ライフサイエンスコーパス: neonatal mouse

1 r maintaining cholesterol homeostasis in the neonatal mouse.

2 rsting in the lumbosacral spinal cord of the neonatal mouse.

3 urally occurring retinal angiogenesis in the neonatal mouse.

4 N region addition, are not available to the neonatal mouse.

5 ired to establish pulmonary infection in the neonatal mouse.

6 hy of spinal motor neurons in the developing neonatal mouse after axotomy.

7 These studies demonstrate that: 1) normal neonatal mouse airway development entails an IL-4Ralpha-

8 tiated keratinocytes in the epidermis of the neonatal mouse and in the bulge area of the adult mouse

9 c neurotransmission was blocked in slices of neonatal mouse and rat hippocampus and neocortex, sPFPs

10 9 Wnt genes and Wnt target gene Axin2 in the neonatal mouse bone by in situ hybridization, and demons

11 KCC2 is partially phosphorylated in neonatal mouse brain and dephosphorylated in parallel wi

12 nfect neurons within specific regions of the neonatal mouse brain and produce a lethal meningoencepha

13 that Ets-1 binds to the DOR promoter in the neonatal mouse brain and that overexpressed Ets-1 can si

14 a trans-activator of the DOR promoter in the neonatal mouse brain and thus may contribute to the deve

15 s into specific regions of the embryonic and neonatal mouse brain in vivo.

16 as a 3.9- and 4.4-kb transcript in adult and neonatal mouse brain total RNA, and in situ hybridizatio

17 t although intraventricular injection of the neonatal mouse brain with adeno-associated virus serotyp

18 ial injection into lateral ventricles of the neonatal mouse brain, a low-affinity AAV4 mutant (AAV4.1

19 After transplantation into the neonatal mouse brain, human ES cell-derived neural precu

20 be difficult to detect in the embryonic and neonatal mouse brain, we used a new transgenic mouse wit

21 functional synapses when transplanted into a neonatal mouse brain.

22 ferent dermal origin after implantation into neonatal mouse brains.

23 aptic depression using a rhythmically active neonatal mouse brainstem slice preparation.

24 calpain-mediated mechanisms of cell death of neonatal mouse C17.2 progenitor cells, transplanted at 2

25 leukin-6 by osteoblasts in organ cultures of neonatal mouse calvaria, and in vivo using a mouse model

26 Neonatal mouse calvariae were cultured in acid (Acid; pH

27 oblastic bone formation in organ cultures of neonatal mouse calvariae, and a neutralizing antibody to

28 lls were obtained by sequential digestion of neonatal mouse calvariae, and cultured with fetal calf s

29 of retinoids on bone resorption in cultured neonatal mouse calvarial bones and their interaction wit

30 It is not clear whether B cells in the neonatal mouse can activate the somatic mutation machine

31 d mechanically integrated ECT using isolated neonatal mouse cardiac cells derived from both wild-type

32 onses in low-density, serum-free cultures of neonatal mouse cardiac myocytes and compared them with r

33 y +8 mV, producing a maximal +34-mV shift in neonatal mouse cardiac myocytes or Chinese hamster ovary

34 ) regulate the intrinsic contraction rate in neonatal mouse cardiac myocytes through distinct signali

35 rising potassium current (I(Kr)) channels in neonatal mouse cardiac myocytes.

36 on were further tested in cultured adult and neonatal mouse cardiac myocytes.

37 R-199a-3p and hsa-miR-590-3p both in primary neonatal mouse cardiomyocytes and in vivo.

38 gested that fusion between WB F344 cells and neonatal mouse cardiomyocytes did not take place.

39 00000117266 led to a significant increase of neonatal mouse cardiomyocytes in G0/G1 phase and reducti

40 was further revealed in primary cultures of neonatal mouse cardiomyocytes.

41 he proximal ANF promoter by ChIP assay using neonatal mouse cardiomyocytes.

42 -induced cardiac cell death were examined in neonatal mouse cardiomyocytes.

43 nslational stability of Na/Ca exchanger 1 in neonatal mouse cardiomyocytes.

44 We have utilized primary cultures from neonatal mouse cerebella in order to determine (i) wheth

45 GRIP infection of neonatal mouse cerebellum in vivo enhances granule cell

46 Sumoylation of FOXP2 in neonatal mouse cerebellum regulates Purkinje cell develo

47 ce tags in heterogeneous primary cultures of neonatal mouse cerebellum that respond to the mitogen So

48 neous waves of activity propagate across the neonatal mouse cerebral cortex and that these waves are

49 Our findings demonstrate that the neonatal mouse cochlea is capable of spontaneous hair ce

50 urified Atoh1-expressing hair cells from the neonatal mouse cochlea.

51 e developed two genetic strategies to ablate neonatal mouse cochlear hair cells in vivo.

52 ther epithelial proteins was detected in the neonatal mouse epidermis lacking periplakin.

53 c cell lines XS52-4D and XS106 (derived from neonatal mouse epidermis), bone marrow-derived dendritic

54 luripotent neural crest-like stem cells from neonatal mouse epidermis, with different potencies, isol

55 sin, is a protective antigen, using a lethal neonatal mouse ETEC challenge model and passive dam vacc

56 MNs) in the thoracolumbar spinal cord of the neonatal mouse exclusively via axons descending ipsilate

57 When Lin(-) BM cells were injected into neonatal mouse eyes, they extensively and stably incorpo

58 Normal neonatal mouse forepaws were imaged by micro-computed to

59 , to mice markedly prolonged the survival of neonatal mouse heart allografts.

60 one major unresolved question is whether the neonatal mouse heart can also regenerate in response to

61 The neonatal mouse heart can regenerate, but only during the

62 Using the neonatal mouse heart cryoinjury and apical resection mod

63 sive analysis of transcriptomes derived from neonatal mouse heart left and right ventricles, a total

64 c transcription factor GATA4 is required for neonatal mouse heart regeneration.

65 tnatal coronary vessels arise de novo in the neonatal mouse heart, rather than expanding from preexis

66 schemic reperfusion in the isolated perfused neonatal mouse heart.

67 o drive angiogenesis and regeneration of the neonatal mouse heart.

68 increase in miR-378 expression in 1-week-old neonatal mouse hearts compared with 16-day-old fetal hea

69 Fetal or neonatal mouse hearts containing proliferating cardiac m

70 Pitx2-deficient neonatal mouse hearts failed to repair after apex resect

71 The introduction of injury models for neonatal mouse hearts has accelerated research on the me

72 solve the entire architecture of large-scale neonatal mouse hearts, revealing the helical orientation

73 quired for the full regenerative capacity of neonatal mouse hearts.

74 CN) channel subunits in pyramidal neurons of neonatal mouse hippocampus using electrophysiological an

75 xpressing dorsal-medial (mpd) neurons of the neonatal mouse hypothalamus.

76 Using a medullary slice preparation from a neonatal mouse, including the site of the neural network

77 Line scan images (2 ms repetition rate) of neonatal mouse inner hair cells filled with the fluoresc

78 Extracts from neonatal mouse lenses contained strong VEID-7-amino-4-tr

79 duction of C/EBPbeta protein isoforms in the neonatal mouse liver is regulated by C/EBPalpha.

80 eroxia-induced biomolecular modifications in neonatal mouse lung fibroblasts (nMLFs).

81 thelial cell ingestion of P. aeruginosa in a neonatal mouse lung infection model led to increased lev

82 e transfer of maternal IgG into the prenatal/neonatal mouse made possible by the beta 2m-dependent Fc

83 ry drive potentials in preBotC neurons using neonatal mouse medullary slice preparations that generat

84 o were expressed by both primary cultures of neonatal mouse microglia and astrocytes exposed to heat-

85 nal injury in the oxygen-induced retinopathy neonatal mouse model (see the related article beginning

86 This report introduces a neonatal mouse model for active protection studies with

87 Using a neonatal mouse model of BPD, we show that hyperoxia incr

88 Using a neonatal mouse model of CCM disease, we show that expres

89 Conscious bioimaging was applied to a neonatal mouse model of cerebral palsy (Hypoxic-Ischaemi

90 We previously described a neonatal mouse model of coxsackievirus B3 (CVB3) infecti

91 In this study, a neonatal mouse model of critical pertussis is characteri

92 well as reduced morbidity and mortality in a neonatal mouse model of disease.

93 otent and substantially more protective in a neonatal mouse model of group B Streptococcus infection

94 Using a neonatal mouse model of HI, mRNA, and protein expression

95 ee of brain damage sustained by animals in a neonatal mouse model of hypoxia-ischemia depends on the

96 antly less virulent than PAO1 in a BALBc/ByJ neonatal mouse model of infection as measured by their a

97 To address this question, we established a neonatal mouse model of influenza infection to test the

98 In this study, we used a neonatal mouse model of ovalbumin (OVA)-induced allergic

99 l neovascularization were determined using a neonatal mouse model of oxygen-induced retinopathy (OIR)

100 munoglobulin G to induce acantholysis in the neonatal mouse model of pemphigus.

101 A neonatal mouse model of pneumonia was used to determine

102 comparing the virulence of fliC mutants in a neonatal mouse model of pneumonia.

103 The neonatal mouse model of retinopathy of prematurity (ROP)

104 We used the neonatal mouse model of rotavirus infection and virus st

105 We used the neonatal mouse model of rotavirus infection to study ext

106 Here we review the use of a neonatal mouse model of RSV infection to mimic severe in

107 accharide inhibited bacterial ingestion in a neonatal mouse model, resulting in increased amounts of

108 In the oxygen-induced ROP neonatal mouse model, retinal neovascularization was dec

109 Using a neonatal mouse model, we previously determined that coxs

110 Using a neonatal mouse model, we previously revealed that mice i

111 dy described in the present paper, we used a neonatal mouse model, which more closely mimics human in

112 ant exfoliative toxin A (rETA) was used in a neonatal mouse model.

113 to elicit protection from GBS infection in a neonatal mouse model.

114 efficacy against C. parvum infection in the neonatal mouse model.

115 dust mite and A alternata were compared in a neonatal mouse model.

116 an inactivated influenza virus vaccine in a neonatal mouse model.

117 lanocyte proliferation and migration using a neonatal mouse model.

118 nd is required for mucosal colonization in a neonatal mouse model.

119 erize FNPB progenitor cell-derived colonies, neonatal mouse mononuclear cells were cultured directly

120 ne transfer of the E83K-GPD1-L mutation into neonatal mouse myocytes markedly attenuated the sodium c

121 ) regulate the intrinsic contraction rate in neonatal mouse myocytes through distinct signaling pathw

122 In > 60 % of neonatal mouse myocytes, a sizable IKs could be measured

123 Using a neonatal mouse NEC model, we examined the changes in int

124 also reveals inhibitory GABA actions in the neonatal mouse neocortex and hippocampus in vivo.

125 rivative D156844 increases SMN expression in neonatal mouse neural tissues, delays motor neuron loss

126 nhance the expression of DOR mRNA in primary neonatal mouse neuronal cells.

127 ished for the first time primary cultures of neonatal mouse olfactory bulb expressing TH and tested w

128 In a neonatal mouse, only strains with intact agr and sarA lo

129 ct of drug-induced fictive locomotion in the neonatal mouse or change gait, motor coordination, or gr

130 ptide, we found that short-term treatment of neonatal mouse ovaries increased nuclear exclusion of Fo

131 Neonatal mouse pathogenesis of virus (TR339) generated f

132 roduced into the medial prefrontal cortex of neonatal mouse pups by electroporation, and the regulati

133 s in neocortical and hippocampal slices from neonatal mouse pups in vitro, but also reveals inhibitor

134 Neonatal mouse pups were exposed to >90% hyperoxia or ro

135 A neonatal mouse retina developmental model was used to st

136 eceptors, we examined the development of the neonatal mouse retina in an organotypic culture system.

137 d terminal processes of cholinergic cells in neonatal mouse retina.

138 ed to test whether such angioblasts exist in neonatal mouse retina.

139 development in dissociated-cell cultures of neonatal mouse retina.

140 Ankyrin-G depletion in neonatal mouse retinas markedly reduced CNG channel expr

141 acetylases (HDACs) in rod differentiation in neonatal mouse retinas, we used a pharmacological approa

142 en together, these results indicate that the neonatal mouse SCN has its full complement of cells, som

143 electron tomography of plastic sections from neonatal mouse skin to visualize the organization of des

144 In neonatal mouse skin, two types of dermal papilla (DP) ar

145 elayed transplantation of SCs generated from neonatal mouse skin-derived precursors (SKP-SCs) promote

146 In neonatal mouse slice preparations that retain the preBot

147 tonic destruction of Dbx1 preBotC neurons in neonatal mouse slices impairs respiratory rhythm but sur

148 ecordings of olfactory receptor neurons from neonatal mouse slices revealed that ATP reduced cyclic n

149 recordings were made from dually innervated neonatal mouse soleus muscle fibers, and quantal content

150 nclude that lumbar locomotor networks in the neonatal mouse spinal cord are targets for modulation by

151 lcium imaging in the in vitro isolated whole neonatal mouse spinal cord preparation to record the act

152 ng during fictive locomotion in the isolated neonatal mouse spinal cord, following earlier work on lo

153 In the neonatal mouse spinal cord, we studied the firing proper

154 ize left-right locomotor coordination in the neonatal mouse spinal cord.

155 l evidence that Mef2c inhibition by Foxp2 in neonatal mouse striatum controls synaptogenesis of corti

156 Using a neonatal mouse system as an appropriate model for human

157 ntine reticulospinal (pRS) projection in the neonatal mouse that mediates synaptic effects on spinal

158 ese data suggest that in the orally infected neonatal mouse, the extraintestinal spread of rotavirus

159 element could activate expression in injured neonatal mouse tissues and was divisible into tissue-spe

160 totransferrin Cre (Ltf-Cre) model and in the neonatal mouse uterus using the progesterone receptor Cr

161 Here we show that HC damage in neonatal mouse utricle activates the Wnt target gene Lgr

162 Neonatal mouse ventricular myocytes were treated with a

163 In neonatal mouse ventricular myocytes, overexpression of C

164 In HeLa cells and neonatal mouse ventricular myocytes, peroxide exposure d

165 a,L) was examined in pertussis toxin-treated neonatal mouse ventricular myocytes.

166 strogen-regulated genes in the uterus of the neonatal mouse, we have isolated a murine homologue of t