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1 al age, low birth weight, preterm birth, and neonatal infections).
2 is, this pathogen remains a leading cause of neonatal infection.
3 smission, and GBS remains a leading cause of neonatal infection.
4 itis is strongly linked to preterm birth and neonatal infection.
5 le in inhibition of antibody responses after neonatal infection.
6 o preterm births, stillbirths, or late-onset neonatal infections.
7 HSV-2 to generate annual numbers of incident neonatal infections.
8 ositive bacterium that is a leading cause of neonatal infections.
9 ease morbidity and mortality associated with neonatal infections.
10 oup B streptococci (GBS), the major cause of neonatal infections.
11 host defenses may attenuate the virulence of neonatal infections.
12 g the most common causes of life-threatening neonatal infections.
13 ) is the foremost bacterial cause of serious neonatal infections.
14 ses from 372 centres, including 107 maternal-neonatal infections, 427 cases of bacteraemia, and 252 c
17 that reported effect measures on the risk of neonatal infection among newborns exposed to maternal in
19 sistant HSV-2 mutants can develop rapidly in neonatal infection and cause clinically significant dise
20 implicated as one of the causative agents in neonatal infection and causes a septicemia thought to be
23 ht represent a therapeutic tool for treating neonatal infections and support the view that breastfeed
24 gnostic methods for identifying maternal and neonatal infection, and regarding optimal prevention and
25 of more severe symptoms such as eye disease, neonatal infection, and, in rare cases, encephalitis.
26 tted, have been implicated in preterm birth, neonatal infections, and chronic lung disease of prematu
30 h (aRR, 1.25; 95% CI, 1.13-1.30), and having neonatal infections (aRR, 1.42; 95% CI, 1.17-1.73), any
31 to term is associated with increased risk of neonatal infection, but immediate delivery is associated
32 hat developed tumors 7 to 8 months following neonatal infection by polyomavirus (PYV) wild-type strai
35 ossible differences or clinical relevance of neonatal infection caused by different biotypes or newer
36 We interpret these data to indicate that neonatal infection causes significant neuronal sequestra
41 rnal antibody concentrations and the risk of neonatal infection has been investigated in US and Afric
44 ished that G10P[11] RVs are a major cause of neonatal infection in Vellore, India, with half of infec
45 ts define an important role for gamma34.5 in neonatal infections in contrast to other studies indicat
46 at emerged as the leading cause of bacterial neonatal infections in Europe and North America during t
49 line treatment of serious community-acquired neonatal infections in rural Bangladesh, which has a mod
51 e role for aberrant neuronal iron storage in neonatal infection-induced disturbances in myelination a
53 e vaccine, and antibody generated by primary neonatal infection is poorly protective against reinfect
62 owel in adult mammals, but their role during neonatal infection of the small bowel is not well establ
64 ne-encoded TCR clonotypes, characteristic of neonatal infection, persisted in the brain, albeit somet
65 ncluded histories of perinatal difficulties, neonatal infections, postnatal brain infections, and tra
66 ternal infection, vertical transmission, and neonatal infection published from January 1, 1960 to Mar
67 size that acute brain inflammation caused by neonatal infection reduces the bioavailability of iron r
70 s suggests that the impact of these cells on neonatal infection risk and progression may be limited.
71 expression analysis of rat brains following neonatal infection showed increased expression of kynure
73 ist, aims to improve scientific reporting of neonatal infection studies, increasing data utility and
74 proved scientific reporting of observational neonatal infection studies, to increase comparability an
76 Thus, our study challenges the notion that neonatal infection susceptibility is due to immune cell-
78 y has decreased the incidence of early-onset neonatal infection, these measures do not prevent ascend
79 on and oral antibiotic therapy for suspected neonatal infections to a basic preventive and promotive
80 further evaluated for potential relevance to neonatal infection, transplantation, and acquired immuno
83 Although few congenital malformations or neonatal infections were seen among exposed neonates, wo
84 vents that occur in the brain as a result of neonatal infection, which likely contribute to cognitive
85 tudied the induction of adaptive immunity to neonatal infection with a murine retrovirus, under condi
86 contrast to infection of mature BALB/c mice, neonatal infection with rhinovirus promotes an IL-25-dri
88 ith ruptured membranes close to term reduces neonatal infection without increasing other morbidity.
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