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1 uperexcitability that follows a conditioning nerve impulse.
2 ure is the basis for saltatory conduction of nerve impulses.
3 l for the rapid and efficient propagation of nerve impulses.
4 vation retards mitochondrial motility during nerve impulses.
5 zations adapted for generation of high-speed nerve impulses.
6 es such as the generation and propagation of nerve impulses.
7 ng axons, which prevents the transmission of nerve impulses.
8 channels are essential for the generation of nerve impulses.
9 s that are essential for the transmission of nerve impulses.
10 and/or such synthesis may be upregulated by nerve impulses.
11 cells, where it mediates the propagation of nerve impulses.
16 , synaptic transmission, and transmission of nerve impulses, all of which are biologically meaningful
18 transmitter ACh in response to an individual nerve impulse and with a brief latency characteristic of
19 s regulate cell division, circadian rhythms, nerve impulses and chemotaxis, and guide the development
20 ransmembrane proteins that are essential for nerve impulses and regulate ion flow across cell membran
21 salivary secretion of SIgA is upregulated by nerve impulses and that sympathetic nerves induce a grea
22 avours the ectopic generation of spontaneous nerve impulses and their ephaptic conduction to adjacent
23 rucial for the generation and propagation of nerve impulses, and as such are widely targeted by toxin
24 Pathways connected to the procession of the nerve impulse are major mechanisms involved in the devel
25 initial segment and nodes of Ranvier, where nerve impulses are generated and propagated, a high dens
27 h cell membranes, producing, for example the nerve impulse, are in many cases composed of four domain
31 e, detection, processing, and propagation of nerve impulses by neurons; contraction and relaxation by
32 se experiments, a single temporally isolated nerve impulse caused the synchronous opening of ATP-gate
33 cytes around axons in the CNS, enables rapid nerve impulse conduction and sustains neuronal health.
37 odium channels (Nav) and thus underpin rapid nerve impulse conduction in the vertebrate nervous syste
39 ough the injury in all treated animals while nerve impulse conduction remained absent in all sham-tre
40 to the cord immediately reversed the loss of nerve impulse conduction through the injury in all treat
41 have been proposed to affect the velocity of nerve impulse conduction; however, direct evidence is la
43 ediates the transduction of sound waves into nerve impulses, depends on the endocochlear potential an
47 thesis was assessed in rats treated with the nerve impulse flow inhibitor gamma-butyrolactone (GBL).
48 ificantly reduced, whereas capsaicin-induced nerve impulses in the skin-nerve preparation increased i
51 ndings demonstrate, for the first time, that nerve impulses or chemical stimulation promote Ca2+ entr
52 al' loci where the myelin terminates and the nerve impulse propagates along the axon by 'saltatory' c
53 rent therapies for neuropathic pain modulate nerve impulse propagation or synaptic transmission; thes
54 We have also observed that, in response to a nerve impulse, synapses with low release probability pri
55 y inner hair cells (IHCs) encode sounds into nerve impulses through fast and indefatigable Ca(2+)-dep
57 leases Ca(2+) from intracellular stores upon nerve impulse to trigger skeletal muscle contraction.
58 inputs within their dendrites and propagate nerve impulses to distant targets through a single axon.
61 myelin sheath surrounding axons ensures that nerve impulses travel quickly and efficiently, allowing
63 ntent (the number of ACh quanta released per nerve impulse) was only approximately 50% of that in con
65 1-S4 voltage-sensing domains responsible for nerve impulses, where interactions with the lipid bilaye
66 anslate acoustic stimuli into 'phase-locked' nerve impulses with frequencies of up to at least 1 kHz.
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