ライフサイエンスコーパス: nervosa

1 a and higher in women recovered from bulimia nervosa).

2 ium effect for ED, large effect for anorexia nervosa).

3 patients with, or recovering from, anorexia nervosa.

4 eir first admission to hospital for anorexia nervosa.

5 nd may be efficacious for youth with bulimia nervosa.

6 n anorexia nervosa and overeating in bulimia nervosa.

7 bulimia nervosa with no history of anorexia nervosa.

8 ing raise the risk for mortality in anorexia nervosa.

9 ental delay, autism, psychosis, and anorexia nervosa.

10 nervosa and promising for adolescent bulimia nervosa.

11 avoid weight gain that characterize bulimia nervosa.

12 flict resolution in adolescents with bulimia nervosa.

13 ty risks, similar to those found in anorexia nervosa.

14 linked to suppression of eating and anorexia nervosa.

15 ng the subtypes anorexia nervosa and bulimia nervosa.

16 ischarge criteria for patients with anorexia nervosa.

17 vosa were unlikely to cross over to anorexia nervosa.

18 desensitized dopaminergic system in bulimia nervosa.

19 osa but were likely to relapse into anorexia nervosa.

20 on the treatment of adolescents with bulimia nervosa.

21 ne how purging disorder differs from bulimia nervosa.

22 sk of premature death in those with anorexia nervosa.

23 disorders, particularly those with anorexia nervosa.

24 e evidence for any rise in rates of anorexia nervosa.

25 ders, including anorexia nervosa and bulimia nervosa.

26 neural reward-effort integration in bulimia nervosa.

27 ive diseases, obesity, bulimia, and anorexia nervosa.

28 eatment of eating disorders such as anorexia nervosa.

29 es (0.30%), and 61 males (0.01%) had bulimia nervosa.

30 were found for anorexia nervosa and bulimia nervosa.

31 eight and duration of amenorrhea in anorexia nervosa.

32 as a risk factor for arrhythmias in anorexia nervosa.

33 ge 1 and the Genetic Consortium for Anorexia Nervosa.

34 c dysfunction in the pathogenesis of bulimia nervosa.

35 ression, and the psychopathology of anorexia nervosa.

36 of psychoanalytic psychotherapy for bulimia nervosa.

37 al therapy (CBT) in the treatment of bulimia nervosa.

38 y, dieting-induced weight gain, and anorexia nervosa.

39 mong women might be up to 4%, and of bulimia nervosa 2%.

40 Crude mortality rates were 4.0% for anorexia nervosa, 3.9% for bulimia nervosa, and 5.2% for eating d

41 itive dose-response association with bulimia nervosa (adjusted hazard ratio = 1.15, 95% confidence in

42 Anorexia nervosa affects 1-4% of women in United States and is th

43 white matter fiber organization in anorexia nervosa after recovery could indicate a biological marke

44 Anorexia nervosa also has large and significant genetic correlati

45 The lifetime prevalence of DSM-5 anorexia nervosa among women might be up to 4%, and of bulimia ne

46 steroid metabolism in subjects with anorexia nervosa (AN) after weight gain have not been elucidated.

47 Patients with anorexia nervosa (AN) and obesity (OB) were investigated in refer

48 Both anorexia nervosa (AN) and obesity are complicated by affective co

49 Anorexia nervosa (AN) and related eating disorders are complex, m

50 ity to reward, yet individuals with anorexia nervosa (AN) are not motivated to eat when starved.

51 rsuit of thinness, individuals with anorexia nervosa (AN) engage in maladaptive behaviors (restrictiv

52 Individuals with anorexia nervosa (AN) engage in relentless restrictive eating and

53 eight is disrupted in patients with anorexia nervosa (AN) for prolonged periods of time.

54 Anorexia nervosa (AN) is a complex and heritable eating disorder

55 Anorexia nervosa (AN) is a complex neuropsychiatric disorder pres

56 Anorexia nervosa (AN) is a condition of severe undernutrition ass

57 Anorexia nervosa (AN) is a devastating psychiatric illness that i

58 Anorexia nervosa (AN) is a serious disorder with high rates of mo

59 Anorexia nervosa (AN) is a serious eating disorder characterized

60 Anorexia nervosa (AN) is a serious eating disorder characterized

61 Anorexia nervosa (AN) is a serious mental illness categorized by

62 Anorexia nervosa (AN) is a serious mental illness with marked mor

63 Anorexia nervosa (AN) is a serious psychiatric illness associated

64 Anorexia nervosa (AN) is a severe psychiatric disorder associated

65 Anorexia nervosa (AN) is an eating disorder characterized by extr

66 Anorexia Nervosa (AN) is characterized by Diagnostic and Statisti

67 Anorexia nervosa (AN) is characterized by extremely low body weig

68 Recovery from osteoporosis in anorexia nervosa (AN) is uncertain.

69 included in the dataset as follows: Anorexia Nervosa (AN) n = 171; Bulimia Nervosa (BN) n = 82; Recov

70 Is starvation in anorexia nervosa (AN) or overeating in bulimia nervosa (BN) a for

71 Individuals with anorexia nervosa (AN) override the drive to eat, forgoing immedia

72 been carried out on iron status in anorexia nervosa (AN) patients.

73 Individuals with anorexia nervosa (AN) restrict eating and become emaciated.

74 me-wide association study (GWAS) of anorexia nervosa (AN) using a stringently defined phenotype.

75 e is a common time for the onset of anorexia nervosa (AN), a condition associated with long-term medi

76 suggested in the pathophysiology of anorexia nervosa (AN), but the role of the endogenous mediators o

77 ations for eating disorders such as anorexia nervosa (AN), in which there is a high prevalence of anx

78 tion has previously been studied in anorexia nervosa (AN), its influence in women with AN on eating d

79 In anorexia nervosa (AN), taste and smell are believed to be anhedon

80 one of the core characteristics of anorexia nervosa (AN), the exact nature of this complex feature i

81 ex eating-related behaviors seen in anorexia nervosa (AN).

82 to inpatient medical treatment for anorexia nervosa (AN).

83 e distortions are a core feature of anorexia nervosa (AN).

84 d genetic correlations (rg) between anorexia nervosa and 159 other phenotypes.

85 sponse in 18 female adolescents with bulimia nervosa and 18 healthy female age-matched subjects durin

86 4.37 (95% CI=2.4-7.3) for lifetime anorexia nervosa and 2.33 (95% CI=0.3-8.4) for bulimia nervosa wi

87 one adolescent female patients with anorexia nervosa and 45 age- and sex-matched healthy volunteers p

88 the most established treatments for bulimia nervosa and binge eating disorder, with stepped-care app

89 and food group intakes of women with bulimia nervosa and binge-eating disorder during pregnancy and c

90 correlations were observed between anorexia nervosa and body mass index, insulin, glucose, and lipid

91 interviewed to assess diagnoses of anorexia nervosa and bulimia nervosa (per DSM-IV and broad criter

92 el, 5-year recovery rates for DSM-5 anorexia nervosa and bulimia nervosa are 69 and 55%, respectively

93 Both anorexia nervosa and bulimia nervosa are associated with increase

94 Anorexia nervosa and bulimia nervosa are common and severe eating

95 ort the longitudinal distinction of anorexia nervosa and bulimia nervosa but do not support the anore

96 lumes were reduced in the recovered anorexia nervosa and bulimia nervosa groups and predicted sensiti

97 Course and outcome studies of both anorexia nervosa and bulimia nervosa show that no significant dif

98 gnostic crossover longitudinally in anorexia nervosa and bulimia nervosa to inform the validity of th

99 Participants who had recovered from anorexia nervosa and bulimia nervosa were studied to avoid confou

100 DNOS, by lowering the threshold for anorexia nervosa and bulimia nervosa, and adding BED as a specifi

101 ected by the diagnostic criteria of anorexia nervosa and bulimia nervosa, which emphasize intrapsychi

102 with an ED, including the subtypes anorexia nervosa and bulimia nervosa.

103 ts with eating disorders, including anorexia nervosa and bulimia nervosa.

104 Similar patterns were found for anorexia nervosa and bulimia nervosa.

105 clarifications and modifications to anorexia nervosa and bulimia nervosa; and the inclusion of binge

106 a genome-wide association study of anorexia nervosa and calculated genetic correlations with a serie

107 ress compared with participants with bulimia nervosa and controls.

108 on affects the risk for anorexia and bulimia nervosa and disordered eating in members of opposite-sex

109 d mortality ratios were elevated for bulimia nervosa and eating disorder not otherwise specified.

110 tios were significantly elevated for bulimia nervosa and eating disorder not otherwise specified; sui

111 had recovered from restricting-type anorexia nervosa and had 1 year of normal weight and regular mens

112 roup (lower in women recovered from anorexia nervosa and higher in women recovered from bulimia nervo

113 re on the development and course of anorexia nervosa and interpreted critical features in light of de

114 stablished treatment for youth with anorexia nervosa and may be efficacious for youth with bulimia ne

115 less likely to exhibit anorexia and bulimia nervosa and more likely to exhibit binge eating disorder

116 f the lumbar spine in patients with anorexia nervosa and normal-weight control subjects and to determ

117 Women with anorexia nervosa and normal-weight control subjects were compared

118 o significantly associated SNPs for anorexia nervosa and only three for educational attainment.

119 contribute to restricted eating in anorexia nervosa and overeating in bulimia nervosa.

120 : stabilization of the incidence of anorexia nervosa and possibly lower incidence rates of bulimia ne

121 first-line treatment for adolescent anorexia nervosa and promising for adolescent bulimia nervosa.

122 rch on molecular genetic studies of anorexia nervosa and provide guidance for future research.

123 increased on the right side in the anorexia nervosa and recovered anorexia nervosa groups and on the

124 nclude genetic correlations between anorexia nervosa and schizophrenia, anorexia and obesity, and edu

125 correlations were observed between anorexia nervosa and schizophrenia, neuroticism, educational atta

126 s are engaged in the development of anorexia nervosa and that stimulus-response learning (that is, ha

127 against schizophrenia, autism, and anorexia nervosa and that these variants may be maintained by new

128 s (0.70%) and 453 males (0.04%) had anorexia nervosa, and 3349 females (0.30%), and 61 males (0.01%)

129 4.0% for anorexia nervosa, 3.9% for bulimia nervosa, and 5.2% for eating disorder not otherwise spec

130 e threshold for anorexia nervosa and bulimia nervosa, and adding BED as a specified eating disorder.

131 lence estimates of anorexia nervosa, bulimia nervosa, and binge-eating disorder were 0.3%, 0.9%, and

132 y 3 variables (any eating disorder, anorexia nervosa, and bulimia nervosa) identified by any lifetime

133 determine whether anorexia nervosa, bulimia nervosa, and eating disorder not otherwise specified are

134 followed them for anorexia nervosa, bulimia nervosa, and eating disorder not otherwise specified unt

135 ass procedures, chronic alcoholics, anorexia nervosa, and restrictive diets.

136 odifications to anorexia nervosa and bulimia nervosa; and the inclusion of binge eating disorder as a

137 rates for DSM-5 anorexia nervosa and bulimia nervosa are 69 and 55%, respectively; little is known ab

138 Both anorexia nervosa and bulimia nervosa are associated with increased mortality.

139 anosis is common, and patients with anorexia nervosa are at risk of various arrhythmias.

140 Anorexia nervosa and bulimia nervosa are common and severe eating disorders (EDs) of

141 eatment trials for adolescents with anorexia nervosa are few.

142 Individuals with anorexia nervosa are known to be ascetic and able to sustain self

143 reatment trials for adolescents with bulimia nervosa are largely absent.

144 dieting behavior characteristic of anorexia nervosa as a well-entrenched habit provides a basis for

145 the same degree in adolescents with bulimia nervosa as in healthy comparison subjects.

146 ivity disorder, alcohol dependence, anorexia nervosa, autism spectrum disorder, bipolar disorder, maj

147 ted eating and weight loss occur in anorexia nervosa because of a failure to accurately recognize hun

148 Women with bulimia nervosa before and during pregnancy and those with binge

149 ew categories are: anorexia nervosa, bulimia nervosa, binge eating disorder, and eating disorder not

150 behaviors (i.e., bulimia nervosa or anorexia nervosa, binge eating/purging type);14 with anorexia ner

151 orexia nervosa (AN) or overeating in bulimia nervosa (BN) a form of addiction?

152 are presented here and compared with bulimia nervosa (BN) data in the World Health Organization (WHO)

153 Bulimia nervosa (BN) has been associated with dysregulation of t

154 lows: Anorexia Nervosa (AN) n = 171; Bulimia Nervosa (BN) n = 82; Recovered AN n = 90; Healthy contro

155 cuits in adolescents and adults with bulimia nervosa (BN), but less is known about the microstructure

156 scent and adult female subjects with bulimia nervosa (BN).

157 Lifetime prevalence estimates of anorexia nervosa, bulimia nervosa, and binge-eating disorder were

158 this study was to determine whether anorexia nervosa, bulimia nervosa, and eating disorder not otherw

159 in 1975-1998 and followed them for anorexia nervosa, bulimia nervosa, and eating disorder not otherw

160 e probable four new categories are: anorexia nervosa, bulimia nervosa, binge eating disorder, and eat

161 d little evidence that the risk for anorexia nervosa, bulimia nervosa, or disordered eating was assoc

162 distinction of anorexia nervosa and bulimia nervosa but do not support the anorexia nervosa subtypin

163 over time; one-third crossed over to bulimia nervosa but were likely to relapse into anorexia nervosa

164 ly-to-severely ill adolescents with anorexia nervosa, but it is costly, and the risks of relapse and

165 in which the marked persistence of anorexia nervosa can be usefully understood as a well-ingrained m

166 se-control cohorts comprising 3,495 anorexia nervosa cases and 10,982 controls, the authors performed

167 brain reward learning in adolescent anorexia nervosa changes with weight restoration.

168 rticipants with purging disorder and bulimia nervosa compared with controls but did not differ betwee

169 unger signals, whereas overeating in bulimia nervosa could represent an exaggerated perception of hun

170 s with chronic treatment-refractory anorexia nervosa, DBS is well tolerated and is associated with si

171 y; the prevalence of DSM-IV or broad bulimia nervosa did not differ in women from opposite- versus sa

172 o resolve conflict, adolescents with bulimia nervosa displayed abnormal patterns of activation in fro

173 Patients with anorexia nervosa exhibit abnormal myocardial repolarization and a

174 7 years, the majority of women with anorexia nervosa experienced diagnostic crossover: more than half

175 e in the treatment of patients with anorexia nervosa following weight restoration.

176 I=3.2-12.1) for those with lifetime anorexia nervosa for >15 to 30 years (10/67 died).

177 0.9-8.3) for patients with lifetime anorexia nervosa for 0 to 15 years (4/119 died), and 6.6 (95% CI=

178 le patients (aged 11-18 years) with anorexia nervosa from six centres in Germany.

179 n 32 weight-recovered subjects with anorexia nervosa from the New York site of the Fluoxetine to Prev

180 ompared with the control group, the anorexia nervosa group exhibited greater brain response 1) for pr

181 a groups and on the left side in the bulimia nervosa group relative to the healthy comparison group.

182 the anorexia nervosa and recovered anorexia nervosa groups and on the left side in the bulimia nervo

183 n the recovered anorexia nervosa and bulimia nervosa groups and predicted sensitivity to reward in al

184 Results Women with anorexia nervosa had higher skewness and kurtosis, lower MPP (P <

185 Conclusion Patients with anorexia nervosa had increased skewness and kurtosis and decrease

186 Although anorexia nervosa has a high mortality rate, our understanding of

187 As bulimia nervosa has been associated with a dysregulated dopamine

188 Anorexia nervosa has been consistently associated with increased

189 Anorexia nervosa has the highest mortality rate of any psychiatri

190 s hypothesize that individuals with anorexia nervosa have an imbalance in information processing, wit

191 l large population-based studies of anorexia nervosa have been conducted in twins; it is possible tha

192 evalence, incidence, and outcome of anorexia nervosa have been limited to cases detected through the

193 ting disorder, anorexia nervosa, and bulimia nervosa) identified by any lifetime diagnoses recorded i

194 with clinical outcome in women with anorexia nervosa in a relapse-prevention trial.

195 efulness of family interventions for bulimia nervosa in adolescents.

196 evidence base for effectiveness for anorexia nervosa in adolescents.

197 nd possibly lower incidence rates of bulimia nervosa in Caucasian North American and Northern Europea

198 increasing rates of binge eating and bulimia nervosa in Hispanic and Black American minority groups i

199 had an independent association with anorexia nervosa in males.

200 subtype predicted fatal outcome for anorexia nervosa in males.

201 Bulimia nervosa in men was too rare to be assessed by zygosity;

202 describe the onset and outcomes of anorexia nervosa in the general population.

203 sses two trials of individuals with anorexia nervosa in which deep brain stimulation of different bra

204 The incidence of anorexia nervosa in women between 15 and 19 years of age was 270

205 Anorexia nervosa is a complex heritable phenotype for which this

206 Anorexia nervosa is a life-threatening complex psychiatric disord

207 Anorexia nervosa is a life-threatening illness.

208 Anorexia nervosa is a perplexing illness marked by low body weigh

209 Anorexia nervosa is a psychiatric disorder of unknown etiology.

210 Anorexia nervosa is a severe psychiatric disorder associated with

211 Anorexia nervosa is a well characterized disorder with remarkable

212 Anorexia nervosa is an important cause of physical and psychosoci

213 A core feature of anorexia nervosa is an over-estimation of body size.

214 Anorexia nervosa is prevalent in adolescents and young adults, an

215 recently weight-restored women with anorexia nervosa, lower percent body fat was associated with poor

216 Individuals who have recovered from anorexia nervosa may have difficulties in differentiating positiv

217 ng the anorexia nervosa subtypes and bulimia nervosa may reflect problems with the validity of the cu

218 The study included 30 women with anorexia nervosa (mean age +/- standard deviation, 26 years +/- 6

219 depression, suicide, schizophrenia, anorexia nervosa, migraine, dementia, and PD.

220 women (N=14), women recovered from anorexia nervosa (N=14) had significantly diminished and women re

221 diminished and women recovered from bulimia nervosa (N=14) had significantly elevated hemodynamic re

222 es of binge eating disorder (N=131), bulimia nervosa (N=17), and anorexia nervosa (N=18).

223 25 years in 1,885 individuals with anorexia nervosa (N=177), bulimia nervosa (N=906), or eating diso

224 N=131), bulimia nervosa (N=17), and anorexia nervosa (N=18).

225 anorexia nervosa (N=24), women with bulimia nervosa (N=19), and healthy comparison women (N=24).

226 women with current restricting-type anorexia nervosa (N=19), women recovered from restricting-type an

227 METHOD: Female adolescents with anorexia nervosa (N=21; mean age, 16.4 years [SD=1.9]) underwent

228 men recovered from restricting-type anorexia nervosa (N=24), women with bulimia nervosa (N=19), and h

229 duals with anorexia nervosa (N=177), bulimia nervosa (N=906), or eating disorder not otherwise specif

230 aths (6.5%) were recorded (lifetime anorexia nervosa, N=14; bulimia nervosa with no history of anorex

231 bulimia nervosa with no history of anorexia nervosa, N=2).

232 For adults with anorexia nervosa, no one specialist treatment has been shown to b

233 ing that appears to be distinct from bulimia nervosa on subjective and physiological responses to a t

234 rrelation between the Eyes Test and anorexia nervosa, openness (NEO-Five Factor Inventory), and diffe

235 eating and purging behaviors (i.e., bulimia nervosa or anorexia nervosa, binge eating/purging type);

236 dies of adolescents and adults with anorexia nervosa or bulimia nervosa that, together, implicate dys

237 al of 216 women with a diagnosis of anorexia nervosa or bulimia nervosa were followed for 7 years; we

238 atment-seeking female patients with anorexia nervosa or bulimia nervosa were interviewed every 6 mont

239 Eighty-five adolescents with bulimia nervosa or eating disorder not otherwise specified were

240 guided self-care in adolescents with bulimia nervosa or eating disorder not otherwise specified.

241 domly assigned to family therapy for bulimia nervosa or individual CBT guided self-care supported by

242 In humans with anorexia nervosa or kwashiorkor, ghrelin and growth hormone are k

243 , 0.8% had partial- or full-criteria bulimia nervosa or purging disorder and 2.9% had partial or full

244 that the risk for anorexia nervosa, bulimia nervosa, or disordered eating was associated with zygosi

245 death among patients with lifetime anorexia nervosa peaked within the first 10 years of follow-up, r

246 ss diagnoses of anorexia nervosa and bulimia nervosa (per DSM-IV and broad criteria).

247 Adolescents with and without bulimia nervosa performed similarly on the task.

248 ularly for adolescent patients with anorexia nervosa, point to the benefits of specialised family-bas

249 Of the five male anorexia nervosa probands, only one was from an opposite-sex twin

250 conducted in which 70 patients with bulimia nervosa received either 2 years of weekly psychoanalytic

251 00 until May 2005, 93 patients with anorexia nervosa received intensive inpatient or day-program trea

252 cerebral glucose metabolism in key anorexia nervosa-related structures at both 6 months and 12 month

253 The pathophysiology of anorexia nervosa remains obscure, but structural brain alteration

254 Patients with anorexia nervosa reported higher levels of perceived coercion and

255 20-60 years and had a diagnosis of anorexia nervosa (restricting or binge-purging subtype) and a dem

256 binge eating/purging type);14 with anorexia nervosa, restricting type; and 13 healthy comparison sub

257 Research in Anorexia Nervosa (RIAN) is a 2-group (FBT and SyFT) randomized tr

258 have consistently demonstrated that anorexia nervosa runs in families.

259 -based interventions for addiction, anorexia nervosa, schizophrenia, and depression.

260 dolescent patients with non-chronic anorexia nervosa seems no less effective than IP for weight resto

261 studies of both anorexia nervosa and bulimia nervosa show that no significant differences exist betwe

262 anxiety, affective regulation, and anorexia nervosa-specific behaviours at 12 months after surgery,

263 a combination of re-nourishment and anorexia nervosa-specific psychotherapy is most effective.

264 r BMI at admission, and restrictive anorexia nervosa subtype predicted fatal outcome for anorexia ner

265 percent body fat, body mass index, anorexia nervosa subtype, waist-to-hip ratio, and serum cortisol

266 of diagnostic "crossover" among the anorexia nervosa subtypes and bulimia nervosa may reflect problem

267 estricting and binge eating/purging anorexia nervosa subtypes over time; one-third crossed over to bu

268 imia nervosa but do not support the anorexia nervosa subtyping schema.

269 Similar results were found for anorexia nervosa (suicide attempts: crude, 4.42 [4.12-4.74] vs ad

270 ] vs adjusted, 2.67 [1.78-4.01]) and bulimia nervosa (suicide attempts: crude, 6.26 [5.73-6.85] vs ad

271 Brain circuits believed to drive anorexia nervosa symptoms can be accessed with surgical technique

272 ease compared with participants with bulimia nervosa (t(76.44) = 2.51; P = .01) and did not differ si

273 ifetime prevalence and incidence of anorexia nervosa than reported in previous studies, most of which

274 represent a phenotype of adolescent anorexia nervosa that does not respond well to treatment.

275 te information about bone health in anorexia nervosa that is independent of that provided with bone m

276 and adults with anorexia nervosa or bulimia nervosa that, together, implicate dysregulation of front

277 ltered striatal dopamine binding in anorexia nervosa, the authors sought to assess the response of th

278 eat is crucial for survival, but in anorexia nervosa, the brain persistently supports reduced food in

279 ward-processing deficits in remitted bulimia nervosa, the purpose of this study was to identify the r

280 ngitudinally in anorexia nervosa and bulimia nervosa to inform the validity of the DSM-IV-TR eating d

281 del helps explain the resistance of anorexia nervosa to interventions that have established efficacy

282 Adults with anorexia nervosa too have a realistic chance of achieving recover

283 ne to Prevent Relapse in Women With Anorexia Nervosa trial.

284 The h(2)SNP of anorexia nervosa was 0.20 (SE=0.02), suggesting that a substantia

285 The lifetime prevalence of DSM-IV anorexia nervosa was 2.2%, and half of the cases had not been det

286 Anorexia nervosa was independently predicted by multiple birth (a

287 Anorexia nervosa was inversely associated with the development of

288 , the prevalence of DSM-IV or broad anorexia nervosa was not significantly different than that of wom

289 Historically, anorexia nervosa was viewed as a disorder primarily influenced by

290 rates, and 5-year recovery rates of anorexia nervosa were calculated on the basis of data from 2,881

291 patients with treatment-refractory anorexia nervosa were enrolled between September, 2011, and Janua

292 h a diagnosis of anorexia nervosa or bulimia nervosa were followed for 7 years; weekly eating disorde

293 le patients with anorexia nervosa or bulimia nervosa were interviewed every 6 months for a median of

294 recovered from anorexia nervosa and bulimia nervosa were studied to avoid confounding effects of alt

295 Women with bulimia nervosa were unlikely to cross over to anorexia nervosa.

296 nsiveness is elevated in adolescent anorexia nervosa when underweight and after weight restoration.

297 tic criteria of anorexia nervosa and bulimia nervosa, which emphasize intrapsychic conflicts such as

298 o understanding the neurobiology of anorexia nervosa, which still remains a mystery and poses a chall

299 authors found that individuals with anorexia nervosa, who make maladaptive food choices to the point

300 ed (lifetime anorexia nervosa, N=14; bulimia nervosa with no history of anorexia nervosa, N=2).

301 ervosa and 2.33 (95% CI=0.3-8.4) for bulimia nervosa with no history of anorexia nervosa.