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1 a- or intracellular action at the frog motor nerve ending.
2  nerve, at a bend in the nerve, and at a cut nerve ending.
3 sible target sites for phorbol esters in the nerve ending.
4 to regulate cytoskeletal organization in the nerve ending.
5 s were placed within 10 microns of the motor nerve ending.
6  resides, with DC dendrites crossing several nerve endings.
7  TRPA1, an excitatory ion channel on sensory nerve endings.
8 ing capsaicin-sensitive perivascular sensory nerve endings.
9 decreases in calcium currents at mouse motor nerve endings.
10 n were immunolabeled to quantitate epidermal nerve endings.
11 ing the basement membrane and branching into nerve endings.
12  treatment causes RII protein to increase at nerve endings.
13 mediators and activation of afferent sensory nerve endings.
14 wnstream of calcium entry at amphibian motor nerve endings.
15 s then distributed to peripheral cholinergic nerve endings.
16 sensory nerve distally toward the peripheral nerve endings.
17 orepinephrine into presynaptic noradrenergic nerve endings.
18 d result from a decreased Ca(2+) influx into nerve endings.
19 ing ligand-gated ion channels in presynaptic nerve endings.
20 l as some intraepidermal and free myelinated nerve endings.
21 igh-affinity monoamine uptake into rat brain nerve endings.
22 lting in elevated neurotransmitter levels in nerve endings.
23 -1) or COX-2 produce hyperalgesia in sensory nerve endings.
24 at surrounds the synaptic vesicle cluster in nerve endings.
25 ter axons that appeared to terminate as free nerve endings.
26  stimulating transmitter(s) for chemosensory nerve endings.
27 al properties from their counterparts in the nerve endings.
28 rphology resembles human mechanosensory free nerve endings.
29 dulate their sensitivity directly in sensory nerve endings.
30 itatory nicotinic ACh receptors on the motor nerve endings.
31 ng specific (vanilloid) receptors on sensory nerve endings.
32 naptic opioid receptors in mammalian central nerve endings.
33 epinephrine release assay in rat hippocampal nerve endings.
34 larization-induced entry of Ca2+ into intact nerve endings.
35 both intact and streptolysin-O permeabilized nerve endings.
36 Capsaicin is a specific activator of sensory nerve endings.
37 logical changes starting at their peripheral nerve endings.
38 al stimuli into the depolarization of distal nerve endings.
39 horing protein for PKC epsilon within intact nerve endings.
40 uroendocrine epithelial cells, and bronchial nerve endings.
41 loss or selective degeneration of peripheral nerve endings.
42 , where it replicated and infected the nodal nerve endings.
43 1 cannabinoid receptors localized on sensory nerve endings.
44 ctor pili muscles, Merkel cells, and sensory nerve endings.
45 TH-negative neurons that form epidermal-free nerve endings.
46  transducer in nociceptive and thermosensory nerve endings.
47 esults in synaptic abnormalities in auditory nerve endings.
48 structure-function studies of vagal afferent nerve endings.
49 t all nerves within the CE terminate as free nerve endings.
50  A(1) adenosine receptors at mammalian motor nerve endings.
51 l-epidermal junction, adjacent to peripheral nerve endings.
52 -like immunoreactive, thin and varicose free nerve endings.
53 ecording of coupled glomus cells and carotid nerve endings.
54 ts differentially innervate the central free nerve endings.
55                At the same time, HSV invades nerve ending-abutting infected cells and is transported
56 ported to be released from cutaneous sensory nerve endings after hapten application, we determined wh
57 roglia in striatum and they protect dopamine nerve endings against drug-induced nerve terminal damage
58  solitary chemosensory cells, and epithelial nerve endings, all of which regenerate after amputation.
59          Substance P labeled a group of free nerve endings along the outer edge of Eimer's organ, ind
60           Pathologic changes in the afferent nerve ending and cell body were evaluated with light and
61 ce of P2 receptor agonists at the peripheral nerve ending and is decreased by the presence of P2 anta
62 ed acetylcholine receptors associated with a nerve ending and were thus considered to form anatomic m
63 apid, transient component observed in 70% of nerve endings and a voltage-activation relationship that
64 onsisting of Merkel cells and Abeta-afferent nerve endings and are localized in fingertips, whisker h
65 e in that they possess properties of central nerve endings and endocrine cells.
66 ody, regulating the excitability of afferent nerve endings and glomus cells (putative chemoreceptor c
67                             Coupling between nerve endings and glomus cells was more complex, When a
68 d hypobaric hypoxia did not change the Em of nerve endings and glomus cells.
69 ion channels, which are expressed in sensory nerve endings and in skin, respond to distinct thermal t
70 ssociation with chromaffin cells, occasional nerve endings and macrophages.
71 the branching of diabetes-suppressed sensory nerve endings and regeneration in the diabetic corneas.
72 n of neurotrophin receptors (Trks) at distal nerve endings and retrograde propagation of Trk activati
73  firefly lantern in cells interposed between nerve endings and the light-producing photocytes.
74 nsporters 1-5, EAATs1-5) located on both the nerve endings and the surrounding glial cells.
75 escued a significant number of VAChT stained nerve endings and treatment of fish with E(2) alone exhi
76 te in mechanically sensitive ion channels in nerve endings, and hearing in mechanically sensitive ion
77 s increased pulpal NGF, sprouting of sensory nerve endings, and increased immunoreactivity for the se
78 n) stimulates abdominal sympathetic afferent nerve endings, and recently have documented increased co
79 ed into those that target the vagal afferent nerve endings, and those that target neural activity wit
80 lation of mechanoreceptors located in the BR nerve-endings, and modulation of the action potential fr
81 kout mice lacking synapsin I and II, sensory nerve endings are normally developed but not stimulated
82  that control the formation and migration of nerve endings are only beginning to be unraveled.
83                             Nasal trigeminal nerve endings are particularly sensitive to oxidants for
84 that Merkel cells rather than Abeta-afferent nerve endings are primary sites of tactile transduction
85         We next asked whether proprioceptive nerve endings are similarly affected in the spinal cord
86                                      Most TG nerve endings are subepithelial, so this colonization im
87  than in slices, suggesting that presynaptic nerve endings are the primary site of inhibition of upta
88          The effects of capsaicin on sensory nerve endings are well known; however, little is known r
89  were able to exclude T cells or sympathetic nerve endings as sources of the injury-modulating catech
90 periodontal ligament had fewer thin branched nerve endings at all ages.
91 epithelial defects, and a reduced density of nerve endings at the center of the cornea.
92                Neurectomy of the intercostal nerve endings at the level of the abdominal wall is an e
93 g the symptomatic phase and after peripheral nerve endings begin to degenerate.
94 mM K+, was 246.1 pS, compared with 213 pS in nerve ending BK channels (P<0.01).
95                           Whilst blockade of nerve ending BK channels markedly slowed the repolarizat
96                        Activation of C-fiber nerve endings by pressure was attributable to stimulatio
97 ne inhibits ACh release from mammalian motor nerve endings by reducing Ca(2+) calcium entry through v
98                 This depolarizes the sensory nerve endings by simultaneously closing M-type potassium
99 of the endogenous Ca2+ buffering capacity of nerve endings by treatment with the mitochondrial Ca2+ u
100 annels or, as is the case at amphibian motor nerve endings, by an effect downstream of Ca(2+) entry.
101 cretion whilst simultaneously decreasing the nerve ending calcium currents that promote evoked releas
102 ng that the distribution of mechanosensitive nerve endings can be inferred by visual inspection of th
103 nd indicate that TRPM3 activation in sensory nerve endings can contribute to neurogenic inflammation.
104               Although free intra-epithelial nerve endings can detect certain lipophilic irritants (e
105  local kallikrein-kinin system in adrenergic nerve endings capable of generating enough bradykinin to
106 mine release in isolated hearts, sympathetic nerve endings (cardiac synaptosomes), and PC12 cells bea
107 ls, and resident cells can overtly stimulate nerve endings, cause long-lasting changes in neuronal ex
108 synaptic depolarizing potential (SDP) in the nerve endings caused by release of an excitatory transmi
109 ble channels in the patch) for cell body and nerve ending channels were similar: 11 vs. 14 mV per e-f
110  (H(3)Rs) are present in cardiac sympathetic nerve endings (cSNE) of animals and humans, where they a
111 otor axons in SOD1(G93A) mice, Ia/II sensory nerve endings degenerate in the absence of obvious alter
112                                 Baroreceptor nerve endings detect acute fluctuations in arterial pres
113  capsaicin, which depletes SP in the sensory nerve endings, eliminates stress-control differences in
114 d during aging in homogenate, cytosol, and a nerve ending-enriched fraction from the hippocampus.
115 staining showed that alpha7 subunit-positive nerve endings enveloped tyrosine hydroxylase-positive gl
116 tivity by using NE released from sympathetic nerve endings evolved early in the history of mammals.
117 llowing the activation of peripheral sensory nerve endings following damage or exposure to inflammato
118             During development, each type of nerve endings forms at different time point.
119 2+) exchange, as measured in intact isolated nerve endings from mouse cortex and in intact varicositi
120 via confocal imaging of isolated presynaptic nerve endings from rat hippocampus and neocortex.
121 9, would stimulate group III and IV afferent nerve endings from the hindlimb of the anesthetized cat.
122 s cell was stimulated, current spread to the nerve ending (GC/NE coupling) was similar in magnitude (
123 different mechanisms for oxygen sensing: the nerve endings generate action potentials in association
124                                   Also, when nerve endings had an Em more negative than -40 mV showed
125 al cell bodies in trigeminal ganglia (TG) to nerve ending in the noses and corneas of infected cattle
126 zed by each other; and (3) DRG neurones with nerve endings in a glycolytic muscle developed greater i
127  likely to be reached at cardiac sympathetic nerve endings in advanced congestive heart failure, prom
128 to protons and capsaicin than did those with nerve endings in an oxidative muscle.
129 cytes and nerve fibers showed intraepidermal nerve endings in contact with melanocytes.
130 gh many rows of myenteric ganglia and formed nerve endings in discrete anatomical layers.
131 lcitonin gene-related peptide-positive, free nerve endings in footpad epidermis.
132 t neurons allowing for bright imaging of the nerve endings in living tissues and suitable for structu
133 nificant alterations at Ia/II proprioceptive nerve endings in muscle spindles before the symptomatic
134          However, analysis of proprioceptive nerve endings in muscles revealed early and significant
135                    Furthermore, encapsulated nerve endings in Pacinian corpuscles also contain reacti
136      The first state directs virus access to nerve endings in peripheral tissue, whereas the second d
137 oteins are found on microvesicles in sensory nerve endings in peripheral tissues.
138  of rapidly adapting nodose ganglion-derived nerve endings in response to mechanical stimuli.
139  Arterial baroreceptors are mechanosensitive nerve endings in the aortic arch and carotid sinus that
140 e ATP is coreleased with NE from sympathetic nerve endings in the heart, we investigated whether ATP
141  pH 6.7 are ASIC(3)-like in DRG neurons with nerve endings in the hindlimb muscles, (2) a greater aci
142 y M2 muscarinic receptors on parasympathetic nerve endings in the lungs decrease release of acetylcho
143 phological identification of spinal afferent nerve endings in the mammalian urinary bladder.
144 fficiently transported retrogradely from the nerve endings in the nose and eye to cell bodies in the
145 ircular and longitudinal muscle cells and to nerve endings in the plexuses.
146       Touch sensitivity in animals relies on nerve endings in the skin that convert mechanical force
147 sults from the excitation of primary sensory nerve endings in the skin, but the underlying molecular
148 soma and along nonmyelinated C-fibers and at nerve endings in the skin.
149         Also, the rapid adaptation of nodose nerve endings in the trachea observed during a mechanica
150 y distinct subsets of mechanically sensitive nerve endings in the trachea/bronchus.
151 ed this approach to identify spinal afferent nerve endings in the upper GI tract of mice.
152            Our results suggest that the free nerve endings innervating Eimer's organ are largely mech
153 esicles, and rates of hormone secretion from nerve endings into the blood and from dendrites into the
154                                The term free nerve ending is perhaps an insufficient and inaccurate d
155  muscarinic receptors on the parasympathetic nerve endings is likely to contribute to increased acety
156  way in which it excites nociceptive sensory nerve endings is still unclear.
157                                      Sensory nerve endings located close to the lumen of the GI tract
158                                  Significant nerve-ending loss occurs before a decrease in cell bodie
159  a majority of PACAP-LI, VIP-LI and VAChT-LI nerve endings making putative synaptic contact onto IMG
160 ]i regulatory properties of neurohypophysial nerve endings may explain both the depletion of peptide
161 that endogenous CGRP concentrated in sensory nerve endings may regulate locally the immune response,
162 e activity represents a direct effect on the nerve ending, mediated by P2X receptors, whereas the lat
163 endent uptake into presynaptic noradrenergic nerve endings, mediated by the norepinephrine transporte
164 terminating in peripheral zones that contain nerve endings mediating distinct perceptions of innocuou
165 es (PCs) are tactile receptors composed of a nerve ending (neurite) that is encapsulated by layers of
166         The surprising coexistence under one nerve ending of separate clusters of receptors that resp
167 s correlated with a block of Ca ion entry at nerve endings of csp mutants.
168 d demonstrate KCNQ3 expression in peripheral nerve endings of cutaneous D-hair follicles.
169 showed intense CGRP immunoreactivity in pulp nerve endings of mutant mice, compared with a gradual de
170                                              Nerve endings of nociceptors (pain-sensing neurons) expr
171 ll processes to form encapsulated lanceolate nerve endings of rapidly adapting mechanoreceptors.
172                                          The nerve endings of rat neurohypophyses were acutely dissoc
173 IC, in several different specialized sensory nerve endings of skin, suggesting it might participate i
174 ation, morphology, and neurochemistry of the nerve endings of spinal afferents that actually detect t
175                               The individual nerve endings of spinal afferents that innervate the uri
176 ), the morphology and location of peripheral nerve endings of spinal afferents that transduce sensory
177 , these results predict that if one depletes nerve endings of synaptic vesicles, one may see a reduct
178 e shown that the VR1 is expressed on sensory nerve endings of the heart.
179 d also in muscle tissue, probably around the nerve endings of the neuromuscular junction.
180 ct and streptolysin-O permeabilized isolated nerve endings of the rat neurohypophysis were studied.
181  by depolarizing stimuli at single, isolated nerve endings of the rat neurohypophysis.
182 tamine causes long-term toxicity to dopamine nerve endings of the striatum.
183 us system (brain, spinal cord and peripheral nerve endings) of behaving mice.
184 e direct stimulation of intraepithelial free nerve endings or indirectly through information transmis
185 lcitonin gene-related peptide from cutaneous nerve endings plays a key role in the local immune aberr
186 sed vesicular [(3)H]glutamate content in the nerve ending preparation synaptosome; this decrease was
187  interaction with the CIRL receptor on these nerve endings resulted in ionic pore formation, generati
188 TE that, in turn, activates TRPV1 on C-fiber nerve endings resulting in depolarization of nerves and
189 this setting, decreased pH(i) in sympathetic nerve endings sequentially leads to a compensatory activ
190                Secretory responses in intact nerve endings showed AA-induced secretion to be sustaine
191 was dependent on the resting [Ca2+]i and the nerve ending size, and was depletable using repetitive d
192 ushy cells of the AVCN receive huge auditory nerve endings specialized for high fidelity neural trans
193 rofilament 200 labeled a more central set of nerve endings, suggesting that these fibers function as
194 pidermal junction next to peripheral sensory nerve endings, suggesting that viral reactivation may oc
195       We previously demonstrated that nodose nerve endings supplying the trachea are exquisitely mech
196 -HT), and norepinephrine (NE) into rat brain nerve endings (synaptosomes) were evaluated.
197 polarization of guinea pig heart sympathetic nerve endings (synaptosomes) with 1 to 100 mmol/L K+ cau
198 ' UTR, is located both in cell bodies and at nerve endings (synaptosomes).
199 -HT), and norepinephrine (NE) into rat brain nerve endings (synaptosomes).
200 5-HT) and norepinephrine (NE) into rat brain nerve endings (synaptosomes).
201 y affected in the spinal cord and found that nerve endings terminating on alpha-motor neurons are aff
202  blood pressure by stimulating renal sensory nerve endings that contain synapsin-positive microvesicl
203 ctions of alpha-latrotoxin on neuroendocrine nerve endings that emanate from central nervous system n
204 is function, we found BNC1 in the lanceolate nerve endings that lie adjacent to and surround the hair
205 n PGP 9.5 immunoreactive intraepidermal fine nerve endings that were normalized after grate removal.
206 tly induced an increase in [Ca2+]i in intact nerve endings, the AA-induced secretory response was lar
207 nsmission of mechanical forces to nociceptor nerve endings thereby reducing pain.
208 te) to the skin activates underlying sensory nerve endings, thereby producing pain, inflammation and
209 2+)-dependent exocytosis in neurohypophysial nerve endings through receptor interaction and insertion
210 tegies that target TRPA1 channels on sensory nerve endings to achieve chemical deterrence.
211 e relocation of activated Trk receptors from nerve endings to cell bodies is required for nuclear sig
212  in Merkel cells, which drive Abeta-afferent nerve endings to fire slowly adapting impulses.
213 t least in part, by sensitizing the afferent nerve endings to ischaemia.
214 cipal neurons, colocalizing with glycinergic nerve endings to mediate fast, phasic IPSCs.
215 f pain by increasing the response of sensory nerve endings to noxious stimuli.
216 via transport of FIV vectors from peripheral nerve endings to sensory ganglia, as evidenced by HuMOR
217 ile TeNT retrograde traffics from peripheral nerve endings to the interneuronal junction, there is li
218 y causing the release of CGRP from cutaneous nerve endings, triggers mast cell release of TNF-alpha,
219 at carotid body type I cells and the apposed nerve endings use different mechanisms for oxygen sensin
220 ngly, a significantly reduced number of free nerve endings was detected in glabrous skin from SNS-gp1
221                    Mechanosensitivity of the nerve endings was quantified using calibrated von Frey f
222 R) causes CGRP to be released from cutaneous nerve endings, we examined whether CGRP participates in
223                                              Nerve endings were also bidirectionally and capacitively
224         Substance P-like immunoreactive free nerve endings were also present in the luminal syringeal
225              The majority of spinal afferent nerve endings were CGRP-immunoreactive.
226    Most FFA3-immunoreactive nerve fibres and nerve endings were cholinergic, colocalized with protein
227 s at the membrane surface in real samples of nerve endings were estimated.
228                               Less commonly, nerve endings were identified in internodal strands, blo
229                                         Most nerve endings were located in detrusor muscle where the
230 entials arriving from single airway afferent nerve endings were monitored extracellularly using a gla
231 entials arriving from single airway afferent nerve endings were monitored extracellularly using a gla
232                       Synaptophysin-positive nerve endings were observed in close apposition to red p
233 the cell body whereas type II is enriched at nerve endings where it is bound to two prominent A kinas
234 a6 subunits are typically found at aminergic nerve endings where they play important roles in nicotin
235  the outer, substance P-positive set of free nerve endings, whereas several afferents differentially
236 group, we blocked 5-HT3 receptors on sensory nerve endings with tropisetron (300 microg kg(-1), I.V.)
237 estruction of both sphincteric myofibers and nerve endings, with a functional incapacity of the damag
238 ly assumed to be located exclusively on free nerve endings within the nasal epithelium, requiring tha
239 s of these sensory neurons terminate as free nerve endings within the oral epithelium.
240 ion of RTX transiently disrupted nociceptive nerve endings, yielding reversible analgesia.

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