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1 gainst subsequent genital HPV infection (ie, natural immunity).
2 rm persistent antibody responses of acquired natural immunity.
3 ividuals from 18 countries that examined HPV natural immunity.
4 cation of robust correlates of protection in natural immunity.
5 ite of herpesvirus vulnerability targeted by natural immunity.
6 an disease and are moderated by pre-existing natural immunity.
7 us evolves rapidly to constantly escape from natural immunity.
8 ass the level of protection achieved through natural immunity.
9 he epidemic area caused residents to acquire natural immunity.
10 Complement is a component of natural immunity.
11 eisseria meningitidis is believed to lead to natural immunity.
14 responses, all of which are associated with natural immunity against Salmonella In vaccinated mice,
15 veloped countries, largely because decreased natural immunity allows for increased susceptibility.
16 lect a broader mechanism that modulates both natural immunity and autoimmunity to other glycotopes.
17 cation of robust correlates of protection in natural immunity and following vaccination against DENV.
18 oorly defined, impeding our understanding of natural immunity and hindering effective vaccine develop
20 r the viral transmissibility, rate of waning natural immunity and rates of progression and clearance
21 en HIF2alpha, the ITPR1-related pathway, and natural immunity and strongly suggest a role for the HIF
24 ion results in robust immunity comparable to natural immunity and vaccine-induced immunity and that t
26 ression allows little opportunity to develop natural immunity, and there is currently no effective an
28 sent a critical component of host defense in natural immunity but also suggest that mast cell functio
29 t the expression of such mast cell-dependent natural immunity can be significantly enhanced by long-t
30 explains the epidemiological observation of natural immunity conferred by carriage of N. lactamica.
31 of emulating the disease-reducing effect of natural immunity could achieve a detectable effect durin
32 bjects who control viral load as a result of natural immunity (elite controller [EC]) or with uninfec
33 ose observed in a population associated with natural immunity.IMPORTANCE The development of effective
35 e a novel immuno-oncology strategy employing natural immunity in the fight against cancers, in partic
36 that may explain the delayed development of natural immunity in the first few years of life and sugg
37 s of cps might be broadly useful to reawaken natural immunity in the highly immunosuppressive microen
39 tervention aimed at effectively boosting our natural immunity, in the form of a host defensive factor
41 l risk factor, and population differences in natural immunity need further investigation to understan
45 mmune responses prevented the acquisition of natural immunity similar to that concurrently acquired b
47 ction is mediated initially by components of natural immunity such as xenoreactive antibodies, comple
48 ization rates generate rapid accumulation of natural immunity that alters the indirect effects of vac
49 ry CD4 TRM cells in immunity to B. pertussis Natural immunity to B. pertussis induced by infection is
50 e safety of IVIg preparations in relation to natural immunity to bacteria and to extend our knowledge
53 we show that CD4(+) T cells are required for natural immunity to Ebola virus infection and that CD4-d
56 The data strongly suggest that the lack of natural immunity to hypervirulent GAS strains in humans
57 rgets of immune neutralization in vitro, and natural immunity to infection is associated with serovar
60 s that have occurred in adults suggests that natural immunity to more common strains does not always
61 er, these findings will provide insight into natural immunity to Mtb and will guide development of no
63 matosis, support the concept that there is a natural immunity to primary granule proteins which can b
67 has largely been achieved in settings where natural immunity to the pathogen results in clearance in
68 s C virus (HCV) infections demonstrated that natural immunity to the virus is induced during primary
72 be substantial effects on chronic pathology, natural immunity, vaccine development strategies, immune
74 he role of complement in mast cell-dependent natural immunity, we examined the responses of complemen
75 -induced immune responses in comparison with natural immunity, we used a panel of broadly neutralizin
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