ライフサイエンスコーパス: neonatal

1 The neonatal 25(OH)D3 concentrations in DBS samples were ass

2 Individuals in the middle quintile of neonatal 25(OH)D3 had lower odds of sustaining a fractur

3 ificantly increase the risk of stillbirth or neonatal, 6-month, or infant mortality, neither overall

4 to opioids in utero and who had signs of the neonatal abstinence syndrome to receive either sublingua

5 Among infants with the neonatal abstinence syndrome, treatment with sublingual

6 Single-center studies suggest that neonatal acute kidney injury (AKI) is associated with po

7 etween-group differences in the incidence of neonatal adverse outcomes or other adverse events.

8 Ctcf in mouse embryonic neurons is lethal by neonatal age, but the effects of CTCF deficiency in post

9 Interpretation: Neonatal AKI is a common and independent risk factor for

10 lar lavage fluid LT levels were increased in neonatal and adult but reduced in juvenile HDM-sensitize

11 In both neonatal and adult hUGT1/Car(-/-) mice, PEITC was unable

12 lt germ-free mice with the cecal contents of neonatal and adult mice, we show that the neonatal micro

13 Central nervous system infection of neonatal and adult rats with Borna disease virus (BDV) r

14 Neonatal and adult T cells differ in their effector func

15 Moreover, both neonatal and adult THS exposure caused a significant inc

16 c brain injury, especially in the vulnerable neonatal and childhood periods.

17 Neonatal and human cord blood CD71(+) cells express argi

18 Furthermore, neonatal and interferon gamma knockout mouse models of C

19 use skin results in reduced proliferation in neonatal and wounded adult epidermis.

20 ion suggests present reproductive, maternal, neonatal, and child health programmes focused on biomedi

21 ne immunization services and other maternal, neonatal, and child health programs in Africa that have

22 We show that the semi-dominant neonatal anemia (Nan) mutation in the EKLF/KLF1 transcri

23 uggest that anesthetic drugs administered to neonatal animals cause widespread neuronal apoptosis and

24 mong patients, 29.5% had a phenotype of ante/neonatal Bartter syndrome (polyhydramnios or diagnosis i

25 Neonatal BCG had no effect on the development of RW befo

26 sis were found to relate to fetal growth and neonatal body composition and thus may be among the earl

27 The neonatal brain is extremely vulnerable to injury during

28 BMP antagonism for response to injury in the neonatal brain.

29 ture placebo-controlled randomized trials of neonatal caffeine are unlikely.

30 ight less than 1,251 g who were treated with neonatal caffeine had improved respiratory function at 1

31 Risk of brain injury is increased during neonatal cardiac surgery, where pre-existing hemodynamic

32 nalysis of MEF2 isoform-specific function in neonatal cardiomyocytes has yielded insight into an unex

33 nstrated that the major effector function of neonatal CD4(+) T cells is to produce CXCL8, a prototypi

34 cyte monolayers currently require the use of neonatal cells with ionic properties that change rapidly

35 , our findings reaffirm that gestational and neonatal challenges can result in long-term programming

36 ression of chemokine (C-C motif) ligand 2 in neonatal cholangiocytes in vitro, and blockade of the co

37 ived cBF volumes were studied in relation to neonatal clinical complications after delivery and intel

38 weight was specifically associated with both neonatal complications (rpart,92 = -.35, p < .001) and a

39 gnificantly mediated the association between neonatal complications and adult cognitive deficits.

40 ations between obesity-related pregnancy and neonatal complications and risks of childhood epilepsy.

41 psy as well as obesity-related pregnancy and neonatal complications were based on information from th

42 is impaired after premature birth and links neonatal complications with long-term cognitive outcome.

43 risks of preterm delivery, asphyxia-related neonatal complications, and congenital malformations, wh

44 ase exacerbation and potential for fetal and neonatal complications.

45 ot explained by obesity-related pregnancy or neonatal complications.

46 birth asphyxia (n=3), septicaemia (n=1), and neonatal convulsion (n=1).

47 This protection was mediated by neonatal crystallizable fragment receptor (FcRn)-depende

48 ion modifications at these candidate loci in neonatal cultured cardiac myocytes after in utero exposu

49 ed a robust adult-like maturation profile of neonatal DCs.

50 w birth weight (0.68; .29-1.57) and fetal or neonatal death (0.24; .04-1.52).

51 Raf deletion caused neonatal death and a significant expansion of the hypert

52 an section, Apgar score <7 at 5 minutes, and neonatal death within 28 days.

53 ed ovum, n=1; intrauterine death, n=2; early neonatal death, n=1; and neonatal death, n=2).

54 e death, n=2; early neonatal death, n=1; and neonatal death, n=2).

55 range of adverse outcomes including fetal or neonatal death, neurodisability, and lifelong risks to t

56 (both in the Foley catheter group) and eight neonatal deaths (n=5 in the misoprostol group and n=3 in

57 rence 0.3%, 95% CI -0.8 to 1.5; p=0.566) and neonatal deaths did not differ significantly between gro

58 y between groups (six [2.0%] vs three [1.0%] neonatal deaths; 1.0, -1.04 to 2.97; p=0.322).

59 ression of UGT1A1 during the early stages of neonatal development is a tightly controlled event invol

60 ion because it supplies preformed lipids for neonatal development, and the assembled LDs are secreted

61 Neonatal diabetes mellitus (NDM) is a rare form of diabe

62 etic etiology of a syndrome characterized by neonatal diabetes, sensorineural deafness, and congenita

63 The major risk factor for neonatal disease is maternal vaginal colonization.

64 y, Cu dysregulation is associated with fatal neonatal disease, liver and cardiac dysfunction, and ane

65 ce the population burden of this devastating neonatal disease.

66 lead to preterm labor, stillbirth, or severe neonatal disease.

67 he single most important risk factor for GBS neonatal disease.

68 exposure of neonates is associated with GBS neonatal disease.

69 DNA was extracted from the neonatal dried blood spot samples obtained from the Dani

70 Current algorithms for management of neonatal early-onset sepsis (EOS) result in medical inte

71 Neonatal encephalopathy (NE) is a leading cause of child

72 minimum of 7000 (UR, 0-19000) presented with neonatal encephalopathy.

73 ts with invasive GBS disease presenting with neonatal encephalopathy.

74 KCNQ2 variants can lead to various forms of neonatal epilepsy.

75 No adverse maternal or neonatal events were identified as associated with botul

76 ry for the induction of persistent AHR after neonatal exposure during rescue assays in mice deficient

77 ocker reversed the progression of AHR in the neonatal exposure model, whereas beta2-adrenoceptor agon

78 ith a mouse model, we examined the effect of neonatal exposure to genistein on gene specific mRNA lev

79 At the end of neonatal exposure, THS-treated male and female mice had

80 irty-eight school-age (8-12 yr) survivors of neonatal extracorporeal membrane oxygenation and/or cong

81 mportant implications for infants exposed to neonatal factors including caesarean delivery, antibioti

82 The endothelial cellular neonatal Fc receptor (FcRn) has been suggested to play a

83 tudy the involvement of immune complexes and neonatal Fc receptor (FcRn) in the emergence of ADAs in

84 offspring by instructing T reg formation via neonatal Fc receptor (FcRn)-mediated transfer and uptake

85 The neonatal Fc receptor FcRn plays a critical role in the t

86 demonstrates that bacterial flora within the neonatal feeding tubes may influence the bacterial colon

87 atory synaptic inputs on to these neurons in neonatal Fmr1 KO mice.

88 ompleted follow-up assessments in a Canadian Neonatal Follow-Up Network clinic at 18 to 21 months.

89 -deleted mice, pregnancy failed by day 10 in neonatal FOXA2-deleted mice lacking uterine glands.

90 l and contained glands, whereas the uteri of neonatal FOXA2-deleted mice were completely aglandular.

91 Neonatal functional connectivity of the ventral attentio

92 The authors hypothesized that neonatal functional connectivity of the ventral attentio

93 Necrotising enterocolitis is a neonatal gastrointestinal inflammatory disease with high

94 Public health interventions preventing neonatal GBS disease are urgently needed for the increas

95 entrations; outcome assessors were masked to neonatal glycemic status.

96 In neonatal gnotobiotic pigs, the icPC22A-S1Delta197 virus

97 Jenny Myers discuss approaches to fetal and neonatal growth assessment.

98 es, which may affect maternal, fetal, and/or neonatal health and physiology.

99 Neonatal health outcomes were significantly improved, wi

100 Secondary outcomes included obstetric and neonatal health outcomes, assessed with all available da

101 intervention centres addressed maternal and neonatal health, child health and nutrition, reproductiv

102 opulation of cardiomyocyte precursors in the neonatal heart and to determine their requirement for ca

103 onstrate the utility of this system to study neonatal hematopoiesis, a developmental stage that has b

104 Neonatal hemorrhagic stroke is more common than previous

105 tations, associated factors, and outcomes of neonatal hemorrhagic stroke.

106 d regional estimates of the annual number of neonatal herpes cases during 2010-15.

107 INTERPRETATION: These neonatal herpes estimates mark the first attempt to quan

108 to obtain global estimates of the number of neonatal herpes infections.

109 Better collection of primary data for neonatal herpes is crucially needed to reduce uncertaint

110 munodeficiency virus, hepatitis B virus, and neonatal herpes simplex virus, from which lessons for th

111 This is the first evidence in humans linking neonatal hippocampal injury to adult dopamine dysfunctio

112 us (RRV) can also lead to biliary atresia (a neonatal human disease) in mice.

113 Novel 96-well in vitro assays using neonatal human monocyte-derived DCs and humanized TLR8 m

114 In addition, neonatal hyperoxia promoted allergic TH responses to hou

115 identify the underlying mechanisms by which neonatal hyperoxia promotes asthma development.

116 0.26 to 0.86; p=0.0157), fewer incidences of neonatal hypoglycaemia (0.45; 0.22 to 0.89; p=0.0250), a

117 Those exposed to neonatal hypoglycemia (280 [58.7%]) did not have increas

118 ansient middle cerebral artery occlusion, or neonatal hypoxic-ischemic brain injury, Mn preferentiall

119 e guidelines for family-centered care in the neonatal ICU, PICU, and adult ICU, we developed an innov

120 gs from an individual primary recipient into neonatal IL-2Rbeta(-/-) mice, the secondary recipients d

121 ntestinal lamina propria (siLP) of adult and neonatal Il7ra(-/-) mice.

122 wo groups in the rates of death or any other neonatal illnesses.

123 icle, we argued that the positive results of neonatal imitation are likely to be by-products of norma

124 &A) propose a biologically plausible view of neonatal imitation based on the analysis of sensorimotor

125 Neonatal imitation illuminates how the initial state eng

126 Our aim here is to shed light on a form of neonatal imitation that goes beyond sensorimotor imitati

127 is eliminates a major source of evidence for neonatal imitation.

128 Keven & Akins (K&A) propose that neonatal "imitation" is a function of newborns' spontane

129 Although minor variations in the neonatal (immediately at birth) microbiota community str

130 ptibility has generally been associated with neonatal immune cell immaturity.

131 This review will consider the aspects of the neonatal immune system which might contribute to the dev

132 de additional support for the novel theme in neonatal immunology that immunosuppression is essential

133 expression analysis of rat brains following neonatal infection showed increased expression of kynure

134 The underlying mechanism of neonatal infection susceptibility has generally been ass

135 Thus, our study challenges the notion that neonatal infection susceptibility is due to immune cell-

136 of more severe symptoms such as eye disease, neonatal infection, and, in rare cases, encephalitis.

137 Neonatal infections are a leading cause of childhood mor

138 We estimated the number of incident neonatal infections by maternal age, and we generated se

139 tted, have been implicated in preterm birth, neonatal infections, and chronic lung disease of prematu

140 ed a significantly modified program in these neonatal iNKT cells that ultimately led to their maligna

141 0.51, 95% CI 0.28 to 0.90; p=0.0210), fewer neonatal intensive care admissions lasting more than 24

142 infancy, cesarean delivery, breast-feeding, neonatal intensive care unit [NICU] admission, and absen

143 ntilation and respiratory support during the neonatal intensive care unit admission.

144 weeks]) with various stages of ROP: 3 in the neonatal intensive care unit and 1 in the operating room

145 ical center among 4 neonates with ROP in the neonatal intensive care unit and in the operating room.

146 inborn babies with type 1 zone 1 ROP at the Neonatal Intensive Care Unit of the Catholic University,

147 To determine the proportion of neonatal intensive care units (NICUs) in 2014 that achie

148 significant PDA was conducted at 3 tertiary neonatal intensive care units and affiliated follow-up p

149 The setting was neonatal intensive care units at The Children's Hospital

150 All neonates admitted to 24 participating neonatal intensive care units from four countries (Austr

151 between January 2006 and December 2013 from neonatal intensive care units in 25 US children's hospit

152 n between 2010 and 2011 who were admitted to neonatal intensive care units participating in the Canad

153 nificant cause of morbidity and mortality in neonatal intensive care units.

154 Retrospective cohort study at tertiary neonatal intensive care units.

155 inical trial performed at 33 US and Canadian neonatal intensive care units.

156 SI; (4) management of staphylococcal BSIs in neonatal intensive care units; and (5) defining the impa

157 s variability as requests for resuscitation, neonatal intensive care, and surgical intervention are b

158 Clinicians were masked to neonatal interstitial glucose concentrations; outcome as

159 maging in conscious, unrestrained mice after neonatal intracranial or intravascular administration of

160 Neonatal invasive candidiasis (IC) presenting in the fir

161 evaluate the feasibility of MEP recording in neonatal lambs and test its validity.

162 re and ameliorating infection in rodents and neonatal lambs.

163 yet this was to our knowledge never done in neonatal lambs.

164 ice and this phenotype is largely rescued by neonatal leptin treatments.

165 ae associated with CLP have been hampered by neonatal lethality.

166 niofacial anomalies, skeletal dysplasia, and neonatal lethality.

167 Neonatal liver disease is an important source of morbidi

168 gist who may be faced with interpretation of neonatal liver imaging.

169 stonia syndrome with frequent and reversible neonatal liver involvement and a strikingly homogenous c

170 No neonatal losses or cases of congenital botulism were rep

171 (PIN) progressed to carcinoma in rats given neonatal low-dose BPA with adult T+E but not in rats giv

172 prenatal iAs exposure and alterations in the neonatal metabolome could reveal critical molecular modi

173 levels were still reduced in hUGT1/Car(-/-) neonatal mice because of ROS induction of intestinal UGT

174 luciferase/eGFP reporter (TFAR) cassettes to neonatal mice enabling longitudinal TFAR profiling by co

175 ransient asymptomatic infection in adult and neonatal mice even at doses 100-fold higher than the LD5

176 However, motor neurons from neonatal mice lacking VIAAT in Renshaw cells received sp

177 virus that causes severe thymic necrosis in neonatal mice, characterized by a loss of CD4(+) T cells

178 oduced fewer symptoms and lower mortality in neonatal mice, resulting in an attenuated form of biliar

179 In neonatal mice, rhesus rotavirus (RRV) can induce biliary

180 pulmonary inflammation but not mortality in neonatal mice, suggesting that death in these mice was n

181 ate locomotor networks in the spinal cord of neonatal mice.

182 as evaluated in vivo by using humanized TLR8 neonatal mice.

183 of neonatal and adult mice, we show that the neonatal microbiota is unable to prevent colonization by

184 curred when Clostridiales were absent in the neonatal microbiota.

185 (ZIKV) infection has led to descriptions of neonatal microcephaly cases.

186 c fluid of ZIKV-positive pregnant women with neonatal microcephaly.

187 dual bias, including survival bias and major neonatal morbidities arising before exposure to ligation

188 natal mortality, but associations with other neonatal morbidities remain understudied.

189 Preterm birth (PTB) is the leading cause of neonatal morbidity and mortality.

190 =3 in the Foley catheter group); and five of neonatal morbidity, comprising birth asphyxia (n=3), sep

191 the risk of stillbirth and infant death and neonatal morbidity.

192 ildren was mediated through asphyxia-related neonatal morbidity.

193 In the mid-2000s, neonatal mortality accounted for almost 40% of deaths of

194 major demographic metrics (adult mortality, neonatal mortality and birthrate) between the two period

195 d deaths to produce estimates of under-5 and neonatal mortality at a resolution of 5 x 5 km grid cell

196 wed significant reductions in stillbirth and neonatal mortality but did not report the overall effect

197 provided significantly greater reductions in neonatal mortality for female neonates compared with mal

198 erogeneity in absolute levels of under-5 and neonatal mortality in 2015, as well as the annualised ra

199 l and child healthcare led to a reduction in neonatal mortality of almost the hypothesized 25% in sma

200 The neonatal mortality rate in Andhra Pradesh was 44 per 1,0

201 n Africa has the world's highest under-5 and neonatal mortality rates as well as the highest naturall

202 ection-associated PTB, 101.10 prevented PTB, neonatal mortality, and fetal brain inflammation.

203 Preterm birth is the leading cause of neonatal mortality, and is frequently associated with in

204 nt Goal 2030 targets for maternal mortality, neonatal mortality, and mortality in children younger th

205 substantial and growing share of under-5 and neonatal mortality, but they are largely neglected in th

206 nificantly increased risk of pregnancy loss, neonatal mortality, or malformation.

207 ated with neurodegeneration and prenatal and neonatal mortality, which could be due to excess cell de

208 cal factor and climate factors for adult and neonatal mortality, while birthrate was not affected by

209 d lead to a reduction of the order of 25% in neonatal mortality.

210 available or feasible in countries with high neonatal mortality.

211 ion of maternal cancer during pregnancy with neonatal mortality.

212 ca achieved marked reductions in under-5 and neonatal mortality.

213 Few data exist for neonatal mortality.

214 The primary outcome was neonatal mortality.

215 ntly 10.7% of under-5 mortality and 15.1% of neonatal mortality.

216 y +8 mV, producing a maximal +34-mV shift in neonatal mouse cardiac myocytes or Chinese hamster ovary

217 R-199a-3p and hsa-miR-590-3p both in primary neonatal mouse cardiomyocytes and in vivo.

218 Sumoylation of FOXP2 in neonatal mouse cerebellum regulates Purkinje cell develo

219 The neonatal mouse heart can regenerate, but only during the

220 In this study, a neonatal mouse model of critical pertussis is characteri

221 acetylases (HDACs) in rod differentiation in neonatal mouse retinas, we used a pharmacological approa

222 to a rapidly progressing and fully penetrant neonatal myeloid disease.

223 ive care units participating in the Canadian Neonatal Network and completed follow-up assessments in

224 tionally inactivated the murine Pten gene in neonatal neural stem/progenitor cells.

225 ation of the Nkx2.5 enhancer and showed that neonatal Nkx2.5+ cardiomyoblasts mature into cardiomyocy

226 Genome-wide expression profile of neonatal Nkx2.5+ cardiomyoblasts showed the absence of s

227 l and could have interesting applications in neonatal nutrition, but also as brain-protective, hepato

228 re early-onset phenotypes, occasionally with neonatal onset epilepsy and developmental impairment, as

229 ligation among preterm infants with adverse neonatal outcomes and neurodevelopmental impairment in e

230 type 1 diabetes is associated with improved neonatal outcomes, which are likely to be attributed to

231 ator groups on maternal GBS colonization and neonatal outcomes.

232 nts to mediate associations between PFAS and neonatal outcomes.

233 miRNA profiling in plasma samples from neonatal PA patients and age-matched control individuals

234 the percentage of polyhormonal cells in the neonatal pancreata of Snord116p-/m+ mice, due primarily

235 group B Streptococcus [GBS]) is an important neonatal pathogen and emerging cause of disease in adult

236 t of bulk motion is reduced in a non-sedated neonatal patient and (b) where the observation of the im

237 38% (MRR, 0.62; 95% CI, .46-.83) within the neonatal period and 16% (0.84; .71-1.00) by age 12 month

238 ; 80 in control clusters), 77 died after the neonatal period and before 18 months (31 in intervention

239 the majority of childhood deaths in the post-neonatal period are caused by infections that can be eff

240 al to the maturation of aerodigestion in the neonatal period but also unlikely to co-occur with imita

241 we observed a very beneficial effect in the neonatal period.

242 for infants with severe infection during the neonatal period.

243 mplications to mortality decreased after the neonatal period; congenital abnormalities remained an im

244 y provides benefits during the perinatal and neonatal period; however, it may not provide additional

245 dy, we investigate early immune responses to neonatal porcine islet (NPI) xenografts compared with rh

246 Only TLN-treated human endothelial cells and neonatal porcine islets prolonged time to clot formation

247 Ex vivo intradermal neonatal porcine skin penetration of VD3 NMP from bilaye

248 ons, are associated with adverse maternal or neonatal pregnancy outcomes.

249 Evidence suggests that repetitive neonatal procedural pain precedes long-term alterations

250 Two of the girls had pronounced neonatal progeroid features and were initially diagnosed

251 ast, cardiomyocytes failed to reactivate the neonatal proliferative network following infarction, whi

252 odissected tissues, as well as expression in neonatal prostate.

253 Consequently, neonatal pups died at birth due to respiratory insuffici

254 METHODS AND Neonatal rat atrial cardiomyocyte monolayers expressing

255 We also used a neonatal rat cardiomyocyte culture system to elucidate t

256 ardial delivery of the progenitor cells into neonatal rat hearts, in vivo incubation and analysis.

257 ability and electrophysiological property of neonatal rat ventricular myocyte (NRVM) cultures.

258 lso observed in hERG-HEK cells as well as in neonatal rat ventricular myocytes treated with the musca

259 THODS AND Patch-clamp recordings of cultured neonatal rat ventricular myofibroblasts revealed that TG

260 Escherichia coli K1 can be replicated in the neonatal rat.

261 and after VA supplementation.Sprague-Dawley neonatal rats (n = 104) were nursed by mothers fed a VA-

262 Further, we found that LCS treated neonatal rats have higher intestinal expressions of Ki67

263 ncluding skin, brain, and adipose tissue, in neonatal rats without and after VA supplementation.Sprag

264 etained efficiently in peripheral tissues of neonatal rats, suggesting that a more frequent, lower-do

265 attach to ciliated cells, it activates human neonatal regulatory B cells, thereby inhibiting immunolo

266 titute of Child Health and Human Development Neonatal Research Network between October 2010 and Janua

267 damage-are also present in several critical neonatal respiratory disorders.

268 eatment (with oral amoxicillin) of suspected neonatal respiratory infections, were linked with tradit

269 blood spot samples obtained from the Danish Neonatal Screening Biobank and genotyped using the Illum

270 s, stem cell transplantation facilities, and neonatal screening programs.

271 infection as well as the lack of large-scale neonatal screening tools.

272 is 89% and 83% for cut-offs of 10ng/mL (for neonatal sepsis and pelvic inflammatory disease) and 30n

273 Precise estimates of neonatal sepsis burden vary by setting.

274 e agent influence the clinical expression of neonatal sepsis.

275 ing efforts designed to reduce the burden of neonatal sepsis.

276 ent facilitates the accumulation of Tregs in neonatal skin and that upon skin entry these cells local

277 , the mechanisms mediating Treg migration to neonatal skin are unknown.

278 , to be preferentially expressed by Tregs in neonatal skin.

279 f development, but also in later fetal/early neonatal stages of maturation.

280 f any postnatal age, including perinatal and neonatal stages, with precise spatiotemporal control but

281 d skin; R(2) = 0.038), this was not true for neonatal stool (meconium; Mann-Whitney P > 0.05), and th

282 c value in terms of congenital infection and neonatal symptomatic disease.

283 The strong age dependency of neonatal systemic infection with Escherichia coli K1 can

284 inded single-center trial was conducted in a neonatal tertiary referral center in Germany.

285 Neonatal testes (P1) were collected for the detection of

286 However, we found neither a neonatal testosterone rise in the boys nor a gender-spec

287 Our results demonstrate that neonatal THS exposure decreases bodyweight and that THS

288 for stem cell function in multiple fetal and neonatal tissues, including the nervous system.

289 eady VA concentrations in rapidly developing neonatal tissues.

290 ing the Tmc1(Bth) allele into the cochlea of neonatal Tmc1(Bth/+) mice substantially reduced progress

291 neurons in the basolateral amygdala (BLA) at neonatal to adult developmental stages in mice.

292 ntation with IgG-IC was sufficient to induce neonatal tolerance.

293 aining the developmental and neural basis of neonatal tongue protrusion has important implications fo

294 Hypoglycemia is common during neonatal transition and may cause permanent neurological

295 uced stress in the heart during the fetal-to-neonatal transition.

296 For stillbirth, low Apgar score, and neonatal unit admission, risk also increased from the 85

297 LPT infants were recruited at birth from the neonatal unit and postnatal wards of the Royal Women's H

298 pgar score <7 at 5 min, and admission to the neonatal unit.

299 adjusted P = 0.13).This study suggested that neonatal vitamin D status does not influence subsequent

300 Neonatal white matter injury (NWMI) is a lesion found in