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1 nd histological analysis demonstrate precise neuro- anatomical parallels between the DS and the Ts65D
2  mechanism by which this deficiency triggers neuro- and cardio-degeneration is unclear.
3 taxia (FRDA), a frequent autosomal recessive neuro- and cardio-degenerative disease.
4                               Currently, the neuro- and cardio-protective effects of CsA (CiCloMulsio
5 in mRNA alters the transcriptome, triggering neuro- and cardiodegeneration in Friedreich's ataxia.
6 ch's ataxia (FRDA) is an autosomal recessive neuro- and cardiodegenerative disorder for which there a
7  current is a novel therapeutic strategy for neuro- and cardioprotection, disorders of smooth muscle
8  ventilation provides ultrafast and potently neuro- and cardioprotective cooling after shockable card
9  of which results in genetic instability and neuro- and ciliopathies.
10 Ps and TBPPs), which have similar or greater neuro- and developmental toxicity compared to BDE 47 in
11             For demonstration, we used three neuro- and glioblastoma cell lines that were cultured as
12 schemia and increase their production of the neuro- and immunomodulatory cytokine tumor necrosis fact
13 rus, causes oxidative stress and progressive neuro- and immunopathology in mice infected soon after b
14  channel 1 has the potential to provide both neuro- and myelo-protective benefits in multiple scleros
15 ss and histopathologic examination by masked neuro- and ocular pathologists.
16 elated brain states, as well as the distinct neuro- and psychodynamic stages (generation and regulati
17                                          The neuro- and reproductive system toxicity of CS2 has been
18  Microglia play important roles in perinatal neuro- and synapto-genesis.
19  IImes may be involved in the degradation of neuro- and vasoactive peptides during pollen-initiated a
20 d their respective attachment sites onto the neuro- and viscerocranium.
21  state of human HFs are subject to profound (neuro-)endocrine regulation by HPT-axis hormones.
22       We show that the gooseberry/gooseberry-neuro (gsb/gsb-n) transcription factor genes act to spec
23                           These clinical and neuro- physiological characteristics suggest diffuse cor
24                             We show that pox-neuro (poxn) mutants lacking taste function in the legs

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