ライフサイエンスコーパス: nerve growth

1 roduced compensatory increases in peripheral nerve growth.

2 oles for corneal KSPGs in regulating corneal nerve growth.

3 uggested enrichment in gene sets involved in nerve growth and general cell development.

4 of Chn1KI/KI mice revealed stalled abducens nerve growth and selective trochlear and first cervical

5 l role in blood vessel formation and affects nerve growth and survival.

6 healing [1], development [2], cell division, nerve growth, and angiogenesis [3].

7 Effects of polySia removal on trigeminal nerve growth behavior were determined in vivo, using exo

8 y proteins that may influence keratocyte and nerve growth cone behavior in the cornea.

9 Effects of purified GAGs on trigeminal nerve growth cone behavior were tested using in vitro ne

10 Nerve growth cone guidance by diffusible attractive and

11 ins that may regulate keratocytes or corneal nerve growth cone immigration interact with corneal GAGs

12 The nerve growth cone is bi-directionally attracted and repe

13 expressions accompany corneal transition to nerve growth cone permissiveness.

14 Nerve growth cones (GCs) are chemical sensors that conve

15 Sensory trigeminal nerve growth cones innervate the cornea in a highly coor

16 t GAGs may direct the movement of trigeminal nerve growth cones innervating the cornea.

17 During cornea development, chick trigeminal nerve growth cones reach the cornea margin at embryonic

18 nd-binding and lattice-binding activities in nerve growth cones, and reveal that the two MT-binding a

19 le cells - fan-like keratocytes, hand-shaped nerve growth cones, polygonal fibroblasts, to name but a

20 d in regulation of neurotransmitter systems, nerve growth/death and gene expression, and subsequently

21 e growth factor-binding protein 2 (IGFBP-2), nerve growth factor (b-NGF), platelet-derived growth fac

22 day, driven by circadian clock control of a nerve growth factor (BDNF) in the inner ear.

23 orcine, bovine, and murine sequences of beta nerve growth factor (beta-NGF).

24 solate low-affinity antibodies to human beta nerve growth factor (hbetaNGF).

25 Human nerve growth factor (hNGF) is an important pharmaceutica

26 and sympathetic neurons is promoted whether nerve growth factor (NGF) activates TrkA receptors on th

27 rch for neuroactive compounds that mimic the nerve growth factor (NGF) activity for the protection ag

28 the preservation of sufficient expression of nerve growth factor (NGF) and activation of the neurotro

29 With expression of nerve growth factor (NGF) and basic fibroblast growth fa

30 ta), tumor necrosis factor alpha (TNFalpha), nerve growth factor (NGF) and brain derived neurotrophic

31 Neurotrophins such as nerve growth factor (NGF) and brain-derived neurotrophic

32 rait loci associated with gene expression of nerve growth factor (NGF) and calneuron 1 (CALN1) genes.

33 The nerve growth factor (NGF) and glial cell line-derived ne

34 The effect of nerve growth factor (NGF) and its receptor (NGFR) in inf

35 Nerve growth factor (NGF) and neurotrophin-3 (NT-3) are

36 ends, can be modulated by luminar release of nerve growth factor (NGF) and neurotrophin-3 (NT-3), res

37 ated by target-derived neurotrophins such as nerve growth factor (NGF) and neurotrophin-3 (NT-3).

38 Nerve growth factor (NGF) and neurotrophin-3 serve as at

39 h cone collapse and loss of actin filaments, nerve growth factor (NGF) and neurotrophin-3 still induc

40 SorCS3 binds the nerve growth factor (NGF) and platelet-derived growth fa

41 Nerve growth factor (NGF) antagonism is on the verge of

42 brain-derived neurotrophic factor (BDNF) and nerve growth factor (NGF) are crucial modulators in the

43 Levels of nerve growth factor (NGF) are elevated in inflamed tissu

44 neurotrophic and neuroprotective actions of nerve growth factor (NGF) are mediated by hypusinated eI

45 In this paper, we identify nerve growth factor (NGF) as a binding partner for MOG a

46 satetraenoic acid (12[S] or 15[S]-HETE), and nerve growth factor (NGF) as positive control.

47 in animals and humans show that blockade of nerve growth factor (NGF) attenuates both malignant and

48 tal apoptotic pathway that is activated when nerve growth factor (NGF) becomes limiting for neuronal

49 TrkA activation by nerve growth factor (NGF) binding the second extracellul

50 esis of SCG neurons at a level comparable to nerve growth factor (NGF) but also doubled the neurite l

51 PC12 pheochromocytoma cell line responds to nerve growth factor (NGF) by exiting from the cell cycle

52 ated the output activity of Ngf encoding the nerve growth factor (NGF) by increasing histone H4 acety

53 Elevating levels of nerve growth factor (NGF) can have pronounced effects on

54 Depriving mouse sympathetic neurons of nerve growth factor (NGF) causes their apoptotic death.

55 Nerve growth factor (NGF) contributes to the development

56 Although nerve growth factor (NGF) controls survival, maturation

57 Sertad1 is also induced in neurons by nerve growth factor (NGF) deprivation and Abeta (beta-am

58 d contributes to neuron death in response to nerve growth factor (NGF) deprivation.

59 the painful arthritic knee joint and whether nerve growth factor (NGF) drives this pathologic reorgan

60 ent a detailed motion analysis of retrograde nerve growth factor (NGF) endosomes in axons to show tha

61 Nerve growth factor (NGF) exerts protective effects on t

62 We show that PC12 cells stimulated with nerve growth factor (NGF) exhibit statistically signific

63 elta), two transcription factors involved in nerve growth factor (NGF) expression and downregulated s

64 timulation of the gastric epithelium induced nerve growth factor (NGF) expression, and in turn NGF ov

65 linergic neurons in both regions depend upon nerve growth factor (NGF) for their survival and maturat

66 ciated haplotype blocks were intronic to the Nerve Growth Factor (NGF) gene (P=0.001, 0.001, 0.002),

67 In 2001, we initiated a clinical trial of nerve growth factor (NGF) gene therapy in AD, the first

68 Nerve growth factor (NGF) has an important role in regul

69 ) at the cell surface, and administration of nerve growth factor (NGF) has been considered and attemp

70 The neurotrophin nerve growth factor (NGF) has been implicated as a key m

71 ng mode of Spz to Toll is similar to that of nerve growth factor (NGF) in complex with the p75 neurot

72 +) BMCs expressed neurotrophins and secreted nerve growth factor (NGF) in conditioned medium.

73 A role for nerve growth factor (NGF) in contributing to increased v

74 taRII-deficient pups showed a lower level of nerve growth factor (NGF) in its mRNA; however, higher l

75 n channel subfamily M, member 8 (TRPM8); and nerve growth factor (NGF) in nasal biopsy specimens.

76 s and becomes functional by the neurotrophin nerve growth factor (NGF) in native brainstem neurons.

77 on (DRG) neurons by increasing expression of nerve growth factor (NGF) in the colon wall.

78 se it is supported by decades of research on nerve growth factor (NGF) in the peripheral nervous syst

79 The endogenous nerve growth factor (NGF) in the urinary bladder regulat

80 Previously, we demonstrated that nerve growth factor (NGF) increased the excitability of

81 e basolateral NHE1 Na(+)/H(+) exchanger with nerve growth factor (NGF) induces actin cytoskeleton rem

82 Nerve growth factor (NGF) induces collateral branching a

83 Nerve growth factor (NGF) influences the key pathologica

84 Nerve growth factor (NGF) influences the survival and di

85 The microinjection of nerve growth factor (NGF) into the cat pontine tegmentum

86 Nerve growth factor (NGF) is a neuropeptide essential fo

87 Nerve growth factor (NGF) is a neurotrophin that is impl

88 Nerve growth factor (NGF) is a potent survival and axon

89 Nerve growth factor (NGF) is a target-derived neurotroph

90 Nerve growth factor (NGF) is critical for the differenti

91 Nerve growth factor (NGF) is elevated in certain chronic

92 Nerve growth factor (NGF) is initially synthesized as a

93 that retrograde signaling by target-derived nerve growth factor (NGF) is necessary for soma-to-axon

94 Nerve growth factor (NGF) is produced in the hippocampus

95 Nerve growth factor (NGF) is the neurotrophin responsibl

96 Nerve growth factor (NGF) levels are highly increased in

97 Nerve growth factor (NGF) monoclonal antibody therapy ha

98 eactivation in medium containing antibody to nerve growth factor (NGF) or delayed reactivation in med

99 We hypothesized that nerve growth factor (NGF) plays a key role in this proce

100 they innervate is regulated by the supply of nerve growth factor (NGF) produced by these tissues.

101 Target-derived nerve growth factor (NGF) promoted expression of its own

102 Here, we report that Nerve Growth Factor (NGF) promotes endocytosis of its Tr

103 Nerve growth factor (NGF) promotes growth, differentiati

104 In sensory neurons, nerve growth factor (NGF) promotes the formation of axon

105 Nerve growth factor (NGF) protein expression and secreti

106 Activation of the high-affinity nerve growth factor (NGF) receptor Trk occurs through mu

107 Activation of the nerve growth factor (NGF) receptor trkA and tissue acido

108 as' disease parasite Trypanosoma cruzi binds nerve growth factor (NGF) receptor TrkA, increasing rece

109 Here, we show that RGS12 associates with the nerve growth factor (NGF) receptor tyrosine kinase TrkA,

110 The nerve growth factor (NGF) receptor, trkA, the tumour sup

111 tic neurons by inhibiting endocytosis of the nerve growth factor (NGF) receptor, TrkA.

112 he administration of neuregulin 1 (NRG1) and nerve growth factor (NGF) recombinant proteins.

113 The neurotrophin nerve growth factor (NGF) regulates neuronal growth, dif

114 Here we demonstrate that nerve growth factor (NGF) regulates the levels of IP(5)

115 s promoted ADRB2-dependent PDAC development, nerve growth factor (NGF) secretion, and pancreatic nerv

116 gic deficit is associated with alteration in nerve growth factor (NGF) signaling and its relation to

117 Facilitation of nerve growth factor (NGF) signaling by the p75 neurotrop

118 This study reports that Rab22 promotes nerve growth factor (NGF) signaling-dependent neurite ou

119 Target tissue-derived nerve growth factor (NGF) signaling-induced gene express

120 to disrupt retrograde axonal trafficking of nerve growth factor (NGF) signals.

121 anges in living colonies of PC12 cells under nerve growth factor (NGF) stimulation for up to 7 days u

122 RK) activity and cell proliferation, whereas nerve growth factor (NGF) stimulation leads to sustained

123 Retrograde trophic signaling of nerve growth factor (NGF) supports neuronal survival and

124 Numerous studies have indicated that nerve growth factor (NGF) supports survival and phenotyp

125 Studies also show that peroxynitrite impairs nerve growth factor (NGF) survival signaling in sensory

126 n ongoing discussion is whether signaling of nerve growth factor (NGF) through its high-affinity rece

127 and impairs neurite outgrowth in response to nerve growth factor (NGF) treatment.

128 egeneration in regards to local secretion of nerve growth factor (NGF) upon carious injury.

129 Nerve growth factor (NGF) was discovered because of its

130 The death of sympathetic neurons after nerve growth factor (NGF) withdrawal requires de novo ge

131 Nerve growth factor (NGF), a classical trophic factor fo

132 ytosis into axon growth cones is enhanced by nerve growth factor (NGF), acting locally on distal axon

133 e treatment with an antibody that sequesters nerve growth factor (NGF), administered when the pain an

134 ptase (TPS1) and mastin, neuromedin-B (NMB), nerve growth factor (NGF), and leukotriene-synthesis enz

135 x8 expression and function were regulated by nerve growth factor (NGF), and the effect of NGF was pot

136 brain derived neurotrophic factor (BDNF) and nerve growth factor (NGF), and this increase is accompan

137 MCF2L regulates a nerve growth factor (NGF), and treatment with a humanize

138 cific enolase, growth-associated protein 43, nerve growth factor (NGF), and tyrosine kinase receptor

139 ophin family of growth factors, comprised of nerve growth factor (NGF), brain derived neurotrophic fa

140 sence of fibroblast growth factor 2 (FGF-2), nerve growth factor (NGF), brain-derived neurotropic fac

141 d protein expression increase in response to nerve growth factor (NGF), concomitant with differentiat

142 pared routes of delivery of the neurotrophin nerve growth factor (NGF), either through a multisynapti

143 /or Ulk2 resulted in impaired endocytosis of nerve growth factor (NGF), excessive axon arborization,

144 ine distinct monoclonal antibodies targeting nerve growth factor (NGF), for which we compare the pred

145 (d) in culture and this can be prevented by nerve growth factor (NGF), GDNF and ARTN, but not NRTN.

146 We show that nerve growth factor (NGF), implicated in the morphogenes

147 Neurotrophins, including nerve growth factor (NGF), increase neurite outgrowth in

148 directly binds acinus, which is regulated by nerve growth factor (NGF), inhibiting its stimulatory ef

149 ociceptive and inflammatory factors, such as nerve growth factor (NGF), interleukin-6 (IL-6), and car

150 However, the prototypic neurotrophin, nerve growth factor (NGF), is not thought to be anterogr

151 by receptor tyrosine kinase ligands such as nerve growth factor (NGF), that involves both Rap1 and P

152 a (TNFalpha), interleukin-1 beta (IL1 beta), nerve growth factor (NGF), the neuronal nuclear protein

153 Pro-nerve growth factor (NGF), the precursor of NGF, is a we

154 ne interleukin 6 (IL-6) and the neurotrophin nerve growth factor (NGF), which are intimately linked t

155 to ischemia, retinal neuronal cells express nerve growth factor (NGF), which can be proangiogenic.

156 oietic cytokine ligands of gp130 but also to nerve growth factor (NGF), which does not bind to gp130-

157 In this study we determined the number of nerve growth factor (NGF)- and interleukin-1beta (IL1bet

158 tors tropomyosin-related kinase A (TrkA) for nerve growth factor (NGF)-beta, TrkB for brain-derived n

159 We discovered that KIF1Bbeta is required for nerve growth factor (NGF)-dependent neuronal differentia

160 This affects the development of nerve growth factor (NGF)-dependent neurons including sy

161 f human CCF astrocytoma cells but stimulated nerve growth factor (NGF)-induced neurite outgrowth from

162 terference RNA methodology strongly enhanced nerve growth factor (NGF)-induced neurite outgrowth in P

163 ative mutants of ATL1 in PC-12 cells inhibit nerve growth factor (NGF)-induced neurite outgrowth.

164 s study, we investigated the effect of ES on nerve growth factor (NGF)-induced neuronal differentiati

165 Egr3 is a nerve growth factor (NGF)-induced transcriptional regula

166 Nerve growth factor (NGF)-induced transport of large ves

167 NF-alpha-TNFR1 forward signaling to suppress nerve growth factor (NGF)-mediated neurite growth, survi

168 oter under basal conditions and also enhance nerve growth factor (NGF)-mediated trkA promoter activat

169 Here, using single-cell image analysis of nerve growth factor (NGF)-stimulated PC12 cells, we iden

170 ouse sympathetic neurons, the target-derived nerve growth factor (NGF)-tropomyosin-related kinase typ

171 the cell surface, similar to treatment with nerve growth factor (NGF).

172 e transmembrane receptor tyrosine kinase for nerve growth factor (NGF).

173 ll neurite extension in response to released nerve growth factor (NGF).

174 ng leptin, insulin, growth hormone (GH), and nerve growth factor (NGF).

175 , basic fibroblast growth factor (bFGF), and nerve growth factor (NGF).

176 anscription factor activation in response to nerve growth factor (NGF).

177 lated local IMPA1 translation in response to nerve growth factor (NGF).

178 at involves depriving sympathetic neurons of nerve growth factor (NGF).

179 p-regulation of the growth-promoting factor, nerve growth factor (NGF).

180 s mediate extension of sympathetic axons via nerve growth factor (NGF).

181 (P<0.0001) and cells positively staining for nerve growth factor (NGF; P<0.0001) in the infarcted bra

182 Significant examples include the nerve growth factor (P = 7.86 x 10(-33)), epidermal grow

183 The precursor of nerve growth factor (proNGF) has been described as a bio

184 We report that the precursor of nerve growth factor (proNGF) is overexpressed in prostat

185 ells showed intense immunoreactivity for pro-nerve growth factor (proNGF), neurotrophin receptor p75

186 ts interaction with proapoptotic ligands pro-nerve growth factor and amyloid-beta peptide.

187 ation around itself, and that high levels of nerve growth factor and axon guidance molecules are reco

188 (2) this interaction is modified by ligands nerve growth factor and beta-amyloid; (3) APP and p75(NT

189 Neurotrophins (nerve growth factor and brain-derived neurotrophic facto

190 ed in intracellular transport and sorting of nerve growth factor and brain-derived neurotrophic facto

191 ed in intracellular transport and sorting of nerve growth factor and brain-derived neurotrophic facto

192 eutralizing antibodies against brain-derived nerve growth factor and ciliary neurotrophic factor.

193 Nerve growth factor and insulin-like growth factor-1 exp

194 tiation and myelination that is regulated by nerve growth factor and its cognate receptor TrkA in a d

195 Nerve growth factor and its TrkA receptor were upregulat

196 d the AP1-mediated transcription promoted by nerve growth factor and modulated the expression of seve

197 E7 to E18 corneal expressions of nerve growth factor and neurotrophin-3 genes were unchan

198 otrophins brain derived neurotrophic factor, nerve growth factor and neurotrophin-3 in the supernatan

199 cription of the key sympathetic neurotrophin nerve growth factor and occurs predominately in extrafol

200 trophin 3, insulin-like growth factor 1, and nerve growth factor and of the nerve growth factor recep

201 F-A-dependent gene transcription elicited by nerve growth factor and serum.

202 on equatorial BM except for higher levels of nerve growth factor and thrombospondin-2 (TSP2) by hES-R

203 tor blockade is independent of brain-derived nerve growth factor and TrkB receptor signaling.

204 mpathetic axon outgrowth that was blocked by nerve growth factor antibodies.

205 (i) Anti-nerve growth factor antibody accelerated the reactivatio

206 e structurally related cystine-knot protein, nerve growth factor beta (NGFbeta), plays an unexpected

207 oclonal antibody, MEDI1912, selected against nerve growth factor binds with picomolar affinity, but u

208 ecal Rab7-siRNA or, indirectly, by reversing nerve growth factor deprivation in peripheral sensory ne

209 le Caspase-6 is activated in axons following nerve growth factor deprivation, microfluidic chamber ex

210 axon-specific but not whole-cell (apoptotic) nerve growth factor deprivation.

211 The present study used nerve growth factor differentiated PC12 cells (NGFDPC12

212 Nerve growth factor engages two structurally distinct tr

213 Choline supplementation increased striatal nerve growth factor expression in wild-type and Mecp2(1l

214 melanocytes and melanogenesis plus FIG4 and nerve growth factor expression, suggesting higher cellul

215 we have demonstrated increased levels of pro-nerve growth factor in cerebrospinal fluid and increased

216 Increased expression of nerve growth factor in injured or inflamed tissue is ass

217 the plasma concentration of norepinephrine, nerve growth factor in the gastric fundus muscularis ext

218 Overexpression of nerve growth factor in the lumbosacral spinal cord induc

219 trograde model derived from experiments with nerve growth factor in the peripheral nervous system.

220 r tyrosine kinase responsive to neurotrophin nerve growth factor in vertebrate nervous systems, to in

221 tor to p75 neurotrophin receptor, blocks pro-nerve growth factor induced apoptosis in cells expressin

222 Nerve growth factor induced mild cold sensitization, con

223 munoprecipitation experiments indicated that nerve growth factor induced the association of endogenou

224 the transcriptional activity of Nurr1 on an nerve growth factor inducible-B response element reporte

225 anticipated, long and extensive neuritis on nerve growth factor induction.

226 which show promise in clinical trials, like nerve growth factor inhibitors and p38 kinase inhibitors

227 onsequence of the increased levels of mature nerve growth factor levels in skin, as revealed by Weste

228 n to motoneurons, depletion of increased pro-nerve growth factor levels or p75 receptor blockade.

229 Second, there was evidence of increased nerve growth factor production and dysregulation of the

230 orward signaling loop in which tumor-derived nerve growth factor promotes enteric tumor innervation,

231 asolateral expression of the apically sorted nerve growth factor receptor (NGFR, p75; extracellular a

232 antage of their restricted expression of the nerve growth factor receptor (p75) in conjunction with f

233 ell sorting based on their expression of the nerve growth factor receptor (p75), a surface marker for

234 he NTRK1 gene that encodes the high-affinity nerve growth factor receptor (TRKA protein).

235 hese (AG879) is a selective inhibitor of the nerve growth factor receptor and human epidermal growth

236 gy domain transcription factor RUNX1 and the nerve growth factor receptor TrkA.

237 re initially marked by the expression of the nerve growth factor receptor TrkA.

238 r hierarchical expression of CD271/p75/NGFR (nerve growth factor receptor) marks cells with enriched

239 sine phosphorylation sequences of either the nerve growth factor receptor, TrkA (tropomyosin receptor

240 We found that the high-affinity nerve growth factor receptor, TrkA, is down-regulated by

241 DHEA was previously shown to bind to the nerve growth factor receptor, tropomyosin-related kinase

242 Adult C57BL/6N and nerve growth factor receptor-deficient mice.

243 rker PGP 9.5 and the Schwann cell marker p75 nerve growth factor receptor.

244 otic properties via mechanisms involving the nerve growth factor receptors (tropomyosin-related kinas

245 factor 1, and nerve growth factor and of the nerve growth factor receptors, tyrosine kinase receptor

246 Ephrins (EFNs), 8 semaphorins (SEMAs), and 2 nerve growth factor receptors.

247 c convergence on gene mutations that disrupt nerve growth factor signaling, upon which sympathetic an

248 sary for PP2A/B'beta-mediated enhancement of nerve growth factor signaling.

249 that this death is not due to a reduction in nerve growth factor synthesis.

250 ons of brain-derived neurotrophic factor and nerve growth factor than NCSC-SCs.

251 uction and dysregulation of the ratio of pro-nerve growth factor to mature nerve growth factor, favou

252 we report that EVT901 reduces binding of pro-nerve growth factor to p75 neurotrophin receptor, blocks

253 ers indicate that PLCbeta induced soon after nerve growth factor treatment associates with TRAX rathe

254 The homodimer NGF (nerve growth factor) exerts its neuronal activity upon b

255 that are also the primary sites of action of nerve growth factor, a powerful modulator of bladder ner

256 at act at receptor tyrosine kinases, such as nerve growth factor, also cause differentiation.

257 diminished levels of inflammatory cytokines, nerve growth factor, and endogenous pruritogens, such as

258 including brain-derived neurotrophic factor, nerve growth factor, and neurotrophin 3.

259 Furthermore, decreased nerve growth factor, decreased c-fos and increased sympa

260 e ratio of pro-nerve growth factor to mature nerve growth factor, favouring p75 receptor expression a

261 including brain-derived neurotrophic factor, nerve growth factor, glial cell-derived neurotrophic fac

262 dy, we determined the interactive effects of nerve growth factor, insulin-like growth factor 1, and e

263 Studies indicate that treatment against nerve growth factor, interleukins, and ischemic-like med

264 inistration of neurotrophic factors (such as nerve growth factor, neurotrophin3, glial-derived neurot

265 er, culture in serum-free media, presence of nerve growth factor, or addition of pituitary adenylate

266 t or the inhibition of adrenergic receptors, nerve growth factor, or brain-derived neurotrophic facto

267 When treated with nerve growth factor, PC12 cells will differentiate over

268 ncluding prostaglandin E(2), bradykinin, and nerve growth factor, that reduce the threshold and incre

269 of the Trk receptors, which are activated by nerve growth factor, there are only two established phos

270 cts neuroprotection by neurotrophins such as nerve growth factor, which bind to p75NTR receptors on M

271 Nerve growth factor-beta (NGF) is essential for the corr

272 ed proteins: a therapeutic IgG1-antibody and nerve growth factor-beta (NGF).

273 ion of Akt and ERK/MAP kinase in response to nerve growth factor-beta (NGF-beta) but not FP6.

274 is was associated with reduced expression of nerve growth factor-beta, indicating less atrial nerve s

275 c acid (PA)-induced lipotoxicity (PA-LTx) in nerve growth factor-differentiated PC12 (NGFDPC12) cells

276 from cardiac synaptosomes and dopamine from nerve growth factor-differentiated PC12 cells in a conce

277 Nerve growth factor-induced Balpha (NGFI-Balpha, Nur77)

278 1 transactivates the Skp2 promoter through a nerve growth factor-induced clone B response element (NB

279 ereas USP21 knockdown in PC12 cells inhibits nerve growth factor-induced neurite outgrowth.

280 siRNA improved cell survival in response to nerve growth factor-induced OL apoptosis.

281 hat increased DNA methylation at the NGFI-A (nerve growth factor-induced protein A) binding site of t

282 hibitory factors (e.g. inhibin alpha and VGF nerve growth factor-inducible), 2) activation of a signa

283 only known Toll receptor ligand is the human nerve growth factor-like cystine knot protein Spatzle.

284 owed a reduction in neurite outgrowth and in nerve growth factor-mediated neuronal differentiation, t

285 t from lactate release and activation of pro-nerve growth factor-p75 receptor signalling are key comp

286 ays 0, 3, 7, and 12 following treatment with nerve growth factor.

287 monoclonal antibody that binds and inhibits nerve growth factor.

288 neurite outgrowth in PC12 cells treated with nerve growth factor.

289 Reactivation can be induced by depletion of nerve growth factor; other commonly used reactivation st

290 of brain-derived neurotrophic factor (BDNF), nerve-growth factor (NGF), neurotrophin-3 (NT-3), fibrob

291 brain-derived neurotrophic factor (BDNF) and nerve-growth factor (NGF).

292 ved regions were found near color vision and nerve-growth genes, consistent with purifying selection

293 chemical and biophysical cues for increasing nerve growth has been investigated, including neurotroph

294 zation as well as an induction of peripheral nerve growth in grafted areas compared with samples not

295 ntial for implementing optogenetics to drive nerve growth in specific cell populations.

296 nd that abrogation of VEGF signaling reduces nerve growth in vitro and in vivo.

297 ter myocardial infarction, a situation where nerve growth is hindered by age-related influences and s

298 ibited skin edema, keratinocyte hyperplasia, nerve growth, leukocyte infiltration, and antihistamine-

299 m microarray of 85 extracellular epitopes of nerve growth-related proteins.

300 VEGF-mediated nerve growth was measured in a trigeminal ganglia explan