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1 lti-omics and immunity - basic tenets of the neoplastic process.
2 n pressure in the continuum of ERG dependent neoplastic process.
3 interrogate the contribution of PERK in the neoplastic process.
4 pectively, including genes implicated in the neoplastic process.
5 een this pattern and its function during the neoplastic process.
6 hat only a subset of these contribute to the neoplastic process.
7 ent pathways that play a pivotal role in the neoplastic process.
8 develop a fully-penetrant myeloproliferative neoplastic process.
9 associated with genes that contribute to the neoplastic process.
10 ip with transcriptional silencing during the neoplastic process.
11 ns, such as MM-1 and Mxi1, can influence the neoplastic process.
12 affecting gene expression and promoting the neoplastic process.
13 hat LOH at these loci may occur later in the neoplastic process.
14 proteins believed to play a key role in the neoplastic process.
15 ded critical insights into their role in the neoplastic process.
16 relevant alteration to the initiation of the neoplastic process.
17 r kills more Americans yearly than any other neoplastic process.
18 ers, suggesting further spreading during the neoplastic process.
19 the importance of the primary cilium in this neoplastic process.
20 athways that modulate both developmental and neoplastic processes.
21 s are at the crossroads of developmental and neoplastic processes.
22 the functional roles played by Id1 in human neoplastic processes.
23 several T-box genes have been implicated in neoplastic processes.
24 biopsy revealed no evidence of infectious or neoplastic processes.
25 e intracellular TG content on autoimmune and neoplastic processes.
26 whether this molecule is expressed in these neoplastic processes.
27 nary tract tumors, implicating this virus in neoplastic processes.
28 ol mechanisms and may in fact be involved in neoplastic processes.
29 ink a novel ABC family member to the hepatic neoplastic process, a role that may be recapitulated in
30 sociated with both the initial stages of the neoplastic process and the acquisition of malignancy can
31 es assessing the molecular components of the neoplastic process and utilizing the data for cancer cla
32 is induced in a variety of inflammatory and neoplastic processes and is believed to play an importan
33 rk, establish the integral role for NANOG in neoplastic processes and shed light on its mechanisms of
34 ew insights into how myc participates in the neoplastic process, and how additional mutations can pro
35 of ornithine decarboxylase (ODC) during the neoplastic process, and to determine whether induction o
36 f vascular permeability, which is altered in neoplastic processes because of release of angiogenic fa
37 the development of anal cancer, but that the neoplastic process becomes irreversible over time with p
38 n prostate tumor cells may contribute to the neoplastic process by activating C-MYC and by abrogating
39 mble of tumor suppressors and modifiers of a neoplastic process can be usefully analyzed in respect t
40 tory process - cicatrical pemphigoid, benign neoplastic process - chondroma, malignant neoplastic - s
42 It is hoped that these agents inhibit the neoplastic process either alone or in combination with o
43 ells, is an indispensable participant in the neoplastic process, fostering proliferation, survival an
45 gic process with a focus on inflammatory and neoplastic processes, identifying pertinent positives an
47 translocation, a potent driving force of the neoplastic process in general and hematopoietic malignan
49 To examine the relationship of SPEM to the neoplastic process in the H. felis -infected C57BL/6 mou
50 he temporal bone as well as inflammatory and neoplastic processes in the temporal bone region (1).
51 phenomena that play a critical role in many neoplastic processes, including silencing of tumor suppr
52 ar what their respective contribution to the neoplastic process is, as well as to what extent they in
54 is, the development of a microvasculature by neoplastic processes, is a critical component of the dev
55 duct of cervical carcinogenesis and (ii) the neoplastic process may be more extensive than currently
56 enetic alteration similar to that present in neoplastic processes may be responsible for the pathogen
57 ocyte hyperplasia, an early event during the neoplastic process, might begin before liver cirrhosis d
60 in this cancer, the role of PAX-FKHR in the neoplastic process remains largely unknown in a progenit
61 that early molecular steps in this and other neoplastic processes represent adaptations in which, thr
62 ispersion of tumors throughout the body is a neoplastic process responsible for the vast majority of
63 ing the importance of RET in development and neoplastic processes, the signal transduction mechanisms
64 t fragile site alterations contribute to the neoplastic process through inactivation of a tumor suppr
65 ) is essential to multiple physiological and neoplastic processes via signaling by its tyrosine kinas
66 A true functional role for DNA-MTase in the neoplastic process would be further substantiated if the
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