ライフサイエンスコーパス: negative staining

1 cimens, 13 with C4d-positive and 18 with C4d-negative staining.

2 ith procapsids but had a novel appearance by negative staining.

3 = 0.045), p53 (adjusted HR for positive vs. negative staining, 1.48 [CI, 1.06 to 2.08]; P = 0.022),

4 (adjusted hazard ratio [HR] for positive vs. negative staining, 1.61 [95% CI, 1.01 to 2.57]; P = 0.04

5 ner domain of actin previously determined by negative staining, also at 3.8 nm radius.

6 y identical to that determined previously by negative staining, although at a radius of 3.8 nm, sligh

7 higher risk of graft failure than those with negative staining and a significantly lower median time

8 To address these questions we have used both negative staining and cryo-EM to generate three-dimensio

9 3-D model of the mouse 16 nm AUM particle by negative staining and electron crystallography.

10 Cross-linking followed by negative staining and electron microscopy suggested a gl

11 Using negative staining and electron microscopy, full-length p

12 n microscopy (and single particle analysis), negative staining and freeze-fracture electron microscop

13 ructural basis of MLCK-actin interactions by negative staining and helical reconstruction.

14 alian heart, and analyzed their structure by negative-staining and electron microscopy.

15 tion of the probes could be monitored by the negative-staining appearance in the fluorescence microsc

16 Using a negative staining approach, maltose phosphorylase, a pho

17 The phage capsids were visualised after negative staining by transmission electron microscopy, a

18 10-20% wider than PolyQKd-33, as measured by negative staining, cryo-electron microscopy, and scannin

19 By negative staining, cryo-EM and scanning transmission EM

20 ick-freeze/deep-etch images and our previous negative staining data indicate that the head domain of

21 associated with responses to nivolumab, but negative staining did not rule out a response.

22 ent ultracentrifugation and visualization by negative staining electromicroscopy demonstrated that th

23 ent ultracentrifugation and visualization by negative staining electron microscopy demonstrated that

24 Negative staining electron microscopy of o-gp140SF162Del

25 fied to near homogeneity, and examination by negative staining electron microscopy revealed large, fl

26 Negative staining electron microscopy revealed that GTD-

27 d HMW1B by size exclusion chromatography and negative staining electron microscopy revealed that the

28 Negative staining electron microscopy showed that FhuA i

29 Negative staining electron microscopy shows that the Chl

30 We have used negative staining electron microscopy to study the effec

31 Using time-resolved negative staining electron microscopy, we show that the

32 rotease sensitivity, spectrofluorometry, and negative staining electron microscopy.

33 ions by gel filtration, CD spectroscopy, and negative-staining electron microscopy (EM).

34 The active fraction was visualized by negative-staining electron microscopy as ring-like parti

35 Visualization of the SEC by negative-staining electron microscopy revealed an anchor

36 bdiffraction-resolution light microscopy and negative-staining electron microscopy revealed that fasc

37 end, compatible with the density observed in negative-staining electron microscopy that depended on t

38 etry (HDX MS), native mass spectrometry, and negative-staining electron microscopy to comprehensively

39 Seen by negative-staining electron microscopy, it presents as a

40 d in the presence of lipid and visualized by negative-staining electron microscopy, the purified Kv1.

41 As seen by negative-staining electron microscopy, these lipoprotein

42 Using negative-staining electron microscopy, we sought Haufen

43 Here we report cryo-electron microscopy and negative-staining electron tomography approaches to imag

44 intracellular fluorescent labels as well as negative staining experiments to measure cell-induced sc

45 ; detection of MUC5AC mRNA using RT-PCR; and negative staining for CK-4, M(1) muscarinic receptor, an

46 Multivariate regression analysis identified negative staining for cytoplasmic FANCD2 as the most sig

47 -old female, both with gingival LP, also had negative staining for ER.

48 ained cells surrounded by cells with weak or negative staining for heparanase.

49 inly proprioceptive neurons, had very low or negative staining for hexokinase I.

50 The epithelial sheet showed negative staining for laminin 5 and collagen VII, but in

51 The remaining stroma showed negative staining for laminin 5, positive linear stainin

52 rescence of the skin of the proband revealed negative staining for the integrin alpha6 and markedly r

53 Electron microscopic analysis after negative staining further revealed that actin filaments

54 Following negative staining, images of the soft-landed complexes r

55 tion conditions and also validate the use of negative staining in investigations of muscle thin filam

56 pproximately 100 A wide fibers visualized by negative staining in the electron microscope, the beta-c

57 The validations by both negative staining (NS) and cryo-electron microscopy (cry

58 ning and electron microscopic examination in negative staining of aged samples of Abeta alone and Abe

59 Negative staining of capsids revealed small patches of h

60 Negative staining of NGF, NT4, BDNF, and TrkB was noted

61 Negative staining of released fibrils showed no evidence

62 Negative staining of the proteins revealed particles con

63 The Old group primarily had negative staining, or Type I and Type II patterns of amy

64 protein, invasive SCCs exhibited a patchy or negative staining pattern.

65 at this type of glycosylation influences the negative-staining pattern of collagen fibrils.

66 Theoretical negative-staining patterns were created based upon the "

67 mparison of the two techniques suggests that negative staining preserves the structure induced by Ca2

68 form fibrils in vitro with a similar amyloid-negative staining property to those of TDP-43 pathogenic

69 riable degrees of artifacts depending on the negative staining protocol.

70 Electron microscopy with negative staining provides further evidence that SAT has

71 gh negative predictive value suggests that a negative staining result indicates that OSSN is relative

72 AI-1 from these column fractions followed by negative staining revealed 25-nm diameter complexes of a

73 Electron microscopy with negative staining revealed an extended, filamentous CHGA

74 Electron microscopy using negative staining revealed that the addition of ATP indu

75 Negative staining reveals that when Ca2+ binds to the he

76 cated variant of FHA by electron microscopy (negative staining, shadowing and scanning transmission e

77 from skinned smooth muscles and observed by negative staining show crossbridges with a 14.5-nm repea

78 Negative staining showed a mixture of straight and twist

79 ectron microscopy using rotary shadowing and negative staining showed that the type I and II receptor

80 meric filaments by electron microscopy after negative staining showed that, remarkably, EspA filament

81 Analysis of the photosynthetic apparatus by negative staining, spectroscopy, and sodium dodecyl sulf

82 We therefore used a time-resolved negative staining technique to determine the time scale

83 ordered unwinding of the DNA was observed by negative staining that appeared to progress through four

84 ron-microscopy techniques, thin sections and negative staining, that enabled answering major question

85 tissue was not corneal, as evidenced by the negative staining to cornea-specific K12 mRNA and protei

86 h 1:1 stoichiometry, [Yfh1]24.[Isu1]24 Using negative staining transmission EM and single particle an

87 Using negative staining transmission EM and single particle an

88 By immunogold labeling and negative staining we have detected in these complexes, m

89 There was negative staining with Congo red.

90 By contrast, most MMIHS tissue gave negative staining with ISH and variable results with ICC