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3 s discussed in thrombotic microangiopathies, necrotizing and crescentic GN, acute tubular necrosis, a
5 ght patients with active, noninfectious, non-necrotizing anterior scleritis with a scleral inflammato
6 ve participants with active, autoimmune, non-necrotizing anterior scleritis with scleral inflammatory
11 We systematically studied the incidence of necrotizing changes in adult patients with pneumococcal
13 Goats developed various effects, including necrotizing colitis, pulmonary edema, and hydropericardi
16 pothesis that endothelial NF-kappaB mediates necrotizing crescentic GN (NCGN) and provides a specific
17 ophil cytoplasmic antibody (ANCA)-associated necrotizing crescentic GN (NCGN) is incompletely underst
19 sgenic mice, evidenced by the development of necrotizing crescentic GN, albuminuria, renal impairment
23 eukocidin (PVL) is most likely causative for necrotizing diseases, but the precise pathogenic mechani
25 liver function and Leigh syndrome (subacute necrotizing encephalomyelopathy) seen in association wit
26 ific acute encephalopathy syndromes (4 acute necrotizing encephalopathy, 1 acute infantile encephalop
27 is required for CN3685 to cause haemorrhagic necrotizing enteritis, apparently because the Agr-like s
28 3 to 1.00; P=0.045) and an increased rate of necrotizing enterocolitis (10.4% vs. 8.0%; relative risk
29 (17.5% [95% CI, 16.5%-18.6%]), and death or necrotizing enterocolitis (19.3% [95% CI, 18.1%-20.4%]).
30 there were increases in deaths attributed to necrotizing enterocolitis (30 [95% CI, 27 to 34] vs. 23
31 urvivors (23.3%, 19.1%, and 11.7%), death or necrotizing enterocolitis (48.1%, 37.1%, and 32.5%), and
32 in mortality (AOR, 0.98; 95% CI, 0.70-1.37), necrotizing enterocolitis (AOR, 0.88; 95% CI, 0.65-1.20)
36 LNT were shown to protect neonatal rats from necrotizing enterocolitis (NEC) and are good therapeutic
41 ant manifestation of severe diseases such as necrotizing enterocolitis (NEC) in neonates or bowel wal
54 rentiation and is involved in development of necrotizing enterocolitis (NEC) of the immature intestin
57 the proinflammatory cascade, is activated in necrotizing enterocolitis (NEC), a devastating condition
58 t of lung disease in the setting of neonatal necrotizing enterocolitis (NEC), a life-threatening gast
59 ure and role of the intestinal leukocytes in necrotizing enterocolitis (NEC), a severe disease affect
61 y infections, otitis media, gastroenteritis, necrotizing enterocolitis (NEC), and sudden infant death
62 the widespread use of plain films to detect necrotizing enterocolitis (NEC), it is considered a time
63 reast milk (HBM) attenuates the incidence of necrotizing enterocolitis (NEC), which remains a leading
64 ession, which can lead to the development of necrotizing enterocolitis (NEC)--a devastating inflammat
65 possible pregnancy-related risk factors for necrotizing enterocolitis (NEC)-associated deaths during
68 riate analysis, SL mortality predictors were necrotizing enterocolitis (NEC; surgical odds ratio, 5.9
70 ve been implicated as a pathogenic factor in necrotizing enterocolitis and inflammatory bowel disease
71 premature newborns has been shown to prevent necrotizing enterocolitis and reduce all-cause mortality
73 ptation to small-bowel resection (SBR) after necrotizing enterocolitis expands absorptive surface are
75 diabetes, nonalcoholic fatty liver disease, necrotizing enterocolitis in very low birth weight infan
76 levance was suggested, as TLR4 activation in necrotizing enterocolitis led to reduced proliferation a
80 CI, 0.58-0.78) and the combined outcomes of necrotizing enterocolitis or death and severe intraventr
81 dio-pulmonary bypass, as well as in neonatal necrotizing enterocolitis or persistent ductus arteriosu
82 r retinopathy of prematurity and surgery for necrotizing enterocolitis or spontaneous intestinal perf
85 ere intraventricular hemorrhage and death or necrotizing enterocolitis was lowest among infants born
87 of the study, just 28.6% of the infants with necrotizing enterocolitis were born into high-level, hig
90 sed sample of 30 566 VLBW infants, 1879 with necrotizing enterocolitis, according to the level of car
91 dities before PDA closure, including sepsis, necrotizing enterocolitis, and a dependence on mechanica
92 aturity, severe intraventricular hemorrhage, necrotizing enterocolitis, and chronic lung disease amon
93 s, hearing loss, bronchopulmonary dysplasia, necrotizing enterocolitis, and severe retinopathy of pre
94 om BEC on premature infants with and without necrotizing enterocolitis, and successfully provided a t
95 iventricular or intraventricular hemorrhage, necrotizing enterocolitis, aspiration, retinopathy of pr
96 he following morbidities: late-onset sepsis, necrotizing enterocolitis, bronchopulmonary dysplasia, p
98 who survived more than 12 hours, were severe necrotizing enterocolitis, infection, bronchopulmonary d
100 hemorrhage, bronchopulmonary dysplasia, and necrotizing enterocolitis, most therapeutic approaches h
101 nography, severe retinopathy of prematurity, necrotizing enterocolitis, or late-onset sepsis) by 36 w
102 of serious infection (sepsis or meningitis), necrotizing enterocolitis, or mortality during the first
103 ystic periventricular leukomalacia, surgical necrotizing enterocolitis, or stage 3 or greater retinop
104 ites of death or bronchopulmonary dysplasia, necrotizing enterocolitis, retinopathy of prematurity, a
106 PN significantly affects risk of mortality, necrotizing enterocolitis, sepsis, chronic lung disease,
107 onary hemorrhage but not with differences in necrotizing enterocolitis, severe bronchopulmonary dyspl
109 etinopathy of prematurity requiring surgery, necrotizing enterocolitis, spontaneous intestinal perfor
112 ntral nervous system injury decreased, while necrotizing enterocolitis-related deaths increased.
128 -specific antigen level developed postbiopsy necrotizing epididymo-orchitis (requiring orchiectomy) a
131 secrete the Rho protein activator cytotoxic necrotizing factor-Y (CNF-Y), but it has been unclear ho
132 ore chronic comorbidities than patients with necrotizing fasciitis (20 [87.0%] vs 17 [54.8%]; P = .02
133 or patients with cellulitis vs patients with necrotizing fasciitis (3 [2-5] vs 5 [3-11]; P = .01), wh
135 known as strep throat) to severely invasive necrotizing fasciitis (also known as the flesh-eating sy
136 n = 23; mean [SD] age, 57.2 [17.7] years) or necrotizing fasciitis (n = 31; mean [SD] age, 54.3 [13.5
139 erile site or from a wound in a patient with necrotizing fasciitis (NF) or streptococcal toxic shock
140 hould maintain a high clinical suspicion for necrotizing fasciitis and distinguish it from more commo
141 than the carriage strain in a mouse model of necrotizing fasciitis and had enhanced growth ex vivo in
147 treptococcal toxic shock syndrome (STSS) and necrotizing fasciitis are the 2 most severe invasive man
148 the reason for ICU admission in 23 patients, necrotizing fasciitis in 31 patients, and other diagnose
149 this report, we present a case of multifocal necrotizing fasciitis in a healthy adult patient, second
156 k thus aimed to address whether CLI improves necrotizing fasciitis outcome by modulating virulence fa
158 a pathogen that causes both invasive (e.g., necrotizing fasciitis) and noninvasive (e.g., pharyngiti
159 GAS), the causative agent of pharyngitis and necrotizing fasciitis, secretes the potent cysteine prot
160 fection (streptococcal toxic shock syndrome, necrotizing fasciitis, septic shock, or GAS cellulitis w
161 h cellulitis and patients with patients with necrotizing fasciitis, Staphylococcus aureus (10 [43.5%]
162 itically ill on admission than patients with necrotizing fasciitis, they have more chronic comorbidit
163 istered as soon as possible to patients with necrotizing fasciitis, while our in vitro studies emphas
186 inflammation in the central nervous system; necrotizing focal myelitis in the cervical spinal cord;
187 en in the pituitary can cause an aggressive (necrotizing) form of hypophysitis through type IV (T-cel
190 T-cell clone to Rag1(-/-) mice induced focal necrotizing glomerulonephritis when glomerular MPO depos
191 V)NC1] develop anti-GBM antibodies and focal necrotizing glomerulonephritis with crescent formation.
193 Using an established murine model of focal necrotizing GN mediated by autoimmunity to MPO (autoimmu
198 inical course is marked by rapidly worsening necrotizing infection, leading to very poor outcomes des
200 istopathological examination revealed severe necrotizing inflammation in various organs, most promine
202 cases of chronic pancreatitis, a progressive necrotizing inflammatory disease that can result in panc
206 microbial defence uniformly develop hypoxic necrotizing lung lesions, widely observed in human TB.
208 ith autoimmune myopathy have a predominantly necrotizing muscle biopsy with minimal lymphocytic infil
211 antibodies found in patients with autoimmune necrotizing myopathies recognize signal recognition part
215 ients with statin-associated immune-mediated necrotizing myopathy and, less commonly, in statin-unexp
218 t evidence now suggests that immune-mediated necrotizing myopathy is not one disease, but can be divi
220 ients with dermatomyositis, polymyositis, or necrotizing myopathy, and 0/20 (0%) age-matched healthy
222 d SLO toxins to virulence in mouse models of necrotizing myositis, bacteremia, and skin and soft tiss
228 n vivo in the course of taurocholate-induced necrotizing pancreatitis in rats and in vitro in rat pan
229 aurocholate into the pancreatic duct induced necrotizing pancreatitis in the head of pancreas and lig
233 into interstitial edematous pancreatitis and necrotizing pancreatitis, (c) distinguish an early phase
241 s aureus (CA-MRSA) causes severe hemorrhagic necrotizing pneumonia associated with high mortality.
242 lveolar capillary destruction in hemorrhagic/necrotizing pneumonia caused by CA-MRSA and offer novel
244 ive sudden, massive loss of lung tissue from necrotizing pneumonia or acute respiratory distress synd
251 nfluenza virus and PVL act together to cause necrotizing pneumonia: an influenza infection activates
252 rgic bronchopulmonary aspergillosis, chronic necrotizing pulmonary aspergillosis, or invasive aspergi
253 ime of conversion and progressed to a severe necrotizing rejection early despite an unaltered baselin
254 inal detachment repair, endophthalmitis, and necrotizing retinitis were excluded, as were those with
255 a, brownish exudates in anterior chamber and necrotizing retinitis within hours despite immediate ini
256 irst IgM test had a greater risk of incident necrotizing retinochoroiditis (hazard ratio = 4.47, 95%
258 al manifestation of T gondii infection, with necrotizing retinochoroiditis occurring months or years
262 ut clinically apparent retinal necrosis] and necrotizing retinochoroiditis) at initial examination (b
267 n vision in patients with scleritis included necrotizing scleritis (odds ratio [OR], 6.63; P<0.001),
270 ocular complications than episcleritis, and necrotizing scleritis is the type of scleritis most ofte
272 esent as highly invasive infections, such as necrotizing skin and soft tissue infections (NSSTI).
273 erans is known to cause Buruli ulcer (BU), a necrotizing skin disease leading to extensive cutaneous
275 virulence contribution in a murine model of necrotizing skin infection is largely driven by its abil
276 lence factor that enables CA-MRSA to produce necrotizing skin infections by allowing the bacteria to
277 isystem autoimmune diseases characterized by necrotizing small- to medium-vessel vasculitis and the p
284 Increased awareness of fungi as a cause of necrotizing soft-tissue infections after a natural disas
285 to more severe forms of infection, including necrotizing stromal keratitis and herpes simplex encepha
287 ity and utility of this approach by rescuing necrotizing tissues and whole limbs using two murine mod
288 n the CNS as well as clear histopathology of necrotizing vasculitis and hemorrhage in the brain.
289 Histopathologic analysis of the skin showed necrotizing vasculitis in biopsy samples from 40 of 50 c
290 dy-associated (ANCA-associated) small vessel necrotizing vasculitis is caused by immune-mediated infl
291 ic infectious trigger of paralyzing systemic necrotizing vasculitis most severely affecting skeletal
293 et vessel wall, which may participate in the necrotizing vasculitis of the kidney during this disease
294 atosis with polyangiitis (GPA) is a systemic necrotizing vasculitis that is associated with granuloma
296 Histology of EGPA shows tissue eosinophilia, necrotizing vasculitis, and eosinophil-rich granulomatou
297 localized in typical PDNS lesions, including necrotizing vasculitis, glomerulonephritis, granulomatou
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