ライフサイエンスコーパス: negative pressure

1 ntilator (20 secs, 14 F catheter, 200 cm H2O negative pressure).

2 us, rather than to loss of responsiveness to negative pressure.

3 essure variability by oscillatory lower body negative pressure.

4 G activity and the GG reflex to upper airway negative pressure.

5 during, and immediately after application of negative pressure.

6 t on the XII reflex response to upper airway negative pressure.

7 gram, where water is both supercooled and at negative pressure.

8 e LMkappaT emerges from the no man's land at negative pressure.

9 ry, and they appear to be unstable except at negative pressure.

10 -tidal CO(2) and a number of intrapharyngeal negative pressures.

11 or bubbles emerging from metastable water at negative pressures.

12 So how do plants transport water under negative pressure?

13 bilization and flow of liquid water at large negative pressures (-1.0 MPa or lower), continuous heat

14 creased cardiac filling by use of lower-body negative pressure (-15 and -30 mm Hg), and after saline

15 whole nerve reflex response to upper airway negative pressure (-20 cm H2O) at any 5HT concentration

16 plasma norepinephrine response to lower body negative pressure (3.0 +/- 0.3 vs. 2.0 +/- 0.2 nmol/l at

17 tral hypovolemia was induced with lower body negative pressure (-60 mm Hg) and pain by the cold press

18 e further reduced by hypovolemia (lower body negative pressure, -60 mm Hg).

19 c retardation of OMP folding places a strong negative pressure against spontaneous incorporation of O

20 By following the maximum range of negative pressure, along with the observed slight decrea

21 Negative pressure also evoked single Cl- channel activit

22 In human embryonic kidney 293 cells, negative pressure also increased the window current (250

23 tive stimuli (e.g. rapidly imposed pulses of negative pressure) also activate these muscles.

24 oximately proportional to the square of peak negative pressure amplitude and were statistically signi

25 Ultrasound at 1 MHz, with a peak negative pressure amplitude of 0.75 MPa, was applied to

26 as brisk GG reflex activation in response to negative pressure (amplitude: +78.5 +/- 28.3 % baseline

27 ry baroreflexes were perturbed by lower body negative pressure and head-down tilt.

28 muscles to respond to rising intrapharyngeal negative pressure and increasing Co(2) during sleep, (3)

29 stimulated activity, or TRPV1, which sensed negative pressure and inhibited activity.

30 ile preload was manipulated using lower body negative pressure and rapid saline infusion to define LV

31 lary wedge pressure and SV during lower body negative pressure and saline loading in 7 men (25+/-2 ye

32 relationship between varying intrapharyngeal negative pressures and genioglossal muscle activation (G

33 ere constructed during decreases (lower body negative pressure) and increases (saline infusion) in ca

34 phy at baseline, -15 and -30 mmHg lower-body negative pressure, and 15 and 30 ml kg(-1) saline infusi

35 echocardiography) at rest, during lower-body negative pressure, and after saline infusion before and

36 sfer with the evaporation of liquid water at negative pressure, and continuous extraction of liquid w

37 ng decreased cardiac filling with lower-body negative pressure, and increased filling with saline inf

38 ers, baseline, with two stages of lower body negative pressure, and repeat baseline with two stages o

39 tracheostomized patient would be exposed to negative pressure, and that high levels of muscle activa

40 us, rather than to loss of responsiveness to negative pressure, and that this wakefulness stimulus ma

41 Negative pressure applied to on-cell membrane patches ac

42 SDK channels were activated in response to negative pressure applied to patches.

43 of TREK-1 channels by mechanical stretch and negative pressure applied to the cell membrane.

44 n the NPR, we quantified GG EMG responses to negative pressure applied to the isolated upper airway i

45 embrane deformation in response to a step in negative pressure applied to the membrane by a micropipe

46 order to encourage admission before force or negative pressures are used.

47 l upper airway respiratory stimuli, possibly negative pressure, are important in mediating the increa

48 Lung volume was altered with continuous negative pressure as applied to the chest wall with a po

49 em surfactants support water transport under negative pressure as explained by the cohesion-tension t

50 Negative pressures associated with multiaxial strain and

51 no systematic reduction in the GG reflex to negative pressure at sleep onset.

52 rm stabilizes interfacial binding due to the negative pressure at the hydrocarbon-water interface.

53 igid head-out shell equipped with a positive/negative pressure attachment for manipulation of extrath

54 We define a negative pressure between the skeleton/membrane connecti

55 non-invasively by externally applying focal negative pressure bilaterally to the neck.

56 able to repair embolized xylem vessels under negative pressure, but its hydraulic vulnerability segme

57 During LHR treatment, two 6-L negative pressure canisters were used to capture 30 seco

58 Application of lower body negative pressure caused progressive reductions of R-R i

59 uscle, ventilatory, and arousal responses to negative-pressure challenges during sleep in 19 healthy

60 th supine treadmill exercise in a lower-body negative pressure chamber (EX group).

61 e line relating SV to PCWP during lower-body negative pressure characterized the steepness of the Sta

62 99Mo/99mTc generators were placed in the negative-pressure clean room to ensure a more efficient

63 t significant portions of inspiration, i.e., negative pressure dependence (Level 4).

64 -S) S-S molecular mode has an unconventional negative pressure dependence, whereas other peaks stiffe

65 a large pressure-driven exchange rate and a negative pressure-dependent activation volume, reflectin

66 binet opposite OR 2, which was maintained at negative pressure differentials, then was poured into bo

67 gth of stay was significantly reduced in the negative pressure dressing group [6.1 vs 14.7 days, P =

68 included, with 25 patients randomized to the negative pressure dressing group and 25 to the standard

69 The effect of prophylactic negative pressure dressing of closed incisional wounds o

70 the abdominal wall are being developed from negative pressure dressing therapies to acellular allogr

71 Prophylactic use of negative pressure dressings for closed laparotomy wounds

72 en to investigate the effect of prophylactic negative pressure dressings on postoperative surgical si

73 ally more collapsible upper airway with more negative pressure driven muscle activation.

74 A novel, simple, negative pressure-driven device with manually operated m

75 A striking result from our simulations is negative pressure-flow correlations observed in several

76 fiber was carried out by applying 11 kPa of negative pressure for 3 s.

77 tained a bullet and responded to positive or negative pressure from the recording pipette were consid

78 y, dissociating the influences of pharyngeal negative pressure, from inspiratory airflow, resistance,

79 during slow (physiological) oscillations in negative pressure generated spontaneously and passively

80 ation of the muscle, combined with increased negative-pressure generation during inspiration.

81 apnea based on pharyngeal closing pressure: negative pressure group (pharyngeal closing pressure les

82 ng the contraction of the vortex core as the negative pressure grows back to positive values, the vor

83 activity and reflex response to upper airway negative pressure in 15 decerebrated, vagotomized, paral

84 bility of the actuator is utilized to create negative pressure in the ampoule and collect ISF.

85 tein effects these changes by decreasing the negative pressure in the headgroup region of the outer l

86 saturation in the vapour phase of water into negative pressures in the liquid phase, stabilization an

87 expressing human embryonic kidney 293 cells, negative pressure increased Na(V) peak currents by 27+/-

88 epiglottic pressures during basal breathing, negative-pressure (iron-lung) ventilation, heliox breath

89 The channel is only active after negative pressure is applied to cell attached patches, c

90 Negative pressure is one potential stimulus for this neu

91 s range of blood gases: (1) the GG reflex to negative pressure is unchanged; (2) slow airway pressure

92 pattern generator activity, intrapharyngeal negative pressure itself modulates genioglossus activity

93 Volume redistributions with lower body negative pressure (LBNP) are similar to those that occur

94 0 min at rest and during 5 min of lower body negative pressure (LBNP) at -10 and -40 mmHg (n = 11).

95 mpathetic activation, produced by lower body negative pressure (LBNP) at -40 mmHg, on cerebrovascular

96 ic nerve activity (SNA) evoked by lower body negative pressure (LBNP) at rest and during moderate-int

97 isengagement of baroreflexes with lower body negative pressure (LBNP) can engage the sympathetic nerv

98 ts undergoing monitoring with the lower body negative pressure (LBNP) device.

99 tral blood volume was reduced via lower-body negative pressure (LBNP) during normothermia, whole-body

100 lex sympathetic activation during lower body negative pressure (LBNP) evoked decreases in muscle oxyg

101 achieved by stepwise increases in lower-body negative pressure (LBNP) in 14 healthy young volunteers.

102 earm vasodilator responses during lower body negative pressure (LBNP) in 21 non-obstructive hypertrop

103 application of -20 and -30 mm Hg lower-body negative pressure (LBNP) in 24 patients with chronic hea

104 ans (n = 20) during a progressive lower body negative pressure (LBNP) protocol designed to cause pres

105 er carotid sinus massage (CSM) or lower body negative pressure (LBNP) received Paxil (20 mg/d) or pla

106 omen underwent an initial maximal lower body negative pressure (LBNP) test to place them into a low (

107 Progressive lower body negative pressure (LBNP) to onset of cardiovascular coll

108 nce was determined by progressive lower-body negative pressure (LBNP) to presyncope.

109 articipants underwent progressive lower-body negative pressure (LBNP) until pre-syncope; end-tidal ca

110 lerance was assessed using graded lower-body negative pressure (LBNP) until the onset of symptoms ass

111 hen a cold pressor test (CPT) and lower body negative pressure (LBNP) were superimposed upon heating.

112 ary wedge pressure (PCWP), during lower-body negative pressure (LBNP) while subjects are normothermic

113 ctions in were accomplished using lower body negative pressure (LBNP), while increases in were accomp

114 n of sympathetic nerves evoked by lower body negative pressure (LBNP).

115 sympathetic activation evoked by lower body negative pressure (LBNP).

116 aemorrhagic challenge imposed via lower-body negative pressure (LBNP).

117 re recorded during nonhypotensive lower body negative pressure (LBNP; -10 mm Hg) and nonhypertensive

118 C, followed by the application of lower body negative pressure (LBNP; -30 mmHg).

119 were also obtained during graded lower body negative pressure (LBNP; activates baroreflex-mediated s

120 Subjects experienced lower-body negative-pressure (LBNP) of 0, 15 and 30 mmHg during nor

121 % NaCl) on endogenously mediated (lower body negative pressure [LBNP]) and exogenously mediated (brac

122 o hypotensive hypovolemic stress (lower body negative pressure [LBNP]) in healthy human males.

123 lk modulus, kappa = 140 +/- 20 kPa; applying negative pressures leads to volumetric expansion of the

124 rodes prevented the shift in kinetics, while negative pressure led to an abrupt shift to fast inactiv

125 (GG) activation in response to upper airway negative pressure may be an important mechanism in the m

126 airway pressure (CPAP) was applied to reduce negative pressure mediated muscle activation.

127 airway pressure (CPAP) was applied to reduce negative pressure-mediated muscle activation).

128 red with control nights before and after the negative pressure nights.

129 nd 37.8 +/- 29.1 events/h during each of two negative pressure nights; p < 0.001) that were associate

130 d pulses of 6-microsecond duration at a peak negative pressure of 15 MPa and a pulse repetition frequ

131 ent loading conditions: baseline, lower-body negative pressures of -15 and -30 mm Hg, and rapid salin

132 before and after the application of topical negative pressures of -50, -70, and -120 mmHg, using las

133 Oscillatory lower body negative pressure (OLBNP) was used to create consistent

134 ressure fluctuations (oscillatory lower body negative pressure, OLBNP) across a range of frequencies

135 The effects of negative pressure on open probability were graded as a f

136 type CaAg(5) phase is found to exhibit large negative pressures on each Ca atom, which are concentrat

137 te emphasis on scientific outputs, and other negative pressures on the scientific enterprise.

138 c changes in airway mechanoreceptor stimuli (negative pressure or flow) were highly correlated with w

139 2 fashion with the combination of lower body negative pressure or not (normovolemia), and ice water o

140 If negative pressure (or another local airway stimulus) wer

141 Hz) decreased progressively with lower body negative pressure (p < .001).

142 hasic and tonic GG EMG, and the GG reflex to negative pressure (Pchoa = -12.5 cm H(2)O).

143 ad reduction using a standardized lower-body negative pressure protocol.

144 ), the correlation with GGEMG was robust for negative pressure (R(2) = 0.98) and less strong for othe

145 onging effects of a sustained square wave of negative pressure (range, -4.0 to -14.9 cmH2O) sufficien

146 of the central circuitry that mediates this negative pressure reflex (NPR).

147 Substantial data support the role of a local negative pressure reflex in modifying genioglossal activ

148 These data suggest that while the negative pressure reflex is able to maintain GGEMG durin

149 ct plane is introduced into one of these two negative-pressure regions, breaking the symmetry equival

150 s to determine which GG premotoneurons relay negative pressure-related information to the hypoglossal

151 rtificial muscles can be driven by fluids at negative pressures (relative to ambient).

152 activity on and off by applying positive and negative pressures, respectively.

153 ly increased their open probability, whereas negative pressure reversibly decreased it.

154 Negative pressure (suction) applied through the pipette

155 th the same properties as those activated by negative pressure, suggesting that the channels were str

156 After completing a maximal lower body negative pressure test to determine level of orthostatic

157 At negative pressure, the fluorescence images are consisten

158 sure ulcers: wound cleansers, repositioning, negative pressure therapy, debridement, enteral and pare

159 Application of negative pressure to cell-attached patches (-20 mmHg) ca

160 s over three isochores at high, ambient, and negative pressures to determine the thermodynamic stabil

161 Progressive lower-body negative pressure (to -50 mm Hg; LBNP) was used to exami

162 e was associated with 23% greater lower body negative pressure tolerance using an active impedance th

163 Lower body negative pressure tolerance was reduced after both condi

164 ngle-atom chain, linked by a tension-driven (negative-pressure) transformation.

165 olunteers by applying progressive lower body negative pressure (under two experimental conditions: a)

166 lled hydrophilic nanochannels under enormous negative pressures up to -7 MPa.

167 d GGEMG during both basal breathing (BB) and negative pressure ventilation (NPV) during wakefulness (

168 itive pressure ventilation (IPPV) to cuirass negative pressure ventilation (NPV) was investigated in

169 Using a model of passive negative pressure ventilation, we have previously report

170 esistances were reduced significantly during negative-pressure ventilation (P < .05 and P < .03, resp

171 Negative-pressure ventilation improved the cardiac outpu

172 Negative-pressure ventilation improves cardiac output in

173 We investigated the effect of negative-pressure ventilation on cardiac output in 11 ch

174 ermittent positive-pressure ventilation, and negative-pressure ventilation was delivered with the Hay

175 pressure ventilation and after 15 minutes of negative-pressure ventilation.

176 ssure generated spontaneously and passively (negative pressure ventilator).

177 Oscillatory lower body negative pressure was applied at six frequencies from 0.

178 Progressive lower body negative pressure was applied in 5-min stages until the

179 We found that the application of negative pressure was associated with the development of

180 he relationship between GGEMG and epiglottic negative pressure was tight across all conditions in bot

181 ffer when a cold pressor test and lower body negative pressure were superimposed upon heating.

182 merged: a current activated in hair cells by negative pressure, with some similarity to the transduct

183 Vascular plants transport water under negative pressure without constantly creating gas bubble

184 Negative pressure wound therapy (NPWT) has become a popu

185 We investigated whether negative pressure wound therapy (NPWT) improves the prop

186 e of this study is to evaluate the effect of Negative Pressure Wound Therapy (NPWT) on closed surgica

187 hanges in the intestines during conventional negative pressure wound therapy (NPWT), and NPWT using a

188 risk [RR], 1.58 [95% CI, 1.20 to 2.08]) and negative pressure wound therapy (RR, 1.49 [CI, 1.11 to 2