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1 ata in order to obtain a complete picture of neural function.
2 t regulation of this pathway is critical for neural function.
3 isruption, which can have adverse effects on neural function.
4 are different from those involved in normal neural function.
5 signaling systems have evolved to stabilize neural function.
6 lecular and neuroimaging levels in exploring neural function.
7 to studying how gender enculturation shapes neural function.
8 n nearly every aspect of normal and abnormal neural function.
9 ogy is a powerful way to analyze C. elegans' neural function.
10 oviding a homeostatic environment for proper neural function.
11 -making and the methodologies used to assess neural function.
12 tribute to the complexity of neurogenesis or neural function.
13 s involved in steroid hormone signalling and neural function.
14 annels are critical regulators of muscle and neural function.
15 proteins, particularly those associated with neural function.
16 s an important role in brain development and neural function.
17 iting the inferences that can be drawn about neural function.
18 ation can be coordinated to achieve constant neural function.
19 ith sensory, motor, or other disabilities of neural function.
20 e large ion fluxes is essential for powering neural function.
21 importance of desensitization for regulating neural function.
22 ted in tumorigenesis, heart development, and neural function.
23 ctively target cancer cells while preserving neural function.
24 in cellular membranes is critical for normal neural function.
25 in an in vivo murine model while preserving neural function.
26 anced both tumor control and preservation of neural function.
27 ial to experience-dependent modifications of neural function.
28 cal synapses and are critically important to neural function.
29 cently been used as cellular-level probes of neural function.
30 maintenance of cardiovascular, skeletal, and neural function.
31 local SWA changes, and these should benefit neural function.
32 r space (ECS) is an important determinant of neural function.
33 e of elucidating how other glycans influence neural function.
34 suggests its unique and nonredundant role in neural function.
35 at particular magnitudes of activity support neural function.
36 promise for investigating genes involved in neural function.
37 ain environment that is essential for proper neural function.
38 c rats with antioxidants has on vascular and neural function.
39 These proteins also regulate many aspects of neural function.
40 the spontaneous onset of labour alter fetal neural function.
41 d under these conditions to maintain optimal neural function.
42 may have a possible modulatory role on renal neural function.
43 ned whether E and/or E+P increased serotonin neural function.
44 tory mechanism for synaptic transmission and neural function.
45 e of rapid and spatially resolved mapping of neural function.
46 s is the first evidence of a role for Wiz in neural function.
47 nal proteome is key to generate and maintain neural function.
48 o microcircuitry is crucial to understanding neural function.
49 al myelination during development influences neural function.
50 cillations across brain networks may play in neural function.
51 e method for quantifying disorders of higher neural function.
52 d flow to adequately couple energy supply to neural function.
53 ve disease can cause devastating deficits in neural function.
54 ninflammatory functions that maintain normal neural function.
55 ally as components of higher-level models of neural function.
56 distinct and important roles, for example in neural function.
57 provide the proper chemical composition for neural function.
58 e and repeated engagement with sound changes neural function.
59 able, reproducible, and predictable baseline neural function.
60 C1-PDE4 modulated and likely to regulate its neural functions.
61 MDAR) is known to mediate many physiological neural functions.
62 hetic devices, but also to shed new light on neural functions.
63 ty of E(2) to impact such a diverse array of neural functions.
64 g, which may contribute to androgen-mediated neural functions.
65 he role of these kinases in a broad range of neural functions.
66 trogenic effects affecting smooth muscle and neural functions.
67 silencing of genes involved in many kinds of neural functions.
68 infected neurones might be a failure of key neural functions.
69 hippocampal formation, indicating a role in neural functions.
70 mpacts of DNA breaks on neurodevelopment and neural functions.
71 he gonads including stem cell-associated and neural functions.
72 ral tissues, suggesting that they might have neural functions.
73 sphingolipids impair protein homeostasis and neural functions.
74 h distinct connectivity, gene expression and neural functions.
75 h alterations in subjective, behavioral, and neural functions.
76 estigate further the impact of deployment on neural functioning.
77 Thus WAVE-1 is required for normal neural functioning.
78 portant functions for normal development and neural functioning.
79 uild biologically grounded models of complex neural function?
81 le homeostasis is believed to be crucial for neural function, a systematic analysis of homeostatic co
83 pendent of drug effects, this study assessed neural function among drug-naive youth with a behavioral
85 , it is remarkable that robust, reproducible neural function and animal behavior can be achieved.
86 dendrocytes, which are vitally important for neural function and axonal integrity, remains unclear.
87 tion of vascular tone is tailored to support neural function and behavior, in this case the drive to
93 ous stem cell populations, in the context of neural function and cardiovascular biology, in metabolis
96 ically important protein required for normal neural function and for the transport of calcium in epit
99 ing a new look at RNA-mediated modulation of neural function and raises the possibility that RNAi mig
100 ling preferences correlate with behavior and neural function and remain stable over multiple days.
102 ond-scale timing of sensory input in shaping neural function and strongly suggest STDP as a relevant
104 d (Abeta) deposition leads to alterations in neural function and ultimately to cognitive decline in A
105 tes, whose associated genes are enriched for neural functions and are functionally repressed by Insv.
107 kephalin signaling pathway regulates various neural functions and can be altered by neurodegenerative
108 ith conserved REST binding are enriched with neural functions and more likely to be upregulated upon
109 orphic brain nuclei underlie gender-specific neural functions and susceptibility to disease, but the
111 errelationships between synaptic morphology, neural function, and behavior; and the evolution of nerv
112 onships among these two aggregated proteins, neural function, and brain structure are poorly understo
113 lopmental activation of Shank3 expression on neural function, and demonstrate a certain degree of con
114 tudies to test the effects of aging, injury, neural function, and dentinal repair on its influx into
115 s in neurobiology is how experience modifies neural function, and how changes in the nervous system p
116 atial processing are intrinsic properties of neural function, and specialized timing mechanisms such
117 ay important roles in epidermal development, neural function, and transcriptional regulation, consist
118 imate relation between vascular activity and neural function, and we will emphasize the various roles
119 complex overlearned maladaptive patterns of neural functioning, and must find a way to ally with dev
120 physical activity (PA), cognitive vitality, neural functioning, and the moderation of these associat
124 l consequences of age-related alterations in neural function are well documented, but less is known a
125 ing neural activations imply that equivalent neural functions are engaged by empathy and direct emoti
130 appropriate environment to allow for proper neural function, as well as protect the CNS from injury
132 o face and gaze stimuli to better understand neural functioning associated with social perception in
133 revealed dysregulated expression of several neural function-associated genes, and PC4 was consistent
134 this test may also result from inhibition of neural functions because of brain hypothermia due to bod
135 ater, may result from dramatic inhibition of neural functions because of severe brain hypothermia.
136 in depression; despite such acute effects on neural function, behavioral modification may require mor
138 n solubility, which in turn might compromise neural function by damaging the neuronal cytoskeleton.
140 n of approximately 1,000 genes important for neural functions by in situ hybridization at a cellular
141 n of both synaptic transmission and auditory neural function can be associated with abnormal loudness
145 injury (including to gray and white matter), neural function (cortical function and cortical connecti
148 microarray analysis on animals with altered neural function due to mutation in npr-1 shows an enrich
149 amined how aging and Abeta deposition affect neural function during memory encoding of visual scenes
150 The ability of sequence variants to affect neural function either alone or in concert may reveal ef
151 minish redundancy and increase efficiency of neural function, fine-tuning of excitatory and inhibitor
152 f MeCP2 in regulation of BDNF expression and neural function, focusing on NG neurons and respiratory
153 aken together, these results suggest a novel neural function for Cdx proteins within the gene regulat
154 ferase; EC 2.4.1.92 [EC])] are revealing key neural functions for the complex gangliosides of brain.
155 possible to dissociate the roles of FMRP in neural function from developmental dysregulation, and th
156 anscription factors, signaling proteins, and neural function genes was analyzed using multi-label con
158 p between the integrity of neural tissue and neural function has not been previously investigated.
161 As limitations in energy supply constrain neural function, high-probability release sites might am
162 elination of the CNS is necessary to restore neural function in a number of demyelinating conditions.
163 reports ERK activity during development and neural function in diverse cell contexts in Caenorhabdit
164 Here we show, using temporal control of neural function in Drosophila, that only short-term appe
170 undant, antipsychotic medications may affect neural function in studies of animals, healthy volunteer
171 function during the day-to-day regulation of neural function in the absence of pathogens and irrespec
173 this study were to investigate vascular and neural function in the cutaneous microcirculation of AL
177 The results provide evidence for compromised neural function in these patients and provide a promisin
178 istic dichotomy between loss and recovery of neural function in visual cortex; CREB function is essen
183 is critical for establishing all aspects of neural function including learning, memory, and locomoti
184 methylation, have been associated with many neural functions including synaptic plasticity, learning
185 ich is believed to control some of essential neural functions, including information processing, work
187 of alcohol and the progression of changes in neural function induced by chronic ethanol consumption t
190 tions provides a better understanding of the neural function involved in information processing.
195 ems, it is poorly understood how lateralized neural function is linked to lateralized gene activity.
201 uitin signaling at synapses is essential for neural function, it has been assumed that synaptic prote
202 re is a factor capable of affecting numerous neural functions, it may be an important variable in the
203 The electrochemical processes that underlie neural function manifest themselves in ceaseless spatiot
204 Estradiol (E(2)) regulates a wide range of neural functions, many of which require activation of es
208 ibular system has access to a broad array of neural functions not typically associated with visuomoto
211 , we used a genetic approach to identify the neural function of DNA-PKcs and the interplay between AT
212 the importance of DNA damage signalling, the neural function of many critical DNA repair factors is u
215 The authors examined sex differences in the neural functioning of adults with ADHD during performanc
219 xistence of potent mechanisms that stabilize neural function, our capacity to learn and remember woul
221 ning attention may normalize emotion-related neural function possibly by engagement of the medial pre
222 broaden our understanding of lateralities in neural function, provide insights into how this laterali
224 he impact of astrocytic CN/NFAT signaling on neural function/recovery after acute injury has not been
226 We find that DMob4 plays a prominent role in neural function, regulating axonal transport, membrane e
229 could underlie aspects of FXS disruption of neural function.SIGNIFICANCE STATEMENT FXS is a leading
230 ehavior must arise because of differences in neural function, status-dependent differences in the und
231 populations, which are involved in numerous neural functions such as sleep, fatigue, anxiety, and co
232 erins are likely to play a role in 'generic' neural functions, such as neurite fasciculation, branchi
233 within the nervous system to maintain normal neural functions, such as neurotransmission, within pred
234 Despite the importance of PI(3,5)P2 for neural function, surprisingly little is known about this
235 We discuss the physiologic role of PNNs in neural function, synaptic assembly, and plasticity as we
236 focuses on molecular and cellular changes in neural function that are produced as adaptations to chro
237 est that UPR signaling is a critical step in neural function that is needed for glutamate receptor as
238 n primates have highlighted abnormalities in neural functioning that might underlie the generation of
239 lar matrix, regulate developmental and adult neural functions that are highly relevant to the pathoge
242 , immune responses, breast cancer, and other neural functions, these studies add to the basic biology
243 ndogenous brain activity can causally affect neural function through field effects under physiologica
244 Homeostatic signaling systems stabilize neural function through the modulation of neurotransmitt
247 ion of protein synthesis might disrupt other neural functions to interfere with memory formation.
248 To investigate this, we disrupted normal neural function via repetitive transcranial magnetic sti
250 o this proinflammatory cytokine might affect neural function, we examined its effects on cultures of
252 3 PUFAs were as follows: 1) their role is in neural function, which is measured only with difficulty
253 drive stable changes in gene expression and neural function, which may cause several symptoms, inclu
254 ion of proteins, show promise in elucidating neural function with greater precision and flexibility.
255 contributing to estrogen neuroprotection, a neural function with potential therapeutic relevance to
256 =5 is correct) to assess the relationship of neural functioning with performance, participants in thi
257 ersity and connectivities fundamental to our neural functions, yet difficulties in interrogating indi
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