD4+Foxp3- T cel

33 disease characterized by inflammation of the nasal mucosa and paranasal sinuses.

34 ed the pathogen-specific IgA response in the nasal mucosa and simultaneously decreased inflammatory c

35 s, and rare epithelial tissues including the nasal mucosa and the ependyma/choroid plexus in the brai

36 2A13 cDNA was prepared by RNA-PCR from human nasal mucosa and was translated using a baculovirus expr

37 ing to M cells, passing between cells of the nasal mucosa, and within lymphatic vessels of the nasal

38 ribution of adaptive response induced in the nasal mucosa appears to be key factors in generating pro

39 nce or mediate clearance of S. aureus on the nasal mucosa are fundamentally undefined.

40 ver, increases in PR8-specific serum IgG and nasal mucosa-associated IgA were detected after removal

41 l of NALT did not elicit changes in serum or nasal mucosa-associated influenza-specific Ig levels.

42 terial spores, phagocytosis of spores in the nasal mucosa-associated lymphoid tissue (NALT) and lungs

43 from the cervical lymph nodes (CLN) and the nasal mucosa-associated lymphoid tissue (NALT) and teste

44 is infections have two portals of entry, the nasal mucosa-associated lymphoid tissue (NALT) and the l

45 ntly reported that GAS preferentially target nasal mucosa-associated lymphoid tissue (NALT) in mice,

46 ficking of iFT from the nasal passage to the nasal mucosa-associated lymphoid tissue (NALT).

47 llowing inoculation, but not in the adjacent nasal mucosa-associated lymphoid tissue (NALT).

48 Nasal mucosa-associated lymphoid tissue and submandibula

49 GFP was detected in the epithelial cells of nasal mucosa, bronchioles, and alveoli for up to 4 days

50 ophils in the superficial compartment of the nasal mucosa, but it had no effect on inflammation in th

51 ly into mice, rM51R-M virus was cleared from nasal mucosa by day 2 postinfection and was attenuated f

52 sal spray on the afferent innervation of the nasal mucosa by monitoring trigeminal nerve activity in

53 Stimulation of the receptors in the nasal mucosa caused reflex apnoea and vasoconstriction i

54 ether human CD49d(+) PMNs are present in the nasal mucosa during acute viral respiratory tract infect

55 ILC2s are recruited to the nasal mucosa during COX-1 inhibitor-induced reactions in

56 2 numbers change in peripheral blood and the nasal mucosa during COX-1 inhibitor-induced reactions in

57 marked influx of inflammatory cells into the nasal mucosa, eosinophils being the predominant cell typ

58 xia, stimulation of the sciatic nerve or the nasal mucosa evoked greater increases in SNA after expos

59 l application of short ragweed pollen to the nasal mucosa followed by a challenge of the ocular mucos

60 ar transport of inhaled pathogens across the nasal mucosa followed by entry into the lymphatic system

61 RNA is expressed at the highest level in the nasal mucosa, followed by the lung and the trachea.

62 immunoglobulin repertoires of blood and the nasal mucosa from aeroallergen-sensitized subjects befor

63 l-transduction molecule, gp130, in olfactory-nasal mucosa from control mice and from 3-day post-OBX m

64 aucity of PrP(Sc) deposition in the oral and nasal mucosa from LRS replication-deficient hosts follow

65 5 mum between a small number of cells of the nasal mucosa in >90% of animals from 5 to 60 min after i

66 n increase in ORN apoptosis or damage to the nasal mucosa in a host with a preexisting prion infectio

67 y, we investigated the role of damage to the nasal mucosa in the shedding of prions into nasal sample

68 is study characterizes S. aureus adhesion to nasal mucosa in vitro and investigates the interaction o

69 LRS in prion neuroinvasion from the oral and nasal mucosa in wild-type and immunodeficient mice and i

70 sa and that the density of this DC subset in nasal mucosa increased significantly after in vivo aller

71 Stimulation of the nasal mucosa increased splanchnic sympathetic nerve disc

72 , showing that the replication of VRP in the nasal mucosa induced the opening of the BBB, allowing pe

73 The oral-nasal mucosa is a primary site of GAS infection, and a m

74 indicate that acetaminophen toxicity in the nasal mucosa is not dependent on hepatic microsomal P450

75 The influx of DCs in the nasal mucosa is not transient, as even higher numbers of

76 vely high prevalence of these viruses in the nasal mucosa is unknown.

77 megalovirus [CMV]) virus inoculations of the nasal mucosa leading to olfactory bulb (OB) infection ac

78 a number of extrahepatic tissues, including nasal mucosa, lung, trachea, brain, mammary gland, prost

79 e is known about lymphocyte responses in the nasal mucosa, lymphocyte accumulation in the nasal mucos

80 nasal mucosa, lymphocyte accumulation in the nasal mucosa, nasal-associated lymphoid tissue (NALT), a

81 matory responses of MSCs isolated from human nasal mucosa (nmMSCs) upon challenge with different Toll

82 ntalized mucosal immune responses within the nasal mucosa of a vertebrate species, a strategy that li

83 ane patches decorate microcapillaries in the nasal mucosa of allergic rhinitis patients.

84 phils, mast cells and dendritic cells in the nasal mucosa of AR animals, compared with diluent treatm

85 tionally rescues the CF phenotype across the nasal mucosa of CF mice and in patient-derived organoids

86 In addition, expression in nasal mucosa of CF mice corrected the Cl- transport defe

87 DCs were also mobilized to the nasal mucosa of children with other viral respiratory in

88 2X agonists stimulate Cl(-) transport across nasal mucosa of cystic fibrosis (CF) patients as well as

89 The tonsils and nasal mucosa of each positive-control pig were swabbed a

90 port in human F508del CFTR lung cells and in nasal mucosa of F508del CF mice.

91 ansepithelial transport of prions across the nasal mucosa of hamsters, some of which occurs rapidly i

92 esults of these studies demonstrate that the nasal mucosa of mice and hamsters is not an absolute ana

93 mucin release and submucosal swelling in the nasal mucosa of mice that depends on cysLTs, as it is ab

94 y infecting olfactory sensory neurons in the nasal mucosa of mice.

95 or ethmoidal nerve (AEN) that innervates the nasal mucosa of muskrats (Ondatra zibethicus).

96 okines expression was amplified in bronchial/nasal mucosa of neutrophilic asthma prone to exacerbatio

97 ials (NMPs) were measured while exposing the nasal mucosa of patients with IR and HC subjects to aero

98 at there was inflammation in portions of the nasal mucosa of the colonized mice but not in the mucosa

99 T cell accumulation peaked in the nasal mucosa on day 7, but peaked slightly earlier in th

100 required that the DNA be administered to the nasal mucosa or ocular surfaces and was not evident afte

101 ed in nasal polyp tissue but not the healthy nasal mucosa or periphery.

102 f the active medication and antigen into the nasal mucosa or to a specific effect of the active medic

103 rosis (CF) patients as well as across non-CF nasal mucosa, P2XRs may provide novel targets for extrac

104 of infection subsequent to infection of the nasal mucosa, remains elusive.

105 Fibroblasts were obtained from nasal mucosa; samples of control subjects (NM-C, n = 8)

106 ion and subsequently given IL-10 DNA via the nasal mucosa, showed diminished Ag-induced delayed type

107 Most neurons (24 of 39) were activated by nasal mucosa stimulation (+65.8 % rise in discharge rate

108 tral C1 area) greatly reduced the effects of nasal mucosa stimulation on SND (-80 %).

109 However they did not reduce the effect of nasal mucosa stimulation on SND.

110 IL-13, and IFN-gamma mRNA expression in the nasal mucosa, suggesting a mixed Th1/Th2 immune response

111 In cells from nasal mucosa, the ratio of arachidonic to docosahexaenoi

112 s well as in other structures, including the nasal mucosa, the vomeronasal organ, the epithelium of t

113 eactivity is an increased sensitivity of the nasal mucosa to various nonspecific stimuli.

114 were prepared from eight different tissues (nasal mucosa, trachea, lung, colon, intestine, pancreas,

115 to test the hypothesis that prions cross the nasal mucosa via M cells.

116 dicate that prions can immediately cross the nasal mucosa via multiple routes and quickly enter lymph

117 Mean capillary density in the nasal mucosa was also approximately 5-fold higher in nas

118 Human nasal mucosa was cultured for 3 d with and without 1 mic

119 Nasal mucosa was immunostained for the co-expression of

120 t of influenza-specific CD4 T cells into the nasal mucosa was not altered by removal of NALT.

121 enge, the accumulation of CD8 T cells in the nasal mucosa was quicker, more intense, and predominantl

122 After the final challenge, the nasal mucosa was removed to produce conditioned medium,

123 ed nanoparticles enhance permeability across nasal mucosa, while retaining the effectiveness of the p

124 Colonization of human nasal mucosa with Staphylococcus aureus sets the stage f

125 H9N2:pH1N1 (P0) virus was restricted to the nasal mucosa, with no virus detected in the trachea or l

126 that CD14(+) monocytes were recruited to the nasal mucosa within hours after local allergen challenge