ライフサイエンスコーパス: negative elongation factor

1 nner, the accumulation of the pause-inducing negative elongation factor.

2 gation complexes are completely resistant to negative elongation factors.

3 ith HIV-1 Tat protein and these positive and negative elongation factors.

4 Depletion through RNA interference of negative elongation factor, a mediator of Pol II stallin

5 ns with TEAD to regulate binding of the NELF negative elongation factor and block SMAD2,3 induction o

6 By phosphorylating negative elongation factors and the C-terminal domain of

7 By phosphorylating negative elongation factors and the C-terminal domain of

8 s we describe are the pausing factors--NELF (negative elongation factor) and DSIF (DRB sensitivity-in

9 rylation of DRB sensitivity-inducing factor, negative elongation factor, and C-terminal domain (CTD)

10 start site upstream transcripts at multiple negative elongation factor-associated genes.

11 Furthermore, we show that negative elongation factor binds to estrogen target prom

12 After Pol II initiates, negative elongation factors cause it to pause in a promo

13 e [DRB]-sensitivity-inducing factor) and the negative elongation factor complex (NELF).

14 ntrolled genes was abolished specifically in negative elongation factor-deficient macrophages.

15 is functional interaction between CE and the negative elongation factor documents a dynamic role of C

16 oncogenic activation of a top candidate RBP, negative elongation factor E (NELFE), via somatic copy-n

17 provides an easy passage for pol II, and the negative elongation factor facilitates termination at th

18 or of BRCA1 (COBRA1), a subunit of the human negative elongation factor, has been shown to repress es

19 E relieves transcriptional repression by the negative elongation factor, indicating a critical role o

20 , Cdk7 inhibition reduced recruitment of the negative elongation factor NELF at start sites.

21 enes are also associated with the polymerase negative elongation factor NELF.

22 the promoter in a complex that includes the negative elongation factor NELF.

23 n of transcript elongation by the complex of negative elongation factor (NELF) and 5,6-dichloro-1-bet

24 Negative elongation factor (NELF) and 5,6-dichloro-1-bet

25 osophila and vertebrates, DSIF together with negative elongation factor (NELF) associates with RNA po

26 P1) mRNA levels and reduced occupancy of the negative elongation factor (NELF) at the p21(CIP1) promo

27 iated transcription is stalled by the host's negative elongation factor (NELF) at the promoter region

28 triggers rapid and transient release of the negative elongation factor (NELF) complex and productive

29 tive elongation by acting as a decoy for the negative elongation factor (NELF) complex upon induction

30 nit (with NELF-A, -C/D, and -E) of the human negative elongation factor (NELF) complex, which partici

31 gene expression requires the pause-inducing negative elongation factor (NELF) complex.

32 BRA1) is a newly characterized member of the negative elongation factor (NELF) complex.

33 The principal negative elongation factor (NELF) contains four polypept

34 s study, we investigated mechanisms by which negative elongation factor (NELF) establishes and mainta

35 The role of the negative elongation factor (NELF) in maintaining HIV lat

36 Negative elongation factor (NELF) induces RNAP II promot

37 The human negative elongation factor (NELF) is a four-subunit prot

38 The Negative Elongation Factor (NELF) is a transcription reg

39 e transcriptional pausing pathway, including negative elongation factor (NELF) that pauses RNA polyme

40 ntrary to current expectations, depletion of negative elongation factor (NELF), a key factor in setti

41 und that PARP-1 ADP-ribosylates and inhibits negative elongation factor (NELF), a protein complex tha

42 azole sensitivity-inducing factor (DSIF) and negative elongation factor (NELF), act as negative trans

43 1 (COBRA1), an integral subunit of the human negative elongation factor (NELF), directly binds to ERa

44 w used to identify two new factors (BRD4 and negative elongation factor (NELF)-E) and to define their

45 erase (Pol) II, which requires the 4-subunit negative elongation factor (NELF).

46 sensitivity inducing factors (DSIF) and the negative elongation factor (NELF).

47 nditionally knock out the b subunit of mouse negative elongation factor (Nelf-b), a key pol II-pausin

48 using, in contrast to depletion of the NELF (negative elongation factor) pausing complex.

49 -RAP to analyze interactions of the EGFP and negative elongation factor subunit E (NELF-E) proteins w

50 made possible by the reversal of effects of negative elongation factors that include NELF and DSIF.

51 Hence, Pcf11 can act as a negative elongation factor to repress RNA Pol II gene ex

52 P-TEFb phosphorylates RNA polymerase II and negative elongation factors to stimulate general transcr

53 f RNA polymerase II (RNA pol II), as well as negative elongation factors, which block elongation by R