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1 a- or intracellular action at the frog motor nerve ending.
2 nerve, at a bend in the nerve, and at a cut nerve ending.
3 sible target sites for phorbol esters in the nerve ending.
4 to regulate cytoskeletal organization in the nerve ending.
5 s were placed within 10 microns of the motor nerve ending.
6 resides, with DC dendrites crossing several nerve endings.
7 TRPA1, an excitatory ion channel on sensory nerve endings.
8 ing capsaicin-sensitive perivascular sensory nerve endings.
9 decreases in calcium currents at mouse motor nerve endings.
10 n were immunolabeled to quantitate epidermal nerve endings.
11 ing the basement membrane and branching into nerve endings.
12 treatment causes RII protein to increase at nerve endings.
13 mediators and activation of afferent sensory nerve endings.
14 wnstream of calcium entry at amphibian motor nerve endings.
15 s then distributed to peripheral cholinergic nerve endings.
16 sensory nerve distally toward the peripheral nerve endings.
17 orepinephrine into presynaptic noradrenergic nerve endings.
18 d result from a decreased Ca(2+) influx into nerve endings.
19 ing ligand-gated ion channels in presynaptic nerve endings.
20 l as some intraepidermal and free myelinated nerve endings.
21 igh-affinity monoamine uptake into rat brain nerve endings.
22 lting in elevated neurotransmitter levels in nerve endings.
23 -1) or COX-2 produce hyperalgesia in sensory nerve endings.
24 at surrounds the synaptic vesicle cluster in nerve endings.
25 ter axons that appeared to terminate as free nerve endings.
26 stimulating transmitter(s) for chemosensory nerve endings.
27 al properties from their counterparts in the nerve endings.
28 rphology resembles human mechanosensory free nerve endings.
29 dulate their sensitivity directly in sensory nerve endings.
30 itatory nicotinic ACh receptors on the motor nerve endings.
31 ng specific (vanilloid) receptors on sensory nerve endings.
32 naptic opioid receptors in mammalian central nerve endings.
33 epinephrine release assay in rat hippocampal nerve endings.
34 larization-induced entry of Ca2+ into intact nerve endings.
35 both intact and streptolysin-O permeabilized nerve endings.
36 Capsaicin is a specific activator of sensory nerve endings.
37 logical changes starting at their peripheral nerve endings.
38 al stimuli into the depolarization of distal nerve endings.
39 horing protein for PKC epsilon within intact nerve endings.
40 uroendocrine epithelial cells, and bronchial nerve endings.
41 loss or selective degeneration of peripheral nerve endings.
42 , where it replicated and infected the nodal nerve endings.
43 1 cannabinoid receptors localized on sensory nerve endings.
44 ctor pili muscles, Merkel cells, and sensory nerve endings.
45 TH-negative neurons that form epidermal-free nerve endings.
46 transducer in nociceptive and thermosensory nerve endings.
47 esults in synaptic abnormalities in auditory nerve endings.
48 structure-function studies of vagal afferent nerve endings.
49 t all nerves within the CE terminate as free nerve endings.
50 A(1) adenosine receptors at mammalian motor nerve endings.
51 l-epidermal junction, adjacent to peripheral nerve endings.
52 -like immunoreactive, thin and varicose free nerve endings.
53 ecording of coupled glomus cells and carotid nerve endings.
54 ts differentially innervate the central free nerve endings.
56 ported to be released from cutaneous sensory nerve endings after hapten application, we determined wh
57 roglia in striatum and they protect dopamine nerve endings against drug-induced nerve terminal damage
58 solitary chemosensory cells, and epithelial nerve endings, all of which regenerate after amputation.
61 ce of P2 receptor agonists at the peripheral nerve ending and is decreased by the presence of P2 anta
62 ed acetylcholine receptors associated with a nerve ending and were thus considered to form anatomic m
63 apid, transient component observed in 70% of nerve endings and a voltage-activation relationship that
64 onsisting of Merkel cells and Abeta-afferent nerve endings and are localized in fingertips, whisker h
66 ody, regulating the excitability of afferent nerve endings and glomus cells (putative chemoreceptor c
69 ion channels, which are expressed in sensory nerve endings and in skin, respond to distinct thermal t
71 the branching of diabetes-suppressed sensory nerve endings and regeneration in the diabetic corneas.
72 n of neurotrophin receptors (Trks) at distal nerve endings and retrograde propagation of Trk activati
75 escued a significant number of VAChT stained nerve endings and treatment of fish with E(2) alone exhi
76 te in mechanically sensitive ion channels in nerve endings, and hearing in mechanically sensitive ion
77 s increased pulpal NGF, sprouting of sensory nerve endings, and increased immunoreactivity for the se
78 n) stimulates abdominal sympathetic afferent nerve endings, and recently have documented increased co
79 ed into those that target the vagal afferent nerve endings, and those that target neural activity wit
80 lation of mechanoreceptors located in the BR nerve-endings, and modulation of the action potential fr
81 kout mice lacking synapsin I and II, sensory nerve endings are normally developed but not stimulated
84 that Merkel cells rather than Abeta-afferent nerve endings are primary sites of tactile transduction
87 than in slices, suggesting that presynaptic nerve endings are the primary site of inhibition of upta
89 were able to exclude T cells or sympathetic nerve endings as sources of the injury-modulating catech
97 ne inhibits ACh release from mammalian motor nerve endings by reducing Ca(2+) calcium entry through v
99 of the endogenous Ca2+ buffering capacity of nerve endings by treatment with the mitochondrial Ca2+ u
100 annels or, as is the case at amphibian motor nerve endings, by an effect downstream of Ca(2+) entry.
101 cretion whilst simultaneously decreasing the nerve ending calcium currents that promote evoked releas
102 ng that the distribution of mechanosensitive nerve endings can be inferred by visual inspection of th
103 nd indicate that TRPM3 activation in sensory nerve endings can contribute to neurogenic inflammation.
105 local kallikrein-kinin system in adrenergic nerve endings capable of generating enough bradykinin to
106 mine release in isolated hearts, sympathetic nerve endings (cardiac synaptosomes), and PC12 cells bea
107 ls, and resident cells can overtly stimulate nerve endings, cause long-lasting changes in neuronal ex
108 synaptic depolarizing potential (SDP) in the nerve endings caused by release of an excitatory transmi
109 ble channels in the patch) for cell body and nerve ending channels were similar: 11 vs. 14 mV per e-f
110 (H(3)Rs) are present in cardiac sympathetic nerve endings (cSNE) of animals and humans, where they a
111 otor axons in SOD1(G93A) mice, Ia/II sensory nerve endings degenerate in the absence of obvious alter
113 capsaicin, which depletes SP in the sensory nerve endings, eliminates stress-control differences in
114 d during aging in homogenate, cytosol, and a nerve ending-enriched fraction from the hippocampus.
115 staining showed that alpha7 subunit-positive nerve endings enveloped tyrosine hydroxylase-positive gl
116 tivity by using NE released from sympathetic nerve endings evolved early in the history of mammals.
117 llowing the activation of peripheral sensory nerve endings following damage or exposure to inflammato
119 2+) exchange, as measured in intact isolated nerve endings from mouse cortex and in intact varicositi
121 9, would stimulate group III and IV afferent nerve endings from the hindlimb of the anesthetized cat.
122 s cell was stimulated, current spread to the nerve ending (GC/NE coupling) was similar in magnitude (
123 different mechanisms for oxygen sensing: the nerve endings generate action potentials in association
125 al cell bodies in trigeminal ganglia (TG) to nerve ending in the noses and corneas of infected cattle
126 zed by each other; and (3) DRG neurones with nerve endings in a glycolytic muscle developed greater i
127 likely to be reached at cardiac sympathetic nerve endings in advanced congestive heart failure, prom
132 t neurons allowing for bright imaging of the nerve endings in living tissues and suitable for structu
133 nificant alterations at Ia/II proprioceptive nerve endings in muscle spindles before the symptomatic
136 The first state directs virus access to nerve endings in peripheral tissue, whereas the second d
139 Arterial baroreceptors are mechanosensitive nerve endings in the aortic arch and carotid sinus that
140 e ATP is coreleased with NE from sympathetic nerve endings in the heart, we investigated whether ATP
141 pH 6.7 are ASIC(3)-like in DRG neurons with nerve endings in the hindlimb muscles, (2) a greater aci
142 y M2 muscarinic receptors on parasympathetic nerve endings in the lungs decrease release of acetylcho
144 fficiently transported retrogradely from the nerve endings in the nose and eye to cell bodies in the
147 sults from the excitation of primary sensory nerve endings in the skin, but the underlying molecular
153 esicles, and rates of hormone secretion from nerve endings into the blood and from dendrites into the
155 muscarinic receptors on the parasympathetic nerve endings is likely to contribute to increased acety
159 a majority of PACAP-LI, VIP-LI and VAChT-LI nerve endings making putative synaptic contact onto IMG
160 ]i regulatory properties of neurohypophysial nerve endings may explain both the depletion of peptide
161 that endogenous CGRP concentrated in sensory nerve endings may regulate locally the immune response,
162 e activity represents a direct effect on the nerve ending, mediated by P2X receptors, whereas the lat
163 endent uptake into presynaptic noradrenergic nerve endings, mediated by the norepinephrine transporte
164 terminating in peripheral zones that contain nerve endings mediating distinct perceptions of innocuou
165 es (PCs) are tactile receptors composed of a nerve ending (neurite) that is encapsulated by layers of
169 showed intense CGRP immunoreactivity in pulp nerve endings of mutant mice, compared with a gradual de
171 ll processes to form encapsulated lanceolate nerve endings of rapidly adapting mechanoreceptors.
173 IC, in several different specialized sensory nerve endings of skin, suggesting it might participate i
174 ation, morphology, and neurochemistry of the nerve endings of spinal afferents that actually detect t
176 ), the morphology and location of peripheral nerve endings of spinal afferents that transduce sensory
177 , these results predict that if one depletes nerve endings of synaptic vesicles, one may see a reduct
180 ct and streptolysin-O permeabilized isolated nerve endings of the rat neurohypophysis were studied.
184 e direct stimulation of intraepithelial free nerve endings or indirectly through information transmis
185 lcitonin gene-related peptide from cutaneous nerve endings plays a key role in the local immune aberr
186 sed vesicular [(3)H]glutamate content in the nerve ending preparation synaptosome; this decrease was
187 interaction with the CIRL receptor on these nerve endings resulted in ionic pore formation, generati
188 TE that, in turn, activates TRPV1 on C-fiber nerve endings resulting in depolarization of nerves and
189 this setting, decreased pH(i) in sympathetic nerve endings sequentially leads to a compensatory activ
191 was dependent on the resting [Ca2+]i and the nerve ending size, and was depletable using repetitive d
192 ushy cells of the AVCN receive huge auditory nerve endings specialized for high fidelity neural trans
193 rofilament 200 labeled a more central set of nerve endings, suggesting that these fibers function as
194 pidermal junction next to peripheral sensory nerve endings, suggesting that viral reactivation may oc
197 polarization of guinea pig heart sympathetic nerve endings (synaptosomes) with 1 to 100 mmol/L K+ cau
201 y affected in the spinal cord and found that nerve endings terminating on alpha-motor neurons are aff
202 blood pressure by stimulating renal sensory nerve endings that contain synapsin-positive microvesicl
203 ctions of alpha-latrotoxin on neuroendocrine nerve endings that emanate from central nervous system n
204 is function, we found BNC1 in the lanceolate nerve endings that lie adjacent to and surround the hair
205 n PGP 9.5 immunoreactive intraepidermal fine nerve endings that were normalized after grate removal.
206 tly induced an increase in [Ca2+]i in intact nerve endings, the AA-induced secretory response was lar
208 te) to the skin activates underlying sensory nerve endings, thereby producing pain, inflammation and
209 2+)-dependent exocytosis in neurohypophysial nerve endings through receptor interaction and insertion
211 e relocation of activated Trk receptors from nerve endings to cell bodies is required for nuclear sig
216 via transport of FIV vectors from peripheral nerve endings to sensory ganglia, as evidenced by HuMOR
217 ile TeNT retrograde traffics from peripheral nerve endings to the interneuronal junction, there is li
218 y causing the release of CGRP from cutaneous nerve endings, triggers mast cell release of TNF-alpha,
219 at carotid body type I cells and the apposed nerve endings use different mechanisms for oxygen sensin
220 ngly, a significantly reduced number of free nerve endings was detected in glabrous skin from SNS-gp1
222 R) causes CGRP to be released from cutaneous nerve endings, we examined whether CGRP participates in
226 Most FFA3-immunoreactive nerve fibres and nerve endings were cholinergic, colocalized with protein
230 entials arriving from single airway afferent nerve endings were monitored extracellularly using a gla
231 entials arriving from single airway afferent nerve endings were monitored extracellularly using a gla
233 the cell body whereas type II is enriched at nerve endings where it is bound to two prominent A kinas
234 a6 subunits are typically found at aminergic nerve endings where they play important roles in nicotin
235 the outer, substance P-positive set of free nerve endings, whereas several afferents differentially
236 group, we blocked 5-HT3 receptors on sensory nerve endings with tropisetron (300 microg kg(-1), I.V.)
237 estruction of both sphincteric myofibers and nerve endings, with a functional incapacity of the damag
238 ly assumed to be located exclusively on free nerve endings within the nasal epithelium, requiring tha
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