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1 for having higher selectivity for the faster nerve fibers.
2 ion of the primary GC subtypes, even beneath nerve fibers.
3 le, and precise spike generation in auditory nerve fibers.
4 ompare this with functional studies of small nerve fibers.
5 action potentials in cold-sensitive afferent nerve fibers.
6 ormation of limbal blood vessels and corneal nerve fibers.
7 lar to that reported for individual auditory nerve fibers.
8 , trigeminal ganglia (TG) neurons, and their nerve fibers.
9 that relay quality information to gustatory nerve fibers.
10 loss of myelin, the fatty sheath that covers nerve fibers.
11 the neuronal cell bodies of corneal sensory nerve fibers.
12 ns at the nodes of Ranvier of teased sciatic nerve fibers.
13 he postnatal ingrowth of essential autonomic nerve fibers.
14 olecules, such as MAG, present in myelinated nerve fibers.
15 truct the responses of entire populations of nerve fibers.
16 y to reflect pathology in sciatic and tibial nerve fibers.
17 ompare this with functional studies of small nerve fibers.
18 the epineurium and extensive teasing of the nerve fibers; (2) an adaptable drop-plating method for s
19 and in the mean deviation using the GDx-VCC nerve fiber analyzer (Laser Diagnostic Technologies, San
22 fferent features from the firing of auditory nerve fibers and convey that information along separate
23 es can be linked to a particular subgroup of nerve fibers and how these changes are correlated with p
24 e central nervous system (CNS) along sensory nerve fibers and initially entered the simian CNS at lum
25 IVCCM can identify an abnormality in corneal nerve fibers and Langerhans cells in patients with and w
26 process characterized by the infiltration of nerve fibers and macrophages into lesions, plays a pivot
28 recovered function of regenerated peripheral nerve fibers and reinnervated mechanoreceptors may diffe
29 ells detect coincident firing among auditory nerve fibers and transmit signals along monaural pathway
32 coherence tomography (OCT) to assess retinal nerve fibers, and underwent muscle and brain magnetic re
37 se the global effects of aging on myelinated nerve fibers are more complex and profound than those in
38 ed pancreatic islet and exocrine sympathetic nerve fiber area from autopsy samples of patients with t
40 een hair cells and the terminals of cochlear nerve fibers, as seen in confocal analysis of the organ
44 patients with SFN would lose intraepidermal nerve fibers at the distal leg more quickly than at more
45 manipulate an object, populations of tactile nerve fibers become activated and convey information abo
46 ound-shaped DCs were closely associated with nerve fiber branching points, penetrating the basement m
47 ses in a subset of sweet-sensitive gustatory nerve fibers but did not affect other types of fibers, a
48 rane of nociceptive, unmyelinated peripheral nerve fibers, but clarifying the role of sodium channel
49 reshold, small-diameter unmyelinated group C nerve fibers (C-fibers) has limited effects on mechanica
50 est that the pathology of the P2X3 epidermal nerve fibers can be selectively linked to neuropathy, hi
51 Cs trigger peptidergic nociceptive (or pain) nerve fibers, causing an alteration of the respiratory r
52 el of a single mammalian myelinated cochlear nerve fiber coupled to a stimulator-electrode-tissue int
53 e findings indicate that in type 1 diabetes, nerve fiber damage is evident in the subbasal nerve plex
57 epidermal nerve fiber and myelinated dermal nerve fiber densities were quantified in skin biopsies f
58 I, 44% to 92%]; P < .001) and intraepidermal nerve fiber density (4 patients [27%; 95% CI, 8% to 55%]
64 ed to age- and sex-matched reference values; nerve fiber density and branching were significantly dec
65 er, our findings suggest that intraepidermal nerve fiber density and changes in NCV and amplitude mig
66 Secondary outcomes included intraepidermal nerve fiber density and nerve conduction study parameter
67 sis Severity Score (R = -0.354; P = .007 for nerve fiber density and R = -0.283; P = .03 for RNFL thi
68 lity Status Scale (rho = -0.295; P = .03 for nerve fiber density and rho = -0.374; P = .004 for RNFL
72 y nerve action potentials and intraepidermal nerve fiber density had a shorter CNFL (P = .04 and P =
79 ealed that cryolipolysis decreased epidermal nerve fiber density, as well as dermal myelinated nerve
84 acetylcholine transporter (VAchT), and their nerve fiber density, were unchanged after buserelin trea
85 fiber density, as well as dermal myelinated nerve fiber density, which persisted throughout the stud
87 creases in norepinephrine level, sympathetic nerve fiber distribution and beta2-AR expression were ob
88 ic responses) from responses of the auditory nerve fibers (electroneural responses), with separate ex
92 oton microscopy, we observe that sympathetic nerve fibers establish neuro-adipose junctions, directly
94 phrine contents, distribution of sympathetic nerve fibers, expression of beta-adrenergic receptors (b
96 d-rich membrane that ensheaths and insulates nerve fibers, facilitates the rapid conduction of electr
97 Although ATP is necessary for activation of nerve fibers for all taste stimuli, the role of 5-HT is
98 depolarization of acid-sensitive trigeminal nerve fibers, for example, polymodal nociceptors, rather
99 significant reduced thickness in the retinal nerve fiber, ganglion cell, inner plexiform, and outer p
100 s were observed in the inner retinal layers (nerve fiber, ganglion cells, inner plexiform, and inner
101 kin cooling to specifically target cutaneous nerve fibers has the potential to be useful for prolonge
102 e recovery of sensory responses to surviving nerve fibers, homeostatic adjustments in PV-mediated inh
103 ons between cochlear hair cells and auditory nerve fibers; however, there is no clinical test of this
104 showed substantial innervation by trigeminal nerve fibers immunoreactive for calcitonin gene-related
105 of peptidergic and non-peptidergic epidermal nerve fibers in a rat model of nerve injury-induced pain
106 lium innervation by accompanying the sensory nerve fibers in crossing the basement membrane and branc
108 ageal achalasia, and the enteric neurons and nerve fibers in gastrointestinal tract were markedly abn
109 n IL-17c-specific receptor, was expressed on nerve fibers in human skin and sensory neurons in dorsal
116 eration is closely linked to the presence of nerve fibers in the pulp and to the healing mechanism by
119 stics (GDx), Carl Zeiss Meditec, Dublin, CA) nerve fiber indicator (NFI), and Spectralis optical cohe
122 microscopy revealed that peripheral afferent nerve fibers innervating taste buds contain calcitonin g
124 e density of peri-arterial renal sympathetic nerve fibers is lower in distal segments and dorsal loca
125 eption of the primary olfactory and terminal nerve fibers, labeled only for NADPH-d, yielded identica
126 , inner retinal layer (IRL), macular retinal nerve fiber layer (mRNFL), macular ganglion cell layer (
127 On SD OCT, the tumor epicenter was in the nerve fiber layer (n = 47; 100%), with all other retinal
128 ere was significant, progressive loss of the nerve fiber layer (NFL) (0.25 mum/y) and the ganglion ce
129 p the thickness of the peripapillary retinal nerve fiber layer (NFL) and ganglion cell complex (GCC).
130 determine correlations between RPC density, nerve fiber layer (NFL) thickness, and visual field indi
132 to measure optic disc, peripapillary retinal nerve fiber layer (NFL), and macular ganglion cell compl
134 nner inner plexiform layer (IPL) (P = 0.02), nerve fiber layer (P = 0.05), and RPE (P = 0.0001), and
135 ssess the thickness of peripapillary retinal nerve fiber layer (pRNFL) and segmented macular layers,
136 posterior pole and the peripapillary retinal nerve fiber layer (pRNFL) protocols of the Spectralis OC
137 h no significant difference in peripapillary nerve fiber layer (pRNFL) thickness and optic nerve head
138 e with INL cysts had thinner average retinal nerve fiber layer (RNFL) (78.2 +/- 1.8 mum vs 52.0 +/- 4
139 mm Hg (P < .00001) and mean average retinal nerve fiber layer (RNFL) 71.0 +/- 30 mum vs 62.8 +/- 24
141 compare the rates of circumpapillary retinal nerve fiber layer (RNFL) and macular retinal ganglion ce
143 icrocirculation of the peripapillary retinal nerve fiber layer (RNFL) between the hemispheres in eyes
146 etinal ganglion cells (RGC) loss and retinal nerve fiber layer (RNFL) injury: this results in functio
147 e been proposed as an alternative to retinal nerve fiber layer (RNFL) parameters to diagnose glaucoma
149 concentration and the peripapillary retinal nerve fiber layer (RNFL) thickness at presentation (r =
151 herence tomography (OCT) measures of retinal nerve fiber layer (RNFL) thickness have been proposed as
152 cell (DC) density, and peripapillary retinal nerve fiber layer (RNFL) thickness in patients with MS.
153 pregnancy and low birth weight with retinal nerve fiber layer (RNFL) thickness in preadolescent chil
154 h previous treated ROP had decreased retinal nerve fiber layer (RNFL) thickness in the superior and n
155 change of the average peripapillary retinal nerve fiber layer (RNFL) thickness measurements obtained
156 annulus 100 microm in width, and the retinal nerve fiber layer (RNFL) thickness profiles were plotted
158 evaluated on visual acuity (VA) and retinal nerve fiber layer (RNFL) thickness using time-domain (TD
164 the optic nerve and analysis of the retinal nerve fiber layer (RNFL) thickness was performed on each
165 nce tomography (OCT) measurements of retinal nerve fiber layer (RNFL) thickness when using automated
166 T parameters, SD-OCT circumpapillary retinal nerve fiber layer (RNFL) thickness, and optic nerve head
167 with the ONH rim area, peripapillary retinal nerve fiber layer (RNFL) thickness, and the macular gang
168 Field (HVF) mean deviation (MD), and retinal nerve fiber layer (RNFL) thickness, as measured by Cirru
169 defects were compared with regard to retinal nerve fiber layer (RNFL) thickness, drusen morphology, s
170 es, including inferior peripapillary retinal nerve fiber layer (RNFL) thickness, inferior ganglion ce
171 The optic nerve head (ONH) shape, retinal nerve fiber layer (RNFL) thickness, ONH volume, and papi
172 y analysis (PPAA), the peripapillary retinal nerve fiber layer (RNFL) thickness, the ganglion cell la
173 disc margin (DM)-based assessment or retinal nerve fiber layer (RNFL) thickness, yielded higher diagn
175 (OSA) syndrome in the peripapillary retinal nerve fiber layer (RNFL) thicknesses remains unclear.
176 ell (RGC) loss and only females have retinal nerve fiber layer (RNFL) thinning, despite mice of both
177 of peripapillary 3-dimensional (3D) retinal nerve fiber layer (RNFL) volume measurements from spectr
179 al coherence tomography (OCT) of the retinal nerve fiber layer (RNFL), and monocular pattern reversal
180 al coherence tomography (OCT) of the retinal nerve fiber layer (RNFL), and volumetric OCT scans throu
181 obtain objective measurements of the retinal nerve fiber layer (RNFL), optic nerve head, and macula f
182 to investigate the thickness of the retinal nerve fiber layer (RNFL), the ganglion cell layer (GCL),
184 visual pathway damage (peripapillary retinal nerve fiber layer [RNFL] thickness and macular volume) a
185 l bodies and axons, the unmyelinated retinal nerve fiber layer and the myelinated post-laminar axons,
186 between eyes, presence of localized retinal nerve fiber layer and/or neuroretinal rim defects, and d
187 e structural thinning in the macular retinal nerve fiber layer correlates with pulmonary function tes
188 disc ratio and cup shape and missing retinal nerve fiber layer defects and disc hemorrhage were the k
192 Retinal astrocytic hamartomas arose in the nerve fiber layer in every case and demonstrated moth-ea
193 , choroidal neovascular membrane (n = 1), or nerve fiber layer infarction (n = 1) for a mean interval
195 have shown that the circumpapillary retinal nerve fiber layer is an important parameter for glaucoma
196 = 0.65), cup shape (kappaw = 0.65), retinal nerve fiber layer loss (kappaw = 0.69), vertical cup-to-
197 p-disc ratio and failure to identify retinal nerve fiber layer loss, disc hemorrhage, or rim loss wer
198 as associated with overestimation of retinal nerve fiber layer loss, rim loss, vertical cup-disc rati
199 xhibited selective reductions of the retinal nerve fiber layer that correlate with electrophysiologic
200 kness (GC-IPLT), and circumpapillary retinal nerve fiber layer thickness (cpRNFLT) were determined.
201 ), and reduced superior and inferior retinal nerve fiber layer thickness (P = 0.01, respectively) wer
202 are needed to compare peripapillary retinal nerve fiber layer thickness (pRNFLT) measurements taken
203 ial pressure correlated with maximal retinal nerve fiber layer thickness (r = 0.60, P </= .001), maxi
206 racterized BMO-MRW and peripapillary retinal nerve fiber layer thickness (RNFLT) in a normal populati
207 , BMO area (BMOA), and peripapillary retinal nerve fiber layer thickness (RNFLT) were measured with o
208 kness, macular volume, peripapillary retinal nerve fiber layer thickness and choroidal thickness usin
209 ated eyes showed an average temporal retinal nerve fiber layer thickness of 54 mum before injection a
212 g, Disc Damage Likelihood Scale, and retinal nerve fiber layer thickness) were generally not informat
214 al parameters, minimum rim width and retinal nerve fiber layer thickness, in addition to peripapillar
216 me patients despite continued marked retinal nerve fiber layer thinning indistinguishable from that i
217 including severity of IOP elevation, retinal nerve fiber layer thinning, or electrodiagnostic finding
220 ickness and the optic nerve head and retinal nerve fiber layer, and visual field in a dark room with
221 cant thinning of the ganglion cell layer and nerve fiber layer, as well as a thickening of the INL an
222 on automated perimetry, and loss of retinal nerve fiber layer, even among those with good visual acu
223 Positive staining was present within the nerve fiber layer, inner plexiform layer, and inner and
224 afe and does not damage the temporal retinal nerve fiber layer, opening the door next for testing of
225 fractal dimension parameters of the retinal nerve fiber layer, photoreceptor outer segments and reti
228 ts who underwent PRP had diffusely thickened nerve fiber layers (P = 0.024) and diffusely thinned ret
230 ents exhibited significantly reduced corneal nerve fiber length (CNFL-MNF), fiber density (CNFD-MNF),
231 hes/mm2; 95% CI, -28.77 to -7.10; P = .001), nerve fiber length (mean [SE] difference, -3.03 [0.89] m
232 s detected in the cornea of 51% of patients: nerve fiber length was significantly decreased in 44% of
233 Corneal nerve branch density and corneal nerve fiber length were reduced in patients with HIV, bu
234 nsity, corneal nerve branch density, corneal nerve fiber length, corneal nerve fiber tortuosity, and
235 l nerve fiber density, nerve branch density, nerve fiber length, DC density, peripapillary RNFL thick
236 rmal nerve fiber density and total epidermal nerve fiber length/mm(2) were significantly and consiste
237 2% CI, -50.62 to -3.13; P = .01; and corneal nerve fiber length: 28.4 mm/mm2 for the controls vs 21.9
239 rmine whether SIV infection leads to corneal nerve fiber loss, we immunostained corneas for the nerve
242 n of activation in one population of tactile nerve fibers, namely slowly adapting type 1 (SA1) affere
243 associated with neural structures, including nerve fibers, nerve bundles, and muscle spindles, which
244 tors affect the phenotypes of colonic mucosa nerve fibers, neuron differentiation, and fiber outgrowt
245 tudy was to quantify the morphology of small nerve fibers of the cornea of patients with fibromyalgia
247 the prion protein, PrP(Sc), was observed in nerve fibers of the tongue approximately 2 weeks prior t
252 ow that intact motor, sensory, and autonomic nerve fibers/paths are distinctly labeled following a si
253 sing 5-HT3A GFP mice, that 5-HT3 -expressing nerve fibers preferentially contact and receive synaptic
255 nstrates anterograde spread of prions within nerve fibers prior to infection of peripheral synapses (
257 cal responses is driven by one population of nerve fibers (rapidly adapting) whereas the timing of co
258 nodal and axonal injury of intact myelinated nerve fibers, recapitulating pathologic features of huma
261 lothionein II was shown to enhance epidermal nerve fiber regeneration so that it was complete within
262 V-2 reactivation exhibit a higher density of nerve fibers relative to biopsies during virological and
263 chronic variable stress in mice, sympathetic nerve fibers released surplus noradrenaline, which signa
264 comparison between SFOAE delays and auditory nerve fiber responses for the barn owl strengthens the n
265 ener kernel) analyses of chinchilla auditory nerve fiber responses to Gaussian noise to reveal pronou
266 e system-identification analyses of auditory nerve fiber responses to Gaussian noise to uncover prono
268 to the healing mechanism by sprouting of the nerve fiber's terminal branches beneath the carious inju
269 ad no effect on measures of large myelinated nerve fibers, specifically sural or sciatic nerve conduc
272 elevance: Unlike other diseases of the small nerve fibers that cause acral pain syndromes, erythromel
274 of sensory, sympathetic, and parasympathetic nerve fibers that contain classical transmitters plus an
275 he hormone leptin is mediated by sympathetic nerve fibers that directly "envelope" white adipocytes.
277 hologically decreased densities of the small nerve fibers that innervate the epidermis, one hypothesi
278 I taste cells and 5-HT3 -expressing afferent nerve fibers that project to a restricted portion of the
279 re examined for the presence and location of nerve fibers that reacted with a labeled antibody agains
281 fferential susceptibility of small and large nerve fibers to specific metabolic impairments associate
282 for the rectum, nearly half of all extrinsic nerve fibers to the distal colon lack the key immunohist
284 density, corneal nerve fiber length, corneal nerve fiber tortuosity, and corneal Langerhans cell dens
286 ased from type III cells activates gustatory nerve fibers via 5-HT3 receptors, accounting for a signi
287 ir primary excitatory input through auditory nerve fibers via large, axosomatic synaptic terminals ca
288 odulate serotonin-sensitive primary afferent nerve fibers via synaptic connections, enabling them to
290 P2X3 receptor, a marker for non-peptidergic nerve fibers, was not only significantly reduced but cou
291 ne-related peptide, a marker for peptidergic nerve fibers, was not significantly changed on the ipsil
292 not synaptically connected with the afferent nerve fibers, we first analyzed tracer production and tr
293 e mainly present in vulvar segments and most nerve fibers were found in the lamina propria of the cer
295 lamine biosynthetic enzymes, and sympathetic nerve fibers were measured in EAT and subcutaneous adipo
298 abeling both peptidergic and non-peptidergic nerve fibers with the pan-neuronal marker PGP9.5, the ex
300 a selective loss of high-threshold auditory nerve fibers without affecting absolute sensitivity perm
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