ライフサイエンスコーパス: neonatal hypoxia

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1 NG2 cells in a mouse model of DWMI (chronic neonatal hypoxia).

2 hreshold, thus mimicking clinical aspects of neonatal hypoxia.

3 studies demonstrate for the first time that neonatal hypoxia affects the Foxo1/p27(Kip1) pathway dur

4 Neonatal hypoxia alters the development of the hypoxic v

5 Since we hypothesized that neonatal hypoxia alters the hypoxic ventilatory response

6 cimens of patients with PVL and in models of neonatal hypoxia and ischemia.

7 ther evaluated as a treatment strategy after neonatal hypoxia and leaders in the field of neonatology

8 Although the adverse effects of neonatal hypoxia associated with premature birth on the

9 In contrast, neonatal hypoxia had no lasting effect on hypoxic ventil

10 rocyte (OL) regeneration in a mouse model of neonatal hypoxia (HX) that reproduces diffuse white matt

11 Here we demonstrate that neonatal hypoxia impairs the hypoxic ventilatory respons

12 Normoxic ventilation was unaffected by neonatal hypoxia in either sex at 7-9 weeks of age (P >

13 Phrenic responses were unaffected by neonatal hypoxia in rats of either sex (P > 0.05), sugge

14 n response to 12% O(2), male rats exposed to neonatal hypoxia increased ventilation less than untreat

15 the SDF-1/CXCR4 axis in the pathogenesis of neonatal hypoxia-induced cardiopulmonary remodeling and

16 ts of either sex (P > 0.05), suggesting that neonatal hypoxia-induced plasticity occurs between the p

17 on, exosomes derived from neonatal normoxia, neonatal hypoxia, infant hypoxia, and child hypoxia sign

18 Neonatal hypoxia-ischemia (H-I) is the leading cause of

19 ion and NMDAR tyrosine phosphorylation after neonatal hypoxia-ischemia (HI) and investigated the neur

20 lamide in both severe and moderate models of neonatal hypoxia-ischemia (HI) in postnatal day 10 Sprag

21 Neonatal hypoxia-ischemia (HI) is a common cause of brai

22 Neonatal hypoxia-ischemia (HI) is an important clinical

23 delayed and ongoing neurodegeneration after neonatal hypoxia-ischemia (HI), but the mechanisms and t

24 in several models of brain injury, including neonatal hypoxia-ischemia (HI).

25 nisms of cell death account for damage after neonatal hypoxia-ischemia (HI).

26 o investigate the role of TRAIL signaling in neonatal hypoxia-ischemia (HI).

27 ental adult rat focal ischemia and in a mild neonatal hypoxia-ischemia (HI, 90 min hypoxia) rat model

28 We conclude that neonatal hypoxia-ischemia causes highly organized, syste

29 S) shRNA into the hippocampus of rats before neonatal hypoxia-ischemia decreased vulnerability to inj

30 We tested G-CSF in a rat model of neonatal hypoxia-ischemia in postnatal day 7 unsexed rat

31 Neonatal hypoxia-ischemia in the preterm human leads to

32 h to ER stress and hypoxia in vitro and in a neonatal hypoxia-ischemia model in vivo.

33 In vivo, neonatal hypoxia-ischemia reciprocally altered Bcl-x pre

34 Neonatal hypoxia-ischemia was induced by ligation of the

35 oubles surviving tissue in a severe model of neonatal hypoxia-ischemia, a major cause of neonatal dea

36 r protein expression increased rapidly after neonatal hypoxia-ischemia, in concert with cleavage of p

37 In a well characterized model of neonatal hypoxia-ischemia, we demonstrate marked but del

38 iorate moderate to severe ischemic damage in neonatal hypoxia-ischemia.

39 echanism for abnormal neurodevelopment after neonatal hypoxia-ischemia.

40 Neonatal hypoxia/ischemia (H/I) was induced in P6 rat pu

41 rization was reduced by 40% in the retina of neonatal hypoxia model using IRS-1-/- mice.

42 and functional recovery in a swine model of neonatal hypoxia-reoxygenation.

43 We demonstrate that neonatal hypoxia results in hypomyelination and delayed

44 ses in the young animals could be related to neonatal hypoxia tolerance.

45 Using a mouse model of neonatal hypoxia, we demonstrate a biphasic effect on ol

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