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1 s observed in only 1 of 187 patients without necrotizing myopathy.
2 entify novel autoantibodies in patients with necrotizing myopathy.
3 rodiagnostic findings were consistent with a necrotizing myopathy.
4 ntly identified statin-associated autoimmune-necrotizing myopathy.
5 ents with dermatomyositis and the autoimmune necrotizing myopathies.
6 ated with dermatomyositis and the autoimmune necrotizing myopathies.
7 ients with statin-associated immune-mediated necrotizing myopathy and, less commonly, in statin-unexp
8 ients with dermatomyositis, polymyositis, or necrotizing myopathy, and 0/20 (0%) age-matched healthy
11 ren with profiles from children with a known necrotizing myopathy (Duchenne muscular dystrophy), as w
15 in use is associated with an immune-mediated necrotizing myopathy (IMNM), with autoantibodies that re
17 t evidence now suggests that immune-mediated necrotizing myopathy is not one disease, but can be divi
19 antibodies found in patients with autoimmune necrotizing myopathies recognize signal recognition part
20 MB in juvenile DM that focuses on extent of necrotizing myopathy, severity of vasculopathy, and feat
23 ed immune response in most of the autoimmune necrotizing myopathies, which may guide therapeutic opti
24 cificity defines a subgroup of patients with necrotizing myopathy who previously were considered to b
25 that statins may trigger an immune-mediated necrotizing myopathy with many features of polymyositis.
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