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1 PCl(5)-initiated living polymerization of 3 needs to be revised to reflect the key role of the count

2 that the reported structure of baulamycin A needs to be revised, as its spectroscopic data are not i

3 year, the WHO guidelines are inadequate and need to be revised since both HIV-infected and HIV-expos

4 fail, particularly in younger patients, and need to be revised, most commonly for loosening secondar

5 daily persistent headache is inadequate and needs to be revised to capture a significant number of p

6 nd functional properties of this protein and needs to be revised.

7 ia freshwater inputs into the North Atlantic needs to be revised.

8 ent models of S(0) oxidation in the Chlorobi need to be revised to take into account the role of cell

9 ing of sepsis and multiple organ dysfunction needs to be revised, as the uniformly negative results o

10 ATP in the control of adiponectin exocytosis needs to be revised to include an additional ATP-depende

11 n of constitutive expression of this isoform needs to be revised.

12 ctivation of I(CRAC) in RBL-1 cells, then it needs to be revised significantly.

13 istinction between the two forms of learning needs to be revised.

14 s of the products reported in the literature need to be revised.

15 t fully correct vitamin D deficiency and may need to be revised to include even higher dosing of ergo

16 largely by cytotoxic T-lymphocyte assays may need to be revised.

17 odifications of the GFP-like chromophore may need to be revised.

18 urrent models of inter-motif connections may need to be revised.

19 ion that CD133 is a marker of colon CSCs may need to be revised.

20 e in glutamate triggering neuronal death may need to be revised.

21 ventional models of internal entrainment may need to be revised to explain control of the periphery b

22 This explanation may need to be revised if, as suggested herein and if replic

23 sensus criteria for the diagnosis of FTD may need to be revised.

24 The goal of HAART may need to be revised to a lower cutoff than 50 copies/mL.

25 central role of mast cells in this model may need to be revised.

26 urrent strategies for obesity prevention may need to be revised.

27 tional views of modularity in psychology may need to be revised.

28 estimates of continuum cell stiffness might need to be revised upward.

29 National guidelines for stroke might need to be revised on the basis of these results.

30 suggest that some key features of the model need to be revised.

31 Such models need to be revised in the light of new data demonstratin

32 The models need to be revised to account for a pure reduction in th

33 e human leukocyte antigen (HLA) region to MS need to be revised to accommodate these data.

34 These conventions now need to be revised to allow neuroscience to progress uni

35 k factors for peanut allergy, these will now need to be revised to more strongly state that avoidance

36 om mutations in the Lowe syndrome gene OCRL1 need to be revised.

37 ur study suggests that (1) certain paradigms need to be revised, such as the 12 SNP distance as the g

38 cupancy, and so previous views of permeation need to be revised: the presence of an AMFE does not imp

39 d safety record, RDRC's 30-y-old regulations need to be revised to be consistent with current scienti

40 ies that have used AQDS as a marker of steps need to be revised.

41 friction based on these estimates therefore needs to be revised.

42 ed, the emerging data suggest that this view needs to be revised.

43 stimates of foraging carrying capacity would need to be revised downward if Oligochaetes are truly av

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