ライフサイエンスコーパス: nervous system

1 gularity of neuronal activity in the central nervous system.

2 Little is known about its role in the nervous system.

3 minating to colonize the purine-poor central nervous system.

4 the embryonic epidermis to give rise to the nervous system.

5 rain, only isoform-A has been studied in the nervous system.

6 sitive, and selective communication with the nervous system.

7 acity of this agent to penetrate the central nervous system.

8 nsity for metastasis to bone and the central nervous system.

9 tassium channels abundantly expressed in the nervous system.

10 d blood cells is a surrogate for AChE in the nervous system.

11 mutant Ras-induced tumors in the developing nervous system.

12 bition of acetylcholinesterase (AChE) in the nervous system.

13 (MS) is an autoimmune disease of the central nervous system.

14 the role of O-GlcNAc cycling in the central nervous system.

15 ys and, perhaps more generally, their entire nervous system.

16 are also essential for the plasticity of the nervous system.

17 mit axon regeneration in the adult mammalian nervous system.

18 ading to suboptimal treatment in the central nervous system.

19 s following early development of the central nervous system.

20 mission throughout the developing and mature nervous system.

21 rocessing and motor rhythm generation in the nervous system.

22 t the genetic ablation of Lpd to the central nervous system.

23 ate and have well-characterized roles in the nervous system.

24 n result in damage to the developing central nervous system.

25 phalitogenic effector T cells in the central nervous system.

26 etylcholine receptors present in the central nervous system.

27 le fetal and neonatal tissues, including the nervous system.

28 vere congenital malformations of the central nervous system.

29 ative and autoimmune diseases of the central nervous system.

30 tic information from the gut directly to the nervous system.

31 ffects of metabolic disorders on the central nervous system.

32 stating consequences for the function of the nervous system.

33 n of virally transduced cells in the central nervous system.

34 n changes in the developing Drosophila pupal nervous system.

35 ning spatial pattern preferences in a simple nervous system.

36 es essential roles in the functioning of the nervous system.

37 l trials in a wide range of disorders of the nervous system.

38 munication between the immune system and the nervous system.

39 t require microglial function in the central nervous system.

40 ory neurotransmission throughout the central nervous system.

41 receptor abundantly expressed in the central nervous system.

42 lammatory effects, as well as to protect the nervous system.

43 lish precise synaptic connections within the nervous system.

44 for this inflammatory protein in the central nervous system.

45 eukin-8 in the respiratory tract and central nervous system.

46 r organisms to information processing in the nervous system.

47 iple roles in the development of the central nervous system.

48 ation of hepatic functions via the autonomic nervous system.

49 ic composition and function in the mammalian nervous system.

50 kinase in preserving genome stability in the nervous system.

51 oligodendroglial protein in the rat central nervous system.

52 he intrinsic immune sentinels of the central nervous system.

53 xhibits high viral burden within the central nervous system.

54 itch-specific labeled line in the peripheral nervous system.

55 eptide present in the CNS and the peripheral nervous system.

56 rotonin interact at the level of the central nervous system.

57 ring comprehensive genome maintenance in the nervous system.

58 amage and BACE1 up-regulation in the central nervous system.

59 nt and maintenance of central and peripheral nervous systems.

60 of interest in and research into cnidarians nervous systems.

61 ystems, including the immune, metabolic, and nervous systems.

62 are found in both the central and autonomic nervous systems.

63 s, presence of microcephaly or other central nervous system abnormalities, and timing of infection in

64 s fundamental properties with the vertebrate nervous system: action potentials, synaptic transmission

65 myelin development proceeds independently of nervous system activity, increasing evidence supports a

66 Editing is enriched in the nervous system, affecting molecules pertinent for excita

67 appropriate function can be restored to the nervous system after the critical period.

68 tions of the sympathetic and parasympathetic nervous systems, along with potential non-autonomic mech

69 sor and a principal component of SOCE in the nervous system, altered the expression of 131 genes incl

70 bies virus (PRV), that infect the peripheral nervous system and have to travel long distances along a

71 ation between spatially distant areas of the nervous system and in controlling motor circuits.

72 elationship to activation of the sympathetic nervous system and inflammation in 59 patients with cirr

73 ld help reveal fundamental properties of the nervous system and neurological diseases.

74 WHO classification of tumours of the central nervous system and on scientific developments since the

75 CL than in DLBCL located outside the central nervous system and ophthalmic regions.

76 nfluence the development and function of the nervous system and several complex host behaviors.

77 l function are compromised in the Drosophila nervous system and SRS patient cells.

78 d neurohormonal signaling of the sympathetic nervous system and the downstream ET system, respectivel

79 focuses on the etiology of DNA damage in the nervous system and the genome stability pathways that pr

80 from the extracellular fluid in the central nervous system and thus presents an essential obstacle t

81 amine neurotransmitters of the human central nervous system, and is involved in many behavioral respo

82 mation, medication use, tumor of the central nervous system, and no diagnosis.

83 atic vessels, including those in the central nervous system, and Schlemm's canal.

84 y conserved, widely expressed outside of the nervous system, and the target of many environmental che

85 ssing biologically active C3a in the central nervous system, and their respective wild-type controls

86 In the central nervous system, apoE is produced primarily by astrocytes

87 f progenitor cells in the developing enteric nervous system are controlled by molecules such as the s

88 Many networks throughout the nervous system are organized into topographic maps, wher

89 size and the number of neurons in the human nervous system, as well as the cellular and molecular re

90 ustly expressed in the developing and mature nervous system, but its contributions to neural cell cir

91 OGT is enriched in the nervous system, but little is known about its importance

92 read of pathogenic proteins in the mammalian nervous system, but whether nonpathogenic ones spread is

93 Ultrasonography of the central nervous system can reveal inflammatory changes and devel

94 d leukemia, soft-tissue sarcoma, and central nervous system cancer.

95 h incidence of severe defects in the central nervous system caused by human cytomegalovirus (HCMV) co

96 bundant and evolutionarily conserved central nervous system cell type.

97 eous neurotransmission have been observed in nervous system circuits as long as methods have existed

98 of Salmonella to disseminate to the central nervous system (CNS) after oral infection in C57BL/6J mi

99 ry environment is induced within the central nervous system (CNS) after WNV infection, leading to ent

100 e mechanisms by which WNV enters the central nervous system (CNS) and host-factors that are involved

101 The central nervous system (CNS) and its meningeal coverings accommo

102 nce of parallel degeneration of both central nervous system (CNS) and peripheral nervous system (PNS)

103 Infections of the central nervous system (CNS) are often acute, with significant m

104 , we sought to determine whether the central nervous system (CNS) can be infected by KSHV in HIV-posi

105 Tumors residing in the central nervous system (CNS) compromise the blood-brain barrier

106 ene expression that occur across the central nervous system (CNS) during neurological diseases do not

107 nonchordates, amphioxus develops its central nervous system (CNS) from a neural plate that is homolog

108 eous, static magnetic field (SMF) on Central Nervous System (CNS) glial cells are less investigated.

109 ssue-resident macrophages within the central nervous system (CNS) have essential roles in neural deve

110 anding of immune surveillance of the central nervous system (CNS) have repeatedly provoked dismissal

111 Microglia play essential roles in central nervous system (CNS) homeostasis and influence diverse a

112 Central nervous system (CNS) infection and neurological involvem

113 ue to inflammation are a hallmark of central nervous system (CNS) infections with neurotropic pathoge

114 valent health issue that can lead to central nervous system (CNS) inflammation with long-term behavio

115 elected cohort with mainly relapsing central nervous system (CNS) inflammatory diseases.

116 tection devices in reducing ischemic central nervous system (CNS) injury during SAVR.

117 r neuroinflammatory responses during central nervous system (CNS) invasion by trypanosomes and are as

118 chain reaction (PCR) is a marker of central nervous system (CNS) involvement in congenital hCMV infe

119 acute lymphoblastic leukemia (ALL), central nervous system (CNS) involvement is a major clinical con

120 Viral infection of the central nervous system (CNS) is complicated by the mostly irrepl

121 ificantly associated with ocular and central nervous system (CNS) lesions and showed the strongest as

122 Primary central nervous system (CNS) lymphoma (PCNSL) and primary testic

123 gene, Sex-lethal (Sxl), functions in central nervous system (CNS) neurons as part of a relay that spe

124 gnosis is difficult with concomitant central nervous system (CNS) pathology.

125 induced characteristics of the adult central nervous system (CNS) pose barriers to axonal regeneratio

126 Central nervous system (CNS) relapses are an uncommon yet devast

127 d with the development of a manifest central nervous system (CNS) synucleinopathy (odds ratio = 7.1).

128 croglial cells are phagocytes in the central nervous system (CNS) that become activated in pathologic

129 signal to the motor circuits of the central nervous system (CNS) through a series of pathways that i

130 Survivors of central nervous system (CNS) tumors (SHR=4.6, 95% confidence int

131 In the adult mammalian central nervous system (CNS), axonal damage often triggers neuro

132 ctively affects motor neurons in the central nervous system (CNS), causing the adult-onset degenerati

133 e-matter tracts throughout the human central nervous system (CNS), including loss of all commissural

134 s on the input and plasticity of the central nervous system (CNS), which may explain the absence of a

135 The discovery that central nervous system (CNS)-targeted autoreactive T cells requi

136 Notably, the first new central nervous system (CNS)-targeted oligonucleotide-based drug

137 ory cell recruitment at the diseased central nervous system (CNS).

138 defined demyelinating disease of the central nervous system (CNS).

139 ly 30% of the cells in the mammalian central nervous system (CNS).

140 major obstacle for tissue repair in central nervous system (CNS).

141 ally assumed to be a function of the central nervous system (CNS).

142 sential protection mechanisms in the central nervous system (CNS).

143 e disease that targets myelin in the central nervous system (CNS).

144 utes to the neuroinflammation in the central nervous system (CNS).

145 or hyperpolarizing inhibition in the central nervous system (CNS).

146 ntrol autoimmune inflammation in the central nervous system (CNS).

147 including those associated with the central nervous system (CNS).

148 s for a number of cardiovascular and central nervous system conditions, but the current drugs for the

149 RATIONALE: Sympathetic nervous system control of inflammation plays a central r

150 hways between gut microbiota and the central nervous system could include autonomic, neuroendocrine,

151 f the inflammasome in peripheral and central nervous system cytokine/chemokine inflammatory responses

152 econdary to central and, perhaps, peripheral nervous system damage.

153 (SHANK3, CDH10, CSMD1) or genes involved in nervous system development (NYAP2, ST6GAL2, GRM6).

154 7-RGS complexes regulate critical aspects of nervous system development and function.

155 d in neuron-glia interactions during central nervous system development and in hair follicle polarity

156 y is encoded and robustly established during nervous system development.

157 arrhythmia in females when acute sympathetic nervous system discharge was applied in the settings of

158 mia risk in the setting of acute sympathetic nervous system discharge.

159 vestigation included cancer (n = 31; 23.1%), nervous system diseases (n = 26; 19.4%), and injury and

160 ors between those three inflammatory central nervous system diseases in adults and children to suppor

161 ected with human metabolic diseases, central nervous system diseases, and neoplasms.

162 of neuroinflammation in a variety of central nervous system diseases.

163 ial actions may advance our understanding of nervous system disorders and suggest strategies for the

164 hat encompassed the development of autonomic nervous system dysfunction and hypertension.

165 l stem and progenitor cells from the enteric nervous system (ENS) might serve as a source of cells fo

166 The enteric nervous system (ENS) of the gastrointestinal tract contr

167 The enteric nervous system (ENS) regulates numerous gastrointestinal

168 licing of C-Src in the developing vertebrate nervous system evolved to regulate neurogenesis.SIGNIFIC

169 uropeptide hormone oxytocin is a key central nervous system factor in the regulation of food intake a

170 e abnormalities were correlated with central nervous system findings, microcephaly, and the timing of

171 ing axon regeneration to occur in the mature nervous system following axonal injury.

172 imulation to targeted regions of the central nervous system for extended periods.

173 also correlate initial abnormalities of the nervous system found on imaging with postnatal clinical

174 put during locomotion.SIGNIFICANCE STATEMENT Nervous system function depends on the specific excitabi

175 fied behaviors, leaving open questions about nervous system function in the context of natural tasks.

176 of Munc13 autoinhibition profoundly impacts nervous system function in vivo.

177 Nervous system function relies on precise synaptic conne

178 oordinated electrical activity that underlie nervous system function.

179 molecule NCAM, play important roles in many nervous system functions during development and in adult

180 f GPR17 per se as an orchestrator of central nervous system functions, they challenge the utility of

181 nal malaise suggests exercise alters central nervous system functions.

182 cate that vpr-1 activity is important in the nervous system, germ line and intestine.

183 The nervous system has limited capability to regenerate, so

184 e of insulin-responsive GLUT4 in the central nervous system has not been well characterized.

185 however, post-developmental functions in the nervous system have not been elucidated.

186 ophages instruct tissue repair in the liver, nervous system, heart, lung, skeletal muscle, and intest

187 1 envelope glycoprotein 120 in their central nervous system (HIVgp120tg) mount a transient IFNbeta re

188 have been described in patients with central nervous system hypomyelination.

189 ide a paradigm for understanding the enteric nervous system in health and disease.

190 By showing involvement of the peripheral nervous system in MS, this proof-of-concept study may of

191 asticity, but its role may extend beyond the nervous system, in tissues where local changes in transl

192 Central nervous system infection of neonatal and adult rats with

193 Patients with central nervous system injuries, injury requiring medical care i

194 ultrasound on memory impairment and central nervous system injury in a rat model of vascular dementi

195 ence progressive lung disease or cardiac and nervous system involvement (complicated sarcoidosis).

196 64; 95% CI, 1.19-2.27; P = .003), peripheral nervous system involvement (HR, 6.75; 95% CI, 2.31-19.7;

197 RATIONALE: The sympathetic nervous system is a major mediator of heart function.

198 The nervous system is believed to be a network of neurons in

199 The nervous system is emerging as a regulator of malignancy.

200 wann cell (SC) myelination in the peripheral nervous system is essential for motor function, and unco

201 nd distribution of such receptors within the nervous system is not well described.

202 Because the developing nervous system is particularly vulnerable to perturbatio

203 phosphorylation of specific residues in the nervous system is poorly understood.

204 shes several cell types from the rat central nervous system, largely based on the relative proportion

205 and in phrenic motor neurons of the central nervous system led us to address the individual contribu

206 small size of most terminals in the central nervous system, little is known about the regulation of

207 In the mammalian central nervous system, Lpd contributes to neuronal morphogenesi

208 odel, in high-risk cluster the prevalence of nervous system malformation decreased by approximately 2

209 ith a focus on preventing congenital central nervous system malformations.

210 ifications (PTMs) reportedly tied to central nervous system maturation, myelin stability, and the pat

211 Synaptic refinement is a critical step in nervous system maturation, requiring a carefully timed r

212 C4A10 expression and function in the central nervous system may affect the regulation of systemic wat

213 d antitubulin cancer drugs on the peripheral nervous system may help guide clinical evaluations to im

214 The nervous system may include more than 100 residue-specifi

215 including neurons and glia of the peripheral nervous system, melanocytes, and bone and cartilage of t

216 if they had symptomatic or untreated central nervous system metastases, had received anticancer thera

217 g predominantly to categories related to the nervous system, muscle development, and especially to me

218 In the mammalian nervous system, myelin provides electrical insulation fo

219 oligodendrocyte differentiation and central nervous system myelination.

220 ve interactions direct cells to form central nervous system (neural plate) or sensory placodes.

221 ct critical populations of intrinsic cardiac nervous system neurons and alter cardiac repolarization.

222 These cytokines regulate central nervous system neurons to induce sleep.

223 and coregulate membrane potential in central nervous system neurons.

224 Enteric nervous system neuropathy causes a wide range of severe

225 haviors, despite having a physically limited nervous system of 302 neurons, is poorly understood.

226 mportant contribution to the ancient enteric nervous system of early jawless vertebrates, a role that

227 ent neurotoxins, which damages the brain and nervous system of human beings through fish consumption.

228 modification that is abundant in the central nervous system of mammals and which results from 5-methy

229 tivity levels when injected into the central nervous system of mice.

230 In the nervous system of vertebrates, its expression is enriche

231 ugh their presence in amyloid plaques in the nervous systems of affected individuals.

232 ordinately in the same pathway in either the nervous system or spermatogenesis.

233 ic renal failure, involvement of the central nervous system, or death), and interventions (ie, renal

234 A core principle of nervous system organization is the diversification of ne

235 multiple sclerosis lesions and other central nervous system pathologies with prominent myelin injury,

236 ised DNA damage response (DDR) and prominent nervous system phenotypes.

237 myelination represents an important form of nervous system plasticity.

238 The nervous system plays a central role in regulating the st

239 ry neurons, demonstrating that the autonomic nervous system plays a substantial role in HSV pathogene

240 -term viability of neurons in the peripheral nervous system (PNS) and retina.

241 Injury to the peripheral nervous system (PNS) causes a dramatic shift in SC molec

242 central nervous system (CNS) and peripheral nervous system (PNS) in PD.

243 patients presenting with pain or peripheral nervous system (PNS) manifestations, 39% were LGI1-IgG s

244 y and chemosensory neurons of the peripheral nervous system (PNS) must signal to the motor circuits o

245 ulfated N-glycans are involved in peripheral nervous system (PNS) myelination.

246 th sensory neuron clusters of the peripheral nervous system (PNS), and blood cells (hemocytes) requir

247 In the peripheral nervous system (PNS), developmental axon pruning relies

248 cal disorders of both the CNS and peripheral nervous systems (PNS), yet few studies have directly exa

249 control of differentiation of mouse enteric nervous system progenitor cells by EDN3 requires regulat

250 over gastrointestinal functions, the central nervous system provides extrinsic neural inputs that mod

251 Here we show that the peripheral sympathetic nervous system (PSNS) is very underdeveloped in def-defi

252 coordinate various functions in the central nervous system ranging from removing synaptic connection

253 Among these, signals from the sympathetic nervous system regulate HSC egress via its niche, but ho

254 The autonomic nervous system regulates cardiac electrical and mechanic

255 us has potential in the treatment of central nervous system-related pathologies, such as Alzheimer's

256 their functional role in organisms without a nervous system remains unclear.

257 od pressure (BP) is regulated at the central nervous system, renal, and vascular levels, but the cell

258 ciently transduce the central and peripheral nervous systems, respectively.

259 show that primary cells from the rat central nervous system respond differently to photo-toxicity, in

260 Understanding how the peripheral nervous system responds to electro-mechanical stimulatio

261 inflammation is initiated by the sympathetic nervous system, resulting in the elevation of exosome-as

262 Therefore, understanding how the nervous system shapes the immune response could have imp

263 ies suggest that, in addition to its role in nervous system signaling, AChE can also modulate non-neu

264 In many parts of the nervous system, signals pass across multiple synaptic re

265 oskeleton both at the AIS and throughout the nervous system.SIGNIFICANCE STATEMENT Spectrin cytoskele

266 e energy balance, which augments sympathetic nervous system (SNS) activation in response to metabolic

267 hat PTSD patients have augmented sympathetic nervous system (SNS) and haemodynamic reactivity during

268 PTSD patients have an overactive sympathetic nervous system (SNS) that could contribute to cardiovasc

269 Optogenetic activation of the sympathetic nervous system (SNS) upregulates NE uptake by SAMs and s

270 dipocytes, immune cells, and the sympathetic nervous system (SNS), wherein CB1 plays a key regulatory

271 es of the development and evolution of human nervous system specializations.

272 at successive relays in the parasympathetic nervous system strongly resemble each other despite the

273 and for treating diseases of the peripheral nervous system, such as chronic nausea, vomiting, pain,

274 bility and additional central and peripheral nervous system symptoms but an absence of frontonasal or

275 pressants, antipsychotics, and other central nervous system-targeted medications) are increasingly us

276 Autonomic nervous system testing revealed no abnormalities in hear

277 d pathogenic modifications to the peripheral nervous system that elevate metastatic capacity.

278 structure and function and have potential as nervous system therapeutics as well.

279 ituitary-adrenal (HPA) axis, the sympathetic nervous system through the greater splanchnic nerve (GSN

280 The nervous system thus seems to partition the learning of o

281 tate; testicular; kidney; bladder; brain and nervous system; thyroid; mesothelioma; Hodgkin lymphoma;

282 This suggests the ability of the central nervous system to concurrently learn operating the BMI w

283 biogenic amine neurotransmitters through the nervous system to control and maintain pairing with fema

284 targeted different sites within the central nervous system to restore motor function following spina

285 ginated from different levels of the central nervous system, to the different compartments.

286 The central and peripheral nervous system transcriptomes of the spider Cupiennius s

287 l age increased risk of leukemia and central nervous system tumors, older paternal age was not associ

288 d to the development of benign and malignant nervous system tumors.

289 Here we test if a mechanism in the central nervous system underlies thermal TCE.

290 oth correlative and causal evidence that the nervous system uses millisecond-scale variations in the

291 o neuronal classes in the C. elegans central nervous system, using VGLUT-pHluorin to monitor synaptic

292 tissue (BAT) is regulated by the sympathetic nervous system via beta3-adrenergic receptors (beta3-AR)

293 ug delivery of macromolecules to the central nervous system via the intrathecal route.

294 bundantly expressed in the mammalian central nervous system, where it regulates intracellular calcium

295 about expression of UBE3A in the peripheral nervous system, where loss of maternal UBE3A might contr

296 on-activated Na(+) channels expressed in the nervous system, where they are involved in learning, fea

297 abnormal activity of the cardiac sympathetic nervous system, which are established risk factors in se

298 quired demyelinating syndrome of the central nervous system will have a monophasic disease course, wi

299 2a is rapidly distributed within the central nervous system with a very favourable brain/blood ratio.

300 ommon, though 1 case did involve the central nervous system with few symptoms.