ライフサイエンスコーパス: necrotic tissue

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1 n (the time points at which cells migrate to necrotic tissues).

2 ans of selectively responding to bacteria or necrotic tissue.

3 was the sole microbial isolate from debrided necrotic tissue.

4 localized to the microvessels bordering pan necrotic tissue.

5 agnosis of the PET-positive mass showed only necrotic tissue.

6 expressed ICAM-1 mRNA and extended into the necrotic tissue.

7 Histologically confirmed necrotic tissue (432 voxels) did not demonstrate any obs

8 Debridement (removal of necrotic tissue and foreign bodies from the wound) at di

9 ly significantly different from the ratio in necrotic tissue and from each other.

10 han healing infarcts, owing to resorption of necrotic tissue and maturation of scar, but infarct size

11 sequent time points revealed liquefaction of necrotic tissue and replacement with granulation tissue.

12 1 is released from activated immune cells or necrotic tissues and acts as a damage-associated molecul

13 IAIP and HMW-HA colocalized with histones in necrotic tissues and areas that displayed neutrophil ext

14 d in a number of tissues and is prevalent in necrotic tissues and granulomata.

15 ning myocytes are unable to reconstitute the necrotic tissue, and the post-infarcted heart deteriorat

16 we found that most bacteria were located in necrotic tissue as large colonies far (750 micro m) from

17 ere was one major complication: Intraluminal necrotic tissue attached to the bladder, which was remov

18 findings to determine the characteristics of necrotic tissue by postreperfusion MCE.

19 Surgery included debridement of necrotic tissue, carpal tunnel decompression, and extern

20 may be caused by the combined effect of the necrotic tissue (cecal ligation, [CL]) and other microbi

21 imal treatment relies on surgical removal of necrotic tissues covered by a split-skin graft.

22 ic cells specialize in cross-presentation of necrotic tissue-derived epitopes to directly activate cy

23 Necrotic tissue did not enhance.

24 sminogen deficiency impedes the clearance of necrotic tissue from a diseased hepatic microenvironment

25 is 100%, but reduced to 80% if targeting of necrotic tissue from previous transurethral resections o

26 e of this study was to assess the ability of necrotic tissue, in the presence or absence of low-dose

27 Defective clearance of necrotic tissue interferes with amelioration of tissue i

28 model of macrophage loss following uptake of necrotic tissue is proposed to explain macrophage deplet

29 races each individually specializing on the necrotic tissues of a different cactus species.

30 low activity on SPECT and the percentage of necrotic tissue on immunohistochemistry.

31 verdose induces massive hepatocyte necrosis, necrotic tissue releases high mobility group B1 (HMGB1)

32 Necrotic tissue releases high mobility group B1 (HMGB1),

33 stress gradient, but changed after bone and necrotic tissue resorption.

34 In infected and necrotic tissue, the Fc-binding proteins were removed fr

35 w muscle synthesis, the human heart replaces necrotic tissue with deposition of a noncontractile scar

36 Pretreatment of apoptotic and necrotic tissue with Klenow enzyme with or without added

37 otomy and resection of this area showed only necrotic tissue with no viable melanoma cells.

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