ライフサイエンスコーパス: nerve fibre

1 eurons arises from myelinated or umyelinated nerve fibres.

2 signals are conveyed onto auditory afferent nerve fibres.

3 d was observed mainly along GBSM bundles and nerve fibres.

4 arge population of nociceptive-like afferent nerve fibres.

5 re closely apposed to substance P-containing nerve fibres.

6 nduce neurotransmitter release onto auditory nerve fibres.

7 ctivates not only efferent but also afferent nerve fibres.

8 eneration and transmission in CNS myelinated nerve fibres.

9 nd the sound-evoked activity of the auditory nerve fibres.

10 rae of Schwann cells related to unmyelinated nerve fibres.

11 al activation of nociceptive primary sensory nerve fibres.

12 due to earlier remyelination of demyelinated nerve fibres.

13 release of transmitter from post-ganglionic nerve fibres.

14 d show that there are no discrete inhibitory nerve fibres.

15 ared not to be associated with noradrenaline nerve fibres.

16 e-sensitive auditory neuropathy) or auditory nerve fibres.

17 of both slowly and rapidly adapting afferent nerve fibres.

18 tect the effects of ischaemia on sympathetic nerve fibres.

19 inated C-fibres and thinly myelinated Adelta nerve fibres.

20 ion of ASIC1 channels on capsaicin-sensitive nerve fibres.

21 sm for physiological tuning of thermosensory nerve fibres.

22 n excites cutaneous group III and IV sensory nerve fibres.

23 ted ischaemic from non-ischaemic sympathetic nerve fibres.

24 iated with at least one substance P-positive nerve fibre, 32% were associated with at least two, 2% w

25 s nerve biopsies revealed loss of myelinated nerve fibres (86%), increased regenerative clusters (50%

26 s have been documented in lung vagal sensory nerves fibres, a rigorous comparison of their expression

27 ents, indicating that they were dependent on nerve fibre activation.

28 The first recordings of vagal afferent nerve fibre activity were performed by Paintal in the ea

29 MPTP lesion in mice, and of lesioned sciatic nerve fibres after nerve crush in rats.

30 t two, 2% were associated with more than two nerve fibres and 1% with none.

31 irt and P2X3 receptor co-localize in bladder nerve fibres and heterologous Pirt expression significan

32 Most FFA3-immunoreactive nerve fibres and nerve endings were cholinergic, colocal

33 potential amplitude, loss of intraepidermal nerve fibres and significant degeneration of myelinated

34 nglioside and Gal(beta1-3)GalNAc moieties in nerve fibres and their relationship to voltage-gated sod

35 by the activation of small diameter afferent nerve fibres and therapeutic effects on the associated v

36 egeneration, remyelination of severed spinal nerve fibres, and functional recovery.

37 oked part of the physiological repertoire of nerve fibres, and here they are interpreted in terms of

38 patially associated with HSCs and adrenergic nerve fibres, and highly express HSC maintenance genes.

39 width dependence similar to that of auditory nerve fibres, and yield significantly greater coding eff

40 ightly myelinated and unmyelinated autonomic nerve fibres are selectively targeted.

41 It is concluded that these sympathetic nerve fibres are sensitive to ischaemia, and that VRCs p

42 n studies and skin biopsy for intraepidermal nerve fibre assessment.

43 toreceptor distributions(3,4) and individual nerve fibre bundles(5) in the living human retina.

44 d not only by widespread loss of myelin from nerve fibres, but also by widespread inflammation in the

45 ise to V-shaped tuning functions in auditory nerve fibres, but by the level of the inferior colliculu

46 coincident activation of groups of auditory nerve fibres by broadband transient sounds, compensating

47 Activation of afferent vagus nerve fibres by endotoxin or cytokines stimulates hypoth

48 tivation of cardiac parasympathetic efferent nerve fibres by stimulation of the cervical vagus is ass

49 Sensory nerve fibres can detect changes in temperature over a re

50 y by direct electrical stimulation of tibial nerve fibres, confirming that centrally mediated mechani

51 citability of the shortest latency (fast, F) nerve fibres, consistent with hyperpolarization.

52 Although multiple causes of small nerve fibre degeneration have been reported, including v

53 back were immunostained and imaged to assess nerve fibre density and neuropeptide content.

54 vealed in six of them reduced intraepidermal nerve fibre density consistent with small fibre neuropat

55 h a significant reduction in intra-epidermal nerve fibre density in plantar hindpaw skin, and produce

56 Evaluation of intraepidermal nerve fibre density showed a striking loss in patients (

57 was assessed by the return of intraepidermal nerve fibre density through regenerative regrowth.

58 Epidermal nerve fibre density was found to be profoundly reduced i

59 The intraepidermal nerve fibre density was markedly reduced with 90.5% of p

60 Nerve fibre density was not altered in the circular musc

61 Intra-epidermal nerve fibre density was significantly reduced compared t

62 d in rectal sensory fibres, and to correlate nerve fibre density with sensory abnormalities.

63 C and there is a reduction in intraepidermal nerve fibre density, comparable to that seen in herpes z

64 lead to a clear reduction in intraepidermal nerve fibre density, which was independent of electrodia

65 wed a significant decrease in intraepidermal nerve fibre density.

66 om the trunk neural tube and associated with nerve fibres, differentiate into neurons within the gut

67 Duodenal vagal afferent nerve fibre discharge was significantly increased by clo

68 A rich supply of nerve fibres displaying immunoreactivity against nitric

69 ecordings were made of afferent discharge in nerve fibres dissected from the vagus nerve, which respo

70 y drive spontaneous discharge in nociceptive nerve fibres during inflammation.

71 s kindred, and suggest that small peripheral nerve fibre dysfunction due to this mutation may have co

72 on model and to define the rate of epidermal nerve fibre (ENF) regeneration first in healthy control

73 Both types of nerve fibres expressed substance P, but only non-vessel-

74 from increased numbers of polymodal sensory nerve fibres expressing TRPV1.

75 cultured sensory neurons and intact sensory nerve fibres from TRPM8-deficient mice exhibit profoundl

76 to accelerate and guide the formation of new nerve-fibre growth.

77 w, brain and spinal cord injuries that sever nerve fibres have resulted in a degree of incurable func

78 equency response of the innervating auditory nerve fibres However, the data supporting these concepts

79 slips of a cervical vagus were dissected and nerve fibres identified that displayed discharge pattern

80 In rectal hypersensitivity, nerve fibres immunoreactive to TRPV1 were increased in m

81 ofilament staining confirmed the presence of nerve fibres in both regions.

82 etect membrane depolarization of sympathetic nerve fibres in human patients when autonomic neuropathy

83 We identified nerve fibres in the outer third of the annulus fibrosus

84 -induced sweating, and prevented the loss of nerve fibres in the skin and reduction of neuropeptide c

85 s in affected skin correlated with decreased nerve fibres in the subepidermis, e.g. axon-reflex flux

86 onal membrane potential of human sympathetic nerve fibres in vivo.

87 hanisms of action include desensitization of nerve fibres (in the case of capsaicin) and postsynaptic

88 uation of non-myelinated, but not myelinated nerve fibres, in sensory neuropathies.

89 osites along excitatory and inhibitory motor nerve fibres increased and decreased respectively, leadi

90 application can produce a uniform epidermal nerve fibre injury that is safe and well tolerated, and

91 lpha-synuclein aggregations are also seen in nerve fibres innervating the gastro-intestinal tract.

92 Immunohistochemical studies localize P2X3 to nerve fibres innervating the urinary bladder of wild-typ

93 Single nerve fibres innervating tooth pulp were isolated from f

94 cycles can distinguish whether a sympathetic nerve fibre is depolarized or not.

95 e assessed the role of peripapillary retinal nerve fibre layer (pRNFL) thickness and macular volume i

96 study to evaluate the correlation of retinal nerve fibre layer (RNFL) and macular thickness with seru

97 We analysed retinal nerve fibre layer (RNFL) defects in eyes with normal cir

98 The retinal nerve fibre layer (RNFL) is composed largely of unmyelin

99 reoscopic optic nerve head (ONH) and retinal nerve fibre layer (RNFL) photography and imaging with Sc

100 with control eyes, the peripapillary retinal nerve fibre layer (RNFL) showed thinning in MSON eyes (m

101 Retinal nerve fibre layer (RNFL) thickness has been associated w

102 The primary outcome was retinal nerve fibre layer (RNFL) thickness in the affected eye a

103 Retinal nerve fibre layer (RNFL) thickness is related to the axo

104 o corresponding localised regions of retinal nerve fibre layer (RNFL) thickness measured by optical c

105 is (MS) by capturing thinning of the retinal nerve fibre layer (RNFL).

106 The relationship between nerve fibre layer anatomy and the pattern visual evoked

107 Changes were seen not only in the retinal nerve fibre layer and ganglion cell layer, but also in t

108 ociated with reduced apoptosis and increased nerve fibre layer and inner plexiform layer thicknesses.

109 ce of retrograde degeneration of the retinal nerve fibre layer and to ascertain if such patients may

110 Although these confirm the damage to retinal nerve fibre layer beyond what is detected by standard vi

111 f the thickness of the peripapillary retinal nerve fibre layer by optical coherence tomography has be

112 to define the temporal evolution of retinal nerve fibre layer changes and to estimate sample sizes f

113 The evolution of retinal nerve fibre layer changes in the affected eye fitted wel

114 ned the visual field scotoma and the retinal nerve fibre layer defect in the corresponding area.

115 ifying optic nerve and peripapillary retinal nerve fibre layer defects, with different efficacy and l

116 onfirm that there is thinning of the retinal nerve fibre layer following both congenital and acquired

117 rst time progressive thinning of the retinal nerve fibre layer following occipital lobe/optic radiati

118 Thinning of the peripapillary retinal nerve fibre layer has been detected in patients with opt

119 n optic neuritis, and in imaging the retinal nerve fibre layer in both optic neuritis and multiple sc

120 ptical coherence tomography-measured retinal nerve fibre layer loss after 6 months is a suitable outc

121 cept trials of neuroprotection using retinal nerve fibre layer loss as the outcome measure.

122 ymptom onset and the rate of ongoing retinal nerve fibre layer loss decreasing thereafter.

123 visual dysfunction beyond that explained by nerve fibre layer loss.

124 ciated with the extent of concurrent retinal nerve fibre layer loss.

125 affected eyes, whereas peripapillary retinal nerve fibre layer oedema was observed in affected eyes (

126 s not related to the extent of acute retinal nerve fibre layer swelling but was significantly associa

127 mpaired axonal transport (implied by retinal nerve fibre layer swelling) are associated with visual d

128 primary outcome is the peripapillary retinal nerve fibre layer thickness (RNT) in microns.

129 ssion r = 0.54, P < 0.001) was found between nerve fibre layer thickness and elapsed time since injur

130 line relationship was found between time and nerve fibre layer thickness in micrometres over a period

131 The retinal nerve fibre layer thickness may be used as a biological

132 g lesions of the occipital lobe, the retinal nerve fibre layer thickness measured by optical coherenc

133 fected and affected eyes rather than retinal nerve fibre layer thickness of the affected eye alone.

134 linically unaffected fellow eye, the retinal nerve fibre layer thickness of the affected eye was sign

135 At presentation, increased retinal nerve fibre layer thickness was associated with impaired

136 Colour-contrast sensitivity and retinal nerve fibre layer thickness were measured in subgroups.

137 Peripapillary retinal nerve fibre layer thickness, measured by optical coheren

138 sion, visual fields, macular volume, retinal nerve fibre layer thickness, or optic nerve magnetisatio

139 Retinal nerve fibre layer thinning is usually evident within 3 m

140 f the tumour showed proliferation in retinal nerve fibre layer with normal structure of underlying re

141 ds on examination of the optic disc, retinal nerve fibre layer, and visual field.

142 entation, yielded thicknesses of the retinal nerve fibre layer, the ganglion cell layer plus inner pl

143 e of optic neuritis, thinning of the retinal nerve fibre layer, which indicates axonal loss, is obser

144 assessing the optic nerve and peripapillary nerve fibre layer.

145 y of 0.17 versus -0.1) and a thinner retinal nerve fibre layer.

146 imarily focused on evaluation of the retinal nerve fibre layer.

147 tection algorithms to judge axon loss in the nerve fibre layer.

148 The local nerve-fibre layer determined patch size, and quantal mye

149 ysed the position, area and thickness of the nerve-fibre layer in 60 patches of retinal myelin in 47

150 Nerve-fibre layer thickness was obtained from an atlas o

151 edominantly macular thinning (normal retinal nerve fibre-layer thickness with average macular thickne

152 Retinal nerve fibre loss in congenital homonymous hemianopia in

153 This study was performed to assess cutaneous nerve fibre loss in conjunction with temperature and swe

154 ade trans-synaptic degeneration of the optic nerve fibres occurs.

155 noreactivity is greatly increased in colonic nerve fibres of patients with active inflammatory bowel

156 in most neurons, including primary afferent nerve fibres of the pain pathway.

157 lized facial structure that is innervated by nerve fibres of the somatosensory system.

158 VRCs of human sympathetic nerve fibres of the superficial peroneal nerve innervati

159 or 2 times the threshold for most excitable nerve fibres) of the superficial radial (SR) and ulnar (

160 n target innervation and the relationship of nerve fibre pathology to sensory symptoms and signs.

161 ion is an endogenous process in the afferent nerve fibres, perhaps linked to random channel activity

162 Expression of nerve fibres positive for GDNF (p=0.001) and tyrosine ki

163 the acid-sensing ion channel (ASIC) family; nerve fibre recordings have shown ASIC2 and ASIC3 null m

164 We find that the activity of fin ray nerve fibres reflects the amplitude and velocity of fin

165 e effect of different factors on the rate of nerve fibre regeneration and investigated whether such a

166 on and the lack of target specificity during nerve fibre regrowth interfere with a good functional re

167 magnitude, whereas the firing rate of optic nerve fibres spans less than two.

168 n seen in ontogeny and preceded regenerating nerve fibres, suggesting that enhancement of blood vesse

169 activates CCK A receptors on vagal afferent nerve fibre terminals, which in turn initiate a vago-vag

170 Our finding of isolated nerve fibres that express substance P deep within diseas

171 infrared signals are detected by trigeminal nerve fibres that innervate specialized pit organs on th

172 A1 channels as infrared receptors on sensory nerve fibres that innervate the pit organ.

173 ls in regulating the excitability of sensory nerve fibres that mediate mechanical pain.

174 n the regions of the auditory and vestibular nerve fibres, the neural and abneural limbs adjacent to

175 dators by activating TRP channels on sensory nerve fibres to elicit pain and inflammation.

176 bcortical strokes is due to the inability of nerve fibres to regenerate.

177 l nerve, to determine whether differences in nerve fibre type or location affect the level of abnorma

178 The average density of epidermal nerve fibres was greatly diminished in the calf and back

179 e labelling of both neuronal cell bodies and nerve fibres was observed in the paraventricular nucleus

180 Single pulpal nerve fibres were electrically stimulated at just above

181 Nerve fibres were electrically stimulated using single o

182 molecular architecture of dermal myelinated nerve fibres were examined using immunohistochemistry an

183 Some scattered positive neurons and nerve fibres were found in many hypothalamic nuclei incl

184 Sympathetic nerve fibres were identified by studying their response

185 t P2X2 immunoreactivity-positive neurons and nerve fibres were localized in many hypothalamic nuclei.

186 cell bodies were strongly positive, but few nerve fibres were positive.

187 nilloid receptor 1 (VR1)-containing afferent nerve fibres were present on the epicardial surface of t

188 he samples from back-pain patients, isolated nerve fibres were seen in the discal matrix.

189 mage.In seven animals, 10 single intradental nerve fibres were selected that responded to hydrostatic

190 The time constants of motor and sensory nerve fibres were studied in normal human ulnar nerves b

191 ased spontaneous activity of single auditory nerve fibres, while concanavalin A had no effect, sugges

192 secreted from axons and reach glial cells in nerve-fibre (white-matter) tracts?

193 estigation showed focal swellings of retinal nerve fibres with neurofilament disruption.

194 on cells and central and peripheral auditory nerve fibres within the cochlea.

195 rostimulation (INMS) we stimulated groups of nerve fibres, within individual fascicles proximal to th