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1 l dose of DENV2 and mice immunized with OVA (negative control).
2 efold prolongation of survival compared with negative controls).
3 ter myocardial infarction (P </= 0.05 versus negative controls).
4 ith a second cohort of patients without CNV (negative controls).
5 oculation and no antifungal supplementation (negative controls).
6 antigen (cases) and children whose stool was negative (controls).
7 skeletal muscle using Kcne3(-/-) tissue as a negative control.
8 atient's tophus and steroid crystals used as negative control.
9 in, together with GSK4028 as an enantiomeric negative control.
10 cy by using PEG to perform a hitherto absent negative control.
11 /cm(2) (n = 6), and six flanks served as the negative control.
12  chemical shift perturbations similar to the negative control.
13  complexes purified from wild type mice as a negative control.
14 ean wheal diameter >/= twice the diameter of negative control.
15 , or with the binding-null D355N mutant as a negative control.
16 ritoneal injections of Mir375 inhibitor or a negative control.
17 atypical bHLHs, which likely keep HBI1 under negative control.
18 isoform-specific knockout mice was used as a negative control.
19 rface showed no capture in comparison to the negative control.
20                UV-irradiated RSV served as a negative control.
21 nd MHC-II proteins significantly more than a negative control.
22  treatments containing the AgNPs, Ag(+), and negative control.
23 urgical intervention were also included as a negative control.
24 ems: three dynamic and one steady-state as a negative control.
25 t-chain fatty acids produced compared to the negative control.
26 N versus HLL (without cytotopic tail) versus negative control.
27 munoaffinity-purified sample, but not in the negative control.
28 teria after a 24h fermentation compared to a negative control.
29  mice with Kv1.3 gene-targeted deletion as a negative control.
30  in 26 libraries from three blood donors and negative controls.
31 mplitudes and axon counts when compared with negative controls.
32 atients, 657 hospital controls, and 334 test-negative controls.
33 hemistry using well-established positive and negative controls.
34 pulmonary function comparable to the healthy negative controls.
35 m patients without malignancies were used as negative controls.
36 gher in HIV-positive cases compared with HIV-negative controls.
37 ratio)] for vaccination in cases versus test-negative controls.
38 ra mussel in 94% of spiked samples and 0% of negative controls.
39 an follow-up, 3.8 years) matched 1:10 to HIV-negative controls.
40 ve for Plasmodium or Babesia species and 151 negative controls.
41 ancy analysis by including both baseline and negative controls.
42 on when challenged with allergen compared to negative controls.
43 e identified as cases and 1,202 as endoscopy-negative controls.
44 CD14(++)CD16(+) monocytes, compared with HIV-negative controls.
45                 Unstimulated WBCCs served as negative controls.
46 higher CX3CR1 levels than monocytes from HIV-negative controls.
47 rols and 53% (95% CI, 24%-72%) using the ARI-negative controls.
48 -CoV-positive patients (cases) with MERS-CoV-negative controls.
49 o of vaccination in cases compared with test-negative controls.
50  of HML-2 RNA in total PBMCs compared to HIV-negative controls.
51 WT and C57BL/6 IL-17RA KO animals serving as negative controls.
52 HIV type 1 RNA load, <50 copies/mL), and HIV-negative controls.
53  both HIV-positive groups, compared with HIV-negative controls.
54                   Unfined wines were used as negative controls.
55 e as an inexhaustible source of high-quality negative controls.
56 cell cycle-related analytes along with their negative controls.
57 ld create bias in VE studies using influenza-negative controls.
58 nfirmed influenza A virus infection and test-negative controls.
59  also studied concomitantly, as positive and negative controls.
60 , including the use of specific positive and negative controls.
61  by the scaffold compared to the activity of negative controls.
62 in and streptavidin components and served as negative controls.
63 ed HIV-infected patients were matched to HIV-negative controls.
64  comparing rotavirus case patients with test-negative controls.
65 ected patients compared with appropriate HIV-negative controls.
66 ccine receipt in influenza cases versus test-negative controls.
67 pdm09 and 305 B(Victoria) cases and 926 test-negative controls.
68 tages of SIV infection and compared with SIV-negative controls.
69 omen naive to antiretroviral therapy and HIV-negative controls.
70 ontrols, and 52% (95% CI, 26%-69%) with test-negative controls.
71 , 2, and 5-8) and viral loads, including two negative controls.
72 sitive) and 1,941 HCV-negative (HCV antibody-negative) controls.
73  matter (DM) intake, respectively; and T5, a negative control (0% additives).
74  subjects in The Gambia, West Africa: 10 HIV-negative controls, 10 HIV-2-infected subjects with low v
75 vary VZV DNA was detected in 0 of 20 healthy negative controls, 11 of 16 positive controls with zoste
76 /males; mean age = 71 +/- 10) and 29 amyloid-negative controls (15/14 females/males; mean age = 69 +/
77 (VE point estimates range = 51%-76%) and ARI-negative controls (48%-76%).
78 terventions: 5 mL of Normosol-R fluid alone (negative control), 5 mL of 2% nonoxynol-9 (N-9) gel, and
79 .9 months; IQR 6.6-11.1), 317 rotavirus-test-negative controls (9.4 months; 6.9-11.9), and 380 commun
80 t of 6 mm or more (wheal diameter, above the negative control), a food-specific serum IgE concentrati
81                                              Negative control AAA animals (N = 15) underwent daily sa
82 talizations compared to 64% (97/152) of test-negative controls; adjusted vaccine effectiveness was 71
83 ncing studies, we propose the Adaptive False-Negative Control (AFNC) procedure that can include a lar
84  on 204 rotavirus case patients and 601 test-negative controls aged 5-23 months, the VE of 2 RV1 dose
85 iving suppressive cART and 79 comparable HIV-negative controls, aged >/=45 years, from the Comorbidit
86  were a total of 1517 CHD cases and 2671 CHD-negative controls, all with type 2 diabetes.
87 e activity in percentage of mean activity of negative controls allows comparison of the different tec
88 positive control) and cysteine-less protein (negative control) allows for the quantification of free
89        Simultaneous analysis of positive and negative controls, along with the assay reagents specifi
90 equences in K562 cells were more active than negative controls, although surprisingly, Weak Enhancer
91 ug/mL) in sugar syrup, pure sugar syrup as a negative control and 48 ng/mL imidacloprid as a positive
92 l organisations of PspA corresponding to its negative control and effector functions.
93 ed form of beta1a490-508 peptide was used as negative control and produced negligible effects on Ca(2
94 operties: 1) conditional independence of the negative control and the outcome (given modeled variable
95              Nonfirearm injuries served as a negative control and were not associated with California
96 ral blood flow differed between amyloid-beta-negative controls and -positive subjects in different di
97  = 0.05) when comparing cases with endoscopy-negative controls and 0.84 (95% CI: 0.56, 1.26; Ptrend =
98 e interval [CI], 5%-67%) using the influenza-negative controls and 53% (95% CI, 24%-72%) using the AR
99  signals were observed in 3/32 (9.4%) of the negative controls and also in few samples containing Myc
100 ral blood mononuclear cells (PBMCs) from HIV-negative controls and HIV-1-infected patients were colle
101  which are estimated from positive controls, negative controls and housekeeping genes embedded in the
102 ed to 58% and 63% vaccinated among influenza-negative controls and matched ARI-negative controls, res
103 nt decreases were observed for the prior two negative controls and no discrete events were observed f
104 es from HIV-positive individuals relative to negative controls and to plasma.
105 0%-70% gamma-chain expression (vs. < 10% for negative control) and a corresponding increase in HbF.
106 respectively, and parental, mock-transduced (negative controls) and tumors expressing Ang-1 or Ang-2
107  the use of a PENTA-containing served as the negative control, and a Methacryloyloxidecyl dihydrogenp
108                            OVA was used as a negative control, and mice were inoculated intranasally
109 h giant CMN with no nodules were included as negative controls, and 6 patients with melanomas arising
110 pare treatment groups with both baseline and negative controls, and analyze the microbiota changes do
111  differential site occupancy in positive and negative controls, and comparisons of estimated site occ
112 oesophageal adenocarcinoma [n=19]), relevant negative controls, and replicates on the Illumina MiSeq
113 usly on day 105-110 of pregnancy, while four negative-control animals underwent mock challenge.
114 tudy effects of these three antibodies and a negative control antibody (HJ3.4) on Tau aggregate uptak
115                                 Fortunately, negative controls are largely bait independent.
116                                              Negative controls are sometimes used in epidemiologic pr
117  active tuberculosis and TST-positive or TST-negative controls, as well as between active untreated a
118 monoinfected recipients and HIV-negative/HCV-negative controls at both times.
119 re similar between elite controllers and HIV-negative controls but significantly lower in ART recipie
120 C/C binding sites in yeast Cdh1 to relay the negative control by Cdk1 phosphorylation of the ubiquiti
121 n humoral responses is due to the absence of negative control by immune regulation.
122 TGFbeta signalling derives from positive and negative control by Ldb2a.
123 ined in the cytoplasm but remain under tight negative control by the Asi proteins.
124 imary antibodies were tested in positive and negative controls by immunohistochemistry and uniplex IF
125  Transmutation tool allows for the design of negative controls by permitting scrambling, reversing, c
126 ontrols were challenged with IL-31 and NaCl (negative control) by skin prick testing.
127 eory and simulations show that including the negative control can reduce residual confounding, if our
128 es but who test negative for rotavirus (test-negative controls) can be considered a suitable alternat
129 ng respective MVs, which were confirmed with negative controls, CD41 and CD235a; 2) anti-CD144 (EC ma
130                                          The negative control cells without any chemical receptor exp
131 d intracranial hypertension (n = 5), and the negative control cohort consisted of otherwise healthy p
132 is was mirrored by 39 (DO34) but also by the negative control compound 40 (DO53) (which does not inhi
133 , or DFO on inhibiting STAT3 activation, the negative control compound di-2-pyridylketone 2-methyl-3-
134 designed and synthesized two new, orthogonal negative control compounds, BRD9715 and BRD8461, which l
135                                              Negative controls consisted of 50 isolates that produced
136 ed by comparing a SPAM modified surface to a negative control, consisting of similar material where t
137                        These methods include negative controls, cross-contextual designs, instrumenta
138  confirmed cases of fungal meningitis and 66 negative control CSF specimens.
139 thms either find large numbers of fusions in negative control data or suffer from low sensitivity det
140 er, our model detected no introgression in a negative control data set.
141                            Using a case-test-negative control design, we estimated the vaccine effect
142 rol for skin penetration) in comparison to a negative control device.
143                             Use of influenza-negative controls did not generate a biased estimate of
144 ool that generates putative nonbinding (i.e. negative control) DNA sequences for one or more proteins
145 lytical approaches such as the use of family negative controls (e.g. comparing the effects of materna
146      This eliminated the need for a separate negative control, effectively minimizing chip-to-chip an
147 ect estimators from models that included the negative control exposure (ozone concentrations 1 day af
148  extensions to the interesting and important negative control exposure approach for partial confoundi
149                               One requires a negative control exposure with 2 key properties: 1) cond
150 ure time series, while the second combines a negative control exposure with a negative control outcom
151 e that estimators from models that include a negative control exposure with these 2 properties tend t
152 e both past and future exposures as multiple negative control exposures to further attenuate confound
153 ive in biochemical and cellular assays, as a negative control for chemical biology studies.
154 is to use a set of 'background' sequences as negative controls for evaluating the false-positive rate
155              We used mock-infected tissue as negative controls for ex-vivo experiments and influenza
156 ient midribs of ppcesa3/8 knockouts provided negative controls for the structural characterization of
157 tested positive (cases) and those who tested negative (controls) for influenza virus.
158 ool specimen that tested positive (cases) or negative (controls) for rotavirus by enzyme immunoassay.
159 positive control) and Alzheimer's disease (a negative control) for comparison.
160                           Hence, aggregating negative controls from multiple AP-MS studies can increa
161 ndiabetic CHD cases and 1637 nondiabetic CHD-negative controls from the Nurses' Health Study and Heal
162 tive control genes and much greater than the negative control genes.
163                              Exposure to the negative controls, gold and polyethylene, did not signif
164 itive control group drug), and esomeprazole (negative control group drug).
165 us VE studies that used traditional and test-negative control groups and compared VE estimates obtain
166 stimates using both the traditional and test-negative control groups closely approximated the true VE
167 luations that used both traditional and test-negative control groups were similar regardless of contr
168                                              Negative-control groups were immunized i.m. and s.c. wit
169 igonucleotides" against miR-494 (GSO-494) or negative control (GSO-control).
170                            Compared with HCV-negative controls, HCV-seropositive participants had an
171 ed with HIV-negative/hepatitis C virus (HCV)-negative controls, HIV monoinfected recipients had simil
172                            Compared with HIV-negative controls, HIV-infected recipients had significa
173 nd was significantly lower than PS among HIV-negative controls (HR, 1.34; 95% CI, 1.08 to 1.68; P<0.0
174                                              Negative controls (i) without glucose, (ii) with an irre
175 ing of bacterial 16S rDNA in the presence of negative controls identified D. pigrum in three cases.
176 cytotoxicity activity similar to that of the negative control IgG2 in a CD20(+) human Raji lymphoma c
177 tribution parameters using (i) internal chip negative controls, (ii) out-of-band Infinium I probe int
178 and, therefore, has often been included as a negative control in dietary studies, whereas the effect
179 ificant and rapid cell death compared with a negative control in most melanoma cell lines tested.
180                                              Negative control in two-component signal transduction re
181 tants should be employed as more appropriate negative controls in future investigations of KLK7, espe
182 kin test (TST)-positive controls, and 39 TST-negative controls in The Gambia.
183 l DNA in incubated blood samples but also in negative controls, indicating that the amplified sequenc
184           Two further groups received either negative control inoculum (group 4a,b) or were inoculate
185                                              Negative control is believed to involve competition of R
186 nother, and the use of specific positive and negative controls is essential for the interpretation of
187 ith a considerable nonspecific shift band in negative control lanes from which protein extracts were
188         While no M. canettii was detected in negative controls, M. canettii-infected mice yielded gra
189 latform, we exposed established positive and negative control materials to the neuronal networks in a
190 m, 0.49 +/- 0.042), which was similar to the negative control MDA-MB-231 xenografts (percent ID per g
191 fects were observed after DMM surgery in Cre-negative control mice, including articular cartilage deg
192  (Agc1CreER) KO mice compared to that in Cre-negative control mice.
193                   In contrast, all immunized negative-control mice developed PcP.
194  and endocervical cells transfected with miR-negative control, miR-143 or miR-145 were used in cell p
195               Participants consisted of gene-negative controls (n = 16) and prodromal Huntington's di
196 0), cases exposed to M. bovis (n = 424), and negative controls (n = 38).
197 rformed in three groups of Lewis (LEW) rats: negative controls (n = 4), local MSCs (epineural) inject
198              Pigs were randomly allocated to negative controls (n = 6), positive controls (n = 8), in
199 ted rejection with endarteritis; n=101), and negative controls (no diagnostic rejection; n=103).
200                                              Negative controls (no prebiotic) of both systems showed
201             Thus, ATF3 converges with CSL in negative control of CAF activation with epigenetic chang
202 sights into the coordination of positive and negative control of gene expression upon salt stress.
203 key novel regulator in both the positive and negative control of miR-155 in the inflammatory response
204                                          The negative control of PRC expression by GSK-3 was consiste
205                         We conclude that the negative control of SV and FV tonoplast channel activity
206 functions as a tumor suppressor by its novel negative control of the EMT, achieved through regulating
207  subtilis, many adaptive genes are under the negative control of the global transition state regulato
208 anscription of these genes is also under the negative control of the NO-sensitive NsrR repressor.
209 Proteomic data highlighted the potential for negative control of the production of phosphatidylinosit
210 expression of SUT1 and SUT2 in turn is under negative control of the transcription factor Ste12.
211 avirus vaccine, compared with 1434 rotavirus-negative controls of whom 856 (60%) received two doses,
212                    The use of rotavirus test-negative controls offers an efficient and cost-effective
213 fter TLR4 triggering and exerts an important negative control on LPS-dependent TNF production in MCs
214  combines a negative control exposure with a negative control outcome for joint indirect adjustment o
215                                      Thus, a negative control outcome found to be empirically associa
216 ng an exposure-based indicator rather than a negative control outcome to partially correct for confou
217            Childhood cognition was used as a negative control outcome.
218 We also collected caregiver-reported data on negative control outcomes (cough/cold and scrapes/bruise
219 nificant difference in the prevalence of the negative control outcomes between study groups that woul
220                                              Negative control outcomes of myocardial infarction (MI)
221 imilar increase in risk was seen for the two negative control outcomes related to sun exposure (HR =
222                   Here we discuss the use of negative control outcomes to detect and adjust for confo
223 , and colorectal cancer were investigated as negative control outcomes to exclude bias.
224 se data suggest that the N terminus exerts a negative control over basal as well as agonist-stimulate
225 tivity as a BtrS antagonist and exerts tight negative control over T3SS genes.
226 d greater ferritin formation compared to the negative control (p<0.05).
227 ctively, vs 7.0 x 10(9) neutrophils/L in EIA-negative controls (P < .0001) and 7.9 x 10(9) neutrophil
228 T recipients with suppressed viremia and HIV-negative controls (P < .01).
229  brain abscess samples, and no archaea in 71 negative controls (P < 10-6).
230 compared with 1 W/cm(2) pulses (P = .02) and negative controls (P = .002).
231 C. difficile toxins, compared with levels in negative controls (P = .008) We also investigated the ro
232  early ART remained elevated relative to HIV-negative controls (P = .02).
233 icromol/L (95% CI, 1.8 to 35.4) in untreated negative controls (P<0.001).
234 eritis, 79% in positive controls, and 91% in negative controls (P=0.01).
235  participant data (IPD) meta-analysis, and a negative-control (paternal BMI) to examine the associati
236            Using cut points derived from the negative control patients, OCT parameters yielded 89% se
237     Children with cranial pathology, but not negative control patients, underwent direct intraoperati
238 lable, well-defined archive of positive- and negative-control patients.
239 ween influenza-positive (case) and influenza-negative (control) patients with pneumonia, controlling
240 ssessed using I-1 liposomes as compared with negative control peptide liposomes (NCP).
241  from the selected family, TP2, as well as a negative control peptide, ONEG, from the same library.
242 oth suspended and biofilm culture versus the negative control PEU films releasing nothing.
243                           In contrast, three negative control phenotypes (height, inflammatory bowel
244  no development of positive reactions in the negative-control pool.
245 based on machine learning using positive and negative control populations.
246                             PP2, but not the negative control, PP3, blocked BDNF's suppressive effect
247 ground instead of the much smaller number of negative control probes on the platforms (n = 32 for Hum
248 al primary pancreatic fibroblast cells (HPF; negative controls) produced MDSC-promoting cytokines [in
249 y nonspecific interactions using one or more negative-control purifications, but many small-scale AP-
250  dermal papilla cells served as positive and negative controls respectively.
251 additives (SOcontrol) served as positive and negative controls, respectively.
252  influenza-negative controls and matched ARI-negative controls, respectively.
253 -beta- and PBS-treated cells as positive and negative controls, respectively.
254 et of highly conserved genes as positive and negative controls, respectively.
255 fected MCF-7 cells were used as positive and negative controls, respectively.
256 ve dose (TCID50)/ml, as well as the no-virus negative-control sample.
257  (n = 24), or suspected (n = 11) CJD and 104 negative control samples (54 CSF and 50 OM).
258 dentical set of 20 standardized positive and negative control samples, including human universal refe
259 patients and 19 control subjects, along with negative control samples.
260 s comparable to those measured in blanks and negative control samples.
261 ermined based on 8 (6 HPV-positive and 2 HPV-negative) control samples.
262 l proteins, 10,000 point mutants, and 30,000 negative control sequences.
263 men that were unreactive with alpha-actinin (negative control sera) failed to detect any of the epito
264 eight rats were divided into four groups: 1) negative control (sham operated); 2) periodontal disease
265 pecimens (108 well-characterized CPO and 142 negative controls spiked onto negative rectal swabs) wer
266 design and utility of different positive and negative control strategies.
267                                     The test-negative control study design is the basis for observati
268 d compared to those in young and elderly HIV-negative control subjects.
269 st) were equivalent in identifying sera from negative-control subjects (99% and 100% specificity, res
270 morphogenesis and was indistinguishable from negative controls, suggesting that it is a null mutation
271 able-length guides per gene and thousands of negative controls targeting non-functional, non-genic re
272 a total of 93 clinical samples, including 21 negative controls, the results of which allow assembly o
273             This study provides positive and negative controls, titrated standards, and a guide for s
274 eal 2 mm or larger than that elicited by the negative control to any of 10 inhalant or food allergens
275  to estimate ammonia kinetic parameters, (2) negative controls to account for biomass reactivity, (3)
276 ses were 2.8-fold more likely than norovirus-negative controls to be secretors (P < .001) in a logist
277 eding was used as a falsification end point (negative control) to evaluate for potential confounding.
278 s without antipsychotic properties served as negative control treatments.
279  the PD-L1-positive tumor, whereas the PD-L1-negative control tumor showed little tracer retention.
280  recipient mice then either received saline (negative control), unsorted MHC-mismatched PBMC (positiv
281 ifferent clades/sequence types (STs) and EIA-negative controls using Cox and normal regression adjust
282             An outcome is said to be a valid negative control variable to the extent that it is influ
283 stically similar adjusted VE using influenza-negative controls (VE point estimates range = 51%-76%) a
284 ositive system that promotes molting and the negative control via the critical weight checkpoint prov
285 justed vaccine effectiveness using rotavirus-negative controls was 57% (95% CI 40-68) for two doses a
286 ng for green fluorescence protein (GFP, as a negative control) was observed.
287                                         As a negative control, we evaluated the combination of lansop
288 mparing amyloid-beta-positive subjects with -negative controls, we found reductions of cerebral blood
289                               Cases and test-negative controls were children who presented with a dia
290 es against HLA antigens and from 16 antibody-negative controls were filtered.
291 and 5 hexon antigens along with positive and negative controls were printed in an array format onto t
292                                         Four negative controls were tested and classified as negative
293 EMD unfractionated compound and positive and negative controls, were tested for their ability to indu
294  viral antigen divided by the mean OD of the negative control when reacted with viral antigen, were h
295 udy exemplifies the crucial need of relevant negative controls when developing, and using, bioinforma
296 n of PTM binding pocket variants as matching negative controls, which is not possible with antibodies
297 1 rotavirus-positive cases and 298 rotavirus-negative controls who had under-5 card-confirmed vaccina
298 misspecification, and 2) associations of the negative control with uncontrolled confounders and the o
299 graphs taken a few minutes apart, which were negative controls with no progression.
300 cially available sweet drinks (including two negative controls) with high levels of complexity (sugar

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