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1 se in the size of myelinated fibres on sural nerve biopsy.
2 ted and 2 uninfected animals underwent sural nerve biopsy.
3 from patients, due to the invasive nature of nerve biopsies.
4 analysed PMP22 messenger RNA levels in sural nerve biopsies.
5 oteins measurement, skin, muscular and sural nerve biopsies.
7 was a uniform feature in a total of 21 sural nerve biopsies and 'onion bulb' formations and/or thin m
8 abetic neuropathy progression in human sural nerve biopsies and describe their potential utility in c
9 s of patients with CMT1A and CMTX, including nerve biopsy, and conclude that coexistent inflammatory
11 nal screening for the PMP22 and P0 genes and nerve biopsy are therefore merited in patients with a ch
18 physiologic data were evaluated, and a sural nerve biopsy from one affected child was examined by imm
20 ircumstances replace the more invasive sural nerve biopsy in the morphological and molecular evaluati
22 europathies are difficult to distinguish and nerve biopsy may be required to demonstrate an inflammat
23 o the findings in younger patients, in their nerve biopsies, myelin thickness tended to be relatively
25 152) at the protein and mRNA levels in sural nerve biopsies of patients with chronic inflammatory dem
26 PMP22 messenger RNA levels in skin and sural nerve biopsies of patients with CMT1A compared with rela
28 nts with post-surgical neuropathies received nerve biopsies, of which 21 demonstrated increased infla
29 GAN mutations as an alternative to invasive nerve biopsy or systematic sequencing of the GAN gene.
36 clear imaging, electroencephalography, sural nerve biopsies, sleep evaluation and neuropsychometric t
42 and electrophysiological investigations and nerve biopsies were carried out on 61 patients shown to
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