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1 ntigens, forming ligands for cytotoxic T and natural killer cell receptors.
2 mmunity are Patr-B variants that engage with natural killer cell receptors.
6 noglobulins, T-cell receptors, interleukins, natural killer-cell receptors and pheromone receptors.
7 ions involving immune evasion included eight natural killer cell receptors, four CC chemokines, three
8 y49A, an inhibitory C-type lectin-like mouse natural killer cell receptor, functions through interact
10 ximately 150 microm), but only weakly to the natural killer cell receptor ligand HLA-E (K(d) >/= 1 mm
11 he possible basis for the specificity of the natural killer cell receptor Ly-49A for several related
12 antigens and reduced ligation of activating natural killer-cell receptors may explain the loss of gr
14 e 6q25.1, encoding activating ligands of the natural killer cell receptor NKG2D that have not previou
16 several encode unique or strong ligands for natural killer cell receptors, now represent more than h
17 te immune receptors on iIELs, such as NKG2D (natural killer-cell receptor), often bind to non-classic
19 ved a substantial number of cells expressing natural killer cell receptor protein 1 (NKR-P1), a marke
20 a significant degree of divergence from the natural killer cell receptors that comprise the natural
21 ynapse with MHC class I bound to its cognate natural killer cell receptor, whereas particles larger t
22 es that coexpress a semiinvariant T cell and natural killer cell receptors, which are particularly ab
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