ライフサイエンスコーパス: neonatal mice

1 s, and no equivalent function was evident in neonatal mice.

2 ated gene) K(+) current in Purkinje cells of neonatal mice.

3 ulate locomotor output in the spinal cord of neonatal mice.

4 of caged glutamate in medullary slices from neonatal mice.

5 -4497 did not influence Inhba mRNA levels in neonatal mice.

6 e a mutation to explain the sld phenotype in neonatal mice.

7 roles for leptin receptor (LepRb) signals in neonatal mice.

8 re pathogenic in a passive transfer model in neonatal mice.

9 onocytes in adults, with similar findings in neonatal mice.

10 to the nucleus of alveolar macrophages from neonatal mice.

11 foundly mitigated induced NEC-like injury in neonatal mice.

12 mu2 amino acid 208 modulates myocarditis in neonatal mice.

13 ansmit MCMV from latent mothers to breastfed neonatal mice.

14 e and children and causes biliary atresia in neonatal mice.

15 a are markedly more vulnerable to axotomy in neonatal mice.

16 tor neurons when injected intravenously into neonatal mice.

17 ed defective migration into the lungs of the neonatal mice.

18 y enhancing regulation of the UGT1A1 gene in neonatal mice.

19 ing rhythmically active in vitro slices from neonatal mice.

20 as evaluated in vivo by using humanized TLR8 neonatal mice.

21 the cartilage of mineralizing vertebrae from neonatal mice.

22 lymphatic development in multiple organs of neonatal mice.

23 ion was tested with congenic C57BL/10 H-2(d) neonatal mice.

24 an in vitro medullary slice preparation from neonatal mice.

25 eloped that was successful in both adult and neonatal mice.

26 ha2 were attenuated in diarrhea induction in neonatal mice.

27 fetus, as well as higher mortality rates in neonatal mice.

28 l enterotoxin capable of causing diarrhea in neonatal mice.

29 tors (SAIs) that innervated the back skin of neonatal mice.

30 a transcutaneous, intrahepatic injection in neonatal mice.

31 , are expressed within the preBotC region of neonatal mice.

32 the period of active glomerular formation in neonatal mice.

33 election of a virus that caused paralysis in neonatal mice.

34 enous delivery of 1x10(11) vector genomes in neonatal mice.

35 l-induced neurodegeneration in the brains of neonatal mice.

36 more virulent than the Wyeth strain virus in neonatal mice.

37 promoted both type 1 and type 2 responses in neonatal mice.

38 ave examined alveolar macrophage activity in neonatal mice.

39 a has been shown in experiments conducted in neonatal mice.

40 isolated from many species, as well as from neonatal mice.

41 te as well as in the calvaria osteoblasts of neonatal mice.

42 in fragment C (Frag C) as a model antigen in neonatal mice.

43 l line-derived neurotrophic factor (GDNF) to neonatal mice.

44 into the subretinal space of LCA8-like model neonatal mice.

45 ivery to adult mice and systemic delivery to neonatal mice.

46 i-influenza virus response in both adult and neonatal mice.

47 d the frequency of iBALT to that observed in neonatal mice.

48 to induce TH1 responses after vaccination of neonatal mice.

49 in the injured hearts of both zebrafish and neonatal mice.

50 duct when microinjected into the kidneys of neonatal mice.

51 ant within a subunit tuberculosis vaccine in neonatal mice.

52 1-MMP expression pattern seen in rabbits and neonatal mice.

53 lungs, spleen, and lymph nodes of adult and neonatal mice.

54 ate locomotor networks in the spinal cord of neonatal mice.

55 fore or after RSV administration in adult or neonatal mice.

56 sive transfer of human anti-Dsg IgG4 mAbs to neonatal mice.

57 n by the protozoan Cryptosporidium parvum in neonatal mice.

58 rocedure to induce cardiac injury in 1-d-old neonatal mice.

59 ction, and improved survival above wild-type neonatal mice.

60 Transgenic (TG) and wild-type (WT) neonatal mice (10 mice per group) were exposed either to

61 thetic ligand for TLR-3, was administered to neonatal mice 14 h before cerebral HI.

62 In neonatal mice A alternata exposure induced higher serum

63 4Ralpha ASOs during primary RSV infection in neonatal mice abolished the pulmonary dysfunction normal

64 f AAV8-CTLA-4Ig and AAV8-CD40Ig vectors into neonatal mice achieved a synergistic effect and the best

65 Fetal monocytes transferred to the lung of neonatal mice acquired an AMF phenotype via defined deve

66 CD4-deficient neonatal mice adoptively transferred with CD4(+) cells f

67 e, HVEM was sufficient to mediate disease in neonatal mice after direct intracranial inoculation, and

68 h the recombinant HBc-E2 particles protected neonatal mice against lethal EV71 and CA16 infections.

69 Moreover, chronic ghrelin exposure in neonatal mice also attenuated leptin-induced STAT3 signa

70 t-natal life, we ablated Ihh in cartilage of neonatal mice and assessed the consequences on temporoma

71 Blocking IL-13 after AAD was established in neonatal mice and did not reduce remodeling or IL-33 lev

72 nized by cell type, observed in RSV-infected neonatal mice and draw comparisons (when possible) to hu

73 ogically enriched CD71(+) erythroid cells in neonatal mice and human cord blood have distinctive immu

74 iation of cultured ENS progenitor cells from neonatal mice and humans.

75 entially homed to extrahepatic bile ducts of neonatal mice and invaded their epithelial lining.

76 nfluenza virus is qualitatively different in neonatal mice and may contribute to an increased morbidi

77 Thus, AFB1-induced HCC development in neonatal mice and p53+/- mice may be due to "immature" c

78 In cold thermosensors of neonatal mice and rats, ATP receptors were functionally

79 t5b-CA) allowed for Treg cell development in neonatal mice and restored Treg cell numbers in Cd28(-/-

80 h a novel model of tendon regeneration using neonatal mice and show that neonates heal via formation

81 D4(+) T cells are required for protection of neonatal mice and that this protection may not require C

82 These populations began to expand in neonatal mice and, upon malignant transformation, result

83 are expressed widely throughout the brain of neonatal mice, and genetic deletion of both AC1 and AC8

84 ased contractility during acute infection of neonatal mice, and persistent viral infection in the hea

85 The SSEA-1(+) PSCs were highly prevalent in neonatal mice, and they were rare in adult mice.

86 egeneration among model organisms, including neonatal mice, appear remarkably similar.

87 In conclusion, liver myeloid cells in neonatal mice are an important source of proinflammatory

88 Unlike adult mammals, zebrafish and neonatal mice are capable of heart regeneration followin

89 We find that IL-18-null neonatal mice are highly protected from polymicrobial se

90 However, we recently reported that neonatal mice are highly resistant to orogastric infecti

91 Neonatal mice are highly vulnerable to influenza and onl

92 Neonatal mice are lymphopenic within the first week of l

93 We report that neuromuscular junctions in neonatal mice are significantly more resistant to both h

94 Undernutrition in neonatal mice, as well as in weaned mice, intensified in

95 IL-4Ralpha were administered intranasally to neonatal mice at the time of primary infection.

96 levels were still reduced in hUGT1/Car(-/-) neonatal mice because of ROS induction of intestinal UGT

97 Evoked postsynaptic currents, recorded from neonatal mice between postnatal day 1 (P1) and P4, incre

98 tetanus toxoid conjugate (dV-TT), and 98% of neonatal mice born to dams vaccinated with dV-TT survive

99 and fusion (F, pMSINH-FdU) glycoproteins in neonatal mice born to naive and measles-immune mothers.

100 We found that allergen exposure in neonatal mice, but not in adult mice, elevated the level

101 with fibroblasts derived from embryonic and neonatal mice, but remained similar for fibroblasts from

102 etinal neovascularization was established in neonatal mice by exposing them to 75% O(2) at postnatal

103 NF-alpha-/- and wild-type (WT) control C57B6 neonatal mice by exposure to 75% oxygen between postnata

104 Oxygen-induced retinopathy was generated in neonatal mice by exposure to 75% oxygen from postnatal d

105 ypoxia-induced cardiopulmonary remodeling in neonatal mice, by decreasing progenitor cell recruitment

106 We found that the hearts of 1-day-old neonatal mice can regenerate after partial surgical rese

107 al development, and both adult zebrafish and neonatal mice can regenerate cardiac muscle after injury

108 Thymectomy of neonatal mice can result in the development of autoimmun

109 We tested the hypothesis that neonatal mice carrying a null mutation for neuronal nitr

110 SPDEF or FOXA3 in airway epithelial cells in neonatal mice caused goblet cell differentiation, sponta

111 Sodium valproate (VPA) administered to neonatal mice causes cognitive and motor deficits simila

112 We show that neonatal mice challenged with arenavirus Tacaribe (TCRV)

113 virus that causes severe thymic necrosis in neonatal mice, characterized by a loss of CD4(+) T cells

114 n of T(reg) cells in the periphery of normal neonatal mice closely paralleled that seen for IL-2Rbeta

115 clearance of the organisms from the lungs of neonatal mice compared to adults.

116 nificantly lower in the lungs of PC-infected neonatal mice compared with adults.

117 anti-tetanus toxoid (TT) vaccine response of neonatal mice could be enhanced by retinoic acid (RA), a

118 oreover, the administration of disulfiram to neonatal mice decreased in vivo cell proliferation in th

119 Remarkably, neutrophil depletion in neonatal mice decreased the expression of the B cell act

120 numbers of facial motoneurons are doubled in neonatal mice deficient in either ZPK/DLK (zipper protei

121 Neonatal mice deficient in IkappaB-alpha, an inhibitor o

122 infection was sufficient to transfer MCMV to neonatal mice, demonstrating that breast milk was a sour

123 ction of antagomirs specific to miR-26a into neonatal mice derepressed both Smad expression and activ

124 summary, the myocardial cryoinjury model in neonatal mice described here is a useful tool for cardia

125 It indicates that, with time, neonatal mice develop full protection after intranasal i

126 srupted, lipid absorption was perturbed, and neonatal mice died within days of birth.

127 Subcutaneous injection of jacalin into neonatal mice drastically reduced PF IgG deposition at t

128 However, in neonatal mice EMG instrumentation could induce stress, a

129 homolog (PTEN) in the sensorimotor cortex of neonatal mice enables regeneration of corticospinal trac

130 luciferase/eGFP reporter (TFAR) cassettes to neonatal mice enabling longitudinal TFAR profiling by co

131 mmatory mediators, and oxidative stress when neonatal mice encounter severe hyperbilirubinemia.

132 ransient asymptomatic infection in adult and neonatal mice even at doses 100-fold higher than the LD5

133 However, both old and especially neonatal mice exhibited significant decreases in TCR div

134 cardiomyocyte ablation, we demonstrated that neonatal mice expand a population of embryonic-derived r

135 vels are significantly increased in lungs of neonatal mice exposed to hyperoxia.

136 Neonatal mice exposed to normoxia or hypoxia were random

137 ng during ongoing locomotor-like activity in neonatal mice expressing archaerhopsin-3 (Arch), halorho

138 In neonatal mice expressing normal amounts of PHD2, exposur

139 stridiales, but not Bacteroidales, protected neonatal mice from pathogen infection and abrogated inte

140 and neonatal beta(7)(-/-) mice and adult and neonatal mice given blocking Abs to alpha(4)beta(7), MAd

141 IL-33 levels were quantified in neonatal mice given inhaled house dust mite (HDM), and t

142 We found that rotavirus infection of neonatal mice has a unique tropism to bile duct cells, a

143 f postnatal window for heart regeneration in neonatal mice has led to the establishment of surgical m

144 SCD2-deficient (Scd2-/-) neonatal mice have a skin permeability barrier defect an

145 Hyperoxia-exposed neonatal mice have increased caspase-1 activation, IL1be

146 logically identified skin sensory neurons in neonatal mice have shown that this region also receives

147 Neonatal mice have the capacity to regenerate their hear

148 We have studied the airway bioelectrics of neonatal mice homozygous for a null allele of Tmem16a (T

149 s/site, or a total of 500,000 cells) into 18 neonatal mice homozygous for a targeted deletion (knocko

150 Our findings demonstrate in neonatal mice how gut flora synergizes with poly(I:C) to

151 ed in increased clearance of the organism in neonatal mice; however, there was decreased expression o

152 Injection of scFvs into neonatal mice identified 2 pathogenic scFvs that caused

153 Thus, in neonatal mice, IL-12, which accumulates in the environme

154 Neonatal mice immunized intranasally with VP6/LT(R192G)

155 We found that neonatal mice immunized only once with the attenuated st

156 Neonatal mice immunized with a single dose of vaccine in

157 Inhibition of DDX3 activity or expression in neonatal mice impaired dendritic outgrowth and spine for

158 usceptibility to allergic airways disease in neonatal mice in a model of maternal transmission of ast

159 erve motor activity in medullary slices from neonatal mice in conditions where periods between succes

160 ex vivo somatosensory system preparations in neonatal mice in the hope that their small size might al

161 del for the study of cardiac regeneration in neonatal mice in which cryoinjury is used to induce hear

162 r constructs in transgenic mouse embryos and neonatal mice, in this report we define a minimal 1400 b

163 Surprisingly, in adult frogs and mice and in neonatal mice, in vivo and in vitro, the stereocilia wer

164 Neonatal mice incapable of classical or terminal complem

165 Leptin injection in neonatal mice increased bone morphogenic protein (BMP) s

166 ed with mature mice, rhinovirus infection in neonatal mice increased lung IL-13 and IL-25 production,

167 st BP180, Dsg1, and Dsg3, when injected into neonatal mice, induce the BP, PF, and PV disease phenoty

168 Neonatal mice injected with poly I:C were found to have

169 Neonatal mice injected with rabbit anti-mouse BP180 (mBP

170 Additionally, in wild-type neonatal mice inoculated intracranially, HSV antigen did

171 . sp. muris (PC) organisms from the lungs of neonatal mice is delayed due to failure of initiation of

172 Inducible endothelial Mekk3 knockout in neonatal mice is lethal due to multiple intracranial hae

173 ility of neuromuscular junctions observed in neonatal mice is not determined by the maturity of the s

174 parvum, CCL20 production by the intestine of neonatal mice is reduced by a mechanism independent both

175 tory response to CO(2), we hypothesized that neonatal mice lacking 60-70% of their 5-HT neurones (Pet

176 In studies with neonatal mice lacking access to bacterial sphingolipids,

177 liferation was not significantly impaired in neonatal mice lacking AQP1, as quantified in flat-mounte

178 lacking CCM2 and in the lymphatic vessels of neonatal mice lacking HEG1 were associated with abnormal

179 However, motor neurons from neonatal mice lacking VIAAT in Renshaw cells received sp

180 on with respiratory syncytial virus (RSV) in neonatal mice leads to exacerbated disease if mice are r

181 inhibitory effect on angiogenesis process in neonatal mice lung following exposure to hyperoxia.

182 In 1-wk-old neonatal mice, MF59 recruits and activates APCs, efficie

183 In neonatal mice motoneurons excite Renshaw cells by releas

184 Upon exposure to Ag on the day of birth, neonatal mice mount balanced primary Th1 and Th2 respons

185 Our study demonstrates that in neonatal mice NLF significantly limits mitochondrial cal

186 s in the nerves dissected from embryonic and neonatal mice of both sexes respond to the application o

187 Remarkably, neonatal mice of either the BALB/c or C57BL/6 mouse stra

188 tinal gene expression profiles in 17-day-old neonatal mice on a 2% LCPUFA feeding paradigm to identif

189 Interestingly, upon i.p. injection into neonatal mice, only the insulin B-chain(15-23)-reactive

190 adult cells is diminished after transfer to neonatal mice or after co-culture with neonatal splenocy

191 n addition, the ablation of CD71(+) cells in neonatal mice, or the decline in number of these cells a

192 etinal vasculature, and imply that in normal neonatal mice, oxygen from the retinal circulation may p

193 ere investigated in tissue slices taken from neonatal mice (P0-P4).

194 Neonatal mice, partially hepatectomized adult mice, and

195 he ARH to other parts of the hypothalamus in neonatal mice, paying particular attention to the innerv

196 In wild-type neonatal mice (postnatal day 0-1), we show that intraven

197 In neonatal mice, prior to P7, heart tissue can be regenera

198 Reovirus induces myocarditis in neonatal mice, providing a tractable model system for in

199 and formation of "miniventral rootlets." In neonatal mice, PTN protected the facial motor neurons ag

200 Neonatal mice raised on antibiotics or lacking the toll-

201 Intracerebroventricular ASO injection in neonatal mice recapitulates SMA-like progressive motor d

202 Neonatal mice receiving the adult CD8(+) T cells had sig

203 tion of adenovirus expressing mutant TLR4 to neonatal mice reduced the severity of NEC and increased

204 Reducing SMN in neonatal mice resulted in a classic SMA-like phenotype.

205 compounds (D152344, D153249 and D156844) to neonatal mice resulted in a dose-dependent increase in S

206 Ghrelin blockade in neonatal mice resulted in enhanced ARH neural projection

207 ells into the external granule cell layer of neonatal mice resulted in the extension of parallel fibe

208 Intracranial delivery of AAV to neonatal mice resulted in widespread neuronal delivery.

209 oduced fewer symptoms and lower mortality in neonatal mice, resulting in an attenuated form of biliar

210 In neonatal mice, rhesus rotavirus (RRV) can induce biliary

211 Olig1-null neonatal mice showed significant hypomyelination after m

212 oral administration of recombinant CCL20 to neonatal mice significantly reduced the parasite load by

213 In neonatal mice, Sox2-null inner pillar cells (IPCs, a sub

214 fficient (Epas1+/-) and wild-type (Epas1+/+) neonatal mice subjected to an oxygen-induced retinopathy

215 Neonatal mice subjected to either increased or decreased

216 e (TG) emulsions provides neuroprotection in neonatal mice subjected to hypoxic-ischemic (H/I) brain

217 Neonatal mice suckled by OMV- or sham-immunized dams wer

218 Th2 cells were observed in IFN-alpha-treated neonatal mice, suggesting dual mechanisms of action.

219 pulmonary inflammation but not mortality in neonatal mice, suggesting that death in these mice was n

220 on of the left ventricular apex in 2-day-old neonatal mice, sympathetic nerve structures, which envel

221 of morphological changes occurred earlier in neonatal mice than in young adults.

222 of 1.8 h, with oxygen) was more virulent in neonatal mice than was GBS grown under suboptimal invasi

223 n which human glial cells are engrafted into neonatal mice that are both immunodeficient and deficien

224 Neonatal mice that had in utero and early-life vitamin D

225 Here we use optical methods to show in live neonatal mice that waves of spontaneous retinal activity

226 Here we show in neonatal mice, that neither reduction of sensory input t

227 e observed transmission of infection between neonatal mice, the first report of B. pertussis transmis

228 In neonatal mice, the heart can regenerate fully without sc

229 In neonatal mice, the majority of peripheral T lymphocytes

230 In neonatal mice, the microbiota induced TNF-alpha and IL-1

231 On subcutaneous injection to neonatal mice, the TLR8 agonist-adjuvanted Ag85B peptide

232 Injection of AAV9-GFP into neonatal mice through the facial vein results in extensi

233 T(reg) cell development depend upon IL-2 in neonatal mice, thymus production, and subsequent expansi

234 In utricles from neonatal mice, time-lapse recordings in the vicinity of

235 Retinopathy was induced by exposing neonatal mice to 75% oxygen from postnatal day (P) 7 to

236 urthermore, FcRn expression was required for neonatal mice to absorb TNP-specific IgE when fed as IgG

237 use white matter injury produced by exposing neonatal mice to chronic hypoxia-a paradigm that mimics

238 slices from wild-type (WT) and Lmx1b(f/f/p) neonatal mice to differentiate autoreceptor versus heter

239 ced propensity for weanling as compared with neonatal mice to form iBALT in response to acute LPS exp

240 Exposure of neonatal mice to hyperoxia enhanced sphingosine-1-phosph

241 Thus, exposure of neonatal mice to PC-bearing pneumococci significantly re

242 compounds attenuated growth and survival of neonatal mice to similar extents and the pathophysiologi

243 y through cranial windows in male and female neonatal mice to test the hypothesis that dynamics of de

244 ng plasmid was injected into the vitreous of neonatal mice undergoing the oxygen-induced retinopathy

245 Neonatal mice, unlike adults, lack factors required for

246 he destruction of extrahepatic bile ducts of neonatal mice using an experimental model of biliary atr

247 lerate FDC maturation and immune response in neonatal mice, using a pneumococcal polysaccharide of se

248 llowing intravenous injection into fetal and neonatal mice, using control uninjected age-matched mice

249 W:I-A(b)-specific naive T cells in different neonatal mice varied significantly in generation of Th1,

250 Brain remodeling after HI+/-MSC treatment in neonatal mice was analyzed using diffusion tensor magnet

251 ly returned to adult levels, TB clearance in neonatal mice was not associated with hepatic UGT1A1 exp

252 urprisingly, CD5 expression on B-1a cells of neonatal mice was only minimally compromised.

253 sequencing in naive CTL of differently aged neonatal mice was performed, which demonstrated differen

254 receptor in GUS transport across the BBB in neonatal mice, we compared brain uptake of phosphorylate

255 the neurogenic subventricular zone (SVZ) of neonatal mice, we deleted Tsc1 and generated olfactory l

256 role of PSCs in the bronchial epithelium of neonatal mice, we developed an enzyme-based digestion me

257 By using gene-deleted neonatal mice, we examined the contributions of the inna

258 immunological basis of these observations in neonatal mice, we found that antibody responses to infec

259 Using this endogenous regeneration model in neonatal mice, we have found that noggin treatment inhib

260 C57BL/6 adult and neonatal mice were challenged with C. rodentium, and a p

261 separate experiments, FcRn(+/-) or FcRn(-/-) neonatal mice were gavage fed TNP-specific IgE as IgG(1)

262 Neonatal mice were injected intramuscularly with a human

263 Neonatal mice were injected intravenously with a Moloney

264 At birth, the in utero-labeled neonatal mice were injected subcutaneously with 3H-TdR (

265 Neonatal mice were inoculated with a recombinant CVB3 ex

266 Neonatal mice were maintained in a 75% oxygen atmosphere

267 The alterations noted in neonatal mice were not observed in adult mice upon re-ex

268 Outbred CD1 neonatal mice were orally gavaged with one of two strain

269 Neonatal mice were primed with meningococcus serotype C

270 Immediately following birth, neonatal mice were prone to develop exaggerated airway e

271 After hyperoxia-treated neonatal mice were returned to ambient room air, retinal

272 Neonatal mice were subjected to 75% to 85% oxygen from p

273 Neonatal mice were subjected to a control diet (CTR) or

274 When neonatal mice were subjected to ischemia-driven retinopa

275 When neonatal mice were subjected to ischemia-induced retinop

276 ection beyond the lungs was also detected in neonatal mice, which may contribute to the observed syst

277 -stimulated IGF-1 expression in the liver of neonatal mice, which plays a key role in the development

278 al denervation impairs heart regeneration in neonatal mice, which was rescued by the administration o

279 zation, here we investigate vocalizations of neonatal mice with a reduction or absence of these compo

280 Treatment of neonatal mice with a single systemic dose of ASO partial

281 how that B1 phenotype B cells are present in neonatal mice with B cell-specific KLF2 deficiency, sugg

282 ties between beta1 integrin-deleted mice and neonatal mice with defective Hedgehog signaling led to t

283 Neonatal mice with disrupted LepRb-->ERK signaling displ

284 Treatment of neonatal mice with exogenous TNF-alpha resulted in incre

285 studies in livers from affected children and neonatal mice with experimental biliary atresia have sho

286 ted organotypic cortical slice cultures from neonatal mice with green fluorescent protein (GFP) drive

287 ective inhibition of hepatic GH signaling in neonatal mice with impaired expression of IGF-1 and IGFB

288 rated that knockdown of the miR-15 family in neonatal mice with locked nucleic acid-modified anti-miR

289 Here, we report that oral infection of neonatal mice with low doses of virulent Y. enterocoliti

290 ZDR/Wor rats, adult mice with I/R injury, or neonatal mice with OIR were injected within the vitreous

291 during sleep were clearly observed in these neonatal mice with or without EMG instrumentation.

292 Infection of neonatal mice with reovirus T3 Dearing (T3D), the protot

293 d strain of virus, experimental infection of neonatal mice with reoviruses can induce fatal encephali

294 Inoculation of neonatal mice with rhesus rotavirus followed by multista

295 Infection of neonatal mice with some reovirus strains produces a dise

296 c release in neurons following infection of neonatal mice with T3 reoviruses.

297 ced lung injury was significantly reduced in neonatal mice with targeted deletion of Chrm2, relative

298 IL-1beta production and skin inflammation in neonatal mice with the CAPS-associated Nlrp3 mutation.

299 were intravenously administered to fetal and neonatal mice, with noninjected age-matched mice used as

300 ed HSCs preferentially homed to the liver in neonatal mice yet showed balanced homing to the liver an