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1 interact with the endothelium, resulting in necrotizing vasculitis.
2 of bronchiolar epithelial cell necrosis, and necrotizing vasculitis.
3 ing polyradiculoneuropathy and from systemic necrotizing vasculitis.
4 ration of blood vessel walls indicative of a necrotizing vasculitis.
5 vels associated with development of systemic necrotizing vasculitis.
6 ctures and evidence of glandular atrophy and necrotizing vasculitis.
8 Histology of EGPA shows tissue eosinophilia, necrotizing vasculitis, and eosinophil-rich granulomatou
9 ely overlap with those of the other forms of necrotizing vasculitis, and no single clinical or histol
10 localized in typical PDNS lesions, including necrotizing vasculitis, glomerulonephritis, granulomatou
11 Histopathologic analysis of the skin showed necrotizing vasculitis in biopsy samples from 40 of 50 c
12 is, granulomatous inflammation, and systemic necrotizing vasculitis, including necrotizing arteritis
13 dy-associated (ANCA-associated) small vessel necrotizing vasculitis is caused by immune-mediated infl
16 ic infectious trigger of paralyzing systemic necrotizing vasculitis most severely affecting skeletal
18 et vessel wall, which may participate in the necrotizing vasculitis of the kidney during this disease
20 atosis with polyangiitis (GPA) is a systemic necrotizing vasculitis that is associated with granuloma
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