ライフサイエンスコーパス: nephrotic syndrome

1 and provide insight into the pathogenesis of nephrotic syndrome.

2 e in podocytes might promote pathogenesis of nephrotic syndrome.

3 deteriorating renal function or untreatable nephrotic syndrome.

4 4 (ADCK4) gene that cause steroid-resistant nephrotic syndrome.

5 ot benefit clinical outcome in children with nephrotic syndrome.

6 ed structure, the disruption of which causes nephrotic syndrome.

7 tal interstitial lung disease and congenital nephrotic syndrome.

8 glomeruli, mimicking the phenotype of human nephrotic syndrome.

9 -based consortium studying steroid-resistant nephrotic syndrome.

10 s with idiopathic membranous nephropathy and nephrotic syndrome.

11 and of patients with Finnish-type congenital nephrotic syndrome.

12 bitors in children with resistant idiopathic nephrotic syndrome.

13 rarenal defects but still exhibit congenital nephrotic syndrome.

14 eading to the filtration barrier defects and nephrotic syndrome.

15 podocyte-secreted Angptl4 has a key role in nephrotic syndrome.

16 ases the risk for high-grade proteinuria and nephrotic syndrome.

17 s of proteinuria and rates of progression to nephrotic syndrome.

18 ide (LPS)-treated mice, a model of transient nephrotic syndrome.

19 mg/dl); three (30%) patients presented with nephrotic syndrome.

20 d, lead to inherited FSGS and/or the related nephrotic syndrome.

21 ltrastructural changes and steroid-sensitive nephrotic syndrome.

22 sclerosis as a cause of steroid-unresponsive nephrotic syndrome.

23 as to the treatment of steroid-unresponsive nephrotic syndrome.

24 ocytes in a number of experimental models of nephrotic syndrome.

25 increased in patients with steroid-sensitive nephrotic syndrome.

26 ss effacement and lipopolysaccharide-induced nephrotic syndrome.

27 ch model Pierson syndrome, a rare congenital nephrotic syndrome.

28 pathophysiology and treatment mechanisms of nephrotic syndrome.

29 te gene expression during the development of nephrotic syndrome.

30 e in children with primary steroid-resistant nephrotic syndrome.

31 several months, but they developed a lethal nephrotic syndrome.

32 e pathogenesis of proteinuria and congenital nephrotic syndrome.

33 ted in autosomal recessive steroid-resistant nephrotic syndrome.

34 ectively, in children with steroid-resistant nephrotic syndrome.

35 foot process (FP) effacement and LPS-induced nephrotic syndrome.

36 s are the standard of care for most forms of nephrotic syndrome.

37 lex, and this connection is disrupted in the nephrotic syndrome.

38 model in which to study the pathogenesis of nephrotic syndrome.

39 in-induced experimental kidney diseases with nephrotic syndrome.

40 agnosis and care of patients with congenital nephrotic syndrome.

41 podocyte injury is typically associated with nephrotic syndrome.

42 ceptors may be novel therapeutic targets for nephrotic syndrome.

43 is, and 1 with congenital hypothyroidism and nephrotic syndrome.

44 ot processes, comparable to that seen in the nephrotic syndrome.

45 cause autosomal recessive steroid-resistant nephrotic syndrome.

46 nts who present with renal insufficiency and nephrotic syndrome.

47 EPHRIN, a protein associated with congenital nephrotic syndrome.

48 jection was accompanied by resolution of the nephrotic syndrome.

49 th lupus nephritis, diabetic nephropathy, or nephrotic syndrome.

50 ypothesized to be the cause of the patient's nephrotic syndrome.

51 mutation is an important cause of hereditary nephrotic syndrome.

52 th frequently relapsing or steroid-dependent nephrotic syndrome.

53 rats with puromycin aminonucleoside-induced nephrotic syndrome.

54 contribution of PCSK9 to the dyslipidemia of nephrotic syndrome.

55 ameliorates dyslipidemia in a mouse model of nephrotic syndrome.

56 SK9 in mediating the hypercholesterolemia of nephrotic syndrome.

57 eful for predicting relapse in patients with nephrotic syndrome.

58 philia due to a factor VIII autoantibody and nephrotic syndrome.

59 KANK1), KANK2, and KANK4 in individuals with nephrotic syndrome.

60 merular capillary tuft, podocyte injury, and nephrotic syndrome.

61 everity of the acquired hypercoagulopathy of nephrotic syndrome.

62 cytes that is dysfunctional in some types of nephrotic syndrome.

63 a severity, consistent with reports in human nephrotic syndrome.

64 aminonucleoside and adriamycin rat models of nephrotic syndrome.

65 markers or therapeutic targets in persistent nephrotic syndrome.

66 5.7% vs 2.6%; aHR, 2.27; 95% CI, 1.32-3.89), nephrotic syndrome (1.3% vs 0.1%; aHR, 15.7; 95% CI, 2.9

67 Of children with idiopathic nephrotic syndrome, 10%-20% fail to respond to steroids

68 hose patients with available data, 92% had a nephrotic syndrome, 64% had AKI, and 67% had a documente

69 At presentation, 49% had nephrotic syndrome, 68% had renal insufficiency, and 77%

70 logy Damage Index, the greatest frequency of nephrotic syndrome (8.9%) was observed in patients in cl

71 nt in children with glucocorticoid-sensitive nephrotic syndrome, a disorder with minimal known indepe

72 Nephrotic syndrome, a malfunction of the kidney glomerul

73 gene are a major cause of steroid-resistant nephrotic syndrome, a severe human kidney disorder.

74 ed protein (CD2AP) develop renal failure and nephrotic syndrome about 4 weeks after birth and die aro

75 hallmarks of Pierson syndrome are congenital nephrotic syndrome accompanied by ocular abnormalities,

76 Patients with biopsy-proven PMN and nephrotic syndrome after 6 months of nonimmunosuppressiv

77 phrotic syndrome in patients with congenital nephrotic syndrome after transplantation may lead to imp

78 rstanding of recurrent disease in congenital nephrotic syndrome after transplantation.

79 Glomeruli isolated from mice with nephrotic syndrome also had increased expression of IL-1

80 pine in 60 children and adolescents with the nephrotic syndrome and 195 control subjects.

81 ldren affected by sporadic steroid-resistant nephrotic syndrome and 38 patients who exhibited a simil

82 obtained from (1) the urine of children with nephrotic syndrome and carrying potentially pathogenic m

83 merulosclerosis (FSGS) is a leading cause of nephrotic syndrome and end-stage renal disease worldwide

84 pathologic lesion that frequently causes the nephrotic syndrome and ensuing renal failure.

85 /d were enrolled at first biopsy for primary nephrotic syndrome and followed.

86 ilial nephropathy in which steroid-sensitive nephrotic syndrome and glomerular deposits of fibrillar

87 erular disorder, manifests clinically with a nephrotic syndrome and has a high propensity for recurre

88 tified in a child with encephalomyopathy and nephrotic syndrome and in a younger sibling with only ne

89 on bilateral transperitoneal nephrectomy for nephrotic syndrome and laparoscopic nephrectomy for Wilm

90 The incidence of nephrotic syndrome and leukopenia was also lower in clus

91 essive sodium retention seen in experimental nephrotic syndrome and liver cirrhosis.

92 phila orthologs of human monogenic causes of nephrotic syndrome and observed conservation of the cent

93 l target for therapeutic intervention in the nephrotic syndrome and other proteinuric diseases.

94 -year-old man with cystic fibrosis developed nephrotic syndrome and progressed to end-stage renal fai

95 a clinicopathologic entity characterized by nephrotic syndrome and progression to ESRD.

96 ng anti-PLA2R antibody levels associate with nephrotic syndrome and progressive loss of kidney functi

97 CD2AP-deficient mice develop nephrotic syndrome and renal failure caused by glomerulo

98 se of a 61-year-old woman who presented with nephrotic syndrome and renal impairment.

99 ide new insights into the molecular basis of nephrotic syndrome and sensorineural deafness.

100 ive genes have been found to cause inherited nephrotic syndromes and FSGS.

101 Two patients presented with nephrotic syndromes and one patient with an IgG4-related

102 scribe an infant with severe Leigh syndrome, nephrotic syndrome, and CoQ(10) deficiency in muscle and

103 in steroid-dependent or frequently relapsing nephrotic syndrome, and halted disease-associated growth

104 ry diseases; 2.77 (1.49-5.15) for nephritis, nephrotic syndrome, and nephrosis; 4.08 (1.38-12.08) for

105 itis), and inflammatory (glomerulonephritis, nephrotic syndrome, and osteoarthritis) disorders.

106 c approach to sepsis-associated hypotension, nephrotic syndrome, and other sight-threatening and life

107 odel with which to study the pathogenesis of nephrotic syndrome, and ROS formation may be a pathomech

108 vents; partial and complete remission of the nephrotic syndrome; and a composite of doubling of serum

109 Common causes of nephrotic syndrome are diabetic nephropathy, minimal cha

110 Patients with idiopathic nephrotic syndrome are initially treated with corticoste

111 naling events contributing to development of nephrotic syndrome are not well defined.

112 Childhood nephrotic syndromes are most commonly caused by one of t

113 the underlying disease, the severity of the nephrotic syndrome (as assessed by serum albumin concent

114 terations in children with sporadic forms of nephrotic syndrome associate with resistance to steroids

115 ers, previous immobility, surgery or trauma, nephrotic syndrome, associated tumor, inflammatory disea

116 subset of CD2AP(-/-) mice exhibiting severe nephrotic syndrome, associated with systemic illness.

117 nhibitors may be beneficial in patients with nephrotic syndrome-associated hypercholesterolemia.

118 k4 in zebrafish and Drosophila recapitulated nephrotic syndrome-associated phenotypes.

119 Neph1-deficient mice develop nephrotic syndrome at birth, indicating the importance o

120 lassic pathway and the highest prevalence of nephrotic syndrome at disease onset.

121 hypercoagulopathy to identify patients with nephrotic syndrome at highest risk for thrombotic diseas

122 tion of a subset of patients with congenital nephrotic syndrome at increased risk of recurrence who a

123 ) collagen developed massive albuminuria and nephrotic syndrome, because of subepithelial deposits of

124 -LAMB2 missense mutation leads to congenital nephrotic syndrome but only mild extrarenal symptoms; th

125 orted to reduce proteinuria in patients with nephrotic syndrome, but mechanisms remain unknown.

126 s a well-described model of human idiopathic nephrotic syndrome, but the mechanism of PAN's effect is

127 e to FFAs may function in the development of nephrotic syndrome by amplifying the effects of proteinu

128 sults suggest that the R246Q mutation causes nephrotic syndrome by impairing secretion of laminin-521

129 In children, sporadic nephrotic syndrome can be related to a genetic cause, bu

130 Other causes of isolated nephrotic syndrome can be subdivided into two major cate

131 Patients with nephrotic syndrome can develop nephrocalcinosis, which a

132 patients with congenital red hair color and nephrotic syndrome caused by idiopathic membranous nephr

133 insufficiency syndrome and steroid-resistant nephrotic syndrome caused by loss-of-function mutations

134 Here, using mouse models of human nephrotic syndrome caused by mutant laminin beta2 protei

135 These data show that diseases that cause nephrotic syndrome change glomerular protein permeabilit

136 nting in early life, Finnish type congenital nephrotic syndrome (CNF) and a form of autosomal recessi

137 Congenital nephrotic syndrome (CNS), a rare entity, is known to pre

138 omal recessive SRNS presenting as congenital nephrotic syndrome (CNS).

139 Lamb2(-/-) mice abrogates the development of nephrotic syndrome, correlating with a greatly extended

140 In nephrotic syndrome, damage to the podocytes of the kidne

141 erosis (FSGS) is a common form of idiopathic nephrotic syndrome defined by the characteristic lesions

142 ests that immunomodulatory therapies used in nephrotic syndrome directly target the podocytes.

143 ding patients with congestive heart failure, nephrotic syndrome, diuretic resistance, or generalized

144 proteinuria, edema and, in steroid-resistant nephrotic syndrome, end-stage kidney disease.

145 were similar to those reported in congenital nephrotic syndrome, Finnish type, that is caused by muta

146 with frequently relapsing steroid-sensitive nephrotic syndrome (FR-SSNS).

147 ith chronic renal insufficiency (CRI) or the nephrotic syndrome frequently manifest diuretic resistan

148 , together with the zebrafish model of human nephrotic syndrome generated by plce1 knockdown, open ne

149 histomorphometry and sequencing of Mendelian nephrotic syndrome genes were performed.

150 Steroid-resistant nephrotic syndrome has a poor prognosis and often leads

151 se, a major life-threatening complication of nephrotic syndrome, has been associated with proteinuria

152 c, thrombotic microangiopathic syndromes and nephrotic syndrome have been described.

153 Genetic studies in children with familial nephrotic syndrome have identified mutations in genes th

154 However, relevant pathomechanisms of nephrotic syndrome have not been studied in nephrocytes.

155 eral nephrectomy in patients with congenital nephrotic syndrome have permitted transplantation to be

156 anous nephropathy (MN) is a leading cause of nephrotic syndrome in adults and a significant cause of

157 Membranous nephropathy is a common cause of nephrotic syndrome in adults and can be primary or secon

158 nephropathy (MN) is the most common cause of nephrotic syndrome in adults, and one-third of patients

159 d as one of the leading causes of idiopathic nephrotic syndrome in adults, particularly among African

160 phropathy (MN), a relatively common cause of nephrotic syndrome in adults, were considered idiopathic

161 nephropathy (MN) is the most common cause of nephrotic syndrome in adults, with an uncertain clinical

162 (MCD) is the etiology of 10%-25% of cases of nephrotic syndrome in adults.

163 ranous nephropathy (MN) is a common cause of nephrotic syndrome in adults.

164 is the leading cause of steroid-unresponsive nephrotic syndrome in childhood.

165 yndrome (SSNS) accounts for >80% of cases of nephrotic syndrome in childhood.

166 Idiopathic nephrotic syndrome in children is commonly associated wi

167 In conclusion, nephrotic syndrome in children with truncating or homozy

168 The nephrotic syndrome in early post-transplantation period

169 ragm length, such as occur in minimal-change nephrotic syndrome in humans, in animals treated with pu

170 d mutations in podocyte-specific genes cause nephrotic syndrome in humans.

171 or the alpha3beta1 ligand laminin result in nephrotic syndrome in murine models.

172 SGPL1, displayed a phenotype reminiscent of nephrotic syndrome in nephrocytes.

173 ievable, would likely lessen the severity of nephrotic syndrome in patients carrying LAMB2 mutations.

174 blood stem-cell transplantation improves the nephrotic syndrome in patients with AL amyloidosis-assoc

175 New findings regarding recurrence of nephrotic syndrome in patients with congenital nephrotic

176 glomerular diseases and induces remission of nephrotic syndrome in patients with diverse glomerulopat

177 on of the presentation of acute rejection as nephrotic syndrome in the literature.

178 tients, and immune-mediated pancytopenia and nephrotic syndrome in the recipient of a double UCB tran

179 target genes, reported as possible causes of nephrotic syndrome, in a cohort of 31 children affected

180 in the epidemiology of steroid-unresponsive nephrotic syndrome, in particular focal segmental glomer

181 er similar changes occur with glomerulopathy/nephrotic syndrome, in which high-circulating/filtered l

182 The main manifestations of nephrotic syndrome include proteinuria, hypoalbuminemia,

183 ity and mortality associated with protracted nephrotic syndrome, including ESRD.

184 The relapse rate of nephrotic syndrome increased significantly after adminis

185 g nephrin lead to proteinuria and congenital nephrotic syndrome, indicating that nephrin is essential

186 -brain barrier integrity was correlated with nephrotic syndrome, indicating that these effects are di

187 of podocyte disorders in cases of idiopathic nephrotic syndrome (INS) are complex and remain incomple

188 multiorgan disorder that included congenital nephrotic syndrome, interstitial lung disease, and epide

189 Idiopathic nephrotic syndrome is a chronic relapsing disease for mo

190 Nephrotic syndrome is a common disorder in adults and ch

191 In conclusion, nephrotic syndrome is associated with an increased filtr

192 It has long been recognized that nephrotic syndrome is associated with an increased risk

193 Nephrotic syndrome is associated with several medical co

194 Nephrotic syndrome is associated with up-regulation of t

195 Nephrotic syndrome is characterized by increased triglyc

196 Nephrotic syndrome is characterized by massive proteinur

197 Steroid-resistant nephrotic syndrome is characterized by podocyte dysfunct

198 Diuretic responsiveness in patients with the nephrotic syndrome is limited by avid Na(+) reabsorption

199 e type of glomerular disease that causes the nephrotic syndrome is necessary for appropriate treatmen

200 nk between proteinuria and hyperlipidemia in nephrotic syndrome is not known.

201 Steroid-unresponsive nephrotic syndrome is often characterized by persistent

202 Nephrotic syndrome is recognized by the presence of prot

203 The pathogenesis of nephrotic syndrome is unclear.

204 membranous nephropathy, a common form of the nephrotic syndrome, is an antibody-mediated autoimmune g

205 tl4 secreted from podocytes in some forms of nephrotic syndrome lacks normal sialylation.

206 is dissociation, wild-type mice with induced nephrotic syndrome maintained an intact blood-brain barr

207 that leak through the abnormal glomerulus in nephrotic syndrome may affect tubular transport by inter

208 pic correlations of patients with congenital nephrotic syndrome may not only modify our understanding

209 eatment for the initial episode of childhood nephrotic syndrome may reduce relapse rate, but whether

210 e of two idiopathic diseases: minimal-change nephrotic syndrome (MCNS) and focal segmental glomerulos

211 (mean serum creatinine, 3.6 mg/dl) and full nephrotic syndrome (mean 24-h urinary protein excretion,

212 ritis, type I membranoproliferative GN), and nephrotic syndrome (minimal change/FSGS, membranous neph

213 nt CGD and DM (n=1), and steroid therapy for nephrotic syndrome (n=1).

214 o heart disease, cancer, diabetes, nephritis/nephrotic syndrome/nephrosis, chronic lower respiratory

215 mutations profile in Brazilian children with nephrotic syndrome (NS) and to determine a genotype-phen

216 Nephrotic syndrome (NS) is a genetically heterogeneous g

217 Nephrotic syndrome (NS) is associated with hyperlipidemi

218 Nephrotic syndrome (NS) is characterized by structural c

219 Nephrotic syndrome (NS) is divided into steroid-sensitiv

220 Response to loop diuretics in patients with nephrotic syndrome (NS) is subnormal.

221 Nephrotic syndrome (NS) occurs when the glomerular filtr

222 Rituximab induces nephrotic syndrome (NS) remission in two-thirds of patie

223 Nephrotic syndrome (NS) represents the association of pr

224 nefits of genetic screening of patients with nephrotic syndrome (NS) to diagnose monogenic causes, re

225 Glucocorticoids are the primary therapy for nephrotic syndrome (NS), but have serious side effects a

226 have been identified as monogenic causes of nephrotic syndrome (NS), but important knowledge gaps ex

227 Patients with nephrotic syndrome (NS), even with normal GFR, often dis

228 Nephrotic syndrome (NS), the association of gross protei

229 ducing remissions of proteinuria, relapse of nephrotic syndrome occurred significantly more often aft

230 of steroid-dependent or frequently relapsing nephrotic syndrome of minimal change disease (MCD), mesa

231 ce of either nephrin (as in human congenital nephrotic syndrome of the Finnish type, NPHS1) or NEPH1.

232 HS1 gene encoding nephrin lead to congenital nephrotic syndrome of the Finnish type.

233 as the gene whose mutations cause congenital nephrotic syndrome of the Finnish type.

234 as either primary or adjunctive therapy for nephrotic syndrome or other diseases treated with glucoc

235 Children with nephrotic syndrome or renal transplants have easily reco

236 PCSK9 and plasma lipids were studied in nephrotic syndrome patients before and after remission o

237 ckdown of kank2 in zebrafish recapitulated a nephrotic syndrome phenotype, resulting in proteinuria a

238 ephrin and podocin cause two types of severe nephrotic syndrome presenting in early life, Finnish typ

239 he model eliminated the association with the nephrotic syndrome (ratio, 0.99; 95 percent confidence i

240 nucleoside-induced injury (designed to mimic nephrotic syndrome-related injury), as determined by bot

241 relative risk 4.79; 95% CI 2.71 to 8.46) and nephrotic syndrome (relative risk 7.78; 95% CI 1.80 to 3

242 iltration, many of the disease mechanisms of nephrotic syndrome remain unresolved.

243 athogenic circulating permeability factor in nephrotic syndrome remains to be confirmed, we propose i

244 The cause of idiopathic nephrotic syndrome remains unknown, but evidence suggest

245 boembolic events with oral anticoagulants in nephrotic syndrome requires a careful case-by-case analy

246 Idiopathic nephrotic syndrome resistant to standard treatments rema

247 months later, the patient suddenly developed nephrotic syndrome resistant to steroid therapy 1 week a

248 D in relapse and in remission, patients with nephrotic syndrome resulting from other glomerular disea

249 Nephrotic syndrome results from a breakdown of the kidne

250 odels of glomerular injury and patients with nephrotic syndrome revealed that calpain-induced talin1

251 Steroid-dependent nephrotic syndrome (SDNS) carries a high risk of toxicit

252 cation of patients with steroid-unresponsive nephrotic syndrome, severity of disease, progression and

253 Patients with nephrotic syndrome showed a decrease in plasma cholester

254 r an early childhood onset recessive form of nephrotic syndrome (SRN1).

255 racterized by the combination of early-onset nephrotic syndrome (SRNS) and microcephaly with brain an

256 yme Q10 biosynthesis cause steroid-resistant nephrotic syndrome (SRNS) as part of multiorgan involvem

257 Steroid-resistant nephrotic syndrome (SRNS) causes 15% of chronic kidney d

258 Steroid-resistant nephrotic syndrome (SRNS) causes 15% of chronic kidney d

259 s reduced in patients with steroid-resistant nephrotic syndrome (SRNS) due to monogenic disorders.

260 n of single-gene causes of steroid-resistant nephrotic syndrome (SRNS) has furthered the understandin

261 Steroid-resistant nephrotic syndrome (SRNS) is a frequent cause of end-sta

262 Steroid-resistant nephrotic syndrome (SRNS) is a frequent cause of progres

263 Steroid-resistant nephrotic syndrome (SRNS) is the second most frequent ca

264 Steroid-resistant nephrotic syndrome (SRNS) leads to end-stage renal disea

265 Steroid-resistant nephrotic syndrome (SRNS), a frequent cause of chronic k

266 Steroid-resistant nephrotic syndrome (SRNS), a heterogeneous disorder of t

267 ding of pathomechanisms in steroid-resistant nephrotic syndrome (SRNS), not even a gene locus is know

268 cause autosomal recessive steroid-resistant nephrotic syndrome (SRNS).

269 Steroid-sensitive nephrotic syndrome (SSNS) accounts for >80% of cases of

270 cocorticoid, children with steroid-sensitive nephrotic syndrome (SSNS) have almost normal adult heigh

271 Disease mechanisms of steroid-sensitive nephrotic syndrome (SSNS) remain unknown.

272 tasets derived from 90 black subjects in the Nephrotic Syndrome Study Network (NEPTUNE), stratified b

273 ogenic NS genes in 312 participants from the Nephrotic Syndrome Study Network and 61 putative control

274 diseases and in other human pathologies with nephrotic syndromes such as HIV and Hantavirus infection

275 lacking B7-1 are protected from LPS-induced nephrotic syndrome, suggesting a link between podocyte B

276 f rituximab, a B cell-depleting antibody, in nephrotic syndrome suggests a pathogenic role of B cells

277 his the first report of a molecular cause of nephrotic syndrome that may resolve after therapy.

278 s central to proteinuric states, such as the nephrotic syndrome, the murine adriamycin nephrosis mode

279 In a cohort of 106 patients with nephrotic syndrome, there were 63 relapses during the 12

280 ldren (9 months to 17 years) presenting with nephrotic syndrome to either 3 months of prednisolone fo

281 ong the glomerular basement membranes, and a nephrotic syndrome, two additional immunizations were ne

282 trolled trial in 31 children with idiopathic nephrotic syndrome unresponsive to the combination of ca

283 relationships among different components of nephrotic syndrome use naturally occurring pathways and

284 Nephrotic syndrome was reported in a highly-sensitized p

285 which are mutated in two types of congenital nephrotic syndrome, was pivotal in establishing the podo

286 CD2AP(-/-) mice normally die by 6 weeks from nephrotic syndrome, we used mice that also express a CD2

287 n during organogenesis results in congenital nephrotic syndrome, whereas VEGF164 overexpression after

288 be single-gene defects-eg, steroid-resistant nephrotic syndrome, which is caused by podocin mutations

289 therapy for patients with steroid-resistant nephrotic syndrome with ADCK4 mutations.

290 epsilon gene (PLCE1) as causing early-onset nephrotic syndrome with end-stage kidney disease.

291 Pierson syndrome is a congenital nephrotic syndrome with eye and neurologic defects cause

292 notypic variance of patients with congenital nephrotic syndrome with nephrin and podocin mutations re

293 ients with first manifestation of idiopathic nephrotic syndrome with no immune complexes can improve

294 cause Pierson syndrome, a severe congenital nephrotic syndrome with ocular and neurologic defects.

295 cause Pierson syndrome, a severe congenital nephrotic syndrome with ocular and neurologic defects.

296 Pierson syndrome is a congenital nephrotic syndrome with ocular and neurological defects

297 THSD7A antibodies into mice induced a severe nephrotic syndrome with proteinuria, weight gain, and hy

298 100 consecutive IMN patients with persistent nephrotic syndrome with rituximab.

299 anous nephropathy is a common cause of adult nephrotic syndrome, with recent evidence suggesting that

300 plant patient who presented with early onset nephrotic syndrome without change in serum creatinine, w