ライフサイエンスコーパス: nephrolithiasis

1 ith PHPT, of whom 265 (8%) were symptomatic (nephrolithiasis).

2 al insights to stop the growing incidence of nephrolithiasis.

3 re previously reported to be associated with nephrolithiasis.

4 TRPV5 channel activity and protects against nephrolithiasis.

5 is inappropriate in patients with recurrent nephrolithiasis.

6 eceptor (VDR) in target tissues; and calcium nephrolithiasis.

7 itical for understanding the pathogenesis of nephrolithiasis.

8 Hypercalciuria is a major risk factor for nephrolithiasis.

9 id peroxidation during hyperoxaluria-induced nephrolithiasis.

10 hyperoxaluria and idiopathic calcium oxalate nephrolithiasis.

11 ic metabolic acidosis, nephrocalcinosis, and nephrolithiasis.

12 ces, paraesthesias, hyperbilirubinaemia, and nephrolithiasis.

13 n and improved accuracy in the evaluation of nephrolithiasis.

14 with HIV infection, has been associated with nephrolithiasis.

15 provide a suitable model of human hereditary nephrolithiasis.

16 cidence of hyperoxaluria and calcium oxalate nephrolithiasis.

17 se stone recurrence in patients with calcium nephrolithiasis.

18 es have been associated with a lower risk of nephrolithiasis.

19 st 2 L of urine per day to prevent recurrent nephrolithiasis.

20 ted sequelae, including nephrocalcinosis and nephrolithiasis.

21 rt an adverse effect of high temperatures on nephrolithiasis.

22 ed claudin-14 associated with hypercalciuric nephrolithiasis.

23 risk of radiation exposure to patients with nephrolithiasis.

24 scribes a 60-year-old patient with bilateral nephrolithiasis.

25 roxaluria or even idiopathic calcium oxalate nephrolithiasis.

26 promising adjuncts for preventing recurrent nephrolithiasis.

27 lly considered a poor experimental model for nephrolithiasis.

28 variant(s) are candidate risk modifiers for nephrolithiasis.

29 Sixty-one percent had nephrolithiasis.

30 alate increases the risk for calcium oxalate nephrolithiasis.

31 e dietary oxalate as a major risk factor for nephrolithiasis.

32 e 2 diabetes at increased risk for uric acid nephrolithiasis.

33 ons as causing a recessive Mendelian form of nephrolithiasis.

34 cal prevention, and surgical intervention of nephrolithiasis.

35 contribute to the hypercalciuria and calcium nephrolithiasis.

36 conducted of 45,619 men without a history of nephrolithiasis.

37 ke seems to increase the risk of symptomatic nephrolithiasis.

38 ally invasive techniques in the treatment of nephrolithiasis.

39 stinuria is the commonest inherited cause of nephrolithiasis (~1% in adults; ~6% in children) and is

40 red with the high-dose scan were as follows: nephrolithiasis, 91%; ureterolithiasis, 94%; obstruction

41 Nephrolithiasis, a condition in which urinary supersatur

42 ntribute to the recent increase in pediatric nephrolithiasis, a definite underlying cause remains elu

43 ue from normal rats and rats developing CaOx nephrolithiasis after challenge with ethylene glycol.

44 n mRNA expression in rat kidneys during CaOx nephrolithiasis after challenge with ethylene glycol.

45 luoroscopy used during surgical treatment of nephrolithiasis also contributes to patient radiation ex

46 yndrome have resulted in increasing rates of nephrolithiasis among women, decreasing the male-to-fema

47 association analysis using 624 patients with nephrolithiasis and 1008 control subjects.

48 ations describing the link between pediatric nephrolithiasis and bone metabolism.

49 X-linked nephrolithiasis and engineered deficiencies in some othe

50 Patients with recurrent calcium nephrolithiasis and fasting hypercalciuria have a higher

51 Patients with recurrent calcium nephrolithiasis and idiopathic fasting hypercalciuria (u

52 nderstand that a relationship exists between nephrolithiasis and low BMD.

53 important factors in the pathophysiology of nephrolithiasis and low bone density.

54 The increased oxalate excretion can cause nephrolithiasis and nephrocalci-nosis and can, in some c

55 est that knowledge of the molecular cause of nephrolithiasis and nephrocalcinosis may have practical

56 may account for the lower incidence of both nephrolithiasis and osteoporosis in black women.

57 nced CT (5-mm section width, no overlap) for nephrolithiasis and other causes of twinkling artifact.

58 between the induction of hyperoxaluria/CaOx nephrolithiasis and the expression of the bikunin gene i

59 Of these, 7 (3%) had nephrolithiasis and the other 12 (5%) had previously und

60 ydrate, the most common solid phase in human nephrolithiasis, and also inhibits the nucleation, growt

61 n guide therapy to prevent nephrocalcinosis, nephrolithiasis, and potentially, CKD.

62 olecular-weight proteinuria, hypercalciuria, nephrolithiasis, and renal failure.

63 t may increase the risk of nephrocalcinosis, nephrolithiasis, and renal insufficiency.

64 cribing further uses of alpha-antagonists in nephrolithiasis, and reporting improvements in extracorp

65 have been described as being associated with nephrolithiasis, and these mutations explain about 15% o

66 l: 0.66, 0.90) positive predictive value for nephrolithiasis anywhere in the kidneys at CT.

67 RECENT FINDINGS: Patients with nephrolithiasis are at risk for significant radiation ex

68 e of bacteria in the pathogenesis of calcium nephrolithiasis are discussed.

69 nd was detectable for long-bone fracture and nephrolithiasis as well as among children.

70 dney stone cases, suggesting that additional nephrolithiasis-associated genes remain to be discovered

71 o establish the relationship between calcium nephrolithiasis, bone densitometry scoring, and bone min

72 's disease, also known as X-linked recessive nephrolithiasis, but the effects of diuretics on calcium

73 h ambient temperatures are a risk factor for nephrolithiasis, but the precise relationship between te

74 Obesity is a strong risk factor for nephrolithiasis, but the role of physical activity and c

75 ncrease of 1.6-2.2 million lifetime cases of nephrolithiasis by 2050, representing up to a 30% increa

76 rted that Uromodulin (UMOD) protects against nephrolithiasis by upregulating the renal calcium channe

77 t in the denosumab to teriparatide group had nephrolithiasis, classified as being possibly related to

78 Three disorders of hypercalciuric nephrolithiasis (Dent's disease, X-linked recessive neph

79 Advances in the management of nephrolithiasis depend on combined efforts of clinicians

80 ques and the renal papillae in patients with nephrolithiasis, detailing genetic discoveries related t

81 stone formation, excessive uptake results in nephrolithiasis due to hypocitraturia.

82 ant advances have been made in understanding nephrolithiasis from single gene defects, the understand

83 tudies have identified slc26a6 as an oxalate nephrolithiasis gene in the mouse.

84 ient population is all adults with recurrent nephrolithiasis (>/=1 prior kidney stone episode).

85 (n=12), patients (n=12) with hypercalciuric nephrolithiasis had significantly decreased levels of ur

86 The incidence of pediatric nephrolithiasis has been steadily increasing for the pas

87 Recent studies in nephrolithiasis have investigated why stones form, impro

88 in adults, the trends occurring in pediatric nephrolithiasis have not been studied rigorously, which

89 ry and pharmacologic management of recurrent nephrolithiasis in adults.

90 Type 2 diabetes is a risk factor for nephrolithiasis in general and has been associated with

91 LC34A1 gene can lead to hypophosphatemia and nephrolithiasis in humans remains unknown.

92 citrate, or allopurinol to prevent recurrent nephrolithiasis in patients with active disease in which

93 The main risk factor for uric acid nephrolithiasis in patients with type 2 diabetes is a lo

94 relation between oxalate intake and incident nephrolithiasis in the Health Professionals Follow-up St

95 Nephrolithiasis incidence is increasing in children and

96 Recurrent nephrolithiasis is a burden to the individual patient as

97 Nephrolithiasis is a not infrequent complication of preg

98 These findings may partly explain why nephrolithiasis is a predominantly male disease.

99 Nephrolithiasis is a prevalent condition with a high mor

100 linicians look for the underlying causes for nephrolithiasis is imperative to direct management.

101 e disease, the best management for recurrent nephrolithiasis is likely a combination of surgical and

102 Nephrolithiasis is often associated with increased super

103 Calcium nephrolithiasis is the most common form of renal stone d

104 rtrophy, complicated by nephrocalcinosis and nephrolithiasis, is reported here.

105 Nephrolithiasis (kidney stones) affects 5-10% of adults

106 Xp11.22, are associated with hypercalciuric nephrolithiasis (kidney stones) in the Northern European

107 terized by hypercalciuria, nephrocalcinosis, nephrolithiasis, low molecular weight proteinuria, Fanco

108 ldren and 166 adults) from 268 families with nephrolithiasis (n=256) or isolated nephrocalcinosis (n=

109 Nephrolithiasis occurred in 12 of 33 patients (36%).

110 ninvasive first-line therapy for millions of nephrolithiasis patients, has not improved substantially

111 parathyroid hyperplasia with a high rate of nephrolithiasis, persistent and recurrent HPT.

112 d and Drug Industry is paramount to reducing nephrolithiasis rates and its complications.

113 are of the amount of radiation patients with nephrolithiasis receive.

114 lly explain the high human susceptibility to nephrolithiasis relative to that of mouse.

115 velopment was evaluated after adjustment for nephrolithiasis risk factors.

116 l imaging method for patients with suspected nephrolithiasis should be computed tomography (CT) or ul

117 Patients symptomatic with nephrolithiasis, significant osteoporosis, bone pain, an

118 expressed in Xenopus oocytes and in reported nephrolithiasis susceptibility.

119 n have a higher incidence of calcium oxalate nephrolithiasis than women.

120 nce is raised by factors unique to pediatric nephrolithiasis that could expose an affected child to m

121 a change in the current trends of pediatric nephrolithiasis that is characterized by a significant i

122 opportunities to learn more about pediatric nephrolithiasis, thereby fueling the much-needed researc

123 inkling artifact is commonly associated with nephrolithiasis, this finding is relatively insensitive

124 d to the emergency department with suspected nephrolithiasis to undergo initial diagnostic ultrasonog

125 ing dysfunction, flank pain, abdominal pain, nephrolithiasis, urinary tract infection and decreased b

126 is review describes the relationship between nephrolithiasis, vascular disease and metabolic syndrome

127 Nephrolithiasis was predictive of PTx (OR 2.94 vs asympt

128 ditional genes whose mutations are linked to nephrolithiasis, we performed targeted next-generation s

129 ups; no cases of hypercalcemia and 1 case of nephrolithiasis were reported in the placebo group.

130 Kidney stones (nephrolithiasis), which affect 12% of males and 5% of fe

131 scribed for patients with idiopathic calcium nephrolithiasis, who account for > 80% of new diagnoses

132 .S. population living in high-risk zones for nephrolithiasis will grow from 40% in 2000 to 56% by 205

133 e cost increase associated with this rise in nephrolithiasis would be $0.9-1.3 billion annually (year

134 ithiasis (Dent's disease, X-linked recessive nephrolithiasis (XRN), and X-linked recessive hypophosph