ライフサイエンスコーパス: nefiracetam

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1 ls were inhibited rather than potentiated by nefiracetam.

2 related to the cognitive enhancing action of nefiracetam.

3 d protein kinases, we studied their roles in nefiracetam action on the NMDA receptor by the whole-cel

4 at these protein kinases are not involved in nefiracetam action.

5 pertussis toxin for 24 h also abolished the nefiracetam action.

6 The cognition-enhancing drug, nefiracetam ameliorated the acquisition of CRs in older

7 experiment was undertaken to explore whether nefiracetam ameliorates conditioning via the hippocampus

8 Results suggest that nefiracetam ameliorates learning via the hippocampus.

9 Although nefiracetam and aniracetam inhibited alpha 7-type curren

10 cted to elucidate the mechanism of action of nefiracetam and aniracetam on neuronal nicotinic acetylc

11 t nnAChRs are an important site of action of nefiracetam and G(s) proteins may be its crucial target.

12 ted under two drug conditions (10 or 0 mg/kg nefiracetam) and 4 lesion conditions (bilateral hippocam

13 h-induced current was greatly accelerated by nefiracetam, and this effect could not be reversed by wa

14 Nefiracetam at 1 nM and aniracetam at 0.1 nM reversibly

15 current-voltage relationships revealed that nefiracetam at 10 nM largely eliminated voltage-dependen

16 Nefiracetam at 100 microM suppressed the currents induce

17 Nefiracetam at very high concentrations (approximately 1

18 Nefiracetam (DM-9384) is a new pyrrolidone nootropic dru

19 The effects of nefiracetam (DM-9384) on the neuronal nicotinic acetylch

20 ostin C, and peptide 19--63) did not prevent nefiracetam from potentiating alpha 4 beta 2-type curren

21 Nefiracetam had a dual effect on ACh-induced currents: i

22 with bilateral hippocampectomy treated with nefiracetam learned like vehicle-treated rabbits.

23 Nefiracetam markedly increased the saturating responses

24 r's disease (AD), these results suggest that nefiracetam may ameliorate conditioning in AD as it amel

25 ic acetylcholine and NMDA receptors and that nefiracetam modulates the glycine binding site of the NM

26 Thus, the nefiracetam modulation of the neuronal nicotinic ACh rec

27 It was concluded that nefiracetam potentiated NMDA currents not by acting as a

28 viously demonstrated that the nootropic drug nefiracetam potentiates the activity of both nicotinic a

29 The nefiracetam potentiating action was not affected by 24-h

30 The nefiracetam potentiation of NMDA currents was inhibited

31 but not G(i)/G(o) proteins, are involved in nefiracetam potentiation.

32 tor calphostin C (0.5 microM), abolished the nefiracetam stimulation of the ACh receptor.

33 The three groups of nefiracetam-treated rabbits with intact hippocampus acqu

34 Nefiracetam treatment did not affect the PKA activity.

35 In immunoblotting analysis, nefiracetam treatment increased the PKCalpha activity wi

36 The stimulating effect of nefiracetam was clearly observed at a concentration of 1

37 dependent Mg2+ block and that this action of nefiracetam was sensitive to PKC inhibition.

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