ライフサイエンスコーパス: nerve conduction velocity

1 x 10(-9)), indicating that INPP4B regulates nerve conduction velocity.

2 e exhibit impaired motor function and slower nerve conduction velocity.

3 al hyperalgesia, cold allodynia, and sciatic nerve conduction velocity.

4 ich, in turn, has a significant influence on nerve conduction velocity.

5 es myelin outfoldings that impair the normal nerve conduction velocity.

6 nset of functional changes such as decreased nerve conduction velocity.

7 d progressive demyelination and a slowing of nerve conduction velocity.

8 ore there were any detectable alterations in nerve conduction velocity.

9 o results in a severe decrease in peripheral nerve conduction velocity.

10 elinating neuropathy and to severely reduced nerve conduction velocity.

11 did not induce the phenomenon of slowing of nerve-conduction velocity.

12 rological deficit and had profoundly reduced nerve conduction velocities.

13 g of axons, delayed myelination, and reduced nerve conduction velocities.

14 xtremities and displayed a mild reduction of nerve conduction velocities.

15 of onset and the degree of slowing of motor nerve conduction velocities.

16 quadriceps muscle contractility, and sciatic nerve conduction velocities.

17 by significant slowing of motor and sensory nerve conduction velocities.

18 ional attachment at the paranode and reduces nerve conduction velocity 3-fold.

19 een a ratio of 0.8 and 1.5, histological and nerve conduction velocity abnormalities are observed, bu

20 d were associated with a 25-30% reduction in nerve conduction velocity, all of which were completely

21 a markedly low birth weight, very slow motor nerve conduction velocities and a general decrease in th

22 muscle action potential amplitudes, improved nerve conduction velocities and ameliorated muscle stren

23 ice experienced significant deterioration in nerve conduction velocities and IENF density and develop

24 on protein fully restored motor- and sensory-nerve conduction velocities and maintained the axonal ca

25 (R98C/R98C) mice develop weakness, abnormal nerve conduction velocities and morphologically abnormal

26 g, mitochondrial DNA deletions, reduction of nerve conduction velocities and of heat sensation, and b

27 al recovery as assessed by motor and sensory nerve conduction velocities and sciatic functional index

28 al defects, transgenic mice also have slower nerve conduction velocity and defects in their responses

29 ical deficits were associated with decreased nerve conduction velocity and increased sensory threshol

30 2 was positively correlated with decreasing nerve conduction velocity and increasing pain severity.

31 ripheral neuropathy characterized by reduced nerve conduction velocity and myelin outfoldings and inf

32 abetic nerve associated with improvements of nerve conduction velocity and pathologic changes.

33 Nerve conduction velocity and psychophysical data were a

34 s of large-diameter myelinated axons, slowed nerve conduction velocity and reduced amplitude of compo

35 s accompanied by decreased motor and sensory nerve conduction velocity and reduced compound muscle ac

36 orphology and impaired transmission, reduced nerve conduction velocities, and a loss of large-diamete

37 rial nutritive blood flow, motor and sensory nerve conduction velocities, and metabolic abnormalities

38 ep-tendon reflexes, sensory impairment, slow nerve conduction velocities, and nerve demyelination.

39 itol supplementation on nerve Na+-K+-ATPase, nerve conduction velocity, and myelinated nerve fiber pa

40 , Na+/K+ -ATPase, vasoactive prostaglandins, nerve conduction velocity, and pathologic changes.

41 onduction velocity, hindlimb digital sensory nerve conduction velocity, and sciatic nerve concentrati

42 f memory, attention, visuomotor function, or nerve conduction velocities (average z scores were very

43 eripheral neuropathy characterized by slowed nerve conduction velocity, axon loss, and distinctive my

44 METHODS AND We found that motor and sensory nerve conduction velocities, blood flow, and capillary d

45 th significant lowering of motor and sensory nerve conduction velocity but no alteration in the mean

46 nerve fibers, specifically sural or sciatic nerve conduction velocities, but significantly improved

47 inhibited a diabetes-induced defect in ulnar nerve conduction velocity, but neither agent was found t

48 TDs, produced significant deficits in caudal nerve conduction velocity, caudal amplitude and digital

49 People with severe PAD have poorer peroneal nerve conduction velocity compared with people with mild

50 by significant slowing of motor and sensory nerve conduction velocities, compared with nondiabetic c

51 Ulnar and median nerve conduction velocities confirmed the clinical diagn

52 DM, we observed that, in addition to reduced nerve conduction velocity, db/db mice also develop PNH.

53 it normalizes polyols and reduces the motor nerve conduction velocity deficit by 59% relative to dia

54 lgesia, tactile allodynia, motor and sensory nerve conduction velocity deficits, and reduction in tib

55 ropathy that presents in adults, with normal nerve conduction velocities, designated as a 'CMT2' form

56 KEY POINTS: Changes in nerve conduction velocity following an impulse (i.e. vel

57 ytostatic agents and a moderate reduction of nerve conduction velocity for cisplatin and paclitaxel.

58 syndrome, but not healthy controls, sensory nerve conduction velocity for Digits 2 and 3 was slower

59 Composite motor nerve conduction velocity for the median, ulnar, and per

60 Sciatic motor nerve conduction velocity, hindlimb digital sensory nerv

61 Here we measured nerve conduction velocities in a rabbit model of limb le

62 yses also reveal that the reduction of motor nerve conduction velocities in affected patients is prop

63 g the formation of compact myelin and normal nerve conduction velocities in the homozygous state.

64 e associated with a significant reduction in nerve conduction velocity in the optic nerve.

65 nd 21b,d all produce a reversible slowing of nerve-conduction velocity in the mice, the onset of whic

66 ct) techniques, such as electromyography and nerve conduction velocity measurement, do not reliably p

67 vascular relaxation precede slowing of motor nerve conduction velocity (MNCV) and decreased sciatic n

68 ld) clearly developed manifest sciatic motor nerve conduction velocity (MNCV) and hind-limb digital s

69 and axonal loss, which underlie slowed motor nerve conduction velocity (MNCV) and reduced compound mu

70 Endoneurial blood flow and motor nerve conduction velocity (MNCV) were impaired in all gr

71 ic with streptozotocin, and changes in motor nerve conduction velocity (MNCV), mechanical and thermal

72 al abnormalities of EDN, i.e., reduced motor nerve conduction velocity (MNCV), total and endoneurial

73 voluntary locomotor activity, reduced motor nerve conduction velocities (MNCVs) and muscle atrophy.

74 ozygous R98C animals, but it did not improve nerve conduction velocity, myelin thickness, G-ratios or

75 Transplantation significantly improved tail nerve conduction velocity, Na(+)-K(+)-ATPase activity, a

76 uced diabetic rats with decreased peripheral nerve conduction velocities (NCV).

77 used on either behaviors or acute changes in nerve conduction velocity (NCV) and amplitude, but great

78 They develop nerve conduction velocity (NCV) and sensory deficits pri

79 The nerve conduction velocity (NCV) of nondiabetic male Wist

80 Nerve conduction velocity (NCV) significantly improved i

81 in transducing high glucose into deficits in nerve conduction velocity (NCV) that are characteristic

82 on nerve laser Doppler blood flow (NLDF) and nerve conduction velocity (NCV) were assessed after 1-16

83 heral neuropathy (DPN) is marked by impaired nerve conduction velocity (NCV), reduced nerve blood flo

84 and FA values were correlated to electrical nerve conduction velocities (NCVs) with Pearson correlat

85 Patients with CMT1A typically have slowed nerve conduction velocities (NCVs), reduced compound mot

86 Sural nerve myelinated fiber density (MFD), nerve conduction velocities (NCVs), vibration perception

87 Effects of each drug on caudal and digital nerve conduction velocity, nerve amplitude, and sciatic

88 Motor nerve conduction velocities of the fastest fibers are se

89 abetes-induced deficits in sensory and motor nerve conduction velocity (P < 0.05).

90 Peroneal motor nerve conduction velocity (p=0.03) and M-wave amplitude

91 ow, significantly improved motor and sensory nerve conduction velocity, prevented the development of

92 ents from six pedigrees (n = 38) had reduced nerve conduction velocities ranging from 7.5 to 27.0m/se

93 l content was elevated (P < 0.001) and motor nerve conduction velocity reduced (P < 0.05) in galactos

94 layed maturation of nodes of Ranvier, slowed nerve conduction velocity, reduced muscle mass and reduc

95 All strategies prevented loss of nerve conduction velocity resulting from STZ-induced dia

96 Motor and sensory digital nerve conduction velocities, sciatic nerve indexes of ox

97 rt internodal lengths and associated reduced nerve conduction velocity seen in the absence of periaxi

98 Motor nerve conduction velocities showed normal values (48-53

99 (R/S)-1 and (R)-1 partially prevented motor nerve conduction velocity slowing in a mouse model of ty

100 elocity (MNCV) and hind-limb digital sensory nerve conduction velocity (SNCV) deficits, thermal hypoa

101 Diabetes caused a decrease in nerve conduction velocity, thermal hypoalgesia, and a re

102 rats fed high-fat diets caused a decrease in nerve conduction velocity, thermal hypoalgesia, and intr

103 Upper limb motor nerve conduction velocity was 19.9 m/s +/- 1.3 (SEM), ra

104 Motor nerve conduction velocity was moderately reduced in the

105 young mice with short mutant Schwann cells, nerve conduction velocity was reduced and motor function

106 Sciatic nerve conduction velocity was significantly decreased in

107 Caudal nerve conduction velocity was significantly slower in mi

108 erences in neurobehavioral assessments or in nerve conduction velocity when compared with children wh

109 tions in sciatic motor and saphenous sensory nerve conduction velocity, which were 62 and 78% correct