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1 as reviewed for previous cases of C albicans necrotizing fasciitis.
2 aryngitis and invasive infections, including necrotizing fasciitis.
3 d in a murine model of group A streptococcal necrotizing fasciitis.
4 ith shock and organ failure, with or without necrotizing fasciitis.
5 lethal infection in a murine model of human necrotizing fasciitis.
6 and gas are valuable in assessing suspected necrotizing fasciitis.
7 sed virulence in a nonhuman primate model of necrotizing fasciitis.
8 and increased virulence in a mouse model of necrotizing fasciitis.
9 uding streptococcal toxic shock syndrome and necrotizing fasciitis.
10 cause illnesses ranging from pharyngitis to necrotizing fasciitis.
11 rom a nonhuman primate experimental model of necrotizing fasciitis.
12 infections such as toxic shock syndrome and necrotizing fasciitis.
13 type virulence phenotype in a mouse model of necrotizing fasciitis.
14 e-threatening invasive infections, including necrotizing fasciitis.
15 irulence in animal models of pharyngitis and necrotizing fasciitis.
16 red from nonhuman primates with experimental necrotizing fasciitis.
17 od and reduced virulence in a mouse model of necrotizing fasciitis.
18 d we review literature on this rare cause of necrotizing fasciitis.
19 tions to life-threatening infections such as necrotizing fasciitis.
20 n nonhuman primate models of pharyngitis and necrotizing fasciitis.
21 litis and to compare them with patients with necrotizing fasciitis.
22 Streptococcus anginosus constellatus causing necrotizing fasciitis.
23 P53 is a primary isolate from a patient with necrotizing fasciitis.
24 the virulence phenotype in a mouse model of necrotizing fasciitis.
25 nificantly less virulent in a mouse model of necrotizing fasciitis.
26 tia marcescens is an extremely rare cause of necrotizing fasciitis.
27 ignificant virulence factors in our model of necrotizing fasciitis.
28 ey pathogen-host interactions underlying GAS necrotizing fasciitis.
29 roat) to life-threatening infections such as necrotizing fasciitis.
30 bearing on the molecular pathogenesis of GAS necrotizing fasciitis.
31 life-threatening invasive infections such as necrotizing fasciitis.
32 aureus (MRSA) is a rarely reported cause of necrotizing fasciitis.
33 d to be less severely ill than patients with necrotizing fasciitis.
34 o life-threatening invasive disease, such as necrotizing fasciitis.
35 sing a range of diseases from pharyngitis to necrotizing fasciitis.
37 ore chronic comorbidities than patients with necrotizing fasciitis (20 [87.0%] vs 17 [54.8%]; P = .02
38 or patients with cellulitis vs patients with necrotizing fasciitis (3 [2-5] vs 5 [3-11]; P = .01), wh
41 known as strep throat) to severely invasive necrotizing fasciitis (also known as the flesh-eating sy
43 hould maintain a high clinical suspicion for necrotizing fasciitis and distinguish it from more commo
44 than the carriage strain in a mouse model of necrotizing fasciitis and had enhanced growth ex vivo in
46 t fever, streptococcal toxic shock syndrome, necrotizing fasciitis and myositis, and the post-infecti
47 vestigated the role of SpyCEP in S. pyogenes necrotizing fasciitis and respiratory tract infection in
48 ngitis to severe invasive infections such as necrotizing fasciitis and streptococcal toxic shock synd
49 tients with a variety of diseases, including necrotizing fasciitis and toxic shock syndrome, were ana
58 a pathogen that causes both invasive (e.g., necrotizing fasciitis) and noninvasive (e.g., pharyngiti
61 s of streptococcal bacteremia, myositis, and necrotizing fasciitis are presented and compared with th
62 treptococcal toxic shock syndrome (STSS) and necrotizing fasciitis are the 2 most severe invasive man
63 ding pharyngitis, impetigo, toxic shock, and necrotizing fasciitis, as well as the postinfection sequ
64 cus aureus has been a very uncommon cause of necrotizing fasciitis, but we have recently noted an ala
66 3 clone significantly underrepresented among necrotizing fasciitis cases has a unique frameshift muta
69 tis, streptococcal toxic shock syndrome, and necrotizing fasciitis) caused by six distinct M types of
70 iabetic man developed into rapidly spreading necrotizing fasciitis despite antimicrobial therapy and
71 e report on two cases of fatal monomicrobial necrotizing fasciitis due to Acinetobacter baumannii, an
76 d retired fisherman with rapidly progressive necrotizing fasciitis from Photobacterium (Vibrio) damse
78 the reason for ICU admission in 23 patients, necrotizing fasciitis in 31 patients, and other diagnose
79 this report, we present a case of multifocal necrotizing fasciitis in a healthy adult patient, second
81 We report an unusually severe case of MRSA necrotizing fasciitis in a previously undiagnosed AIDS p
83 arrants a high index of suspicion for fungal necrotizing fasciitis in the setting of wound infection
87 c shock syndrome (strep TSS) with associated necrotizing fasciitis is a rapidly progressive process t
95 e destruction in group A streptococcal (GAS) necrotizing fasciitis/myonecrosis often necessitates ext
96 n = 23; mean [SD] age, 57.2 [17.7] years) or necrotizing fasciitis (n = 31; mean [SD] age, 54.3 [13.5
97 with clinical and intraoperative findings of necrotizing fasciitis, necrotizing myositis, or both.
98 tease activity in vivo and recapitulated the necrotizing fasciitis-negative phenotype of the Deltamts
99 ssociated with invasive infections including necrotizing fasciitis (NF) and express a potent DNase (S
103 treptococcal toxic shock syndrome (STSS) and necrotizing fasciitis (NF) express numerous virulence fa
105 erile site or from a wound in a patient with necrotizing fasciitis (NF) or streptococcal toxic shock
108 n cause severe infections that can result in necrotizing fasciitis or even death in otherwise healthy
109 type 3 isolate recovered from a patient with necrotizing fasciitis, or with isogenic gene replacement
110 k thus aimed to address whether CLI improves necrotizing fasciitis outcome by modulating virulence fa
112 ma samples collected from a patient with GAS necrotizing fasciitis post-IVIG infusions markedly inhib
114 GAS), the causative agent of pharyngitis and necrotizing fasciitis, secretes the potent cysteine prot
115 fection (streptococcal toxic shock syndrome, necrotizing fasciitis, septic shock, or GAS cellulitis w
116 is endemic, empirical treatment of suspected necrotizing fasciitis should include antibiotics predict
117 h cellulitis and patients with patients with necrotizing fasciitis, Staphylococcus aureus (10 [43.5%]
119 itically ill on admission than patients with necrotizing fasciitis, they have more chronic comorbidit
120 ns in 20 patients with pathologically proved necrotizing fasciitis were reviewed retrospectively for
121 diseases, including toxic shock syndrome and necrotizing fasciitis, which are both associated with si
122 with the bacteria can result in devastating necrotizing fasciitis, which can progress to septicemia.
123 istered as soon as possible to patients with necrotizing fasciitis, while our in vitro studies emphas
125 ptococcal toxic shock syndrome cases and one necrotizing fasciitis without shock, were treated with i
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