ライフサイエンスコーパス: nerve

1 DRS is secondary to absence of the abducens nerve.

2 at supplies the syrinx, the tracheosyringeal nerve.

3 the lateral rectus muscle by the oculomotor nerve.

4 communication after damage to the peripheral nerve.

5 omas relative to normal non-neoplastic optic nerve.

6 on potential discharge from colonic afferent nerves.

7 early into intratumoral, chemokine-secreting nerves.

8 HL tendon and the medial and lateral plantar nerves.

9 from the wound and distal regions of severed nerves.

10 variable morphology of the pre-otic cranial nerves.

11 complete transection of the Octopus pallial nerves.

12 tide (CGRP), a marker of nociceptive sensory nerves.

13 nd gp130 were expressed only in intratumoral nerves.

14 The sensory nerve action potentials (SNAPs) remained dispersed and a

15 were profiled against capsaicin in a sensory nerve activation assay and in vivo potency established a

16 illatory breathing and increased sympathetic nerve activity (SNA) are associated with increased arrhy

17 Using direct measurements of sympathetic nerve activity and pharmacological manipulation of blood

18 Recordings of phrenic nerve activity in female Sprague Dawley rats (3-4 months

19 ellular pH regulation is vital to neurons as nerve activity produces large and rapid acid loads in pr

20 Bilateral phrenic nerve activity was recorded in anesthetized and ventilat

21 S of anesthetized rats increased sympathetic nerve activity, whereas functionally related enzymes tha

22 od capable of detecting all organophosphorus nerve agent (OPNA) adducts to human butyrylcholinesteras

23 yl ethylphosphonate (DEEP, a simulant of the nerve agent sarin) of at least 5 times higher than a sim

24 during the hydrolysis of a chemical warfare nerve agent simulant over a polyoxometalate catalyst.

25 nditions, 1 catalyzes both hydrolysis of the nerve agent simulant, diethyl cyanophosphonate (DECP) an

26 atrace quantitation of a phosphoryl fluoride nerve agent surrogate.

27 d wearable wireless tattoo and textile-based nerve-agent vapor biosensor systems offer considerable p

28 id warning regarding personal exposure to OP nerve-agent vapors in variety of decentralized security

29 Toxicity from acute exposure to nerve agents and organophosphorus toxicants is due to ir

30 s 7-9 (OP), akin in size and shape to G-type nerve agents, form inclusion complexes with baskets 1-3

31 age-related mechanism along the cholinergic nerve-airway smooth muscle (ASM) axis that underlies pro

32 The vagal nerve also projects to the commissural nucleus of Cajal,

33 the fact that tumors do not form in injured nerves, although they contain proliferating Schwann cell

34 ion in dorsal root ganglia (DRG) and sciatic nerve and abundance of Schwann cells.

35 iated signaling between IHC and the auditory nerve and also balanced differences in decay kinetics be

36 d in increased concentrations in the sciatic nerve and dorsal root ganglia of oxaliplatin treated mic

37 ng adrenal gland, where they detach from the nerve and form postsynaptic neuroendocrine chromaffin ce

38 erved voltage-gated sodium channels (NaV) of nerve and muscle, causing paralysis.

39 over, immunostainings and dot blots of optic nerve and myelin showed that expression of Rtn4b is very

40 Because ATP is released extracellularly by nerve and other tissue injury, we hypothesize that injec

41 Optic nerve and retinal abnormalities were the most frequent f

42 Stimulation of the gastrocnemius nerve and sural nerve revealed significant convergence o

43 This cross-talk between nerves and endothelial metabolism could potentially be t

44 d Schwann cells acutely purified from intact nerves and from the wound and distal regions of severed

45 rode directly through the neuronal sheath of nerves and ganglia in insects.

46 ed cells that build Bungner bands in injured nerves and that such cells can transform to myelin cells

47 DTI-MRN covered proximal (sciatic nerve) and distal (tibial nerve) nerve segments of the l

48 ts from the bladder (primarily in the pelvic nerve) and the urethra (in the pudendal and pelvic nerve

49 ent) and normal cerebellar, sensory, cranial nerve, and autonomic function.

50 se in the accumulation of neutrophils in the nerve, and elevated phagocytosis by neutrophils.

51 that affect the brain-gut axis via the vagus nerve, and then travel to higher centers, compromising t

52 athy, which primarily affects the peripheral nerves, and transthyretin cardiomyopathy (TTR-CM), which

53 ria core with matrix molecules, fibroblasts, nerves, and vessels.

54 on neural population responses for auditory nerve (ANF) input and SBC output to assess the influence

55 pathway inhibitor (HPI), and that gustatory nerves are a source of sonic hedgehog (Shh) for taste bu

56 nflammation, but the possibility that airway nerves are dysfunctional has not been fully explored.

57 d ST266 accumulated in rodent eyes and optic nerves, attenuated visual dysfunction, and prevented ret

58 l evoked calcium signals in mammalian tibial nerve axons using an in vitro mouse model with a dextran

59 was a uniform feature in a total of 21 sural nerve biopsies and 'onion bulb' formations and/or thin m

60 input before movement prevented BTP, whereas nerve block after movement failed to reverse BTP.

61 movement-induced afferent input by saphenous nerve block before, but not after, hindlimb movement blo

62 with kilohertz electrical stimulation (KES) nerve block to preferentially activate efferent pathways

63 and electroactive tissues, such as cardiac, nerve, bone, cartilage, and skeletal muscle.

64 ers/mm2; 95% CI, -11.04 to -2.52; P = .002), nerve branch density (mean [SE] difference, -17.94 [5.45

65 Stalled abducens nerve bundles did not reach the orbit, resulting in seco

66 sions not only were found in impinged spinal nerves but also were associated with nonspinal causes of

67 In vitro, the afferent nerve calyx surrounding type I hair cells causes unstabl

68 st time, that the excitation of a peripheral nerve can be accomplished by 12-ns PEF without electropo

69 ATP, released from perivascular sympathetic nerves, causes vasoconstriction largely via P2X1 recepto

70 Although the location of the nerve cell bodies of spinal afferents is well known to r

71 ion, the virus remains inactive or latent in nerve cells that sense the region where that infection o

72 consistent with a single origin of the gut, nerve cells, and muscle cells in the stem lineage of eum

73 chondrial damage and induce demyelination of nerve cells.

74 Cells residing within peripheral nerves collaborate with cancer cells to enable PNI, but

75 atment also restored the duration of sensory nerve compound potentials.

76 In all cases, large fibre nerve conduction studies were normal.

77 and axonal loss, which underlie slowed motor nerve conduction velocity (MNCV) and reduced compound mu

78 This defect is associated with slowed nerve conduction, which could contribute to behavioral d

79 ICANCE STATEMENT Although injured peripheral nerves contain repair Schwann cells that provide signals

80 Branches of the same nerve containing different levels of Htt show distinct p

81 ts widespread glial responses in the ventral nerve cord (VNC).

82 distribution, including expression in radial nerve cords, circumoral nerve ring, digestive system, tu

83 to differentiate motor and sensory axons on nerve cross sections.

84 Optic nerve crush rescued the circadian period of Myk/+ behavi

85 nce when the motor neurons are challenged by nerve crush.

86 dditionally, we found 5 novel loci for optic nerve cup area and 6 for disc area.

87 ing regeneration, the clinical outcome after nerve damage is frequently poor.

88 Corneal nerve damage produced by aging, diabetes, refractive sur

89 timulation of rat segmental dorsal cutaneous nerves (DCNs) evokes the nociceptive intersegmental cuta

90 and draw critical parallels to mechanisms of nerve degeneration and regeneration in the CNS and in th

91 the loss of CCR2, injured Ccr2(-/-) sciatic nerves demonstrate prolonged expression of neutrophil ch

92 nerve regeneration and substance P-positive nerve density in both wounded and unwounded eyes compare

93 with gemcitabine reduced NGF expression and nerve density, and increased survival of LSL-Kras(+/G12D

94 Hypertonic saline did not reduce nerve density, but did cause qualitative changes in nerv

95 s regarding the relationship between corneal nerve density, morphology, and function.

96 ters positively correlated with intratumoral nerve density.

97 Schwann cells in developing nerves depend on extrinsic signals from the axon and fro

98 simplex virus (HSV) reactivation, peripheral nerve destruction and sensory anesthesia are rare.

99 Nerve diameter was measured as a morphometric criterion.

100 The distribution of CGRP-positive nerves did not differ significantly between the distal e

101 were nystagmus associated with retinal/optic nerve disease in 23 (32.4%), idiopathic or congenital mo

102 euronal cells is common in retinal and optic nerve disease.

103 ATP is also released from sensory-motor nerves during antidromic reflex activity, to produce rel

104 Sensory nerves emanating from the dorsal root extensively innerv

105 Significant nerve-ending loss occurs before a decrease in cell bodie

106 ), the morphology and location of peripheral nerve endings of spinal afferents that transduce sensory

107 The majority of spinal afferent nerve endings were CGRP-immunoreactive.

108 (3'UTR) landscapes after unilateral sciatic nerve entrapment (SNE) injury in rats.

109 iled and complete description of the cranial nerves exists for this species.

110 Cutaneous nerves extend throughout the dermis and epidermis and co

111 Phox2b-tdTomato labels nerve fascicles in the tongue of the developing embryo a

112 larly be associated with a loss of epidermal nerve fiber density (ENFD).

113 Corneal nerve fiber density (mean [SE] difference, -6.78 [2.14]

114 Epidermal nerve fiber density, the primary outcome, is a measureme

115 iking response that would be produced in the nerve fiber innervating that receptor.

116 p the thickness of the peripapillary retinal nerve fiber layer (NFL) and ganglion cell complex (GCC).

117 To quantify retinal nerve fiber layer (RNFL) changes in patients with multip

118 defects were compared with regard to retinal nerve fiber layer (RNFL) thickness, drusen morphology, s

119 olds, hyperopic refractive error shifts, and nerve fiber layer infarcts.

120 al parameters, minimum rim width and retinal nerve fiber layer thickness, in addition to peripapillar

121 including severity of IOP elevation, retinal nerve fiber layer thinning, or electrodiagnostic finding

122 Positive staining was present within the nerve fiber layer, inner plexiform layer, and inner and

123 hes/mm2; 95% CI, -28.77 to -7.10; P = .001), nerve fiber length (mean [SE] difference, -3.03 [0.89] m

124 manipulate an object, populations of tactile nerve fibers become activated and convey information abo

125 showed substantial innervation by trigeminal nerve fibers immunoreactive for calcitonin gene-related

126 yme neuronal nitric oxide synthase (nNOS) in nerve fibers of the murine vaginal wall.

127 V-2 reactivation exhibit a higher density of nerve fibers relative to biopsies during virological and

128 Further, these 5-HT3 -expressing nerve fibers terminate in a restricted central-lateral p

129 hologically decreased densities of the small nerve fibers that innervate the epidermis, one hypothesi

130 re examined for the presence and location of nerve fibers that reacted with a labeled antibody agains

131 me, is a measurement of the density of small nerve fibers within the epidermis.

132 P2X3 receptor, a marker for non-peptidergic nerve fibers, was not only significantly reduced but cou

133 ion of the primary GC subtypes, even beneath nerve fibers.

134 , trigeminal ganglia (TG) neurons, and their nerve fibers.

135 vealed in six of them reduced intraepidermal nerve fibre density consistent with small fibre neuropat

136 cycles can distinguish whether a sympathetic nerve fibre is depolarized or not.

137 It is concluded that these sympathetic nerve fibres are sensitive to ischaemia, and that VRCs p

138 equency response of the innervating auditory nerve fibres However, the data supporting these concepts

139 onal membrane potential of human sympathetic nerve fibres in vivo.

140 VRCs of human sympathetic nerve fibres of the superficial peroneal nerve innervati

141 tect the effects of ischaemia on sympathetic nerve fibres.

142 Optic nerve findings included hypoplasia with the double-ring

143 strong associations with sciatic and tibial nerve findings.

144 ly high levels during high rates of auditory nerve firing, or that calcium-dependent processes involv

145 ouse model in which a portion of the sciatic nerve from one hind limb was transected at postnatal day

146 n microscopy, we show that injury of sciatic nerves from mice of either sex triggers extensive and un

147 and consists of rerouting motor and sensory nerves from the residual limb towards intact muscles and

148 mpatible with restoration of myelination and nerve function after injury.

149 local versus distal sites may improve median nerve function at the wrist by somatotopically distinct

150 of the aneurysm, a full recovery of cranial nerve function was achieved.

151 he development and maintenance of peripheral nerve function.

152 ificant long-term adverse effects on sciatic nerve functions.

153 Nerve growth factor (NGF) antagonism is on the verge of

154 brain-derived neurotrophic factor (BDNF) and nerve growth factor (NGF) are crucial modulators in the

155 The neurotrophin nerve growth factor (NGF) has been implicated as a key m

156 n channel subfamily M, member 8 (TRPM8); and nerve growth factor (NGF) in nasal biopsy specimens.

157 We discovered that KIF1Bbeta is required for nerve growth factor (NGF)-dependent neuronal differentia

158 ative mutants of ATL1 in PC-12 cells inhibit nerve growth factor (NGF)-induced neurite outgrowth.

159 Furthermore, decreased nerve growth factor, decreased c-fos and increased sympa

160 hibitory factors (e.g. inhibin alpha and VGF nerve growth factor-inducible), 2) activation of a signa

161 ervous system through the greater splanchnic nerve (GSN), and elevation of pro-inflammatory cytokines

162 ry approach to stimulating nerve repair in a nerve-guidance scaffold was used to explore the regenera

163 o noxious mechanical and chemical stimuli in nerve-gut preparations in mouse, or following antagonism

164 l, and behavioral analyses were performed in nerves harvested from each group at different survival t

165 DiGeorge syndrome (DGS), cataract and optic nerve head drusen (ONHD).

166 e structural changes of the macula and optic nerve head in the free eyes of unilateral cured retinobl

167 along anomalous communications in the optic nerve head induce migration of fluid into the adjacent r

168 lium, outer part of the retina and the optic nerve head within 24-hours, in both groups of animals.

169 ling and other immune responses in the optic nerve head.

170 e lateral rectus by fibers of the oculomotor nerve in DRS is secondary to absence of the abducens ner

171 unexpected role for SOX2 and parasympathetic nerves in generating the acinar lineage that has broad i

172 tics and distributions of peptide-containing nerves in the mouse vagina are unknown.

173 g technologies that allow imaging of corneal nerves in vivo are spawning questions regarding the rela

174 Labeling along the antennal nerve, in projections of the dorsal lobe and in the gnat

175 ensity, but did cause qualitative changes in nerves including enlarged varicosities that were also se

176 In conclusion, medial and lateral plantar nerve injuries did not occur more frequently, even after

177 ological in neurodegenerative conditions and nerve injuries.

178 In this study, we performed optic nerve injury in adult naked mole-rats, the longest livin

179 derstand the mechanism of recovery following nerve injury in this species we investigated the process

180 Here, we report that peripheral nerve injury increases expression of the DNA methyltrans

181 Nerve injury induces changes in gene transcription in do

182 Mechanistically, peripheral nerve injury induces DNA demethylation and upregulation

183 otected from hypersensitivity in two sciatic nerve injury models.

184 nerve might mimic the stimulatory effect of nerve injury on the regenerative state of the primary se

185 derate to severe chronic pain resulting from nerve injury or disorder, affects 6.9%-10% of the global

186 namic mechanical hypersensitivity induced by nerve injury or inflammation in mice by ablating a group

187 in RGCs, including in a mouse model of optic nerve injury, and show that the same pathway is active i

188 here is consensus that, distal to peripheral nerve injury, myelin and Remak cells reorganize to form

189 elopment of SC lineage and during peripheral nerve injury, so we sought to study their functional pro

190 actors as important regulators of tumor- and nerve injury-associated pain.

191 evels are suppressed in the spinal cord in a nerve injury-induced neuropathic pain mouse model.

192 glion (DRG) neurons, which may contribute to nerve injury-induced neuropathic pain.

193 le and female mice, neither before nor after nerve injury.

194 tic nerve fibres of the superficial peroneal nerve innervating the dorsum of the foot were recorded b

195 l cochlear nucleus (DCN) integrates auditory nerve input with a diverse array of sensory and motor si

196 ful comparisons between different peripheral nerve interfaces.

197 dge directly above the entrance of the optic nerve into the bony canal.

198 Of 178 patients without optic nerve involvement (tumor >1 disc diameter from optic ner

199 patients were more likely to have peripheral nerve involvement, an intact circulating immunoglobulin,

200 subjects without DPN might indicate that the nerve is affected by diabetes.

201 ever, although the repopulation of the optic nerve lesion site by astrocytes was significantly delaye

202 To detect and quantify peripheral nerve lesions in multiple sclerosis (MS) by magnetic res

203 neuropathic pain behaviors in the rat spinal nerve ligation (SNL) model.

204 canals, that include parts of the trigeminal nerve; many branches of this complex terminate on the ex

205 size that injection of ATP into a peripheral nerve might mimic the stimulatory effect of nerve injury

206 Data on the cranial nerve morphology of tadpoles are scarce, and only one ot

207 d by Schwann cells that regulates peripheral nerve myelination via its cognate receptor ADAM22 expres

208 While in 1-month-old Nf1 mutant nerve neither SCs nor macrophages significantly differed

209 proximal (sciatic nerve) and distal (tibial nerve) nerve segments of the lower extremity.

210 y colocalize with SMIT1 and SMIT2 at sciatic nerve nodes of Ranvier and in axon initial segments, and

211 ressing cells are prominent in motor cranial nerve nuclei, and some scattered cells lie in the preopt

212 ion and biopsied the frontal lobes and optic nerves of a macaque experimentally infected with variant

213 ed DRGs (defined as DRGs with injured spinal nerves) of living SNL rats.

214 rolling and adhesion in veins near the optic nerve (ON) head at 9 hours after ON injury.

215 based study, the majority of isolated fourth nerve palsies were presumed congenital, even though they

216 The incidence of fourth nerve palsy and the frequency of each etiology were calc

217 e the incidence of isolated, presumed fourth nerve palsy in a defined population, and to report the f

218 sed to identify all cases of isolated fourth nerve palsy in Olmsted County, Minnesota, USA diagnosed

219 quent, and in no case was an isolated fourth nerve palsy the presenting sign of an intracranial tumor

220 n eye movement dysfunctions such as abducens nerve palsy.

221 ar matrix remodeling in other, related optic nerve pathological states, we found decreased expression

222 that myelin clearance in the injured sciatic nerve proceeds unhindered in the Ccr2(-/-) mouse model.

223 The ulnar and median nerves proximal to the elbow joint were activated transc

224 6 mul of 150 mum ATP into female rat sciatic nerve quadrupled the number of axons growing into a lesi

225 o preparations based on paired extracellular nerve recordings and videography to identify central and

226 ntation of iPSC-NCSCs accelerated functional nerve recovery with the involvement of stem cell differe

227 nvestigated the process of axon regrowth and nerve regeneration after complete transection of the Oct

228 eneration studies may propel improvements in nerve regeneration and draw critical parallels to mechan

229 a significant increase in corneal epithelial nerve regeneration and substance P-positive nerve densit

230 GC development, promotes long-distance optic nerve regeneration in adult rats of both sexes.

231 We first confirmed that PEDF + DHA increased nerve regeneration in the mouse cornea.

232 However, nerve regeneration studies require long-term recovery of

233 rm hospital" allows us to perform the entire nerve regeneration studies, including on-chip axotomy, p

234 ck-in mice reportedly accelerates peripheral nerve regeneration via increased MAP1B phosphorylation a

235 Similarly, optic nerve regeneration was completely unaffected, although r

236 onal mechanism linking C5aR and C5L2 in pulp nerve regeneration, which may be useful in future dentin

237 T/A) in GSK3(S/A) RGCs further boosted optic nerve regeneration, with axons reaching the optic chiasm

238 rylation of CRMP2 in RGCs and improved optic nerve regeneration.

239 an immunomodulatory approach to stimulating nerve repair in a nerve-guidance scaffold was used to ex

240 how PNI is driven in part by corruption of a nerve repair program.

241 g and myelinating SCs in the later phases of nerve repair, resulting in slowed axon regeneration, cut

242 se conversions are believed to be central to nerve repair.

243 determined the impact of such activation on nerve repair.

244 n events from the glossopharyngeal and vagus nerves, respectively.

245 stimulation exacerbated the loss of auditory nerve response in aged animals but attenuated the loss i

246 ent with a NO-GC stimulator altered auditory nerve responses but did not affect OHC function and hear

247 deography to identify central and peripheral nerves responsible for nociception and sensitization of

248 ulation of the gastrocnemius nerve and sural nerve revealed significant convergence of muscle and cut

249 expression in radial nerve cords, circumoral nerve ring, digestive system, tube feet and innervation

250 al (sciatic nerve) and distal (tibial nerve) nerve segments of the lower extremity.

251 at LIF promoted the differentiation of glial nerve sheath Schwann cells and induced their migration b

252 Malignant peripheral nerve sheath tumors (MPNSTs) are devastating sarcomas fo

253 myxofibrosarcomas, and malignant peripheral nerve sheath tumors are characterized by complex genomic

254 affin-embedded specimens of human peripheral nerve sheath tumors.

255 ly, we introduce a preparation of peripheral nerve slices for patch-clamp recordings.

256 Understanding how nerves spontaneously innervate tissues or regenerate sma

257 anti-inflammatory therapy via cervical vagus nerve stimulation (cVNS) one should selectively activate

258 tic force in response to submaximal rates of nerve stimulation in situ producing significantly higher

259 Hypoglossal nerve stimulation is a useful second-line therapy in pat

260 We examined the effects of CHL at auditory nerve synapses onto bushy cells in the mouse anteroventr

261 a homeostatic, adaptive response of auditory nerve synapses to reduced activity.

262 ction and sensory conduction studies by near-nerve technique, including tactile stimulation of mechan

263 rsts last seconds; however, the increases in nerve terminal Ca(2+) driven by neuropeptides can persis

264 e both capable of evoking large increases in nerve terminal Ca(2+) Increases in Ca(2+) driven by spik

265 We report, for the first time, a nerve terminal impairment in SV trafficking selectively

266 Localization to the nerve terminal suggests a role in neurotransmitter relea

267 ecific effects were found on the presynaptic nerve terminals at the neuromuscular junction level, but

268 euronal activity when ATP consumption within nerve terminals increases drastically.

269 numerous neurological diseases, its role at nerve terminals is poorly understood.

270 Abnormal accumulation of Abeta at nerve terminals leads to synaptic pathology and ultimate

271 we have performed live Ca(2+) imaging in the nerve terminals of gonadotropin-releasing hormone neuron

272 al accumulation of intermediate filaments in nerve terminals of the neuromuscular synapse and improve

273 ce that during action potential (AP) firing, nerve terminals rely on the glucose transporter GLUT4 as

274 ivity-driven Ca(2+) influx and exocytosis at nerve terminals.

275 s such as TMEM16a in GPCR-activation of itch nerve terminals.

276 ral endings of the branch of the hypoglossal nerve that supplies the syrinx, the tracheosyringeal ner

277 opism, whether AAV can distribute to sensory nerves that innervate the bone or skeletal tissue has no

278 volvement (tumor >1 disc diameter from optic nerve), the mean (SD) largest basal diameter was 18.1 (1

279 FICANCE STATEMENT After injury to peripheral nerves, the myelin and Remak Schwann cells distal to the

280 ke timing, or phase locking, in the auditory nerve (time code), and the spatial distribution of respo

281 Nerve tissue contains a high density of chemical synapse

282 nd mitochondrial membrane potential in these nerve tissues.

283 sibilities in rats after crushing the tibial nerve (TN), and using Vesicular GLUtamate Transporter 1

284 SCPs migrate along the visceral motor nerve to the vicinity of the forming adrenal gland, wher

285 and the urethra (in the pudendal and pelvic nerves) to maintain continence or initiate micturition.

286 tasis by cancers that preferentially exploit nerve tract migration routes.

287 Injury of CNS nerve tracts remodels circuitry through dendritic spine

288 ny human glandular cancers metastasize along nerve tracts, but the mechanisms involved are generally

289 r the number of neurons changed in a sciatic nerve transection model of neuropathic pain or in the Co

290 After nerve transection, Ia afferent synapses and stretch refl

291 ough some serotypes of AAV are known to have nerve tropism, whether AAV can distribute to sensory ner

292 Neurofibromas are benign peripheral nerve tumors driven by NF1 loss in Schwann cells (SCs).

293 whereby afferent light signals in the optic nerve ultimately drive iris-sphincter-muscle contraction

294 or a height greater than 8 mm when the optic nerve was involved.

295 d demyelination of the spinal cord and optic nerves, we also show that thinly remyelinated axons with

296 Animals were sacrificed and corneal nerves were examined using immunocytochemistry and three

297 However, CGRP-positive nerves were significantly more superficial in mucosa fro

298 oducing macrophages were enriched in invaded nerves, which was associated with increased local tumor

299 ere recorded simultaneously from the sciatic nerve with a 16-contact cuff electrode.

300 s, indicating that rather than acting on the nerves within the pancreas slice, CCK cellular actions d