ライフサイエンスコーパス: nerve conduction

1 roduction with the increased energy needs of nerve conduction.

2 ical illness and early recovery and assessed nerve conduction.

3 L-NNA abolished C-peptide effects on nerve conduction.

4 al phenotype with only a slight reduction in nerve conduction.

5 entifying the effects of these antibodies on nerve conduction.

6 ubjects and 11 diabetic patients with normal nerve conduction.

7 (4-AP) nor tetraethylammonium alters normal nerve conduction.

8 the sheath as an insulating layer for proper nerve conduction.

9 rain, muscle, immune, metabolism, mucoid and nerve conduction.

10 oligodendrocytes is essential for saltatory nerve conduction.

11 xhibit neurological abnormalities and slower nerve conduction.

12 approach to identify novel genes controlling nerve conduction.

13 rength, proprioception, tendon reflexes, and nerve conductions.

14 ssive peripheral neuropathy characterized by nerve conduction abnormalities as well as extensive musc

15 after transplantation, a composite score of nerve conduction abnormalities, an independent measure o

16 endent sodium channel antagonist that blocks nerve conduction, abolished both the oxidation current a

17 lar decompression leads to rapid recovery of nerve conduction across the indented root, a phenomenon

18 kinase, muscle strength and function, motor nerve conduction, activities of daily living, and erecti

19 They appear to be involved in nerve conduction and cell adhesion, but their precise ph

20 e-deficient (Cgt(-/-)) mice exhibit aberrant nerve conduction and display virtually no HSPC egress fr

21 tion, as well as electrophysiologic studies (nerve conduction and electrochemical skin conductance) a

22 ropathy as indicated by changes in behavior, nerve conduction and histology.

23 ne as this dose only prevented the change in nerve conduction and hot-plate latencies.

24 Two patients had temporal dispersion of nerve conduction and irregularity of conduction slowing,

25 rements in motor/sensory neuron myelination, nerve conduction and muscle function; however, the mecha

26 d time-dependently prevented the deficits in nerve conduction and nerve blood flow and biochemical ab

27 axons, as well as the functions of myelin in nerve conduction and neural circuits, and consider its p

28 ibition, prevented demyelination, leading to nerve conduction and neuromuscular performance recovery

29 Moreover, although deficits in nerve conduction and neuromuscular transmission were obs

30 the brain, myelin is important in regulating nerve conduction and neurotransmitter release by providi

31 Schwann cell dystroglycan results in slowed nerve conduction and nodal changes including reduced sod

32 orming oligodendrocytes facilitate saltatory nerve conduction and support neuronal functions in the m

33 n is essential for the rapidity of saltatory nerve conduction, and also provides trophic support for

34 xamination scores, visual analog pain scale, nerve conduction, and quantitative sensory testing.

35 cipants underwent standardized examinations, nerve conduction, and skin biopsy at 3 sites along the l

36 pinal motor axons, causing pain, inefficient nerve conduction, and the ectopic migration of oligodend

37 central autonomic regulation and sympathetic nerve conduction, and the norepinephrine transporter (NE

38 integrity of the myelin sheath and defective nerve conduction as indicated by morphological and elect

39 cal muscles, following recovery from chronic nerve conduction block.

40 oss of ion channel segregation, and impaired nerve conduction, but the mechanisms regulating their in

41 s and respond to stress or injury, and rapid nerve conduction can provide instantaneous input for mod

42 Peripheral motor and sensory nerve conduction, central motor conduction, a clinical n

43 eks of CNTF treatment significantly improved nerve conduction compared with untreated diabetic rats a

44 ts were classified into five groups by motor nerve conduction criteria; 69% were demyelinating, 3% ax

45 indices of abnormality of motor and sensory nerve conduction decreased significantly at all interval

46 nd was associated with 63% prevention of the nerve conduction defect and complete prevention of struc

47 ly corrected thermal hypoalgesia and sensory nerve conduction deficit without affecting motor nerve c

48 sciatic motor and hind-limb digital sensory nerve conduction deficits and sciatic nerve energy state

49 hermore, electrophysiological studies reveal nerve conduction deficits in the spinal cord of these mu

50 They also had motor and sensory nerve conduction deficits, tactile allodynia, and therma

51 resulted in significant normalization of the nerve conduction deficits.

52 schemia and axonal dwindling in the reported nerve conduction deficits.

53 Optic nerve conduction delays depend on RGC axon conduction ve

54 rupts paranodal junction assembly and delays nerve conduction during early development in mice.

55 patients who underwent electromyography and nerve conduction examinations, evidence of peripheral ne

56 otoxic effects of vincristine as measured by nerve conduction, gait, response to noxious stimuli and

57 showed normal large fibre sensory and motor nerve conduction; however, skin biopsy showed a signific

58 Thus, slower peripheral nerve conduction in carpal tunnel syndrome corresponds t

59 Rapid nerve conduction in myelinated nerves requires the clust

60 s may be linked to demyelination and reduced nerve conduction in peripheral neuropathies.

61 fully correlate with the presence of delayed nerve conduction in the median nerve.

62 We conclude they do not block nerve conduction in the terminals.

63 s and adhesion molecules essential for rapid nerve conduction in vertebrates are differentially targe

64 models displayed a significant decrement in nerve conduction, increase in tail distal motor latency

65 results in accelerated recovery of saltatory nerve conduction, increased innervation as detected by n

66 The motor and sensory nerve conduction indices increased significantly at all

67 Impairment of nerve conduction is common in neurodegenerative and neur

68 Upon demyelination, saltatory nerve conduction is disrupted, and patients experience n

69 Huxley's seminal discovery that the crux of nerve conduction is ion flow across cellular membranes.

70 neurological and reproductive deficits: CNS nerve conduction is slowed, hindlimb weakness is conspic

71 eeks, measured as recovery of affected optic nerve conduction latency using full-field visual evoked

72 both local to the wrist (i.e. median sensory nerve conduction latency) and in the brain (i.e. digit 2

73 The abnormality of attributes of nerve conduction may be used to validate the dysfunction

74 ociated with severe paroxysmal pain in which nerve conduction may have a key role.

75 blood flow (NBF) vs. other factors in motor nerve conduction (MNC) slowing in short-term diabetes wa

76 By promoting rapid, efficient nerve conduction, myelination also made possible the dev

77 In addition to increasing the speed of nerve conduction, myelination has emerged as a source of

78 mechanism by which PMP22 deficiency affects nerve conduction not through removal of myelin, but thro

79 ith the blood chemistry panel and peripheral nerve conduction of Chinese farmers.

80 In all, 155 were tested for nerve conduction of sensory and motor median nerve laten

81 ptoms had only a 40% sensitivity for delayed nerve conduction on latency testing.

82 Scrambled C-peptide was without effect on nerve conduction or perfusion.

83 B-12, holotranscobalamin, or cB-12 with any nerve conduction outcome.

84 ed no significant deleterious effects on any nerve conduction parameter measured and caused milder, l

85 on was used to investigate associations with nerve conduction parameters in participants without poly

86 Improvements in peripheral nerve conduction positively correlated with cytokine-ind

87 Other measures including nerve conduction potentials did not improve.

88 ellular location of Caspr was disrupted, and nerve conduction properties remained immature.

89 eurological examination and abnormalities of nerve conduction, quantitative sensory tests, and quanti

90 Patterns of hand symptoms were compared with nerve conduction results and associations weighted back

91 s of capacitance and serious disturbances in nerve conduction, severely damaging brain function.

92 etic and galactose-induced motor and sensory nerve conduction slowing and nerve energy failure that w

93 s have an increased risk for developing both nerve conduction slowing and small sensory fiber neuropa

94 limb digital sensory, but not sciatic motor, nerve conduction slowing and thermal and mechanical hypo

95 et of 8 weeks of galactose feeding prevented nerve conduction slowing in a dose-dependent manner.

96 ry nerve terminals, thermal hypoalgesia, and nerve conduction slowing in diverse rodent models of dia

97 e conduction deficit without affecting motor nerve conduction slowing.

98 To meet the energy demands of nerve conduction, small mitochondria are transported to

99 A subset of patients underwent nerve conduction studies (n = 13).

100 mination, needle electromyography (EMG), and nerve conduction studies (NCS) were performed before and

101 ertaken in 108 type 2 diabetic patients with nerve conduction studies (NCS), HbA(1c) levels, and tota

102 Neurophysiological measures included nerve conduction studies (NCS), quantitative sensory tes

103 cal clinical presentations, demyelination on nerve conduction studies (p = 0.0005), and difficult ide

104 f peripheral nerve lesions routinely involve nerve conduction studies and electromyography.

105 and a global symptom assessment, as well as nerve conduction studies and occurrence of new plantar f

106 l examination, quantitative sensory testing, nerve conduction studies and skin biopsy for intraepider

107 exopathy was made after electromyography and nerve conduction studies and the etiology of radiculopat

108 a lesion site precisely, where fractionated nerve conduction studies are not applicable.

109 ited, however, by problems inherent in using nerve conduction studies as a criterion standard.

110 Nerve conduction studies assessing median nerve sensory

111 l syndrome should be investigated first with nerve conduction studies but consideration should be giv

112 rical irritability; 13 patients had abnormal nerve conduction studies but four of these had long-stan

113 s of peripheral nerve cannot proceed without nerve conduction studies but particular interest has bee

114 is also disagreement about whether pudendal nerve conduction studies can be used to predict outcome

115 ropsychological testing remained stable, and nerve conduction studies have shown improvement.

116 ralysis in 10 percent; in all of the latter, nerve conduction studies indicated an axonal polyneuropa

117 Nerve conduction studies localised abnormalities to the

118 Nerve conduction studies revealed no evidence of neuropa

119 Electromyography/nerve conduction studies showed evidence for a mild myop

120 Nerve conduction studies showed normal large fibre senso

121 Serial nerve conduction studies showed progressively reduced am

122 Nerve conduction studies showed severe axonal sensorimot

123 ropathy caused by an H10P MPZ mutation whose nerve conduction studies suggested severe axonal loss bu

124 Nerve conduction studies were also performed, and neurop

125 Nerve conduction studies were normal, but increased jitt

126 In all cases, large fibre nerve conduction studies were normal.

127 Results of nerve conduction studies were retrieved and assessed.

128 Blood examination and nerve conduction studies were unremarkable.

129 urological function by clinical examination, nerve conduction studies, and autonomic function tests i

130 n, functional and walking disability scores, nerve conduction studies, and muscle biopsies are report

131 Physical examinations, questionnaires, nerve conduction studies, and NFTs, including the Jebsen

132 ength, tendon reflexes, vibration sense, and nerve conduction studies, and reduced intraepidermal ner

133 ile capacity and performed electromyography, nerve conduction studies, and vastus lateralis biopsies

134 Clinical examinations, nerve conduction studies, and vibratory perception thres

135 Nerve conduction studies, needle electromyography, singl

136 Neuroimaging, nerve conduction studies, neurological examinations, and

137 rs were studied, using clinical examination, nerve conduction studies, perimetry, optical coherence t

138 Nerve conduction studies, quantitative sensory testing,

139 y skin biopsy, quantitative sensory testing, nerve conduction studies, screening of genomic DNA for m

140 h measurements may be comparable to those of nerve conduction studies, though their prognostic value

141 erties and are complementary to conventional nerve conduction studies.

142 commonly than motor axonal polyneuropathy on nerve conduction studies.

143 questionnaire, neurological examination and nerve conduction studies.

144 sts for the evaluation of the motor unit are nerve conduction studies/electromyography (NCS/EMG) and

145 Toxicity assessment included nerve-conduction studies (NCS) and radionuclide assessme

146 Among 46 patients in whom nerve-conduction studies and electromyography were perfo

147 The nerve conduction study (NCS) and electromyography (EMG)

148 cale scores, greater reduction of upper limb nerve conduction study amplitudes, more frequent occurre

149 forms (7.7% vs. 5.3%; p=0.63) with a single nerve conduction study as compared with when serial elec

150 luded intraepidermal nerve fiber density and nerve conduction study parameters.

151 smission at neuromuscular junctions, whereas nerve conduction, synaptic structure, and spontaneous sy

152 questionnaire on current hand symptoms, and nerve conduction testing in samples of subjects with and

153 e neuropathy score version 1 or 2 and slower nerve conductions than the other groups, and severity in

154 Digital nerve conduction underwent a dose-dependent improvement

155 Nerve conduction, vascular and airway dynamics, and prop

156 f memory, attention, visuomotor function, or nerve conduction velocities (average z scores were very

157 voluntary locomotor activity, reduced motor nerve conduction velocities (MNCVs) and muscle atrophy.

158 uced diabetic rats with decreased peripheral nerve conduction velocities (NCV).

159 and FA values were correlated to electrical nerve conduction velocities (NCVs) with Pearson correlat

160 Patients with CMT1A typically have slowed nerve conduction velocities (NCVs), reduced compound mot

161 Sural nerve myelinated fiber density (MFD), nerve conduction velocities (NCVs), vibration perception

162 a markedly low birth weight, very slow motor nerve conduction velocities and a general decrease in th

163 muscle action potential amplitudes, improved nerve conduction velocities and ameliorated muscle stren

164 ice experienced significant deterioration in nerve conduction velocities and IENF density and develop

165 on protein fully restored motor- and sensory-nerve conduction velocities and maintained the axonal ca

166 (R98C/R98C) mice develop weakness, abnormal nerve conduction velocities and morphologically abnormal

167 g, mitochondrial DNA deletions, reduction of nerve conduction velocities and of heat sensation, and b

168 al recovery as assessed by motor and sensory nerve conduction velocities and sciatic functional index

169 Ulnar and median nerve conduction velocities confirmed the clinical diagn

170 Here we measured nerve conduction velocities in a rabbit model of limb le

171 yses also reveal that the reduction of motor nerve conduction velocities in affected patients is prop

172 g the formation of compact myelin and normal nerve conduction velocities in the homozygous state.

173 Motor nerve conduction velocities of the fastest fibers are se

174 ents from six pedigrees (n = 38) had reduced nerve conduction velocities ranging from 7.5 to 27.0m/se

175 Motor nerve conduction velocities showed normal values (48-53

176 orphology and impaired transmission, reduced nerve conduction velocities, and a loss of large-diamete

177 rial nutritive blood flow, motor and sensory nerve conduction velocities, and metabolic abnormalities

178 ep-tendon reflexes, sensory impairment, slow nerve conduction velocities, and nerve demyelination.

179 METHODS AND We found that motor and sensory nerve conduction velocities, blood flow, and capillary d

180 nerve fibers, specifically sural or sciatic nerve conduction velocities, but significantly improved

181 by significant slowing of motor and sensory nerve conduction velocities, compared with nondiabetic c

182 ropathy that presents in adults, with normal nerve conduction velocities, designated as a 'CMT2' form

183 Motor and sensory digital nerve conduction velocities, sciatic nerve indexes of ox

184 xtremities and displayed a mild reduction of nerve conduction velocities.

185 of onset and the degree of slowing of motor nerve conduction velocities.

186 by significant slowing of motor and sensory nerve conduction velocities.

187 quadriceps muscle contractility, and sciatic nerve conduction velocities.

188 rological deficit and had profoundly reduced nerve conduction velocities.

189 g of axons, delayed myelination, and reduced nerve conduction velocities.

190 vascular relaxation precede slowing of motor nerve conduction velocity (MNCV) and decreased sciatic n

191 ld) clearly developed manifest sciatic motor nerve conduction velocity (MNCV) and hind-limb digital s

192 and axonal loss, which underlie slowed motor nerve conduction velocity (MNCV) and reduced compound mu

193 Endoneurial blood flow and motor nerve conduction velocity (MNCV) were impaired in all gr

194 ic with streptozotocin, and changes in motor nerve conduction velocity (MNCV), mechanical and thermal

195 al abnormalities of EDN, i.e., reduced motor nerve conduction velocity (MNCV), total and endoneurial

196 used on either behaviors or acute changes in nerve conduction velocity (NCV) and amplitude, but great

197 They develop nerve conduction velocity (NCV) and sensory deficits pri

198 The nerve conduction velocity (NCV) of nondiabetic male Wist

199 Nerve conduction velocity (NCV) significantly improved i

200 in transducing high glucose into deficits in nerve conduction velocity (NCV) that are characteristic

201 on nerve laser Doppler blood flow (NLDF) and nerve conduction velocity (NCV) were assessed after 1-16

202 heral neuropathy (DPN) is marked by impaired nerve conduction velocity (NCV), reduced nerve blood flo

203 abetes-induced deficits in sensory and motor nerve conduction velocity (P < 0.05).

204 Peroneal motor nerve conduction velocity (p=0.03) and M-wave amplitude

205 elocity (MNCV) and hind-limb digital sensory nerve conduction velocity (SNCV) deficits, thermal hypoa

206 ional attachment at the paranode and reduces nerve conduction velocity 3-fold.

207 een a ratio of 0.8 and 1.5, histological and nerve conduction velocity abnormalities are observed, bu

208 al defects, transgenic mice also have slower nerve conduction velocity and defects in their responses

209 ical deficits were associated with decreased nerve conduction velocity and increased sensory threshol

210 2 was positively correlated with decreasing nerve conduction velocity and increasing pain severity.

211 ripheral neuropathy characterized by reduced nerve conduction velocity and myelin outfoldings and inf

212 abetic nerve associated with improvements of nerve conduction velocity and pathologic changes.

213 Nerve conduction velocity and psychophysical data were a

214 s of large-diameter myelinated axons, slowed nerve conduction velocity and reduced amplitude of compo

215 s accompanied by decreased motor and sensory nerve conduction velocity and reduced compound muscle ac

216 th significant lowering of motor and sensory nerve conduction velocity but no alteration in the mean

217 People with severe PAD have poorer peroneal nerve conduction velocity compared with people with mild

218 it normalizes polyols and reduces the motor nerve conduction velocity deficit by 59% relative to dia

219 lgesia, tactile allodynia, motor and sensory nerve conduction velocity deficits, and reduction in tib

220 KEY POINTS: Changes in nerve conduction velocity following an impulse (i.e. vel

221 ytostatic agents and a moderate reduction of nerve conduction velocity for cisplatin and paclitaxel.

222 syndrome, but not healthy controls, sensory nerve conduction velocity for Digits 2 and 3 was slower

223 Composite motor nerve conduction velocity for the median, ulnar, and per

224 e associated with a significant reduction in nerve conduction velocity in the optic nerve.

225 ct) techniques, such as electromyography and nerve conduction velocity measurement, do not reliably p

226 l content was elevated (P < 0.001) and motor nerve conduction velocity reduced (P < 0.05) in galactos

227 All strategies prevented loss of nerve conduction velocity resulting from STZ-induced dia

228 rt internodal lengths and associated reduced nerve conduction velocity seen in the absence of periaxi

229 (R/S)-1 and (R)-1 partially prevented motor nerve conduction velocity slowing in a mouse model of ty

230 Upper limb motor nerve conduction velocity was 19.9 m/s +/- 1.3 (SEM), ra

231 Motor nerve conduction velocity was moderately reduced in the

232 young mice with short mutant Schwann cells, nerve conduction velocity was reduced and motor function

233 Sciatic nerve conduction velocity was significantly decreased in

234 Caudal nerve conduction velocity was significantly slower in mi

235 erences in neurobehavioral assessments or in nerve conduction velocity when compared with children wh

236 d were associated with a 25-30% reduction in nerve conduction velocity, all of which were completely

237 itol supplementation on nerve Na+-K+-ATPase, nerve conduction velocity, and myelinated nerve fiber pa

238 , Na+/K+ -ATPase, vasoactive prostaglandins, nerve conduction velocity, and pathologic changes.

239 onduction velocity, hindlimb digital sensory nerve conduction velocity, and sciatic nerve concentrati

240 eripheral neuropathy characterized by slowed nerve conduction velocity, axon loss, and distinctive my

241 inhibited a diabetes-induced defect in ulnar nerve conduction velocity, but neither agent was found t

242 TDs, produced significant deficits in caudal nerve conduction velocity, caudal amplitude and digital

243 DM, we observed that, in addition to reduced nerve conduction velocity, db/db mice also develop PNH.

244 Sciatic motor nerve conduction velocity, hindlimb digital sensory nerv

245 ozygous R98C animals, but it did not improve nerve conduction velocity, myelin thickness, G-ratios or

246 Transplantation significantly improved tail nerve conduction velocity, Na(+)-K(+)-ATPase activity, a

247 Effects of each drug on caudal and digital nerve conduction velocity, nerve amplitude, and sciatic

248 ow, significantly improved motor and sensory nerve conduction velocity, prevented the development of

249 layed maturation of nodes of Ranvier, slowed nerve conduction velocity, reduced muscle mass and reduc

250 Diabetes caused a decrease in nerve conduction velocity, thermal hypoalgesia, and a re

251 rats fed high-fat diets caused a decrease in nerve conduction velocity, thermal hypoalgesia, and intr

252 tions in sciatic motor and saphenous sensory nerve conduction velocity, which were 62 and 78% correct

253 nset of functional changes such as decreased nerve conduction velocity.

254 d progressive demyelination and a slowing of nerve conduction velocity.

255 ore there were any detectable alterations in nerve conduction velocity.

256 es myelin outfoldings that impair the normal nerve conduction velocity.

257 o results in a severe decrease in peripheral nerve conduction velocity.

258 elinating neuropathy and to severely reduced nerve conduction velocity.

259 x 10(-9)), indicating that INPP4B regulates nerve conduction velocity.

260 e exhibit impaired motor function and slower nerve conduction velocity.

261 al hyperalgesia, cold allodynia, and sciatic nerve conduction velocity.

262 ich, in turn, has a significant influence on nerve conduction velocity.

263 nd 21b,d all produce a reversible slowing of nerve-conduction velocity in the mice, the onset of whic

264 did not induce the phenomenon of slowing of nerve-conduction velocity.

265 trodotoxin-resistant sodium channel, sciatic nerve conduction was at least as sensitive to tetrodotox

266 logical evaluation of both sensory and motor nerve conduction was performed.

267 l examinations of P30 op/op mice showed that nerve conduction was significantly delayed but not block

268 This defect is associated with slowed nerve conduction, which could contribute to behavioral d