ライフサイエンスコーパス: negative potential

1 ethanolamine (PE) in the presence of a trans-negative potential.

2 ss-linked and electrodeposited by applying a negative potential.

3 ociation with wild-type protein and dominant-negative potential.

4 ric state and to shift E degrees ' to a more negative potential.

5 k-state wild-type RC, indicating substantial negative potential.

6 electron at positive potential and 1.5-3 at negative potential.

7 n participate in redox reactions at a rather negative potential.

8 e of the steady-state inactivation to a more negative potential.

9 ilability of Na(V)1.6 is quickly restored at negative potentials.

10 shift of the midpoint voltage (V0.5) to more negative potentials.

11 howed a strong voltage-dependent decrease at negative potentials.

12 onger as spark probability decreased at more negative potentials.

13 each mechanism is **COOH formation at higher negative potentials.

14 (KA) was voltage-dependent, being greater at negative potentials.

15 omposite by shifting the Fermi level to more negative potentials.

16 voltage of half-maximal inactivation to more negative potentials.

17 .45 V vs RHE, but methane is favored at more negative potentials.

18 are irregularly spaced at more positive and negative potentials.

19 re restored when the patches were stepped to negative potentials.

20 ve potentials but enhances N-type current at negative potentials.

21 This resulted in enhanced NMDA responses at negative potentials.

22 MDA) was voltage-dependent, being greater at negative potentials.

23 fts the current-voltage relationship to more negative potentials.

24 rm entry vs voltage relationship toward more negative potentials.

25 out two-fold, and the E(Gly) shifted to more negative potentials.

26 toplasm relieves the block to sperm entry at negative potentials.

27 ing conditions, and was observed at the most negative potentials.

28 d was observed in samples poised at slightly negative potentials.

29 ut development are mainly AMPA-R-mediated at negative potentials.

30 mine from -60 to +60 mV, but failed for more negative potentials.

31 the electrolytes on the electrode surface at negative potentials.

32 hat closes cx46 gap junction hemichannels at negative potentials.

33 PO and promoted lower conductance levels at negative potentials.

34 of the PA current were nearly linear at low negative potentials.

35 k, the rate of unblock is more rapid at more negative potentials.

36 a2+ current-voltage relationship toward more negative potentials.

37 mV, and less pronounced at more positive or negative potentials.

38 cker dissociates following repolarization to negative potentials.

39 a voltage-dependent block by external Na+ at negative potentials.

40 movement but shifted the G(V) curve to more negative potentials.

41 rents, being stimulated less than twofold at negative potentials.

42 two additional reversible reductions at more negative potentials.

43 voltage-gated calcium currents (ICa) to more negative potentials.

44 ch that increased salt concentrations induce negative potentials.

45 the voltage dependence of activation to more negative potentials.

46 rents were evoked even after conditioning at negative potentials.

47 dependent manner with stronger inhibition at negative potentials.

48 y voltage dependent, being most effective at negative potentials.

49 n inward flow of Na(+) through Na/K pumps at negative potentials.

50 annel opening by shifting activation to more negative potentials.

51 urrent and a shift of activation toward more negative potentials.

52 At negative potentials (-0.2V(SCE)), however, the molecules

53 2 currents activated and inactivated at more negative potentials (1 s Vh = -46 mV), showed substantia

54 owire electrode on which UO2 precipitated at negative potential (-1.2 V) improved substantially the d

55 ential perceptual fluency were identified as negative potentials 100-300 ms poststimulus onset.

56 tes ethylene capture, and subsequent dose of negative potential (-2.0 V) induces the release.

57 rred quickly after acidification for a trans-negative potential across the target membrane (i.e., neg

58 E(gly) was found to shift toward more negative potentials after a period of complex spiking or

59 ated by cAMP, and activated at physiological negative potentials, allowing K(+) to enter the mitochon

60 potentials reduced inward currents (i.e. at negative potentials), although this result was highly co

61 This dependence was restricted to negative potentials, although all data were consistent w

62 f divalent ions (Ins) also activated at more negative potential and decayed more slowly in rat.

63 get cell showed that fusion is promoted by a negative potential and hindered by a positive potential.

64 HERG when oocytes were voltage clamped at a negative potential and not pulsed during equilibration w

65 , where the closed channel is more stable at negative potential and the open channel is more stable a

66 decreased during several seconds at strongly negative potentials and (iii) had an underlying unitary

67 a smaller component that activated over more negative potentials and a larger component (L-type Ca2+

68 cken Cx56 hemichannels were mostly closed at negative potentials and application of depolarizing volt

69 f I521C in the outer S4 was enhanced at more negative potentials and at earlier times in these same m

70 voltage dependence of channel gating to more negative potentials and by enhancing the affinity of the

71 d Ca2+ release (VGCR) that activated at more negative potentials and displayed a significantly higher

72 the voltage dependence of activation to more negative potentials and enhancement of maximum conductan

73 41V shifted the activation curve toward more negative potentials and increased the window current, wh

74 , catalytic activity onsets at prohibitively negative potentials and is highly dependent on the natur

75 se connexins is reduced at both positive and negative potentials and is maximal at intermediate volta

76 45.6 hemichannels were predominantly open at negative potentials and rapidly closed in response to ap

77 at suggests a sensitivity to hyperpolarizing negative potentials and that depolarized the cells.

78 inhibition of I(Cl(Ca)) at both positive and negative potentials and there was no increase in current

79 (hERG1, Kv11.1) channels to activate at more negative potentials and to deactivate more slowly than n

80 ells activated and inactivated at relatively negative potentials and was very sensitive to holding po

81 e steady state inactivation of ICa,L to more negative potentials, and (3) elicited a tonic block and

82 e voltage of half-activation (V(12)) to more negative potentials, and decreased unitary conductance.

83 n-voltage relationship, slower activation at negative potentials, and increased inactivation.

84 dent 'leak' current (Ileak) operating at all negative potentials, and, at potentials >= -60 mV, a tim

85 Fusion occurred without leakage for a negative potential applied to the trans side of the plan

86 the electrode array) and reversed polarity (negative potentials applied to the electrode array) for

87 veals a current that activates at relatively negative potentials (approximately-50 mV) and shows rapi

88 ensitive to [Na+]o, but V0.5 shifted to more negative potentials as [Na+]o was reduced.

89 dual shift of the voltage dependence to more negative potentials as well as uncoupling from voltage s

90 of calcium and also on the application of a negative potential, as shown earlier in eukaryotic cells

91 o Boltzmann expressions and ranged over more negative potentials, as compared with the voltage depend

92 the bipyridine ligand and Ru metal center at negative potentials, as well as the inhibition of Ru-Ru

93 the detection of a voltammetric peak at more negative potentials associated with the one-electron red

94 and diazepam increased the amplitude of the negative potential at 45 ms after stimulation (N45) and

95 on sites exist on the ribozyme, and that the negative potential at the active site may help shift the

96 change was found to significantly reduce the negative potential at the active site, as supported by e

97 N1 potentials, with preserved face-specific negative potentials at 170 ms.

98 c protein signal, peak H, observed at highly negative potentials at around -1.8 V (vs Ag/AgCl3 M KCl)

99 N1-DnsSpm was partially relieved at extreme negative potentials at receptors containing NR1(N616G) o

100 low wave repolarization and maintenance of a negative potential between slow waves was also found.

101 alpha1D I(Ca-L) activated at negative potentials (between -60 and -50 mV).

102 a greater number of channels to open at more negative potentials both in the presence and absence of

103 In contrast, at negative potentials, brief sojourns to subconductance st

104 mine (NMDG+) NFA still enhanced I(Cl(Ca)) at negative potentials but the increase of I(Cl(Ca)) on was

105 NFA (100 microM) increased inward current at negative potentials, but inhibited outward current at po

106 1.1a/1b isochronal activation curves to more negative potentials, but shifted the potential of half-m

107 he Cu(111) face is shown to be disordered at negative potentials, but to order with applied positive

108 the voltage dependence of activation to more negative potentials by >20 mV.

109 ut also increases Na channel availability at negative potentials by antagonizing fast inactivation.

110 and I(K1) currents that are active at large negative potentials by CsCl and BaCl2, respectively, did

111 teady-state inactivation was shifted to more negative potentials by increases in submembrane [Ca2+] f

112 potential (E(r)) of I(Cl(Ca)) was shifted to negative potentials by replacing external Cl- with SCN-,

113 el inactivation is shifted reversibly toward negative potentials by synthetic detergents that decreas

114 itive potentials, reversing the trend toward negative potentials caused by agonist.

115 show reduction potentials at similar or less negative potentials compared to the silylethynyl derivat

116 p for rSkM1 Na+ channels was shifted to more negative potentials compared with its G-V relationship w

117 le increased FV currents (up to fivefold) at negative potentials compared with the currents in symmet

118 ed a shift in the reversal potential to more negative potentials, consistent with electrogenic transp

119 with greater inhibition at positive than at negative potentials, consistent with more effective buff

120 The spectral density function of Icat at negative potentials could be described by the sum of two

121 (ICa) was half-activated at about 10 mV more negative potential, decayed slower, was half-inactivated

122 d Au and GC electrodes exhibit a significant negative potential drift during their equilibration in a

123 ting the PEDOT(PSS) surface with an ISM, the negative potential drift is compensated by a positive po

124 se data are consistent with the concept that negative potentials drive GABA and Na+ into the cell, wh

125 ssolution of Pt nanoelectrodes at moderately negative potentials during the ORR.

126 e for channel deactivation increases at more negative potentials (e-fold per 170 mV) whereas activati

127 two chemically reversible processes at very negative potentials, E(1)(1/2)= -0.444 V and E(2)(1/2)=

128 In the case of less negative potentials, e.g., at -0.45 V with a NB (TCNQ) e

129 cal anion and dianion forms occurred at less negative potentials (easier to reduce).

130 eases T-type channel activity selectively at negative potentials, evoking an 11 mV hyperpolarizing sh

131 However, after a negative potential excursion to approximately -2.0 V ver

132 els, L-type channels activated at relatively negative potentials, favoring their opening with EPSP st

133 ERGs of normal subjects contained a slow negative potential following the a- and b-waves, the PhN

134 (<100 nA at positive potential and <25 nA at negative potential for 96% ethanol; < 40 nA at positive

135 t Co(II/I) reduction step occurs at the most negative potential for Co(mnt)(2).

136 similar for flavin, whereas MoR+ had a more negative potential for heme-iron.

137 avage of the S-S bond that avoids the highly negative potential for the first direct electron reducti

138 e disulfide moiety with a substantially more negative potential for the first electron than for the s

139 ctivation relationships were shifted to more negative potentials for channels containing alpha(1B) an

140 annels at positive potentials and unbinds at negative potentials, generating a resurgent Na current a

141 lectrochemical gaps and are reduced at small negative potentials giving LUMO energy levels of -3.57 t

142 nward current increased in amplitude at more negative potentials, had a reversal potential of approxi

143 Inward resurgent currents at strongly negative potentials, however, were larger in reverse tha

144 Cathodic pretreatment of BDD at a negative potential in an acidic environment successfully

145 mal urinary acidification induced by a lumen-negative potential in response to furosemide infusion is

146 A large laterally extended negative potential in the frontal portion of the nitroge

147 (2+) release from lysosomes could generate a negative potential in the lumen to disturb subsequent Ca

148 approach revealed a region of exceptionally negative potential in the major groove surrounding the 2

149 curves for nitro-based SAMs were observed at negative potentials in both forward and reverse scans an

150 antly slows the rate of fast inactivation at negative potentials in heterologously expressed mNa(V)1.

151 Similar LMAS spectra were obtained at more negative potentials in the onset region of photoelectroc

152 ocatalyst (i.e., to produce H(2) at the most negative potential) in this series, even though it has t

153 ift in steady-state inactivation toward more negative potentials; inactivation was faster but was not

154 eas the shift in the conduction band to more negative potentials increases the driving force and favo

155 Recovery from clofilium block was reduced at negative potentials independent of pH, an effect attribu

156 potential across the target membrane (i.e., negative potential inside the target cell) but that a tr

157 In this method, a sufficiently negative potential is applied to the electrode surface t

158 activity does not occur until a ~400 mV more negative potential is present.

159 We conclude that the block to sperm entry at negative potentials is mediated by calcium which crosses

160 ing Mn(I)-COOH complex at significantly more negative potentials is required to achieve fast catalyti

161 of the absence of the more delayed sensitive negative potential, it was sustained, lasting as long as

162 ift of the calcium current I-V curve to more negative potentials, leading to an increase in basal [Ca

163 by shifting the activation curve toward more negative potentials, leaving little room for facilitatio

164 ry from inactivation was relatively rapid at negative potentials (<-80 mV) but was slow at more posit

165 akest test stimuli, ERGs consisted of a slow negative potential maximal approximately 200 ms after th

166 e potentials and Ca2+ effects predominate at negative potentials, may be relevant to the regulation o

167 The leading edge of the first negative potential (N1) is largely shaped by the initial

168 The MRCP negative potential (NP) related to motor task preparatio

169 ts most likely results from reduction of the negative potential of a small pore between the E and PS

170 Mac1 also reduced the negative potential of S. aureus and E. coli membrane wit

171 of considerable implication is the intensely negative potential of the phosphate-binding cleft.

172 tions indicate a lower voltage dependence at negative potentials of the kidney enzyme in comparison w

173 l on the AB face of the beta2 motif opposite negative potential on the AB face of the alpha20 motif a

174 alculations revealed several patches of high negative potential, one of which is present in a cleft n

175 Moreover, at negative potentials open probabilities of EAAT5 anion ch

176 tom of the quinoline ring and the absence of negative potential over the molecular plane are crucial

177 ng quickly followed lipid dye transfer for a negative potential, providing a direct demonstration tha

178 riboflavin is superior in its oxidization in negative potential range, where the number of interferin

179 gative slope conductance was observed in the negative potential range.

180 The two negative potential regions and the positive potential lo

181 Two negative potential regions occur near the hydroxyl oxyge

182 The CEM electrode is held at a negative potential relative to the AEM side; cations/ani

183 tetrahydrofuran at -0.48 V vs NHE, the least negative potential reported for a molecular catalyst.

184 The increase in the negative potential response can be used for detection an

185 The latter gives a greater negative potential response due to the presence of the h

186 in slow conduction and blocks, whereas more negative potentials resulted in faster conduction.

187 Less negative potentials resulted in slow conduction and bloc

188 As intensity was increased, the negative potential saturated but the positive potential

189 c input was also found to induce spontaneous negative potentials (SNPs) rapidly in the tectum.

190 ltage dependence of ICa,L activation to more negative potentials so that ICa,L was always present wit

191 develops as the sum of exponentials, tauh at negative potentials speeds and then slows with more posi

192 A negative potential step applied at the generator produce

193 In addition, in oxygenated solutions a negative potential step at the generator produces hydrog

194 activation of Ca2+ current shifted to a more negative potential, suggesting stronger depression of hi

195 nd an increased sensitivity to Ba2+ block at negative potentials, suggesting that mKir4.2 forms funct

196 contrast, compound 2(2+) is reduced at less negative potential than 1 and at the dimethyl bipyridini

197 ction of the H-bond complex occurs at a less negative potential than that of U(H)(+), leading to reve

198 unstable red anionic semiquinone with a more negative potential than the hydroquinone.

199 , which allows them to activate at even more negative potentials than C-terminally long-splice varian

200 to produce channels that opened at even more negative potentials than control, suggesting the presenc

201 iac sodium channels open and close over more negative potentials than do skeletal muscle sodium chann

202 activation, induces channels to open at more negative potentials than normal, and increases current m

203 yzed glucose oxidation at substantially more negative potentials than pure platinum in enzyme-free vo

204 e current activated more rapidly and at more negative potentials than the alpha-DTX-insensitive curre

205 e significantly larger and activated at more negative potentials than the control.

206 s 7-10 pS, and it activated at slightly more negative potentials than the I channel; its deactivation

207 age sensors from KV6.4 subunits move at more negative potentials than the voltage sensors belonging t

208 bunits form channels that are active at more negative potentials than wild-type channels.

209 lectrodes-a counter electrode held at highly negative potential that serves as the cathode, and two a

210 3 days, kainate induced an inward current at negative potentials that recovered to baseline levels im

211 ways exist in skeletal muscle: one active at negative potentials that requires store depletion (store

212 larised potentials (> +5 mV) whereas at more negative potentials the current amplitude was enhanced.

213 At negative potentials the probability of opening (Po) was

214 Upon application of a negative potential, the dsDNA denatures into its constit

215 and of TiO(2) shifts 59 mV/pH unit to a more negative potential, thereby decreasing the driving force

216 be expelled from the nanotubes by applying a negative potential, this provides a route for reversibly

217 or a second one-electron reduction at a less negative potential to form a dianionic species.

218 ysis at the reported potential and at a more negative potential to speed up the reaction, it appears,

219 By applying a negative potential to the electrode, the air-stable Cu(I

220 ton and two-electron transition occurring at negative potential to the organic pyranopterin ligand sy

221 ing of mDia1 from its autoinhibited state at negative potentials to its activated state at positive p

222 d outward current occurs only after applying negative potentials to the cell.

223 cesses: electrochemically polarizing MoS2 at negative potentials (vs RHE) in acidic media or immersin

224 When negative potential was applied to membrane at the side o

225 The duration of the negative potential was reduced to normal during rapid st

226 The slope conductance at negative potentials was 12 pS.

227 f Ca2+ channel recovery from inactivation at negative potentials was increased dramatically by Ba2+ s

228 oM Ca2+, the maximal channel availability at negative potentials was similar despite a shift in the v

229 The EECC mechanism, operative at more negative potentials, was isolated through use of a weak

230 f hemichannel opening and mean open times at negative potentials, was observed in (Cx56 + Cx45.6) cRN

231 ture, while in other studies relatively more negative potentials were needed to achieve higher curren

232 I(KSper), setting the membrane potential to negative potentials where Ca2+ entry via I(CatSper) is m

233 hanism following cyclic voltammetry scans to negative potentials where reduction occurs at the pi* le

234 uced current and 5HT uptake both increase at negative potentials, where 5HT carries approximately 5%

235 protamine requires approximately 0.2 V more negative potentials, where a potentiometric super-Nernst

236 nel currents had an increased Po compared to negative potentials which was associated with increased

237 , the 4-aminopyridine-induced GABA-dependent negative potentials, which appeared to trigger the ictal

238 activated single inward channel currents at negative potentials, which had a slope conductance of 2-

239 K(+) channel conductance is low than at more negative potentials (wild-type channels), where total K(

240 ltage dependence of charge movements to more negative potentials, with apparent affinity constants (K

241 oltage for half-maximal inactivation to more negative potentials without affecting the half-maximal v

242 ctivation (V0.5) approximately 18 mV to more negative potentials without affecting the maximal conduc

243 aphy (ECochG) showed prolonged low amplitude negative potentials without auditory nerve compound acti

244 idinium-derivative-carried inward current at negative potentials without Na(+) and K(+).