ライフサイエンスコーパス: nerve growth factor

1 ays 0, 3, 7, and 12 following treatment with nerve growth factor.

2 monoclonal antibody that binds and inhibits nerve growth factor.

3 neurite outgrowth in PC12 cells treated with nerve growth factor.

4 otrophin receptor p75NTR; both of which bind nerve growth factor.

5 dritic compartment, similar to brain-derived nerve growth factor.

6 factor pathway is the gene encoding the VGF nerve growth factor, a peptide precursor previously show

7 that are also the primary sites of action of nerve growth factor, a powerful modulator of bladder ner

8 at act at receptor tyrosine kinases, such as nerve growth factor, also cause differentiation.

9 ts interaction with proapoptotic ligands pro-nerve growth factor and amyloid-beta peptide.

10 ation around itself, and that high levels of nerve growth factor and axon guidance molecules are reco

11 (2) this interaction is modified by ligands nerve growth factor and beta-amyloid; (3) APP and p75(NT

12 Neurotrophins (nerve growth factor and brain-derived neurotrophic facto

13 ed in intracellular transport and sorting of nerve growth factor and brain-derived neurotrophic facto

14 ed in intracellular transport and sorting of nerve growth factor and brain-derived neurotrophic facto

15 eutralizing antibodies against brain-derived nerve growth factor and ciliary neurotrophic factor.

16 Nerve growth factor and insulin-like growth factor-1 exp

17 tiation and myelination that is regulated by nerve growth factor and its cognate receptor TrkA in a d

18 Nerve growth factor and its TrkA receptor were upregulat

19 d the AP1-mediated transcription promoted by nerve growth factor and modulated the expression of seve

20 E7 to E18 corneal expressions of nerve growth factor and neurotrophin-3 genes were unchan

21 otrophins brain derived neurotrophic factor, nerve growth factor and neurotrophin-3 in the supernatan

22 cription of the key sympathetic neurotrophin nerve growth factor and occurs predominately in extrafol

23 trophin 3, insulin-like growth factor 1, and nerve growth factor and of the nerve growth factor recep

24 F-A-dependent gene transcription elicited by nerve growth factor and serum.

25 on equatorial BM except for higher levels of nerve growth factor and thrombospondin-2 (TSP2) by hES-R

26 tor blockade is independent of brain-derived nerve growth factor and TrkB receptor signaling.

27 diminished levels of inflammatory cytokines, nerve growth factor, and endogenous pruritogens, such as

28 including brain-derived neurotrophic factor, nerve growth factor, and neurotrophin 3.

29 mpathetic axon outgrowth that was blocked by nerve growth factor antibodies.

30 (i) Anti-nerve growth factor antibody accelerated the reactivatio

31 e growth factor-binding protein 2 (IGFBP-2), nerve growth factor (b-NGF), platelet-derived growth fac

32 day, driven by circadian clock control of a nerve growth factor (BDNF) in the inner ear.

33 e structurally related cystine-knot protein, nerve growth factor beta (NGFbeta), plays an unexpected

34 orcine, bovine, and murine sequences of beta nerve growth factor (beta-NGF).

35 Nerve growth factor-beta (NGF) is essential for the corr

36 ed proteins: a therapeutic IgG1-antibody and nerve growth factor-beta (NGF).

37 ion of Akt and ERK/MAP kinase in response to nerve growth factor-beta (NGF-beta) but not FP6.

38 is was associated with reduced expression of nerve growth factor-beta, indicating less atrial nerve s

39 oclonal antibody, MEDI1912, selected against nerve growth factor binds with picomolar affinity, but u

40 Neurotrophins (nerve growth factor, brain-derived neurotrophic factor,

41 Furthermore, decreased nerve growth factor, decreased c-fos and increased sympa

42 ecal Rab7-siRNA or, indirectly, by reversing nerve growth factor deprivation in peripheral sensory ne

43 le Caspase-6 is activated in axons following nerve growth factor deprivation, microfluidic chamber ex

44 axon-specific but not whole-cell (apoptotic) nerve growth factor deprivation.

45 12 cells from 6-hydroxydopamine but not from nerve growth factor deprivation.

46 The present study used nerve growth factor differentiated PC12 cells (NGFDPC12

47 c acid (PA)-induced lipotoxicity (PA-LTx) in nerve growth factor-differentiated PC12 (NGFDPC12) cells

48 from cardiac synaptosomes and dopamine from nerve growth factor-differentiated PC12 cells in a conce

49 Moreover, nerve growth factor elicits extensive sprouting in p42 s

50 Nerve growth factor engages two structurally distinct tr

51 The homodimer NGF (nerve growth factor) exerts its neuronal activity upon b

52 Choline supplementation increased striatal nerve growth factor expression in wild-type and Mecp2(1l

53 melanocytes and melanogenesis plus FIG4 and nerve growth factor expression, suggesting higher cellul

54 e ratio of pro-nerve growth factor to mature nerve growth factor, favouring p75 receptor expression a

55 including brain-derived neurotrophic factor, nerve growth factor, glial cell-derived neurotrophic fac

56 solate low-affinity antibodies to human beta nerve growth factor (hbetaNGF).

57 Human nerve growth factor (hNGF) is an important pharmaceutica

58 we have demonstrated increased levels of pro-nerve growth factor in cerebrospinal fluid and increased

59 Increased expression of nerve growth factor in injured or inflamed tissue is ass

60 the plasma concentration of norepinephrine, nerve growth factor in the gastric fundus muscularis ext

61 Overexpression of nerve growth factor in the lumbosacral spinal cord induc

62 trograde model derived from experiments with nerve growth factor in the peripheral nervous system.

63 r tyrosine kinase responsive to neurotrophin nerve growth factor in vertebrate nervous systems, to in

64 tor to p75 neurotrophin receptor, blocks pro-nerve growth factor induced apoptosis in cells expressin

65 Nerve growth factor induced mild cold sensitization, con

66 munoprecipitation experiments indicated that nerve growth factor induced the association of endogenou

67 sis; by contrast 5-HT was protective against nerve growth factor-induced apoptosis.

68 Nerve growth factor-induced Balpha (NGFI-Balpha, Nur77)

69 1 transactivates the Skp2 promoter through a nerve growth factor-induced clone B response element (NB

70 ckbeta, but not Nckalpha, completely blocked nerve growth factor-induced neurite outgrowth in PC12 ce

71 ereas USP21 knockdown in PC12 cells inhibits nerve growth factor-induced neurite outgrowth.

72 siRNA improved cell survival in response to nerve growth factor-induced OL apoptosis.

73 hat increased DNA methylation at the NGFI-A (nerve growth factor-induced protein A) binding site of t

74 the transcriptional activity of Nurr1 on an nerve growth factor inducible-B response element reporte

75 hibitory factors (e.g. inhibin alpha and VGF nerve growth factor-inducible), 2) activation of a signa

76 anticipated, long and extensive neuritis on nerve growth factor induction.

77 which show promise in clinical trials, like nerve growth factor inhibitors and p38 kinase inhibitors

78 dy, we determined the interactive effects of nerve growth factor, insulin-like growth factor 1, and e

79 Studies indicate that treatment against nerve growth factor, interleukins, and ischemic-like med

80 onsequence of the increased levels of mature nerve growth factor levels in skin, as revealed by Weste

81 n to motoneurons, depletion of increased pro-nerve growth factor levels or p75 receptor blockade.

82 only known Toll receptor ligand is the human nerve growth factor-like cystine knot protein Spatzle.

83 owed a reduction in neurite outgrowth and in nerve growth factor-mediated neuronal differentiation, t

84 inistration of neurotrophic factors (such as nerve growth factor, neurotrophin3, glial-derived neurot

85 and sympathetic neurons is promoted whether nerve growth factor (NGF) activates TrkA receptors on th

86 rch for neuroactive compounds that mimic the nerve growth factor (NGF) activity for the protection ag

87 Nerve growth factor (NGF) acts through its receptor, Trk

88 the preservation of sufficient expression of nerve growth factor (NGF) and activation of the neurotro

89 With expression of nerve growth factor (NGF) and basic fibroblast growth fa

90 ta), tumor necrosis factor alpha (TNFalpha), nerve growth factor (NGF) and brain derived neurotrophic

91 Neurotrophins such as nerve growth factor (NGF) and brain-derived neurotrophic

92 interleukin-6 (IL-6), interleukin-10 (IL10), nerve growth factor (NGF) and brain-derived neurotrophic

93 rait loci associated with gene expression of nerve growth factor (NGF) and calneuron 1 (CALN1) genes.

94 The nerve growth factor (NGF) and glial cell line-derived ne

95 The effect of nerve growth factor (NGF) and its receptor (NGFR) in inf

96 Nerve growth factor (NGF) and neurotrophin-3 (NT-3) are

97 ends, can be modulated by luminar release of nerve growth factor (NGF) and neurotrophin-3 (NT-3), res

98 ated by target-derived neurotrophins such as nerve growth factor (NGF) and neurotrophin-3 (NT-3).

99 Nerve growth factor (NGF) and neurotrophin-3 serve as at

100 h cone collapse and loss of actin filaments, nerve growth factor (NGF) and neurotrophin-3 still induc

101 SorCS3 binds the nerve growth factor (NGF) and platelet-derived growth fa

102 Nerve growth factor (NGF) antagonism is on the verge of

103 brain-derived neurotrophic factor (BDNF) and nerve growth factor (NGF) are crucial modulators in the

104 Levels of nerve growth factor (NGF) are elevated in inflamed tissu

105 neurotrophic and neuroprotective actions of nerve growth factor (NGF) are mediated by hypusinated eI

106 In this paper, we identify nerve growth factor (NGF) as a binding partner for MOG a

107 satetraenoic acid (12[S] or 15[S]-HETE), and nerve growth factor (NGF) as positive control.

108 in animals and humans show that blockade of nerve growth factor (NGF) attenuates both malignant and

109 tal apoptotic pathway that is activated when nerve growth factor (NGF) becomes limiting for neuronal

110 TrkA activation by nerve growth factor (NGF) binding the second extracellul

111 Nerve growth factor (NGF) binds to TrkA receptor and tri

112 esis of SCG neurons at a level comparable to nerve growth factor (NGF) but also doubled the neurite l

113 PC12 pheochromocytoma cell line responds to nerve growth factor (NGF) by exiting from the cell cycle

114 ated the output activity of Ngf encoding the nerve growth factor (NGF) by increasing histone H4 acety

115 Elevating levels of nerve growth factor (NGF) can have pronounced effects on

116 Depriving mouse sympathetic neurons of nerve growth factor (NGF) causes their apoptotic death.

117 Nerve growth factor (NGF) contributes to the development

118 Although nerve growth factor (NGF) controls survival, maturation

119 Sertad1 is also induced in neurons by nerve growth factor (NGF) deprivation and Abeta (beta-am

120 d contributes to neuron death in response to nerve growth factor (NGF) deprivation.

121 the painful arthritic knee joint and whether nerve growth factor (NGF) drives this pathologic reorgan

122 ent a detailed motion analysis of retrograde nerve growth factor (NGF) endosomes in axons to show tha

123 Nerve growth factor (NGF) exerts protective effects on t

124 We show that PC12 cells stimulated with nerve growth factor (NGF) exhibit statistically signific

125 elta), two transcription factors involved in nerve growth factor (NGF) expression and downregulated s

126 timulation of the gastric epithelium induced nerve growth factor (NGF) expression, and in turn NGF ov

127 linergic neurons in both regions depend upon nerve growth factor (NGF) for their survival and maturat

128 ciated haplotype blocks were intronic to the Nerve Growth Factor (NGF) gene (P=0.001, 0.001, 0.002),

129 In 2001, we initiated a clinical trial of nerve growth factor (NGF) gene therapy in AD, the first

130 Nerve growth factor (NGF) has an important role in regul

131 ) at the cell surface, and administration of nerve growth factor (NGF) has been considered and attemp

132 The neurotrophin nerve growth factor (NGF) has been implicated as a key m

133 ng mode of Spz to Toll is similar to that of nerve growth factor (NGF) in complex with the p75 neurot

134 +) BMCs expressed neurotrophins and secreted nerve growth factor (NGF) in conditioned medium.

135 A role for nerve growth factor (NGF) in contributing to increased v

136 taRII-deficient pups showed a lower level of nerve growth factor (NGF) in its mRNA; however, higher l

137 n channel subfamily M, member 8 (TRPM8); and nerve growth factor (NGF) in nasal biopsy specimens.

138 s and becomes functional by the neurotrophin nerve growth factor (NGF) in native brainstem neurons.

139 K)1 inhibits transient activation induced by nerve growth factor (NGF) in PC12 cells.

140 on (DRG) neurons by increasing expression of nerve growth factor (NGF) in the colon wall.

141 se it is supported by decades of research on nerve growth factor (NGF) in the peripheral nervous syst

142 The endogenous nerve growth factor (NGF) in the urinary bladder regulat

143 Previously, we demonstrated that nerve growth factor (NGF) increased the excitability of

144 e basolateral NHE1 Na(+)/H(+) exchanger with nerve growth factor (NGF) induces actin cytoskeleton rem

145 Nerve growth factor (NGF) induces collateral branching a

146 Nerve growth factor (NGF) induces neurite outgrowth and

147 Nerve growth factor (NGF) influences the key pathologica

148 Nerve growth factor (NGF) influences the survival and di

149 The microinjection of nerve growth factor (NGF) into the cat pontine tegmentum

150 Nerve growth factor (NGF) is a neuropeptide essential fo

151 Nerve growth factor (NGF) is a neurotrophin that is impl

152 Nerve growth factor (NGF) is a potent survival and axon

153 Nerve growth factor (NGF) is a target-derived neurotroph

154 Nerve growth factor (NGF) is critical for the differenti

155 Nerve growth factor (NGF) is elevated in certain chronic

156 Nerve growth factor (NGF) is initially synthesized as a

157 that retrograde signaling by target-derived nerve growth factor (NGF) is necessary for soma-to-axon

158 Nerve growth factor (NGF) is produced in the hippocampus

159 Nerve growth factor (NGF) is the neurotrophin responsibl

160 Nerve growth factor (NGF) levels are highly increased in

161 Nerve growth factor (NGF) monoclonal antibody therapy ha

162 eactivation in medium containing antibody to nerve growth factor (NGF) or delayed reactivation in med

163 We hypothesized that nerve growth factor (NGF) plays a key role in this proce

164 they innervate is regulated by the supply of nerve growth factor (NGF) produced by these tissues.

165 Target-derived nerve growth factor (NGF) promoted expression of its own

166 Here, we report that Nerve Growth Factor (NGF) promotes endocytosis of its Tr

167 Nerve growth factor (NGF) promotes growth, differentiati

168 In sensory neurons, nerve growth factor (NGF) promotes the formation of axon

169 Nerve growth factor (NGF) protein expression and secreti

170 High-affinity nerve growth factor (NGF) receptor (NGFR-TrkA) expressio

171 Activation of the high-affinity nerve growth factor (NGF) receptor Trk occurs through mu

172 Activation of the nerve growth factor (NGF) receptor trkA and tissue acido

173 as' disease parasite Trypanosoma cruzi binds nerve growth factor (NGF) receptor TrkA, increasing rece

174 Here, we show that RGS12 associates with the nerve growth factor (NGF) receptor tyrosine kinase TrkA,

175 The nerve growth factor (NGF) receptor, trkA, the tumour sup

176 tic neurons by inhibiting endocytosis of the nerve growth factor (NGF) receptor, TrkA.

177 he administration of neuregulin 1 (NRG1) and nerve growth factor (NGF) recombinant proteins.

178 Nerve Growth Factor (NGF) regulates chronic inflammatory

179 The neurotrophin nerve growth factor (NGF) regulates neuronal growth, dif

180 Here we demonstrate that nerve growth factor (NGF) regulates the levels of IP(5)

181 s promoted ADRB2-dependent PDAC development, nerve growth factor (NGF) secretion, and pancreatic nerv

182 gic deficit is associated with alteration in nerve growth factor (NGF) signaling and its relation to

183 Facilitation of nerve growth factor (NGF) signaling by the p75 neurotrop

184 nd NGFRAP1 interact with p75NTR and modulate nerve growth factor (NGF) signaling through nuclear fact

185 This study reports that Rab22 promotes nerve growth factor (NGF) signaling-dependent neurite ou

186 Target tissue-derived nerve growth factor (NGF) signaling-induced gene express

187 Retrograde axonal transport of nerve growth factor (NGF) signals is critical for the su

188 to disrupt retrograde axonal trafficking of nerve growth factor (NGF) signals.

189 anges in living colonies of PC12 cells under nerve growth factor (NGF) stimulation for up to 7 days u

190 RK) activity and cell proliferation, whereas nerve growth factor (NGF) stimulation leads to sustained

191 Retrograde trophic signaling of nerve growth factor (NGF) supports neuronal survival and

192 Numerous studies have indicated that nerve growth factor (NGF) supports survival and phenotyp

193 Studies also show that peroxynitrite impairs nerve growth factor (NGF) survival signaling in sensory

194 n ongoing discussion is whether signaling of nerve growth factor (NGF) through its high-affinity rece

195 Nerve growth factor (NGF) treatment induced an increase

196 and impairs neurite outgrowth in response to nerve growth factor (NGF) treatment.

197 egeneration in regards to local secretion of nerve growth factor (NGF) upon carious injury.

198 Nerve growth factor (NGF) was discovered because of its

199 The death of sympathetic neurons after nerve growth factor (NGF) withdrawal requires de novo ge

200 nteraction between MLK3 and GSK-3beta during nerve growth factor (NGF) withdrawal-induced cell death

201 Nerve growth factor (NGF), a classical trophic factor fo

202 tone and that these effects are modulated by nerve growth factor (NGF), a neurotrophin that regulates

203 ytosis into axon growth cones is enhanced by nerve growth factor (NGF), acting locally on distal axon

204 e treatment with an antibody that sequesters nerve growth factor (NGF), administered when the pain an

205 ptase (TPS1) and mastin, neuromedin-B (NMB), nerve growth factor (NGF), and leukotriene-synthesis enz

206 x8 expression and function were regulated by nerve growth factor (NGF), and the effect of NGF was pot

207 brain derived neurotrophic factor (BDNF) and nerve growth factor (NGF), and this increase is accompan

208 MCF2L regulates a nerve growth factor (NGF), and treatment with a humanize

209 cific enolase, growth-associated protein 43, nerve growth factor (NGF), and tyrosine kinase receptor

210 ophin family of growth factors, comprised of nerve growth factor (NGF), brain derived neurotrophic fa

211 To determine which neurotrophins (NTs)-nerve growth factor (NGF), brain-derived neurotrophin (B

212 sence of fibroblast growth factor 2 (FGF-2), nerve growth factor (NGF), brain-derived neurotropic fac

213 d protein expression increase in response to nerve growth factor (NGF), concomitant with differentiat

214 pared routes of delivery of the neurotrophin nerve growth factor (NGF), either through a multisynapti

215 /or Ulk2 resulted in impaired endocytosis of nerve growth factor (NGF), excessive axon arborization,

216 ine distinct monoclonal antibodies targeting nerve growth factor (NGF), for which we compare the pred

217 (d) in culture and this can be prevented by nerve growth factor (NGF), GDNF and ARTN, but not NRTN.

218 We show that nerve growth factor (NGF), implicated in the morphogenes

219 Neurotrophins, including nerve growth factor (NGF), increase neurite outgrowth in

220 directly binds acinus, which is regulated by nerve growth factor (NGF), inhibiting its stimulatory ef

221 ociceptive and inflammatory factors, such as nerve growth factor (NGF), interleukin-6 (IL-6), and car

222 However, the prototypic neurotrophin, nerve growth factor (NGF), is not thought to be anterogr

223 t additional bovine pituitary extract (BPE), nerve growth factor (NGF), or basic fibroblast growth fa

224 by receptor tyrosine kinase ligands such as nerve growth factor (NGF), that involves both Rap1 and P

225 a (TNFalpha), interleukin-1 beta (IL1 beta), nerve growth factor (NGF), the neuronal nuclear protein

226 Pro-nerve growth factor (NGF), the precursor of NGF, is a we

227 ne interleukin 6 (IL-6) and the neurotrophin nerve growth factor (NGF), which are intimately linked t

228 to ischemia, retinal neuronal cells express nerve growth factor (NGF), which can be proangiogenic.

229 oietic cytokine ligands of gp130 but also to nerve growth factor (NGF), which does not bind to gp130-

230 In this study we determined the number of nerve growth factor (NGF)- and interleukin-1beta (IL1bet

231 tors tropomyosin-related kinase A (TrkA) for nerve growth factor (NGF)-beta, TrkB for brain-derived n

232 We discovered that KIF1Bbeta is required for nerve growth factor (NGF)-dependent neuronal differentia

233 This affects the development of nerve growth factor (NGF)-dependent neurons including sy

234 ethyltransferase 3b (Dnmt3b) is required for nerve growth factor (NGF)-induced differentiation of PC1

235 f human CCF astrocytoma cells but stimulated nerve growth factor (NGF)-induced neurite outgrowth from

236 terference RNA methodology strongly enhanced nerve growth factor (NGF)-induced neurite outgrowth in P

237 ative mutants of ATL1 in PC-12 cells inhibit nerve growth factor (NGF)-induced neurite outgrowth.

238 s study, we investigated the effect of ES on nerve growth factor (NGF)-induced neuronal differentiati

239 repressed by DNA methyltransferase 3b during nerve growth factor (NGF)-induced neuronal differentiati

240 Egr3 is a nerve growth factor (NGF)-induced transcriptional regula

241 Nerve growth factor (NGF)-induced transport of large ves

242 NF-alpha-TNFR1 forward signaling to suppress nerve growth factor (NGF)-mediated neurite growth, survi

243 ce, we investigated the role of DeltaNp73 in nerve growth factor (NGF)-mediated neuronal differentiat

244 oter under basal conditions and also enhance nerve growth factor (NGF)-mediated trkA promoter activat

245 Here, using single-cell image analysis of nerve growth factor (NGF)-stimulated PC12 cells, we iden

246 ouse sympathetic neurons, the target-derived nerve growth factor (NGF)-tropomyosin-related kinase typ

247 the cell surface, similar to treatment with nerve growth factor (NGF).

248 e transmembrane receptor tyrosine kinase for nerve growth factor (NGF).

249 ll neurite extension in response to released nerve growth factor (NGF).

250 ng leptin, insulin, growth hormone (GH), and nerve growth factor (NGF).

251 , basic fibroblast growth factor (bFGF), and nerve growth factor (NGF).

252 anscription factor activation in response to nerve growth factor (NGF).

253 lated local IMPA1 translation in response to nerve growth factor (NGF).

254 at involves depriving sympathetic neurons of nerve growth factor (NGF).

255 p-regulation of the growth-promoting factor, nerve growth factor (NGF).

256 s mediate extension of sympathetic axons via nerve growth factor (NGF).

257 (P<0.0001) and cells positively staining for nerve growth factor (NGF; P<0.0001) in the infarcted bra

258 of brain-derived neurotrophic factor (BDNF), nerve-growth factor (NGF), neurotrophin-3 (NT-3), fibrob

259 brain-derived neurotrophic factor (BDNF) and nerve-growth factor (NGF).

260 sting internalization, whereas brain-derived nerve growth factor or bicuculline increased the ratio b

261 er, culture in serum-free media, presence of nerve growth factor, or addition of pituitary adenylate

262 t or the inhibition of adrenergic receptors, nerve growth factor, or brain-derived neurotrophic facto

263 Reactivation can be induced by depletion of nerve growth factor; other commonly used reactivation st

264 Significant examples include the nerve growth factor (P = 7.86 x 10(-33)), epidermal grow

265 t from lactate release and activation of pro-nerve growth factor-p75 receptor signalling are key comp

266 When treated with nerve growth factor, PC12 cells will differentiate over

267 The nerve growth factor precursor (pro-NGF) may function as

268 Image analysis demonstrated that nerve growth factor-primed rat pheochromocytoma cells (P

269 Second, there was evidence of increased nerve growth factor production and dysregulation of the

270 orward signaling loop in which tumor-derived nerve growth factor promotes enteric tumor innervation,

271 The precursor of nerve growth factor (proNGF) has been described as a bio

272 We report that the precursor of nerve growth factor (proNGF) is overexpressed in prostat

273 ells showed intense immunoreactivity for pro-nerve growth factor (proNGF), neurotrophin receptor p75

274 asolateral expression of the apically sorted nerve growth factor receptor (NGFR, p75; extracellular a

275 antage of their restricted expression of the nerve growth factor receptor (p75) in conjunction with f

276 ell sorting based on their expression of the nerve growth factor receptor (p75), a surface marker for

277 he NTRK1 gene that encodes the high-affinity nerve growth factor receptor (TRKA protein).

278 hese (AG879) is a selective inhibitor of the nerve growth factor receptor and human epidermal growth

279 gy domain transcription factor RUNX1 and the nerve growth factor receptor TrkA.

280 re initially marked by the expression of the nerve growth factor receptor TrkA.

281 opaminergic cells, through the activation of nerve growth factor receptor TrkA.

282 r hierarchical expression of CD271/p75/NGFR (nerve growth factor receptor) marks cells with enriched

283 sine phosphorylation sequences of either the nerve growth factor receptor, TrkA (tropomyosin receptor

284 We found that the high-affinity nerve growth factor receptor, TrkA, is down-regulated by

285 DHEA was previously shown to bind to the nerve growth factor receptor, tropomyosin-related kinase

286 Adult C57BL/6N and nerve growth factor receptor-deficient mice.

287 rker PGP 9.5 and the Schwann cell marker p75 nerve growth factor receptor.

288 otic properties via mechanisms involving the nerve growth factor receptors (tropomyosin-related kinas

289 factor 1, and nerve growth factor and of the nerve growth factor receptors, tyrosine kinase receptor

290 Ephrins (EFNs), 8 semaphorins (SEMAs), and 2 nerve growth factor receptors.

291 c convergence on gene mutations that disrupt nerve growth factor signaling, upon which sympathetic an

292 sary for PP2A/B'beta-mediated enhancement of nerve growth factor signaling.

293 that this death is not due to a reduction in nerve growth factor synthesis.

294 ons of brain-derived neurotrophic factor and nerve growth factor than NCSC-SCs.

295 ncluding prostaglandin E(2), bradykinin, and nerve growth factor, that reduce the threshold and incre

296 of the Trk receptors, which are activated by nerve growth factor, there are only two established phos

297 uction and dysregulation of the ratio of pro-nerve growth factor to mature nerve growth factor, favou

298 we report that EVT901 reduces binding of pro-nerve growth factor to p75 neurotrophin receptor, blocks

299 ers indicate that PLCbeta induced soon after nerve growth factor treatment associates with TRAX rathe

300 cts neuroprotection by neurotrophins such as nerve growth factor, which bind to p75NTR receptors on M