ライフサイエンスコーパス: neurocognitive deficit

1 or patients with childhood-onset SLE without neurocognitive deficit.

2 e of the disease, characterized by pervasive neurocognitive deficit.

3 e, notably stunting, immune dysfunction, and neurocognitive deficits.

4 th CNS-directed chemotherapy are at risk for neurocognitive deficits.

5 ral and CNS inflammation, neuronal death and neurocognitive deficits.

6 n certain growth disorders may contribute to neurocognitive deficits.

7 ic leukemia (ALL) exhibit increased rates of neurocognitive deficits.

8 nd neuropsychiatric manifestations including neurocognitive deficits.

9 iation between early anesthetic exposure and neurocognitive deficits.

10 ructural brain anomalies as well as multiple neurocognitive deficits.

11 t of human female carriers have seizures and neurocognitive deficits.

12 elia burgdorferi, often manifests by causing neurocognitive deficits.

13 s a valid model for studying ADHD-associated neurocognitive deficits.

14 ications developed to target ADHD-associated neurocognitive deficits.

15 als with FASDs and related these findings to neurocognitive deficits.

16 difiers and the management and prevention of neurocognitive deficits.

17 preterm (<33 weeks) are at increased risk of neurocognitive deficits.

18 an impact on aspects of disability, such as neurocognitive deficits.

19 f an HSCT are at risk for severe, persistent neurocognitive deficits.

20 Although intellectual and neurocognitive deficits accompany schizophrenia, there a

21 nosis was the most prominent risk factor for neurocognitive deficits among survivors of MB despite re

22 The neurocognitive deficits among survivors of primary centr

23 xamined whether the combination of patients' neurocognitive deficits and criticism by others would pr

24 The study of neurocognitive deficits and psychosocial functioning in

25 ssociated with long-term sequelae, including neurocognitive deficits and secondary neoplasms.

26 ient at deciphering post-concussion residual neurocognitive deficits and thus has a potential clinica

27 4 counts were linked both to the severity of neurocognitive deficits and to discordant resistance pat

28 Secondary malignancies, neurocognitive deficits and treatment failure continue t

29 ed in patients with childhood-onset SLE with neurocognitive deficit, and the reduction was positively

30 Both psychotic symptoms and neurocognitive deficits appear to contribute independent

31 Neurocognitive deficits are among the most promising ind

32 Neuropathy and neurocognitive deficits are common among chronic alcohol

33 This study demonstrates that neurocognitive deficits are common even in recently diag

34 ct transition to psychosis, and determine if neurocognitive deficits are robust or explained by poten

35 e positive effects on neurophysiological and neurocognitive deficits associated with schizophrenia, w

36 gene is thought to have a major role in the neurocognitive deficits associated with Trisomy 21.

37 established that schizophrenic patients have neurocognitive deficits, but it is not known how these d

38 ) exposure is associated with neuromotor and neurocognitive deficits, but the exact mechanism of Mn n

39 In this study, we calculated neurocognitive deficits combining EEG analysis with thre

40 th schizophrenia have taken into account the neurocognitive deficits common to this illness.

41 with OSAS that may help explain some of the neurocognitive deficits described in these children.

42 -term complications such as hypertension and neurocognitive deficits despite early initiation of ther

43 drome (SLOS) is a malformation syndrome with neurocognitive deficits due to mutations of DHCR7 that i

44 uch complications include secondary tumours, neurocognitive deficits, endocrine disorders and growth

45 and late complications (eg, second cancers, neurocognitive deficits, endocrine disorders, and growth

46 M impairments which are thought to be a core neurocognitive deficit found in disorders such as autism

47 onal process formation may contribute to the neurocognitive deficits found in SLOS and may represent

48 Complications include neurocognitive deficits, growth failure, and pulmonary h

49 chological testing confirmed the presence of neurocognitive deficit in 8 patients with childhood-onse

50 Neurocognitive deficit in patients with childhood-onset

51 creased the susceptibility to HIV-associated neurocognitive deficits in both species.

52 her antiglucocorticoid treatments may reduce neurocognitive deficits in major depression.

53 These results suggest that neurocognitive deficits in mental perspective taking may

54 Neurocognitive deficits in patients with hepatitis C vir

55 tic drugs have shown promise in ameliorating neurocognitive deficits in patients with schizophrenia,

56 unologic and genetic factors associated with neurocognitive deficits in SM including 551 SM children,

57 alies, neurodevelopmental abnormalities, and neurocognitive deficits, including intellectual, executi

58 uals and are likely linked to HIV-associated neurocognitive deficits, including those in learning and

59 overall population demonstrated considerable neurocognitive deficits (mean values for all 4 subtests

60 term HIV-1 viral protein exposure on chronic neurocognitive deficits observed in pediatric HIV-1 (PHI

61 Long-term neurocognitive deficits occur in 15-40% of patients, whe

62 5 of the V3 loop was highly correlated with neurocognitive deficit (P < 0.0025, Fisher's exact test)

63 ormal brain imaging scans (P = .04) and with neurocognitive deficits (P = .04).

64 f resistance was associated with severity of neurocognitive deficits (P = 0.07), while low nadir CD4

65 Clinicians should be aware that neurocognitive deficits persist and have an adverse effe

66 ssociated with childhood comorbid disorders, neurocognitive deficits, polygenic risk, and residual ad

67 this review was to determine which, if any, neurocognitive deficits restrict the functioning of schi

68 st that some of the neuronal dysfunction and neurocognitive deficits seen in TSC patients may be attr

69 logy of the illness, and a potential link to neurocognitive deficits shaping the disorder.

70 ymphoblastic leukaemia (ALL) are at risk for neurocognitive deficits that affect development in adole

71 lymphoblastic leukemia (ALL) are at risk for neurocognitive deficits that are associated with treatme

72 unction, may combine with symptoms and other neurocognitive deficits to influence illness presentatio

73 ollowing cardiac surgery range from discrete neurocognitive deficits to severe neurologic injury such

74 the patients with childhood- onset SLE with neurocognitive deficit versus controls or patients with

75 te the pathological processes underlying the neurocognitive deficits, we compared protein expression

76 ts indicate that malnutrition predisposes to neurocognitive deficits, which in turn predispose to per