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1 itions (volume paired with hypoglossal (XII) nerve stimulation).
2 timulation but not F-EPSPs evoked by colonic nerve stimulation.
3 timulation but not F-EPSPs evoked by colonic nerve stimulation.
4 postsynaptic responses elicited by afferent nerve stimulation.
5 vels ([NAD(P)H]m) increase within seconds of nerve stimulation.
6 imary sensory cortex face area due to median nerve stimulation.
7 elicited by bath-applied CCh and cholinergic nerve stimulation.
8 vered to tibialis anterior (TA) MNs by sural nerve stimulation.
9 r inhibition of cytokine production by vagus nerve stimulation.
10 but no increased activation following tibial nerve stimulation.
11 ulation, but reduced activations with tibial nerve stimulation.
12 firing in pulmonary veins by local autonomic nerve stimulation.
13 ed by measuring force in response to femoral nerve stimulation.
14 ter activity evoked in response to selective nerve stimulation.
15 es to acetylcholine, 5-hydroxytryptamine, or nerve stimulation.
16 to assess gamma-knife radiosurgery or vagal nerve stimulation.
17 tional inhibition of TNF production by vagus nerve stimulation.
18 recocious corneal thinning, but augmented T4 nerve stimulation.
19 cipital region with electrodes for occipital nerve stimulation.
20 exercise in response to supramaximal femoral nerve stimulation.
21 d for the soleus H-reflex elicited by tibial nerve stimulation.
22 ease of beta-NAD depends on the frequency of nerve stimulation.
23 taste buds on the rostral 1/3 of the tongue) nerve stimulation.
24 e seen with field potentials evoked by sural nerve stimulation.
25 c input that can also be evoked by olfactory nerve stimulation.
26 lex blinks evoked by supraorbital trigeminal nerve stimulation.
27 oorly responsive to carbachol and transmural nerve stimulation.
28 atable artificial anal sphincter, and sacral nerve stimulation.
29 d and received inhibitory inputs from aortic nerve stimulation.
30 ic ICC or smooth muscle cells in response to nerve stimulation.
31 DeltaQTsingle ) evoked by electrical femoral nerve stimulation.
32 ation of sensory neuron excitability by tail-nerve stimulation.
33 ng anaesthesia (n = 5) instead of repetitive nerve stimulation.
34 epicardial DOR increased during sympathetic nerve stimulation.
35 anced vascular responsiveness to sympathetic nerve stimulation.
36 mpromised the muscle twitch triggered by the nerve stimulation.
37 dominant repolarizing role during repetitive nerve stimulation.
38 s and stretchable multi-electrode arrays for nerve stimulation.
39 in relocalization to synaptic membranes upon nerve stimulation.
40 sensory evoked potentials elicited by median nerve stimulation.
41 timulation but not F-EPSPs evoked by colonic nerve stimulation.
43 imulation protocols were assessed: patterned nerve stimulation (10 pulses at 100 Hz every 1.5 sec), u
44 rhodamine-123, we determined how repetitive nerve stimulation (100 Hz) affects Psi(m) in motor termi
45 is depleted in about 10 s by high-frequency nerve stimulation (30 Hz); the RRP refills in about 1 mi
47 d, ventilated dogs were elicited via sciatic nerve stimulation (50 Hz; 200 ms duration; 1 contraction
48 ponse to bilateral anterior magnetic phrenic nerve stimulation (a pressure <11 cm H2O defined dysfunc
52 tylcholine release in the ventricle on vagal nerve stimulation and a high density of acetylcholine M2
53 e EAA afferents to the LC, including sciatic nerve stimulation and auditory stimuli and the tonic act
55 the carrageenan air pouch model, both vagus nerve stimulation and cholinergic agonists significantly
57 a decremental muscle response to repetitive nerve stimulation and frequently related to postsynaptic
59 paired and decreased in response to unpaired nerve stimulation and is mediated by the opposing action
61 AS, which consisted of peripheral electrical nerve stimulation and subsequent transcranial magnetic s
62 Vasoconstrictor responses to sympathetic nerve stimulation and their sensitivity to metabolic mod
63 a method of transcutaneous mechanical vagus nerve stimulation and then investigated whether this the
64 erminals is more responsive to low-frequency nerve stimulation and this is due to higher cytosolic Ca
65 suspended in an organ bath, and responses to nerve stimulation and to acetylcholine and substance P i
66 al, twitch responses to supramaximal femoral nerve stimulation and transcranial magnetic stimulation
67 hich involves repeated pairing of peripheral nerve stimulation and transcranial magnetic stimulation
68 Somatosensory cortical potentials to median nerve stimulation and visual cortical potentials to reve
69 timulation (SNS) and right and/or left vagus nerve stimulation and was compared with DOR during isopr
70 ve electrical, transcutaneous, or sham vagus nerve stimulation and were followed for survival or euth
72 lation, namely deep brain stimulation, vagal nerve stimulation, and transcranial magnetic stimulation
73 al heat, traction, transcutaneous electrical nerve stimulation, and ultrasonography), spinal manipula
75 eep brain stimulation, vagus, and trigeminal nerve stimulation are effective only in a fraction of th
77 spond best to neuromodulation through sacral nerve stimulation are those with a primary disorder of s
78 euromodulation approaches, such as occipital nerve stimulation, are currently being actively studied
79 nerated during different tasks, or following nerve stimulation, are likely to arise from similar mech
80 lso conditioned by medial gastrocnemius (MG) nerve stimulation at C-T intervals ranging from 4 to 7 m
82 espiratory and bulbar paralysis since birth, nerve stimulation at physiologic rates rapidly decrement
83 ssSEPs were elicited by electrical median nerve stimulation at the left and right wrist, using a s
84 e and shape of the EPSPs evoked by olfactory nerve stimulation at the site of origin (glomerular tuft
85 enal sympathetic denervation, cervical vagal nerve stimulation, baroreflex stimulation, cutaneous sti
87 ained by bilateral anterior magnetic phrenic nerve stimulation, body mass index (BMI) z score, fat ma
88 he amplitude of F-EPSPs evoked by splanchnic nerve stimulation but not F-EPSPs evoked by colonic nerv
89 ic fast EPSPs (F-EPSPs) evoked by splanchnic nerve stimulation but not F-EPSPs evoked by colonic nerv
90 he amplitude of F-EPSPs evoked by splanchnic nerve stimulation but not F-EPSPs evoked by colonic nerv
92 creased activations with handgrip and median nerve stimulation, but reduced activations with tibial n
93 but only reduced contractions evoked by 4 Hz nerve stimulation by approximately 40-60% (n = 4-6) and
95 t bilateral autonomic activity (SNS or vagus nerve stimulation) cause reversible shifts of apex-base
96 dly pairing tones with brief pulses of vagus nerve stimulation completely eliminated the physiologica
100 anti-inflammatory therapy via cervical vagus nerve stimulation (cVNS) one should selectively activate
101 on, transcranial magnetic stimulation, vagal nerve stimulation, deep brain stimulation, electroconvul
109 lammatory pathway." Here, we show that vagus nerve stimulation during endotoxemia specifically attenu
112 on, (3) surgical approaches, including vagus nerve stimulation, epidural electrical stimulation, and
115 y the uncoupling agent carbenoxolone reduces nerve stimulation-evoked catecholamine release in contro
116 of beta-NAD in superfusates of muscles after nerve stimulation exceed ATP by at least 30-fold; unlike
117 lease events increases with the frequency of nerve stimulation; external Ca(2+) entry is required for
121 m signal by nucleus isthmi occurs when optic nerve stimulation follows the ipsilateral nucleus isthmi
122 od and Drug Administration approval of vagus nerve stimulation for recurrent treatment-resistant depr
125 the measurement of the tension generated by nerve stimulation gave evidence of any significant impai
126 ic glutamate receptors (mGluRs) by olfactory nerve stimulation generates slow (2 Hz) oscillations nea
127 t-central gyrus at 13.6-17.5 ms after median-nerve stimulation, gradually slowed down in frequency ar
130 patients with depression or epilepsy, vagal nerve stimulation has been demonstrated to promote weigh
134 owing conditioning by high-frequency sciatic nerve stimulation (HFS) at intensities recruiting C-fibe
135 icroM) inhibited contractions evoked by 4 Hz nerve stimulation in a concentration-dependent manner (I
136 determine the safety and efficacy of sacral nerve stimulation in a large population under the rigors
137 ion attenuated heart rate responses to vagal nerve stimulation in all atria (P < 0.05) and normalized
140 ity after both ipsilateral and contralateral nerve stimulation in Aplysia, (2) a smaller and shorter-
141 nced renal vasoconstrictor response to renal nerve stimulation in CHF is a result of an impairment in
143 Similarly, hypercontractile responses to nerve stimulation in H. polygyrus- and N. brasiliensis-i
147 tic force in response to submaximal rates of nerve stimulation in situ producing significantly higher
150 imary sensory cortex face area during median nerve stimulation in subjects with spinal cord injury co
151 There has been growing interest in sacral nerve stimulation in the management of both overactivity
154 cral neuromodulation and percutaneous tibial nerve stimulation in the treatment of men with urge inco
155 ia in vivo and heart rate responses to vagal nerve stimulation in vitro compared to gene transfer of
156 d IL-13 increased smooth muscle responses to nerve stimulation in wild-type mice, but the effects wer
157 ermore, intracavernous pressure increases to nerve stimulation, in vivo, were reduced by 22% in Slo-/
158 in the direction of DOR, but bilateral vagus nerve stimulation increased and reversed DOR to base-->a
160 icotine, an alpha7 agonist that mimics vagus nerve stimulation, increases proinflammatory cytokine pr
166 When hemorrhage occurs in the presence of nerve stimulation (injury), the relative protection of t
169 incipal functions of the bladder by pudendal nerve stimulation is an exciting prospect for neurorehab
170 The renal vasoconstrictor response to renal nerve stimulation is greater in congestive heart failure
174 A phosphorylation in response to sympathetic nerve stimulation, is a macromolecular complex that incl
175 uptake after the trigeminal and hypoglossal nerves stimulation labeled the bilateral hypoglossal mot
177 hypothesize that left-sided low-level vagus nerve stimulation (LL-VNS) can suppress sympathetic outf
178 stimulation (via a transcutaneous electrical nerve stimulation machine to the lateral forefoot) once
179 neuromodulatory procedures such as occipital nerve stimulation may be effective for the most disabled
180 riceps force in response to magnetic femoral nerve stimulation, measured in 39 patients, was also gen
181 stigated 1) the feasibility of a new phrenic nerve stimulation method allowing early diaphragmatic ac
184 30% hemorrhage, 30% hemorrhage plus afferent nerve stimulation, nerve stimulation alone, surgical con
189 interneurons by testing the effect of ulnar nerve stimulation on motor-evoked potentials (MEPs) elic
190 e that the anti-fibrillatory effect of vagus nerve stimulation on the ventricle is mediated by nitric
191 (10 pulses at 100 Hz every 1.5 sec), uniform nerve stimulation (one pulse every 150 msec), and combin
193 be exploited through either electrical vagus nerve stimulation or administration of alpha7 agonists t
194 tal development and, when activated by optic nerve stimulation or visual stimuli, induce sustained de
199 ageing alters MA reactivity to perivascular nerve stimulation (PNS) and adrenoreceptor (AR) activati
200 sympathetic neurotransmission), perivascular nerve stimulation (PNS) evoked dilatation in Young but n
202 t periodic diaphragm contraction via phrenic nerve stimulation (PNS) substantially reduces MV-induced
205 t (up to 72%) in response to 3 Hz repetitive nerve stimulation pointed towards a neuromuscular transm
207 the mechanisms are poorly understood, vagal nerve stimulation prevents weight gain in response to a
208 cral neuromodulation and percutaneous tibial nerve stimulation prove to be viable, durable options fo
209 he long-term efficacy of percutaneous tibial nerve stimulation (PTNS) in fecal incontinence (FI).
212 ect disrupted emotion circuits include vagal nerve stimulation, rapid transcranial magnetic stimulati
214 The response to hemorrhage was modified by nerve stimulation: Reductions in systemic hemodynamics a
216 arrheal and laxative medications, and sacral nerve stimulation) require validation by randomized, con
220 tion of botulinum toxin, percutaneous tibial nerve stimulation, sacral neuromodulation, and surgical
221 By contrast, movements evoked by facial nerve stimulation showed no such frequency-dependent pro
225 oea-predominant or mixed IBS subtypes sacral nerve stimulation (SNS) alleviates IBS-specific symptoms
226 r sinus rhythm, during bilateral sympathetic nerve stimulation (SNS) and right and/or left vagus nerv
227 tudy aimed to evaluate the outcome of sacral nerve stimulation (SNS) for fecal incontinence at 5 year
230 t viral activation of inflammation and vagal nerve stimulation, suggesting a mechanism by which tiotr
231 iac nerve abolishes TNF suppression by vagus nerve stimulation, suggesting that the cholinergic pathw
234 Together, these results establish that vagus nerve stimulation targeting the inflammatory reflex modu
235 al evidence on the feasibility of a proximal nerve stimulation technique in controlling a variety of
236 the capabilities of a non-invasive proximal nerve stimulation technique in eliciting various hand gr
238 eral low-frequency transcutaneous electrical nerve stimulation (TENS) applied on the first dorsal int
240 slight voluntary contraction and electrical nerve stimulation that each electrode recorded motor uni
241 ring normal bursting activity and antidromic nerve stimulation, the conduction delay over the length
244 easing cerebral perfusion, making trigeminal nerve stimulation (TNS) a promising strategy for TBI man
246 e no recommendation on the use of peripheral nerve stimulation to monitor degree of block in patients
247 of direct muscle responses evoked by sciatic nerve stimulation to pretransection levels over an 8-wk
249 ood flow (CBF) fMRI during unilateral median nerve stimulation to show that the poststimulus fMRI sig
253 eters such as sedation scales and peripheral nerve stimulation, use of intermittent therapy when feas
255 ifically, the authors demonstrate that vagus nerve stimulation (VNS) activates the cholinergic antiin
256 al assessed the safety and efficacy of vagal nerve stimulation (VNS) among patients with HF and a red
258 of this study was to determine whether vagus nerve stimulation (VNS) can enhance the consolidation of
264 nity, and modulation of this reflex by vagus nerve stimulation (VNS) is effective in various inflamma
268 to investigate the effect of cervical vagus nerve stimulation (VNS) on cerebral blood flow (CBF), in
269 ot study was to evaluate the effect of Vagus Nerve Stimulation (VNS) paired with sounds in chronic ti
271 We have previously shown that direct vagus nerve stimulation (VNS) reduces the slope of action pote
272 d cardiac response to bipolar cervical vagus nerve stimulation (VNS) reflects a dynamic interaction b
274 gistry investigated whether adjunctive vagus nerve stimulation (VNS) with treatment as usual in depre
279 with either pudendal sensory nerve or pelvic nerve stimulation was examined in the female rat using c
282 Unilateral electrical superior laryngeal nerve stimulation was used to elicit early (R1) and late
283 agmatic twitch pressure generated by phrenic nerve stimulation, was similar in hypogonadal and eugona
284 e cortical, cervicomedullary, and peripheral nerve stimulation we examined in humans motor-evoked pot
286 ne an energy-efficient waveform for cochlear nerve stimulation, we used a genetic algorithm approach,
288 interval changes in response to right vagal nerve stimulation were also enhanced by exercise in wild
289 ded (i) handgrip; and (ii) median and tibial nerve stimulation were assessed using functional magneti
290 osensitive rVLM neurons evoked by splanchnic nerve stimulation were reduced by EA and then restored w
291 z; 200 ms duration) via supramaximal sciatic nerve stimulation were used to manipulate metabolic rate
292 f release, indicated by a response to single nerve stimulation, whereas Shab channels regulate repeti
294 xcitability by combining paired-pulse median nerve stimulation with recording somatosensory evoked po
295 6) We suggest against the use of peripheral nerve stimulation with train of four alone for monitorin
298 tion frequency-dependent manner during vagus nerve stimulation, with comparable increases seen during
299 concurrent BOLD and CBF responses to median nerve stimulation, with primary signal increases in cont
300 failed, or been denied access to, occipital nerve stimulation within the UK's National Health Servic
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