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1 n factor against three different families of negative-sense RNA viruses.
2 is not a dominant restriction factor against negative-sense RNA viruses.
3 (EBOV) belongs to the group of nonsegmented negative-sense RNA viruses.
4 es with well-analyzed prototype nonsegmented negative-sense RNA viruses.
5 pe of the Bunyaviridae family of tri-partite negative-sense RNA viruses.
6 nus and the Bunyaviridae family of segmented negative-sense RNA viruses.
7 f the Paramyxoviridae family of nonsegmented negative-sense RNA viruses.
15 The RNA synthesis machinery of non-segmented negative-sense RNA viruses comprises a ribonucleoprotein
16 endent RNA polymerase (RdRP) of nonsegmented negative-sense RNA viruses consists of a large catalytic
17 er substantially from the RNP and N of other negative-sense RNA virus families and provide valuable i
22 its in vivo role in host protection from the negative-sense RNA virus influenza virus type A (flu) is
23 ype control mice and primary cells with four negative-sense RNA viruses (influenza A virus [IAV], La
24 ns it is shown to inhibit the replication of negative-sense RNA viruses (influenza viruses A and B, N
25 The proposed basis for the activity against negative-sense RNA viruses is the binding to exposed 5'-
27 s 5 (PIV5) is an enveloped, single-stranded, negative-sense RNA virus of the Paramyxoviridae family.
28 he Mononegavirales, a group of single-strand negative-sense RNA viruses, provides a unique system to
32 we evaluated the ability of IFIT1 to inhibit negative-sense RNA virus replication and pathogenesis bo
37 ment can be exploited to design nonsegmented negative-sense RNA viruses that have characteristics des
39 ructural studies of a prototype nonsegmented negative-sense RNA virus, vesicular stomatitis virus, su
40 te the ability of IFIT1 to have an impact on negative-sense RNA viruses, we infected Ifit1(-/-) and w
41 ean-Congo hemorrhagic fever virus (CCHFV), a negative-sense RNA virus with a 5'-monophosphorylated ge
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