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1 lly, is reported to be rare or absent in the neonatal animal.
2 is the major form expressed in the fetal and neonatal animal.
3 ation of the developing lung in utero and in neonatal animals.
4 bset found in adults is missing in fetal and neonatal animals.
5 ctions of identified skin sensory neurons in neonatal animals.
6 ld induce in the retinocollicular pathway in neonatal animals.
7 s hypothalamic GnRH release in embryonic and neonatal animals.
8 e of the major causes of immunodeficiency in neonatal animals.
9 linked to widespread neuronal cell death in neonatal animals.
10 oform is the only one expressed in the fetal/neonatal animal and showed that expression was limited t
11 in the CNS of adult as well as embryonic and neonatal animals and can differentiate into lineage-rest
12 ve been documented to induce neutrophilia in neonatal animals and human infants, increase the NSP, an
13 utrophils have been consistently reported in neonatal animals and humans and contribute to their susc
14 nts of pure olfactory ensheathing cells from neonatal animals and mixed olfactory cells from both neo
19 lt animals [past postnatal day (P)90] and in neonatal animals as early as P27 formed a dense band in
20 rebral artery myocytes isolated from control neonatal animals but were absent in myocytes from Kir2.1
21 and dysmorphogenesis, which were evident in neonatal animals by anatomical and micro-computed tomogr
22 uggest that anesthetic drugs administered to neonatal animals cause widespread neuronal apoptosis and
25 dicate that in the absence of RFC1 function, neonatal animals die due to failure of hematopoietic org
26 ordial follicles by 30% when administered to neonatal animals during the time of germline cyst breakd
27 We recently demonstrated high-dose BMT in neonatal animals enables chimeric engraftment without to
28 e been at high risk of BSE infection only if neonatal animals had inadvertently ingested contaminated
31 ncy of IRF-3 and IRF-7 was age dependent, as neonatal animals lacking either factor succumbed to infe
33 intrinsic immaturity of the immune system of neonatal animals, neonates are highly sensitive to a var
35 quickly, that rapid turnover occurs only in neonatal animals, only in culture, or only in response t
37 the proteins in all neurons from ganglia of neonatal animals (postnatal days 0-3) and in 85-90 % of
40 ation of beta-catenin in supporting cells in neonatal animals resulted in proliferation of supporting
42 cardiography in all fetal lambs (n = 13) and neonatal animals studied at one and three days of life (
44 wo groups of retrogradely labeled neurons in neonatal animals, those neurons with axons that ascend t
45 e progress of the reformation of this map in neonatal animals under conditions that enhanced the like
46 ed, and single-cell genomic PCR studies in a neonatal animal were used to examine any relationship be
49 sraphisms are progressively resolved and the neonatal animals, while showing signs of scarring and ti
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