ライフサイエンスコーパス: nerve root

1 cells were required for establishing the PP nerve root.

2 the tracer HRP/WGA-HRP on the cut end of the nerve root.

3 um, sacral foramina, and proximal S-1 to S-4 nerve roots.

4 sferase or retrogradely labeled from cranial nerve roots.

5 ing (MRI) evidence of grossly swollen spinal nerve roots.

6 ts in addition to the spinal cord and spinal nerve roots.

7 involvement and contact with spinal cord or nerve roots.

8 er the spinal motor neurons or their ventral nerve roots.

9 oplasmic whole mounts and delipidated spinal nerve roots.

10 plied by C8 and T1 as well as C5, C6, and C7 nerve roots.

11 within the spinal cord; however, five of 16 nerve roots (31.2%) demonstrated moderate wallerian dege

12 al nervous system (CNS) within the segmental nerve root and grow out toward the body wall muscles.

13 The surface of neurons in the eighth nerve root and in neighboring nodes of Ranvier stained f

14 Nerve root and limb-girdle muscle abnormalities were vis

15 toms is sustained after decompression of the nerve root and may also be responsible for the spontaneo

16 ells migrate avidly toward axonal tracts and nerve roots and differentiate into nonmyelinating enshea

17 ive stretch receptors with axons in anterior nerve roots and four more such receptors with axons in p

18 trates spine anatomy and the relationship of nerve roots and intervertebral disks.

19 to peripheral targets through the ganglionic nerve roots and longitudinal projections toward neighbor

20 c pulses elicited the largest responses from nerve roots and motor cortex.

21 nerve involvement suggests vulnerability of nerve roots and terminals where the blood-nerve barrier

22 the degree of axonal degeneration in sensory nerve roots, and improved clinical measures of neuropath

23 and white matter, in the dorsal and ventral nerve roots, and in the roof plate at E13, when neurocan

24 us palsy (Klumpke's) affecting the C8 and T1 nerve roots, and total plexus palsy.

25 um of lesions known to exist in VHL kidneys, nerve roots appear to harbor more wide-spread and morpho

26 plexus avulsion, a traumatic injury in which nerve roots are torn from the spinal cord.

27 n of GAP43(+) PLP(+) precursors in embryonic nerve roots as the cells of origin for these tumors and

28 e than a decade following traumatic brachial nerve root avulsion.

29 Studies of potentials for dorsal spinal nerve root axons to regrow into the spinal cord involved

30 ion, with the final branching pattern of the nerve roots being generated by a secondary condensation.

31 ese required surgery; four were treated with nerve root block; two, with central epidural injection;

32 Selective nerve root blocks with betamethasone and triamcinolone r

33 roots from patients with compression of the nerve root by an overlying blood vessel has revealed foc

34 opathy were more likely to have stenosis and nerve root compression (P < .006).

35 ies and the etiology of radiculopathy due to nerve root compression was excluded.

36 Except in cases with nerve root compression, disk extrusion, or central steno

37 ting either motor neuron or multiple ventral nerve root damage.

38 Similar foci of nerve root demyelination and juxtaposition of axons have

39 In most cases, the trigeminal nerve root demyelination involves the proximal, CNS part

40 utput, which can be monitored from occipital nerve roots, directly determines the rate and duration o

41 d T2 signal intensity and volume of brachial nerve roots do not exclude a diagnosis of ALS and sugges

42 myelination within the brainstem and cranial nerve roots during the progression of MS.

43 n mass with neurobiotin and biocytin through nerve roots, dye transfer was rarely observed.

44 nerves was characterized by a pair of large nerve roots, each of which branched into two major tract

45 Stimulation of the presynaptic nerve root elicited a biphasic synaptic current, includi

46 MRI imaging which demonstrated lumbar spinal nerve root enhancement and clumping or lesions.

47 Vascular and nerve root enhancement increased with triple-dose gadoli

48 alibre of the left trigeminal nerve from the nerve root exit zone in the pons to Meckel's cave.

49 xonal damage to the brainstem and the spinal nerve roots, found in 11 cases but not in controls, indi

50 Examination of trigeminal nerve roots from patients with compression of the nerve

51 Herpes simplex virus (HSV) remains latent in nerve root ganglia of infected persons and is thought to

52 apparent signal-to-noise ratio in the dorsal nerve root ganglion and C6 nerve (P < .001) with the mul

53 neuropile (longitudinal glia, midline glia, nerve root glia).

54 Nerve roots have specialized transition zones that permi

55 steophytic spurring, lordosis, kyphosis, and nerve root impingement.

56 tly located in the brainstem and the cranial nerve roots in addition to the spinal cord and spinal ne

57 ntified in myelinated axons of lumbar spinal nerve roots in rabbit and rat on the basis of RNase sens

58 emin after crush injury of the dorsal spinal nerve roots in rats.

59 of the cerebellum, brainstem, and peripheral nerve roots in spontaneous, as well as actively induced,

60 earch related to surgical repair of proximal nerve root injuries, and emerging potential therapies, w

61 jury than nerve root injury, suggesting that nerve root injury elicits a more robust, centrally media

62 nerve transection distal to the DRG or an L5 nerve root injury proximal to the DRG.

63 l allodynia for peripheral nerve injury than nerve root injury, suggesting that nerve root injury eli

64 echanical contributions for painful cervical nerve root injury.

65 nt of these conditions that have concomitant nerve-root involvement.

66 Previous studies have shown that the PP nerve root is normally pioneered by an O lineage-derived

67 il application, carrageenan inflammation, or nerve-root ligation.

68 lsy (Erb's) affecting the C5, C6, and +/- C7 nerve roots, lower plexus palsy (Klumpke's) affecting th

69 al cord and in motor neuron axons in ventral nerve roots, many of which are eventually lost over time

70 t to the toxic effects of IRE, injury to the nerve roots may be a limiting factor for the use of IRE

71 tive synaptic terminals were enriched around nerve root neurons and octopus cells in the PVCN and wer

72 as in wild-type mice, but end bulbs near the nerve root of je/je animals were smaller than in hearing

73 The cell clusters in the cochlear nerve root of the chinchilla provide the simplest exampl

74 abnormalities in F-wave chronodispersion in nerve roots of a few infected animals; which were absent

75 receptors, we penetrated large axons in the nerve roots of nerve cords from adult leeches with dye-f

76 rons with small caliber axons in the ventral nerve roots of the spinal cord.

77 ial dorsal cochlear nucleus and the cochlear nerve root on the ablated side.

78 ed--the motor neuron in the spinal cord, the nerve root or peripheral nerve, the neuromuscular juncti

79 trigeminal sensory fibres within either the nerve root or, less commonly, the brainstem.

80 In cases of nerve-root or meningeal enhancement, Lyme disease should

81 ng lesion with edema, and three demonstrated nerve-root or meningeal enhancement.

82 Decompression of the nerve root produces rapid relief of symptoms in most pat

83 Paravertebral block of the spinal nerve roots provides similar analgesia to thoracic epidu

84 of type I SGNs, with intense labeling in the nerve root region and posteroventral CN (PVCN).

85 Histopathological evaluation of L4 ventral nerve roots revealed that transgenic expression of the I

86 Accordingly, the C7 nerve root separately underwent chromic gut exposure, 10

87 stological features, were distributed in the nerve roots, spinal cord, and cerebellum.

88 he trigeminal nerve, along with the cervical nerve roots, supplies most of the sensory supply to the

89 In animals with transected peripheral nerve roots, TAxI delivery into motor neurons after peri

90 d early in development are the cranial motor nerve roots that exit the hindbrain, the motor neuron po

91 ngiomesenchymal tumorlets were in the dorsal nerve roots; the anterior roots and cerebellum were less

92 olvement without contact with spinal cord or nerve roots; third group, moderate involvement and conta

93 ellular treatment strategies after extensive nerve root trauma.

94 demonstrated enhancement of the cauda equina nerve roots, trigeminal nerve, and pachymeninges.

95 r magnetic stimulation of the lower thoracic nerve roots (Tw Pga).

96 rest-based analysis was performed to measure nerve root volume and T2 signal intensity.

97 en the IRE electrode and the spinal cord and nerve root was 1.71 mm +/- 0.90 and 8.47 mm + 3.44, resp

98 Increased T2 signal intensity of nerve roots was associated with faster disease progressi

99 ated to the spinal cord surface and emerging nerve roots was observed in images obtained in all contr

100 or wall of the vertebral body or the exiting nerve root were 2.93 mm +/- 0.77 (standard deviation) an

101 odel brachial plexus avulsion in the rat, C8 nerve roots were cut flush to the spinal cord and a pero

102 ity and volume alterations of C5, C6, and C7 nerve roots were observed in patients with ALS (P < .001

103 Cutting both nerve roots, which eliminates both lateral projections,

104 vertebrates is segmented to align the spinal nerve roots with the vertebrae.