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1 s markedly impair filtration function in the nephrocyte.
2 enotype reminiscent of nephrotic syndrome in nephrocytes.
3 diomyopathy that developed in the absence of nephrocytes.
4  nephrotic syndrome have not been studied in nephrocytes.
5 hrin and Neph1, respectively, in pericardial nephrocytes.
6 on of the fluorescent protein in pericardial nephrocytes.
7  rise to cardioblasts, blood progenitors and nephrocytes.
8  in podocytes or the nephrocyte diaphragm in nephrocytes.
9   Expressing the G0 or G1 APOL1 transgene in nephrocytes also impaired the acidification of organelle
10 O-1 (TJP1) and podocin, are expressed in the nephrocyte and form a complex of interacting proteins th
11                                   The insect nephrocyte and the mammalian glomerular podocyte are sim
12 tch that differentiates between the blood or nephrocyte and vascular lineages involves the Notch path
13    Here, we present a functional analysis of nephrocytes and establish an in vivo system to screen fo
14 nd is expressed in cardioblasts, pericardial nephrocytes and hematopoietic progenitors.
15                                   Drosophila nephrocytes and human podocytes share striking similarit
16 irculatory system: cardioblasts, pericardial nephrocytes and lymph gland hematopoietic progenitors, b
17 loss-of-function strategy was used to ablate nephrocytes and then heart function and the hemolymph pr
18 imilarities we describe between invertebrate nephrocytes and vertebrate podocytes provide evidence su
19  and Pannier in cardioblasts and pericardial nephrocytes, and by Serpent in hematopoietic progenitors
20 la Vps8 is highly expressed in hemocytes and nephrocytes, and localizes to early endosomes despite th
21                      Drosophila garland cell nephrocytes are podocyte-like cells and thus provide a p
22 rdioblasts) and excretory cells (pericardial nephrocytes), arises from the cardiogenic mesoderm.
23 re evolutionarily related, and establish the nephrocyte as a simple model in which to study podocyte
24              We conclude that the Drosophila nephrocyte can be used to elucidate clinically relevant
25 ial relevance to humans using the Drosophila nephrocyte-cardiomyocyte system.
26 on of APOL1 risk variants in D. melanogaster nephrocytes caused cell-autonomous accumulation of the e
27                    In conclusion, the insect nephrocyte combines filtration with protein reabsorption
28                                In Drosophila nephrocytes, deficiency of the Pals1 ortholog caused alt
29 ph by uptake of molecules through an SD-like nephrocyte diaphragm (ND) into labyrinthine channels tha
30 structure revealed that the formation of the nephrocyte diaphragm and lacunar structure, which is ess
31 wn as the slit diaphragm in podocytes or the nephrocyte diaphragm in nephrocytes.
32                Furthermore, we find that the nephrocyte diaphragm is completely lost in flies lacking
33  function declined, cell size increased, and nephrocytes died prematurely.
34          RNAi-mediated knockdown of dKank in nephrocytes disrupted slit diaphragm filtration structur
35                              Rendering adult nephrocytes dysfunctional by disrupting their endocytic
36                                We found that nephrocytes efficiently take up secreted fluorescent pro
37 xpression of the APOL1 G0 or G1 transgene in nephrocytes, fly cells homologous to mammalian podocytes
38           We screened for genes required for nephrocyte function and identified two Drosophila genes
39 As transgenic flies with either allele aged, nephrocyte function declined, cell size increased, and n
40 ion of an APOL1 transgene initially enhances nephrocyte function, causing hypertrophy and subsequent
41 ophila KANK homolog (dKank) is essential for nephrocyte function.
42 ing the identification of genes required for nephrocyte function.
43  cells and oenocytes (also called Drosophila nephrocytes) function in taking up waste material from t
44 lencing Cindr in nephrocytes led to dramatic nephrocyte functional impairment and shortened life span
45 und that 85% of these genes are required for nephrocyte functions, suggesting that a majority of huma
46  co-expressed within binucleate garland cell nephrocytes (GCNs) that contribute to detoxification of
47            Here, we show that the Drosophila nephrocyte has molecular, structural, and functional sim
48                 Here we show that the insect nephrocyte has remarkable anatomical, molecular and func
49 data suggest that the Drosophila pericardial nephrocyte is a useful in vivo model to help identify ge
50  shortened life span, as well as collapse of nephrocyte lacunar channels and effacement of nephrocyte
51                                  Ablation of nephrocytes led to a severe cardiomyopathy characterized
52                           Silencing Cindr in nephrocytes led to dramatic nephrocyte functional impair
53 -red fluorescent protein at early stages and nephrocyte loss at later stages.
54 te, but the lack of a functional readout for nephrocytes makes it challenging to study this model of
55       In conclusion, Drosophila garland cell nephrocytes provide a model with which to study the path
56                     Proteomics revealed that nephrocytes regulate the circulating levels of many secr
57 these known NS genes play conserved roles in nephrocytes remains unknown.
58                        Tracer endocytosis by nephrocytes required Cubilin and reflected size selectiv
59                               The Drosophila nephrocyte shares remarkable similarities to the glomeru
60 ephrocyte lacunar channels and effacement of nephrocyte slit diaphragms.
61 d fluorescent protein and combined it with a nephrocyte-specific driver for targeted gene knockdown,
62 ulation was found at garland and pericardial nephrocytes supporting its role in organismal defence an
63 ch are pairs of highly endocytic pericardial nephrocytes that modulate cardiac function by uncharacte
64                          Vps34 deficiency in nephrocytes, the podocyte-like cells of Drosophila melan
65 bsorption is required for the maintenance of nephrocyte ultrastructure and fly survival under conditi
66 rgeted expression of human AMN in Drosophila nephrocytes was sufficient to rescue defective protein u
67    Using RNAi and conditional CRISPR/Cas9 in nephrocytes, we found this pattern depends on the expres
68 pression of dCubilin and dAMN is specific to nephrocytes, where they function as co-receptors for pro
69 ctional genetic screen of Drosophila cardiac nephrocytes, which are equivalents of mammalian podocyte

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