ライフサイエンスコーパス: neonatal seizure

1 metanide might be an effective treatment for neonatal seizures.

2 l mechanism for the early crescendo phase of neonatal seizures.

3 nic acid and flurothyl models of symptomatic neonatal seizures.

4 l and diazepam, two drugs in current use for neonatal seizures.

5 Q channels may be effective for treatment of neonatal seizures.

6 etanide should be useful in the treatment of neonatal seizures.

7 ng the diagnosis, aetiology and treatment of neonatal seizures.

8 irth weight is a significant risk factor for neonatal seizures.

9 f these infants were diagnosed with clinical neonatal seizures.

10 an add-on to phenobarbital for treatment of neonatal seizures.

11 0 minutes, meconium aspiration syndrome, and neonatal seizures, adjusted for maternal height, materna

12 There were 207 cases of clinical neonatal seizures among 116,048 live births (an incidenc

13 is study estimated the incidence of clinical neonatal seizures among infants born between 1992 and 19

14 h, an Apgar score of 3 or less at 5 minutes, neonatal seizure, an umbilical-artery blood pH of 7.05 o

15 c encephalopathy is the most common cause of neonatal seizures and can lead to chronic epilepsy.

16 In one family, the phenotype extends beyond neonatal seizures and includes rolandic seizures, and a

17 le deleterious effects of GABAergic drugs on neonatal seizures and on excitotoxic neuronal injury in

18 ng mutations responsible for benign familial neonatal seizures and/or peripheral nerve hyperexcitabil

19 re often hypotensive, needed intubation, had neonatal seizures, and received a clinical diagnosis of

20 Neonatal seizures are a naturally occurring source of ne

21 Whereas neonatal seizures are a predictor of adverse neurologica

22 Neonatal seizures are most commonly the clinical finding

23 Although neonatal seizures are quite common, there is controversy

24 This is of particular concern for treating neonatal seizures, as early life exposure to drugs such

25 Finally, human hippocampal samples from neonatal seizure autopsy cases also showed an increase i

26 known to be responsible for benign familial neonatal seizures (BFNS).

27 nt compounds inhibit motor manifestations of neonatal seizures but not cortical seizure activity.

28 ear in understanding the basis for a form of neonatal seizures can be attributed to the successful po

29 Neonatal seizures can lead to epilepsy and long-term cog

30 Neonatal seizures can lead to later life epilepsy and ne

31 Neonatal seizures can permanently disrupt neuronal devel

32 GABAergic) drugs, the standard treatment for neonatal seizures, can have excitatory effects in the ne

33 Neonatal seizures caused by hypoxia can be refractory to

34 ificates, death certificates, and a study of neonatal seizures conducted concurrently with this study

35 Neonatal seizures continue to present a diagnostic and t

36 nked lethal disorder involving cleft palate, neonatal seizures, contractures, central nervous system

37 nfant with ISOD who initially presented with neonatal seizures, diffusion restriction noted on magnet

38 Rats with neonatal seizures had a significant reduction in the num

39 Compared with controls, rats subjected to neonatal seizures had impaired learning and decreased ac

40 Although the treatment of neonatal seizures has not significantly changed in the p

41 odent model of the most common form of human neonatal seizures, hypoxia-induced seizures (HS), we aim

42 approaches to the diagnosis and treatment of neonatal seizures, identifies some of the critical facto

43 The incidence of neonatal seizures in Harris County was lower than the in

44 iventricular leukomalacia in the preterm and neonatal seizures in the term infant.

45 s 0-3 at 5 minutes, meconium aspiration, and neonatal seizures increased similarly with maternal BMI.

46 lifetime is found in the neonatal period and neonatal seizures lead to a propensity for epilepsy and

47 Whether better control of neonatal seizures leads to a reduction in neurodisabilit

48 d KV 7.3, are known to cause benign familial neonatal seizures mainly by haploinsufficiency.

49 Neonatal seizures occur frequently, are often refractory

50 luded in the diagnostic workup of refractory neonatal seizures of unknown origin.

51 se to antidepressant exposure; the impact of neonatal seizures on dentate gyrus neurogenesis; and the

52 ted channelopathies, such as benign familial neonatal seizures or peripheral nerve hyperexcitability

53 of an unknown prior diagnosis of epilepsy or neonatal seizures) or classified as not having epilepsy

54 pothesis that neonatal encephalopathy, early neonatal seizures, or both result from early antenatal i

55 nd in children affected with benign familial neonatal seizures (R213W mutation) or with neonatal epil

56 Electroclinical uncoupling of neonatal seizures refers to electrographic seizure activ

57 [corrected] and need for resuscitation, and neonatal seizures-signs commonly attributed to birth asp

58 sted the hypothesis that a single episode of neonatal seizures (sNS) on rat postnatal day (P) 7 perma

59 recent insights about the pathophysiology of neonatal seizures that may provide the foundation for be

60 Using a rodent model of hypoxia-induced neonatal seizures that results in a persistent increase

61 eptic disorders ranging from benign familial neonatal seizures to severe epileptic encephalopathies.

62 In a rodent model of neonatal seizures, we have shown previously that adminis

63 The odds for neonatal seizure were higher and the odds for admission

64 Infants with neonatal seizures were ascertained from four sources: ho

65 Risk factors for neonatal seizures were evaluated in 116,048 infants born

66 ve mutation in KCNQ2 that has a phenotype of neonatal seizures without permanent clinical CNS impairm