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1        There is evidence that MPO-ANCA cause necrotizing and crescentic glomerulonephritis (NCGN) and
2                                              Necrotizing and crescentic GN (NCGN) with a paucity of g
3 s discussed in thrombotic microangiopathies, necrotizing and crescentic GN, acute tubular necrosis, a
4                                              Necrotizing anterior scleritis was more commonly seen in
5 ght patients with active, noninfectious, non-necrotizing anterior scleritis with a scleral inflammato
6 ve participants with active, autoimmune, non-necrotizing anterior scleritis with scleral inflammatory
7 icacy of gevokizumab in the treatment of non-necrotizing anterior scleritis.
8                                              Necrotizing autoimmune myopathy (NAM) is characterized p
9  including rhabdomyolysis and statin-induced necrotizing autoimmune myopathy (SINAM), are rare.
10                                              Necrotizing autoimmune myopathy was idiopathic in half o
11   We systematically studied the incidence of necrotizing changes in adult patients with pneumococcal
12                                              Necrotizing changes in the lungs were seen in 6.6% of a
13   Goats developed various effects, including necrotizing colitis, pulmonary edema, and hydropericardi
14 is central to ANCA-associated vasculitis and necrotizing crescentic glomerulonephritis (NCGN).
15 gan, including the kidneys, where they cause necrotizing crescentic glomerulonephritis.
16 pothesis that endothelial NF-kappaB mediates necrotizing crescentic GN (NCGN) and provides a specific
17 ophil cytoplasmic antibody (ANCA)-associated necrotizing crescentic GN (NCGN) is incompletely underst
18 CA-activated phagocytes cause vasculitis and necrotizing crescentic GN (NCGN).
19 sgenic mice, evidenced by the development of necrotizing crescentic GN, albuminuria, renal impairment
20                        With the emergence of necrotizing/crescentic glomerulonephritis, approximately
21 tional immunizations were needed to induce a necrotizing/crescentic glomerulonephritis.
22 xperimental autoimmune glomerulonephritis or necrotizing/crescentic glomerulonephritis.
23 eukocidin (PVL) is most likely causative for necrotizing diseases, but the precise pathogenic mechani
24            Leigh syndrome (LS) is a subacute necrotizing encephalomyelopathy with gliosis in several
25  liver function and Leigh syndrome (subacute necrotizing encephalomyelopathy) seen in association wit
26 ific acute encephalopathy syndromes (4 acute necrotizing encephalopathy, 1 acute infantile encephalop
27 is required for CN3685 to cause haemorrhagic necrotizing enteritis, apparently because the Agr-like s
28 3 to 1.00; P=0.045) and an increased rate of necrotizing enterocolitis (10.4% vs. 8.0%; relative risk
29  (17.5% [95% CI, 16.5%-18.6%]), and death or necrotizing enterocolitis (19.3% [95% CI, 18.1%-20.4%]).
30 there were increases in deaths attributed to necrotizing enterocolitis (30 [95% CI, 27 to 34] vs. 23
31 urvivors (23.3%, 19.1%, and 11.7%), death or necrotizing enterocolitis (48.1%, 37.1%, and 32.5%), and
32 in mortality (AOR, 0.98; 95% CI, 0.70-1.37), necrotizing enterocolitis (AOR, 0.88; 95% CI, 0.65-1.20)
33      Mental development was most impaired by necrotizing enterocolitis (d = -0.40; P < .001) and meni
34       Motor development was most impaired by necrotizing enterocolitis (d = -0.66; P < .001).
35                                              Necrotizing enterocolitis (NEC) affects up to 10% of pre
36 LNT were shown to protect neonatal rats from necrotizing enterocolitis (NEC) and are good therapeutic
37 d blood cell (RBC) transfusion and anemia to necrotizing enterocolitis (NEC) are conflicting.
38                                              Necrotizing enterocolitis (NEC) continues to be a major
39                                              Necrotizing enterocolitis (NEC) develops in response to
40                                              Necrotizing enterocolitis (NEC) has long remained a sign
41 ant manifestation of severe diseases such as necrotizing enterocolitis (NEC) in neonates or bowel wal
42                                              Necrotizing enterocolitis (NEC) is a devastating disease
43                                              Necrotizing enterocolitis (NEC) is a devastating inflamm
44                                              Necrotizing enterocolitis (NEC) is a devastating intesti
45                                              Necrotizing enterocolitis (NEC) is a major cause of morb
46                                              Necrotizing enterocolitis (NEC) is a major cause of neon
47                                              Necrotizing enterocolitis (NEC) is a severe disease of t
48                                              Necrotizing enterocolitis (NEC) is a severe disease that
49                                              Necrotizing enterocolitis (NEC) is an inflammatory disea
50                                              Necrotizing enterocolitis (NEC) is one of the most serio
51                                              Necrotizing enterocolitis (NEC) is the leading cause of
52                                              Necrotizing enterocolitis (NEC) is the most common and s
53                                              Necrotizing enterocolitis (NEC) is the most common gastr
54 rentiation and is involved in development of necrotizing enterocolitis (NEC) of the immature intestin
55                       The pathophysiology of necrotizing enterocolitis (NEC) remains poorly understoo
56 r is the consequence of intestinal injury in necrotizing enterocolitis (NEC) remains unknown.
57 the proinflammatory cascade, is activated in necrotizing enterocolitis (NEC), a devastating condition
58 t of lung disease in the setting of neonatal necrotizing enterocolitis (NEC), a life-threatening gast
59 ure and role of the intestinal leukocytes in necrotizing enterocolitis (NEC), a severe disease affect
60 y result in antibiotic resistance, fungemia, necrotizing enterocolitis (NEC), and mortality.
61 y infections, otitis media, gastroenteritis, necrotizing enterocolitis (NEC), and sudden infant death
62  the widespread use of plain films to detect necrotizing enterocolitis (NEC), it is considered a time
63 reast milk (HBM) attenuates the incidence of necrotizing enterocolitis (NEC), which remains a leading
64 ession, which can lead to the development of necrotizing enterocolitis (NEC)--a devastating inflammat
65  possible pregnancy-related risk factors for necrotizing enterocolitis (NEC)-associated deaths during
66 diseases characterized by ISC loss including necrotizing enterocolitis (NEC).
67 is supposedly associated with development of necrotizing enterocolitis (NEC).
68 riate analysis, SL mortality predictors were necrotizing enterocolitis (NEC; surgical odds ratio, 5.9
69 pathy of prematurity (stage 3 or higher), or necrotizing enterocolitis (stages 2-3).
70 ve been implicated as a pathogenic factor in necrotizing enterocolitis and inflammatory bowel disease
71 premature newborns has been shown to prevent necrotizing enterocolitis and reduce all-cause mortality
72                                 Infants with necrotizing enterocolitis born into midlevel hospitals (
73 ptation to small-bowel resection (SBR) after necrotizing enterocolitis expands absorptive surface are
74  given enterally may reduce the incidence of necrotizing enterocolitis in preterm infants.
75  diabetes, nonalcoholic fatty liver disease, necrotizing enterocolitis in very low birth weight infan
76 levance was suggested, as TLR4 activation in necrotizing enterocolitis led to reduced proliferation a
77             Late-onset invasive infection or necrotizing enterocolitis occurred in 32% of infants (19
78                                              Necrotizing enterocolitis occurred less frequently in th
79                                              Necrotizing enterocolitis occurred more frequently in in
80  CI, 0.58-0.78) and the combined outcomes of necrotizing enterocolitis or death and severe intraventr
81 dio-pulmonary bypass, as well as in neonatal necrotizing enterocolitis or persistent ductus arteriosu
82 r retinopathy of prematurity and surgery for necrotizing enterocolitis or spontaneous intestinal perf
83                  Interestingly, we find that necrotizing enterocolitis patients also exhibit decrease
84                                 Infants with necrotizing enterocolitis represent a high-risk subgroup
85 ere intraventricular hemorrhage and death or necrotizing enterocolitis was lowest among infants born
86                            The occurrence of necrotizing enterocolitis was significantly higher in th
87 of the study, just 28.6% of the infants with necrotizing enterocolitis were born into high-level, hig
88 nary dysplasia, severe cerebral lesions, and necrotizing enterocolitis).
89 2, periventricular leukomalacia >grade 1, or necrotizing enterocolitis).
90 sed sample of 30 566 VLBW infants, 1879 with necrotizing enterocolitis, according to the level of car
91 dities before PDA closure, including sepsis, necrotizing enterocolitis, and a dependence on mechanica
92 aturity, severe intraventricular hemorrhage, necrotizing enterocolitis, and chronic lung disease amon
93 s, hearing loss, bronchopulmonary dysplasia, necrotizing enterocolitis, and severe retinopathy of pre
94 om BEC on premature infants with and without necrotizing enterocolitis, and successfully provided a t
95 iventricular or intraventricular hemorrhage, necrotizing enterocolitis, aspiration, retinopathy of pr
96 he following morbidities: late-onset sepsis, necrotizing enterocolitis, bronchopulmonary dysplasia, p
97                                              Necrotizing enterocolitis, defined as Bell stage 2 or gr
98 who survived more than 12 hours, were severe necrotizing enterocolitis, infection, bronchopulmonary d
99                               Infections and necrotizing enterocolitis, major causes of mortality and
100  hemorrhage, bronchopulmonary dysplasia, and necrotizing enterocolitis, most therapeutic approaches h
101 nography, severe retinopathy of prematurity, necrotizing enterocolitis, or late-onset sepsis) by 36 w
102 of serious infection (sepsis or meningitis), necrotizing enterocolitis, or mortality during the first
103 ystic periventricular leukomalacia, surgical necrotizing enterocolitis, or stage 3 or greater retinop
104 ites of death or bronchopulmonary dysplasia, necrotizing enterocolitis, retinopathy of prematurity, a
105          It also decreases the prevalence of necrotizing enterocolitis, sepsis, and intraventricular
106  PN significantly affects risk of mortality, necrotizing enterocolitis, sepsis, chronic lung disease,
107 onary hemorrhage but not with differences in necrotizing enterocolitis, severe bronchopulmonary dyspl
108                   No differences in rates of necrotizing enterocolitis, severe bronchopulmonary dyspl
109 etinopathy of prematurity requiring surgery, necrotizing enterocolitis, spontaneous intestinal perfor
110             The frequencies of air leaks and necrotizing enterocolitis, the duration of respiratory s
111                            Chorioamnionitis, necrotizing enterocolitis, white matter injury on crania
112 ntral nervous system injury decreased, while necrotizing enterocolitis-related deaths increased.
113 rain was common to all infants who developed necrotizing enterocolitis.
114 on, a process that may lead to diseases like necrotizing enterocolitis.
115 am, urinary tract, or cerebrospinal fluid or necrotizing enterocolitis.
116 ch were profoundly depleted in newborns with necrotizing enterocolitis.
117 ther this practice reduces the prevalence of necrotizing enterocolitis.
118 so a trend towards a diminished incidence of necrotizing enterocolitis.
119 en microbes and host immune elements such as necrotizing enterocolitis.
120 n of ISCs contributes to the pathogenesis of necrotizing enterocolitis.
121 ncovered, such as the transfusion-associated necrotizing enterocolitis.
122 d incidence of neonatal bacterial sepsis and necrotizing enterocolitis.
123 s such as intestinal atresias, volvulus, and necrotizing enterocolitis.
124 h postnatal intestinal pathologies including necrotizing enterocolitis.
125  achieve the rate from the best quartile for necrotizing enterocolitis.
126 re intraventricular hemorrhage, and death or necrotizing enterocolitis.
127 ities in infants who did and did not develop necrotizing enterocolitis.
128 -specific antigen level developed postbiopsy necrotizing epididymo-orchitis (requiring orchiectomy) a
129                                    Cytotoxic necrotizing factor 1 (CNF1) and hemolysin (Hly) are toxi
130                                    Cytotoxic necrotizing factor 1 (CNF1) and hemolysin (HlyA1) are to
131  secrete the Rho protein activator cytotoxic necrotizing factor-Y (CNF-Y), but it has been unclear ho
132 ore chronic comorbidities than patients with necrotizing fasciitis (20 [87.0%] vs 17 [54.8%]; P = .02
133 or patients with cellulitis vs patients with necrotizing fasciitis (3 [2-5] vs 5 [3-11]; P = .01), wh
134  slightly younger and had a higher degree of necrotizing fasciitis (56% vs 14%).
135  known as strep throat) to severely invasive necrotizing fasciitis (also known as the flesh-eating sy
136 n = 23; mean [SD] age, 57.2 [17.7] years) or necrotizing fasciitis (n = 31; mean [SD] age, 54.3 [13.5
137                                              Necrotizing fasciitis (NF) caused by flesh-eating bacter
138                 Shock frequently complicates necrotizing fasciitis (NF) caused by group A Streptococc
139 erile site or from a wound in a patient with necrotizing fasciitis (NF) or streptococcal toxic shock
140 hould maintain a high clinical suspicion for necrotizing fasciitis and distinguish it from more commo
141 than the carriage strain in a mouse model of necrotizing fasciitis and had enhanced growth ex vivo in
142 mic invasive infections worldwide, including necrotizing fasciitis and toxic shock syndrome.
143 p throat") to invasive conditions, including necrotizing fasciitis and toxic shock syndrome.
144 itis and skin infections to life-threatening necrotizing fasciitis and toxic shock syndrome.
145 sociated with invasive infections, including necrotizing fasciitis and toxic shock syndrome.
146 itis and impetigo to severe outcomes such as necrotizing fasciitis and toxic shock syndrome.
147 treptococcal toxic shock syndrome (STSS) and necrotizing fasciitis are the 2 most severe invasive man
148 the reason for ICU admission in 23 patients, necrotizing fasciitis in 31 patients, and other diagnose
149 this report, we present a case of multifocal necrotizing fasciitis in a healthy adult patient, second
150             Current therapeutic regimens for necrotizing fasciitis include surgical debridement and t
151                                   Periocular necrotizing fasciitis is a rare but potentially devastat
152 terature pertinent to S. marcescens-mediated necrotizing fasciitis is also reviewed.
153                                              Necrotizing fasciitis is often confused for cellulitis a
154         As seen in these 5 cases, periocular necrotizing fasciitis may cause severe visual loss more
155                  We report the first case of necrotizing fasciitis of the chest wall due to infection
156 k thus aimed to address whether CLI improves necrotizing fasciitis outcome by modulating virulence fa
157              We report 5 cases of periocular necrotizing fasciitis resulting in severe vision loss, 3
158  a pathogen that causes both invasive (e.g., necrotizing fasciitis) and noninvasive (e.g., pharyngiti
159 GAS), the causative agent of pharyngitis and necrotizing fasciitis, secretes the potent cysteine prot
160 fection (streptococcal toxic shock syndrome, necrotizing fasciitis, septic shock, or GAS cellulitis w
161 h cellulitis and patients with patients with necrotizing fasciitis, Staphylococcus aureus (10 [43.5%]
162 itically ill on admission than patients with necrotizing fasciitis, they have more chronic comorbidit
163 istered as soon as possible to patients with necrotizing fasciitis, while our in vitro studies emphas
164 e-threatening invasive infections, including necrotizing fasciitis.
165 irulence in animal models of pharyngitis and necrotizing fasciitis.
166 red from nonhuman primates with experimental necrotizing fasciitis.
167 litis and to compare them with patients with necrotizing fasciitis.
168 od and reduced virulence in a mouse model of necrotizing fasciitis.
169 d we review literature on this rare cause of necrotizing fasciitis.
170 tions to life-threatening infections such as necrotizing fasciitis.
171  the virulence phenotype in a mouse model of necrotizing fasciitis.
172 n nonhuman primate models of pharyngitis and necrotizing fasciitis.
173 life-threatening invasive infections such as necrotizing fasciitis.
174 d to be less severely ill than patients with necrotizing fasciitis.
175 Streptococcus anginosus constellatus causing necrotizing fasciitis.
176 P53 is a primary isolate from a patient with necrotizing fasciitis.
177 nificantly less virulent in a mouse model of necrotizing fasciitis.
178 tia marcescens is an extremely rare cause of necrotizing fasciitis.
179 sed virulence in a nonhuman primate model of necrotizing fasciitis.
180  and increased virulence in a mouse model of necrotizing fasciitis.
181 uding streptococcal toxic shock syndrome and necrotizing fasciitis.
182  cause illnesses ranging from pharyngitis to necrotizing fasciitis.
183 rom a nonhuman primate experimental model of necrotizing fasciitis.
184  infections such as toxic shock syndrome and necrotizing fasciitis.
185 type virulence phenotype in a mouse model of necrotizing fasciitis.
186  inflammation in the central nervous system; necrotizing focal myelitis in the cervical spinal cord;
187 en in the pituitary can cause an aggressive (necrotizing) form of hypophysitis through type IV (T-cel
188                          Focal and segmental necrotizing glomerular lesions with crescents, mimicking
189 vasculitis that often manifests as focal and necrotizing glomerulonephritis and renal failure.
190 T-cell clone to Rag1(-/-) mice induced focal necrotizing glomerulonephritis when glomerular MPO depos
191 V)NC1] develop anti-GBM antibodies and focal necrotizing glomerulonephritis with crescent formation.
192                           Pauci-immune focal necrotizing GN (piFNGN) is usually associated with ANCAs
193   Using an established murine model of focal necrotizing GN mediated by autoimmunity to MPO (autoimmu
194 xic effect of NET-related histone release in necrotizing GN.
195 es to myeloperoxidase, and more severe focal necrotizing GN.
196 tion on chest imaging, and poorly formed non-necrotizing granulomas on pathology.
197 al mortality in conjunction with a fulminant necrotizing hepatitis.
198 inical course is marked by rapidly worsening necrotizing infection, leading to very poor outcomes des
199                   These findings explain why necrotizing infections mainly develop in serum-free spac
200 istopathological examination revealed severe necrotizing inflammation in various organs, most promine
201 nephritis, and/or severe systemic multiorgan necrotizing inflammation.
202 cases of chronic pancreatitis, a progressive necrotizing inflammatory disease that can result in panc
203         Over time, the neutrophil-rich acute necrotizing lesions cause the accumulation of more lymph
204                                      Hypoxic necrotizing lesions rendered moxifloxacin less active.
205 arterial smooth muscle cells and deposits in necrotizing lesions.
206  microbial defence uniformly develop hypoxic necrotizing lung lesions, widely observed in human TB.
207           It has a lethal canine equivalent: necrotizing meningoencephalitis.
208 ith autoimmune myopathy have a predominantly necrotizing muscle biopsy with minimal lymphocytic infil
209 s the spectrum of diseases associated with a necrotizing muscle biopsy.
210                              Immune-mediated necrotizing myopathies (IMNM) may be associated with eit
211 antibodies found in patients with autoimmune necrotizing myopathies recognize signal recognition part
212 ated with dermatomyositis and the autoimmune necrotizing myopathies.
213 ents with dermatomyositis and the autoimmune necrotizing myopathies.
214                              Immune-mediated necrotizing myopathy (IMNM) is considered one of the idi
215 ients with statin-associated immune-mediated necrotizing myopathy and, less commonly, in statin-unexp
216                 In a subgroup, an autoimmune necrotizing myopathy develops that persists after discon
217                      One patient with severe necrotizing myopathy died.
218 t evidence now suggests that immune-mediated necrotizing myopathy is not one disease, but can be divi
219                A decade ago, immune-mediated necrotizing myopathy was recognized as a distinct form o
220 ients with dermatomyositis, polymyositis, or necrotizing myopathy, and 0/20 (0%) age-matched healthy
221 ntly identified statin-associated autoimmune-necrotizing myopathy.
222 d SLO toxins to virulence in mouse models of necrotizing myositis, bacteremia, and skin and soft tiss
223 ins are equally impaired in ability to cause necrotizing myositis.
224 ernatively, statins can induce an autoimmune necrotizing myositis.
225 sues became progressively more fibrinous and necrotizing over time.
226       At least 30% of patients with infected necrotizing pancreatitis are successfully treated with c
227         Between 1997 and 2013, patients with necrotizing pancreatitis at the Liverpool Pancreas Cente
228 n vivo in the course of taurocholate-induced necrotizing pancreatitis in rats and in vitro in rat pan
229 aurocholate into the pancreatic duct induced necrotizing pancreatitis in the head of pancreas and lig
230                  Most patients with infected necrotizing pancreatitis require necrosectomy.
231                                        Acute necrotizing pancreatitis was induced by i.p. administrat
232 gastrointestinal tract perforation, 21 acute necrotizing pancreatitis).
233 into interstitial edematous pancreatitis and necrotizing pancreatitis, (c) distinguish an early phase
234                    In patients with infected necrotizing pancreatitis, endoscopic necrosectomy reduce
235 lure; this makes the pancreas susceptible to necrotizing pancreatitis.
236 ea of intra-parenchymal necrosis, indicating necrotizing pancreatitis.
237 g catheter drainage for (suspected) infected necrotizing pancreatitis.
238 for success of catheter drainage in infected necrotizing pancreatitis.
239  2), multifocal lymphadenopathy (n = 2), and necrotizing pneumonia (n = 1).
240 of codY increased the virulence of USA300 in necrotizing pneumonia and skin infection.
241 s aureus (CA-MRSA) causes severe hemorrhagic necrotizing pneumonia associated with high mortality.
242 lveolar capillary destruction in hemorrhagic/necrotizing pneumonia caused by CA-MRSA and offer novel
243                        Staphylococcus aureus necrotizing pneumonia is a life-threatening disease that
244 ive sudden, massive loss of lung tissue from necrotizing pneumonia or acute respiratory distress synd
245                            A rabbit model of necrotizing pneumonia using MRSA clone USA300 was used t
246 with severe skin and soft tissue infections, necrotizing pneumonia, and eye infections.
247 th cystic fibrosis (CF) and can cause severe necrotizing pneumonia, which is often fatal.
248 USA300 is the predominant strain that causes necrotizing pneumonia.
249 ng inflammation and, in some cases, a lethal necrotizing pneumonia.
250  survival outcomes in a rabbit model of MRSA necrotizing pneumonia.
251 nfluenza virus and PVL act together to cause necrotizing pneumonia: an influenza infection activates
252 rgic bronchopulmonary aspergillosis, chronic necrotizing pulmonary aspergillosis, or invasive aspergi
253 ime of conversion and progressed to a severe necrotizing rejection early despite an unaltered baselin
254 inal detachment repair, endophthalmitis, and necrotizing retinitis were excluded, as were those with
255 a, brownish exudates in anterior chamber and necrotizing retinitis within hours despite immediate ini
256 irst IgM test had a greater risk of incident necrotizing retinochoroiditis (hazard ratio = 4.47, 95%
257                   The increased risk of late necrotizing retinochoroiditis associated with isolated f
258 al manifestation of T gondii infection, with necrotizing retinochoroiditis occurring months or years
259                   The incidence of recurrent necrotizing retinochoroiditis was 10.5/100 PY.
260 ar involvement at baseline, the incidence of necrotizing retinochoroiditis was 6.4/100 PY.
261                                     Incident necrotizing retinochoroiditis was more common among thos
262 ut clinically apparent retinal necrosis] and necrotizing retinochoroiditis) at initial examination (b
263 inal whitening), without clinically apparent necrotizing retinochoroiditis.
264 ith focal retinal whitening only and 13 with necrotizing retinochoroiditis.
265           We present a case of hepatosplenic necrotizing sarcoid granulomatosis, a variant form of "c
266                    In general, patients with necrotizing scleritis (n = 15) had a poorer outcome.
267 n vision in patients with scleritis included necrotizing scleritis (odds ratio [OR], 6.63; P<0.001),
268 otentially lethal (OR = 17.41, P=0.007), and necrotizing scleritis (OR = 4.73, P = 0.026).
269 (80.0%) occurred most often in patients with necrotizing scleritis (P<0.0001 for each).
270  ocular complications than episcleritis, and necrotizing scleritis is the type of scleritis most ofte
271                                Patients with necrotizing scleritis may respond to IMT, mainly alkylat
272 esent as highly invasive infections, such as necrotizing skin and soft tissue infections (NSSTI).
273 erans is known to cause Buruli ulcer (BU), a necrotizing skin disease leading to extensive cutaneous
274                       Buruli ulcer (BU) is a necrotizing skin disease most prevalent among West Afric
275  virulence contribution in a murine model of necrotizing skin infection is largely driven by its abil
276 lence factor that enables CA-MRSA to produce necrotizing skin infections by allowing the bacteria to
277 isystem autoimmune diseases characterized by necrotizing small- to medium-vessel vasculitis and the p
278 ococcus (GAS) patients, including sepsis and necrotizing soft tissue infections (NSTIs).
279  research on immunocompromised patients with necrotizing soft-tissue infection (NSTI).
280                                              Necrotizing soft-tissue infection-associated in-hospital
281 n as a potential therapeutic in SAg-mediated necrotizing soft-tissue infection.
282 GAS bacterial infection in a murine model of necrotizing soft-tissue infection.
283                                              Necrotizing soft-tissue infections (NSTI) have high morb
284   Increased awareness of fungi as a cause of necrotizing soft-tissue infections after a natural disas
285 to more severe forms of infection, including necrotizing stromal keratitis and herpes simplex encepha
286 d to control T. gondii infection and died of necrotizing TE before day 77.
287 ity and utility of this approach by rescuing necrotizing tissues and whole limbs using two murine mod
288 n the CNS as well as clear histopathology of necrotizing vasculitis and hemorrhage in the brain.
289  Histopathologic analysis of the skin showed necrotizing vasculitis in biopsy samples from 40 of 50 c
290 dy-associated (ANCA-associated) small vessel necrotizing vasculitis is caused by immune-mediated infl
291 ic infectious trigger of paralyzing systemic necrotizing vasculitis most severely affecting skeletal
292            Polyarteritis nodosa is a form of necrotizing vasculitis of small and medium-sized arterie
293 et vessel wall, which may participate in the necrotizing vasculitis of the kidney during this disease
294 atosis with polyangiitis (GPA) is a systemic necrotizing vasculitis that is associated with granuloma
295           Polyarteritis nodosa is a systemic necrotizing vasculitis with a pathogenesis that is poorl
296 Histology of EGPA shows tissue eosinophilia, necrotizing vasculitis, and eosinophil-rich granulomatou
297 localized in typical PDNS lesions, including necrotizing vasculitis, glomerulonephritis, granulomatou
298 ctures and evidence of glandular atrophy and necrotizing vasculitis.
299  interact with the endothelium, resulting in necrotizing vasculitis.
300 vels associated with development of systemic necrotizing vasculitis.

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