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1 on may produce aberrant axonal sprouting and neuroaxonal dystrophy.
2 l childhood genetic disorders categorized as neuroaxonal dystrophies.
3 A2G6 mutations are associated with infantile neuroaxonal dystrophy and have been reported previously
5 y complete normalization of the frequency of neuroaxonal dystrophy in both sites without altering the
7 or affect the ultrastructural appearance of neuroaxonal dystrophy in diabetic or age-matched control
10 cently identified in patients with infantile neuroaxonal dystrophy (INAD) and neurodegeneration with
12 esions homologous to that of human infantile neuroaxonal dystrophy (INAD), most commonly caused by mu
15 associated with disorders such as infantile neuroaxonal dystrophy, neurodegeneration with brain iron
17 iabetic humans and experimental animals show neuroaxonal dystrophy of autonomic nerve terminals, part
20 f chronically diabetic rats with established neuroaxonal dystrophy (the neuropathological hallmark of
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