ライフサイエンスコーパス: neural plasticity

1 tional and social processing and the role of neural plasticity.

2 tant neural locus of reconsolidation-related neural plasticity.

3 regulate key events in physiology, including neural plasticity.

4 Recovery from stroke engages mechanisms of neural plasticity.

5 TGF-beta has also been implicated in adult neural plasticity.

6 s diverse functions in development and adult neural plasticity.

7 ological processes ranging from evolution to neural plasticity.

8 target genes involved in stress response and neural plasticity.

9 ) in the central nervous system and regulate neural plasticity.

10 rapidly altered during experience-dependent neural plasticity.

11 may play a nonproteolytic role in regulating neural plasticity.

12 eceptors because of their potential roles in neural plasticity.

13 central nervous system (CNS) homeostasis and neural plasticity.

14 cularly cellular and molecular mechanisms of neural plasticity.

15 olarized, suggesting that it plays a role in neural plasticity.

16 model system with which to investigate adult neural plasticity.

17 hat myelin genesis might also be involved in neural plasticity.

18 -studied model system for activity-dependent neural plasticity.

19 arget of rapamycin (mTOR), are implicated in neural plasticity.

20 a dynamic and flexible model of compensatory neural plasticity.

21 neurons involves glutamatergic and GABAergic neural plasticity.

22 example of competitive, experience-dependent neural plasticity.

23 s anchored these results to previous work in neural plasticity.

24 model system with which to investigate adult neural plasticity.

25 oal of identifying novel factors involved in neural plasticity.

26 indicates the involvement of this pathway in neural plasticity.

27 eceptor (AMPAR) trafficking is important for neural plasticity.

28 trix metalloproteinases (MMPs) as markers of neural plasticity.

29 tion can be viewed as a form of drug-induced neural plasticity.

30 eins such as brevican that are implicated in neural plasticity.

31 ent gene regulation that may be critical for neural plasticity.

32 contribute to elucidate how alcohol disrupts neural plasticity.

33 -amblyopic eye could be a model for residual neural plasticity.

34 ng protein (CREB) has been shown to regulate neural plasticity.

35 s exhibit experience-dependent developmental neural plasticity.

36 lucidating the mechanisms and time course of neural plasticity.

37 transmitters is a critical step in long-term neural plasticity.

38 proposed as a mediator of activity-dependent neural plasticity.

39 p190 by integrin and Src, both implicated in neural plasticity.

40 defects in several common in vitro models of neural plasticity.

41 receptor types may be important elements in neural plasticity.

42 cated in central roles in activity-dependent neural plasticity.

43 ith amblyopia retain a significant degree of neural plasticity.

44 ggest an isoform-specific role of SNAP-25 in neural plasticity.

45 ding synapse strength, circuit function, and neural plasticity.

46 h Gp1 mGluR mediates protein translation and neural plasticity.

47 ight into myelination-centered mechanisms of neural plasticity.

48 ritical mediators of transcription-dependent neural plasticity.

49 n neuronal development and activity-mediated neural plasticity.

50 s ostensibly adhere to Hebbian principles of neural plasticity.

51 that VNS-tone pairing can direct therapeutic neural plasticity.

52 y in pre-mRNA quality control is involved in neural plasticity.

53 o provide an important platform for studying neural plasticity.

54 oligodendrocytes, which may facilitate rapid neural plasticity.

55 gulating the molecular mechanisms underlying neural plasticity.

56 model system with which to investigate adult neural plasticity.

57 echanism can underlie such stable changes in neural plasticity?

58 ectrical microstimulation is known to induce neural plasticity [10, 11], and caudate microstimulation

59 The different forms of neural plasticity [7-9] underlying these changes have be

60 an be correlated with cellular mechanisms of neural plasticity, a novel mechanism that may explain th

61 hy subjects and modulates pathophysiological neural plasticity after sensorimotor loss, but the mecha

62 These animals also exhibited abnormal neural plasticity after training, which may be a mechani

63 g a protease with functions in apoptosis and neural plasticity, alters specific social behaviors in m

64 The role of synaptophysin in neural plasticity and ageing should be further examined.

65 ic restriction (CR) has SSRI-like effects on neural plasticity and anxiety-related behavior.

66 tor kinase B (TrkB), have important roles in neural plasticity and are required for antidepressant ef

67 ion in a number of cellular processes (e.g., neural plasticity and circadian rhythms).

68 supported that younger patients have better neural plasticity and clinical recovery than do older pa

69 ng through WAVE-1 complexes is essential for neural plasticity and cognitive behavior.

70 omeostatic signaling is thought to constrain neural plasticity and contribute to the stability of neu

71 asis for understanding Arc's contribution to neural plasticity and disease.

72 plications of such epigenetic regulation for neural plasticity and disorders.

73 s appear to be involved in the regulation of neural plasticity and emotion.

74 en cellular metabolism, gene regulation, and neural plasticity and establish a link between acetyl-Co

75 f functionally linked proteins that regulate neural plasticity and glutamate-mediated synaptic activi

76 sex steroid hormones play in mediating adult neural plasticity and in neuroprotection.

77 eceptor signalling stimulates post-ischaemic neural plasticity and intranasal treatment with C3a rece

78 n (PA) has been proposed as a tool to induce neural plasticity and is used to help neglect rehabilita

79 This process crucially depends on ongoing neural plasticity and keen sensitivity to environmental

80 generated, adult-born neurons contribute to neural plasticity and learning.

81 Thus, dysbindin is essential for adaptive neural plasticity and may link altered homeostatic signa

82 prominent molecular devices for controlling neural plasticity and memory formation.

83 ctive means of restoring a high capacity for neural plasticity and of promoting recovery from the eff

84 and provides insight into the mechanisms of neural plasticity and peripheral regeneration in humans.

85 nvolvement of sleep in protein synthesis and neural plasticity and point to a novel role for sleep in

86 These results reveal a capacity for neural plasticity and recovery that is larger than anyth

87 and with treatment effects in the setting of neural plasticity and recovery.

88 an be pharmacologically modulated to improve neural plasticity and regeneration.

89 novel rehabilitation therapies that enhance neural plasticity and sculpt motor recovery.

90 medication exposure has been shown to alter neural plasticity and shift sensitive periods in percept

91 ulating neural plasticity; low doses enhance neural plasticity and spatial memory behavior, whereas c

92 l activity during sleep mediates large-scale neural plasticity and stabilizes kinematics during early

93 of a significant number of genes related to neural plasticity and stress, as well as the dynamic reg

94 Here, I discuss a new perspective on neural plasticity and suggest how plasticity might be ta

95 Transcripts implicated in neural plasticity and survival are enriched in ventral t

96 nscription factor implicated respectively in neural plasticity and the specific expression of neurona

97 rotonin reuptake inhibitors (SSRI's) enhance neural plasticity and their ability to enhance fear exti

98 ts play distinct roles in activity-dependent neural plasticity and thus can be incorporated along wit

99 a key role in mediating experience-dependent neural plasticity and, thus, creates a link between the

100 channels (CaVs) drive synaptic transmission, neural plasticity, and cardiac contraction.

101 hways associated with cellular regeneration, neural plasticity, and development.

102 em cell biology, developmental neurobiology, neural plasticity, and disease mechanisms.

103 patients, such as impaired memory capacity, neural plasticity, and neurogenic processes.

104 death and neurogenesis, experience-dependent neural plasticity, and sexual differentiation.

105 ychiatric disorders derive from pathological neural plasticity, and studying the mechanisms that unde

106 and current clinical treatments that harness neural plasticity, and we offer perspectives on future d

107 Studies of neural plasticity are also important to understand the n

108 nd the beneficial trophic effects of BDNF on neural plasticity are critical components for drug respo

109 g, but the contribution of these isoforms to neural plasticity are not well understood.

110 orphology and receptor content that underlie neural plasticity are poorly understood.

111 (NO) and cGMP, in long-term potentiation and neural plasticity are well documented.

112 d its receptor and may have implications for neural plasticity arising from such experiences.

113 al prefrontal cortex could reflect change in neural plasticity as a consequence of an intense stimula

114 Studies on neural plasticity associated with brain-machine interfac

115 RGS9 levels contribute to the behavioral and neural plasticity associated with chronic opiate adminis

116 addiction shares striking commonalities with neural plasticity associated with natural reward learnin

117 These findings provide strong evidence for neural plasticity at the level of large-scale networks s

118 A mechanism of neural plasticity based on protein dissociation rather t

119 ent, It also has implications for studies of neural plasticity because a stable baseline permits iden

120 ion (LTP) is an experience-dependent form of neural plasticity believed to involve mechanisms that un

121 Chromatin is a critical regulator of neural plasticity, but basic principles of chromatin fun

122 lloproteinases that cleave lecticans mediate neural plasticity by altering the structure of ECM aggre

123 Our data advance current understanding of neural plasticity by determining the differential effect

124 t a possible mechanism by which ACh augments neural plasticity by directing activity to populations o

125 modulated by experience, which could mediate neural plasticity by optimizing the performance of the c

126 g of the activity-dependent central auditory neural plasticity changes that must certainly generate t

127 3000, including many not typically linked to neural plasticity, compared with <300 following HDACi ad

128 A large body of evidence suggests that neural plasticity contributes to learning and disease.

129 The locus of the neural plasticity could be a subpopulation of directiona

130 es different brain structures and degrees of neural plasticity dependent upon task requirements.

131 osis protease with newly discovered roles in neural plasticity, disrupts attention in mice while pres

132 ependent to NMDAR-independent mechanisms for neural plasticity during aging is associated with better

133 Neural plasticity during development appears to result i

134 nthesis in the amygdala may be essential for neural plasticity during this form of associative learni

135 periods are temporary windows of heightened neural plasticity early in development.

136 coding strategies facilitated by underlying neural plasticity enable the adult brain to learn from v

137 osed and are under investigation to modulate neural plasticity, enhance it when it plays an adaptive

138 Neural plasticity following brain injury illustrates the

139 estigate in vivo functional consequences and neural plasticity following cell death as well as apopto

140 future behavior-based therapies that harness neural plasticity for recovery.

141 ctions, perhaps by regulating the expression neural-plasticity genes such as brain derived neurotroph

142 functional impact of activated microglia on neural plasticity has so far been elusive.

143 Studies of experience-dependent neural plasticity have largely focused on individual syn

144 The behavioral correlates of this seasonal neural plasticity have not been well characterized, part

145 eful changes in afferent input that modulate neural plasticity impact on behavioral markers of perfor

146 is that VNS paired with experience can drive neural plasticity in a controlled and therapeutic direct

147 provides biological evidence for system-wide neural plasticity in auditory experts that facilitates a

148 onatal isolation stress alters bidirectional neural plasticity in BLA-DG synapses, which may help to

149 n (PA) has been proposed as a tool to induce neural plasticity in both healthy participants and patie

150 Significance statement: We investigated neural plasticity in dorsal striatum from rats that were

151 rent input have been evaluated as drivers of neural plasticity in healthy subjects and in small group

152 viability of metabolic mechanisms supporting neural plasticity in hippocampal slices from 24 to 30 mo

153 r mental disorders, is a potent modulator of neural plasticity in humans and has been linked to defic

154 ults are beginning to emerge from studies of neural plasticity in humans.

155 ating the cascade of events underlying adult neural plasticity in ITC.

156 galese Finches, the degree of behavioral and neural plasticity in juvenile and adult birds may be les

157 BDNF is involved in many forms of adult neural plasticity in other systems and is present in the

158 Given the history of NO in mediating neural plasticity in other systems, the results point to

159 ic, treatments that maximise what remains of neural plasticity in patients with progressive multiple

160 Instead, the neural plasticity in sensorimotor areas is sensitive to

161 ervous system development and contributes to neural plasticity in the adult brain.

162 in demyelinating diseases and for increasing neural plasticity in the adult CNS.

163 tion in demyelinating diseases and increased neural plasticity in the adult CNS.

164 valuable opportunity not only for exploring neural plasticity in the adult human brain but also for

165 Neural plasticity in the auditory cortex of trained mice

166 An attractive approach to stimulate neural plasticity in the brain is to transplant stem cel

167 Physical exercise promotes neural plasticity in the brain of healthy subjects and m

168 or studies related to sensory processing and neural plasticity in the brain.

169 mals and provides a mechanism for continuous neural plasticity in the brain.

170 t neural pathways may limit the induction of neural plasticity in the cerebellum and thereby limit th

171 This work furthers our understanding of neural plasticity in the context of adult vision loss.

172 nnectivity, and they provide a mechanism for neural plasticity in the developing and adult nervous sy

173 nges induced by antidepressants may regulate neural plasticity in the diseased brain, providing sympt

174 strates large-scale and seemingly ubiquitous neural plasticity in the ground squirrel brain during to

175 oral estrogen could improve memory and alter neural plasticity in the hippocampus and neocortex of mi

176 uromodulatory receptors, interact to produce neural plasticity in the LA and behavioral fear conditio

177 on of a Pavlovian fear memory and associated neural plasticity in the LA.

178 levels is an essential step in understanding neural plasticity in the mature animal.

179 tive in cocaine sensitization and associated neural plasticity in the mPFC and NAc core.

180 bic pathway and activate common mechanism of neural plasticity in the nucleus accumbens.

181 istically with theta oscillations to promote neural plasticity in the service of learning and memory.

182 Neural plasticity in the song control system of seasonal

183 s play a role in stimulus-induced, long-term neural plasticity in the spinal cord.

184 MPA)) glutamate receptors; the potential for neural plasticity in this pathway is suggested by its ca

185 that endogenous opioids during mating induce neural plasticity in VTA dopamine neurons that appear cr

186 rb mechanisms relevant to activity-dependent neural plasticity, in which neuronal activity activates

187 They are implicated in many forms of neural plasticity including hippocampal long-term potent

188 he mammalian brain exhibits diverse types of neural plasticity, including activity-dependent neurogen

189 cAMP signaling is involved in many forms of neural plasticity, including hypersensitivity of nocicep

190 ted gene expression mediates many aspects of neural plasticity, including long-term memory.

191 to synapses is essential to several forms of neural plasticity, including long-term potentiation (LTP

192 It is proposed that AAE modulates neural plasticity induced by high-frequency hearing loss

193 Neural plasticity induced by stroke can mediate positive

194 , typical of those known to elicit long-term neural plasticity, induced a marked increase in Zif268 e

195 , suggesting that this LC-mediated olfactory neural plasticity, induced under anesthesia, can store a

196 inase II (CaMKII), an important modulator of neural plasticity, interacts with syntaxin-1A to regulat

197 line intake that is optimal for the types of neural plasticity involved in cognitive function.

198 Neural plasticity is crucial for understanding the exper

199 rtant future direction for understanding how neural plasticity is expressed in brain disorders.

200 This neural plasticity is largely mediated by morphological a

201 operties during the developmental epoch when neural plasticity is most pronounced.

202 ation of glutamatergic neurotransmission and neural plasticity is not well understood.

203 One dramatic example of neural plasticity is ongoing neurogenesis in the adult b

204 However, the role of orexin signaling in neural plasticity is poorly understood.

205 Finally, functional neural plasticity is possible even when a cortical lesio

206 ently described, compelling example of adult neural plasticity is the effect of patching one eye for

207 This neural plasticity is the physical basis of associative m

208 An often-overlooked aspect of neural plasticity is the plasticity of neuronal composit

209 issue concerning visual-experience-dependent neural plasticity is whether experience is required only

210 lay an important biphasic role in modulating neural plasticity; low doses enhance neural plasticity a

211 Disruptions in neurodevelopment or neural plasticity may act alone or in combination to bri

212 damage, these findings suggest that adaptive neural plasticity may be enhanced using behavioral manip

213 symptoms, the current findings suggest that neural plasticity may contribute to smaller corpus callo

214 , we review the advances in basic studies of neural plasticity mechanisms in developing and adult ner

215 R1 and mGluR5, elicits translation-dependent neural plasticity mechanisms that are crucial to animal

216 Addictive drugs usurp neural plasticity mechanisms that normally serve reward-

217 Experience alters cortical networks through neural plasticity mechanisms.

218 It is widely held that this type of neural plasticity might involve mechanisms like long-ter

219 en the hypothesis that nucleus basalis gates neural plasticity necessary for instrumental learning.

220 functions such as RNA and protein synthesis, neural plasticity, neurotransmission, and metabolism.

221 However, neither neural plasticity nor a new dimension of sensory experie

222 in the establishment of acquisition-relevant neural plasticity, not simply in the expression of the l

223 ng pathways may contribute to the changes in neural plasticity observed during brain development.

224 ategic and nonsensory factors and implicated neural plasticity occurring in both low- and high-level

225 Within the amygdala and cerebellum, neural plasticity occurs because of convergence of these

226 inal study, we investigated the auditory and neural plasticity of musical learning in 111 young child

227 f this finding for discerning the effects of neural plasticity over and above normal brain maturation

228 mates may be appropriate for preservation of neural plasticity over their longer life span and is rel

229 cruiting the CaMKIIalpha-BDNF-CREB-dependent neural plasticity pathways.

230 Memory and its underlying neural plasticity play important roles in sensory discri

231 Neural plasticity plays a critical role in learning, mem

232 ormation of new memories by interfering with neural plasticity processes in the adult brain.

233 factor (BDNF) is a key positive regulator of neural plasticity, promoting, for example, the actions o

234 Precisely targeted neural plasticity provides a new avenue for the treatmen

235 f the initiation of developmental windows of neural plasticity; pubertal hormones may trigger the ope

236 Both DARPP-32 and neural plasticity regulator activity-regulated cytoskele

237 ulture, ketamine increased expression of the neural plasticity-related protein Arc, and this was prev

238 tical role of orexin signaling in the VTA in neural plasticity relevant to addiction.

239 However, rTMS-induced neural plasticity remains insufficiently understood at t

240 Neural plasticity represents a crucial mechanism of the

241 Although extensive research on neural plasticity resulting from hearing deprivation has

242 ocused nearly exclusively on the analysis of neural plasticity resulting from paired stimuli.

243 y implanted human subjects and suggests that neural plasticity resulting from previous deafness and d

244 ily attended, suggesting that the underlying neural plasticity selectively engages when stimuli are b

245 jor active zone protein presumed to regulate neural plasticity, specifically in the synaptic plasma m

246 vity-dependent spinal stimulation can induce neural plasticity that improves behavioral recovery afte

247 se, such as cocaine, cause stable changes in neural plasticity that in turn drive long-term changes i

248 g protein (CREB) is a critical integrator of neural plasticity that is responsive in a brain region-s

249 vation (MD) is a model of activity-dependent neural plasticity that is restricted to an early critica

250 Homeostatic scaling is a non-Hebbian form of neural plasticity that maintains neuronal excitability a

251 Kindling is a model of the neural plasticity that occurs following stimulation to t

252 ssible and impossible images and demonstrate neural plasticity that predicts behavioral priming for s

253 thin which to conceptualize the drug-induced neural plasticity that underlies the long-term actions o

254 These results reveal a novel form of neural plasticity, that epileptogenic stimulation can se

255 ther in the hippocampus, through homeostatic neural plasticity, the olfactory bulb or the hypothalamu

256 h of which is subject to or an expression of neural plasticity-the capacity of neurons to change thei

257 Through neural plasticity, this extensive movement training shou

258 ng development and is an integral feature of neural plasticity throughout life.

259 thought to interface with the mechanisms of neural plasticity to achieve stable yet flexible neural

260 y input and motor output and (2) controlling neural plasticity to achieve the desired behavior of the

261 ed the long-term potentiation (LTP) model of neural plasticity to assess synaptic function.

262 findings, and hampered the ability to relate neural plasticity to behavior.

263 tial therapeutic application in manipulating neural plasticity to treat a variety of conditions, incl

264 doxical roles for REST/NRSF in neurogenesis, neural plasticity, tumour suppression and cancer progres

265 se that Notch plays an important role in the neural plasticity underlying consolidated memory.

266 minance plasticity in visual cortex, and the neural plasticity underlying learning and memory.

267 omise expression of proteins involved in the neural plasticity underlying learning.

268 abilization molecules, appear to mediate the neural plasticity underlying specific forms of long-term

269 is demonstrated by improved performance and neural plasticity underlying that improvement after slee

270 Adult neurogenesis is thought to provide neural plasticity used in forming and storing new memori

271 te mechanisms underlying this nonassociative neural plasticity using in vivo and in vitro preparation

272 een shown to support cognitive functions and neural plasticity, we generated CD3zeta(-/-) mice in whi

273 C3a receptor signalling in ischaemia-induced neural plasticity, we subjected C3a receptor-deficient m

274 Val66Met SNP alters SSRI-induced changes in neural plasticity, we used wild-type (BDNF(Val/Val)) mic

275 ese functions are often tied to processes of neural plasticity whether in the hippocampus, through ho

276 fines a temperature-driven model of dramatic neural plasticity, which provides a unique opportunity t

277 , we used the monocular deprivation model of neural plasticity, which shares many common mechanisms w

278 ation by PKC represents a novel mechanism of neural plasticity with potentially significant implicati