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1 standardized continuous scores on individual neurocognitive tests.
2 uestionnaire and a battery of 6 standardized neurocognitive tests.
3 h HIV worldwide have poor outcomes on formal neurocognitive tests.
4 affected, and the mice performed normally in neurocognitive tests.
5 on the UPSA, the ADCS-ADL, and a battery of neurocognitive tests.
6 s would be associated with deficits found by neurocognitive tests.
7 DTI on a 3.0 Tesla scanner and a battery of neurocognitive tests.
8 aluated by MRI, neurologic examinations, and neurocognitive tests.
9 and were age >/= 18 years were recruited for neurocognitive testing.
10 tcome was based on clinical observations and neurocognitive testing.
11 is, 18 years [11 to 42 years]) and completed neurocognitive testing.
12 comes (death or severe disability precluding neurocognitive testing: 19% [68/349] vs 18% [63/351] wit
13 ited no sequelae on physical examination and neurocognitive tests a mean of 6.0 years after infection
14 no significant differences in the results of neurocognitive testing among the three treatment groups
16 luations were conducted with age-appropriate neurocognitive testing and quantitative magnetic resonan
17 d between the two groups and correlated with neurocognitive tests and clinical performance in patient
18 nt repeated structural MRI and comprehensive neurocognitive testing, and they were genotyped for four
20 s after their diagnosis, survivors completed neurocognitive testing, another brain MRI, and their par
23 The patients were assessed with a battery of neurocognitive tests at baseline and 12 weeks after begi
24 -resolution structural MRI and comprehensive neurocognitive tests at baseline and 18 months and conti
28 d examined through the use of a computerized neurocognitive test battery that provided measures of ac
30 s, compared the findings to performance on a neurocognitive test battery, and found that N-acetylaspa
33 urocognitive performance, measured by direct neurocognitive tests (Delis-Kaplan Executive Function Sy
36 tly associated with worse performance on all neurocognitive tests except that of sustained attention.
38 confirmed 22q11.2 deletions (N=44) underwent neurocognitive testing following Val(158)Met genotyping
42 nts (67%) with available long-term follow-up neurocognitive testing had severe impairment in at least
43 us cutoffs, a combination of scores on the 3 neurocognitive tests identified 16 (20%) of the mothers
44 itative magnetic resonance imaging (MRI) and neurocognitive testing in multiple sclerosis (MS) patien
48 ribe currently used imaging, functional, and neurocognitive testing modalities and to better understa
50 tinent users have been found to do poorly on neurocognitive tests of attention and motor skills, both
51 exploratory outcomes included performance on neurocognitive tests of executive function, memory, atte
59 ld TBI and vestibular symptoms had decreased neurocognitive test scores (P < .05) and FA values in th
60 lar convergence insufficiency had diminished neurocognitive test scores (P < .05) and FA values in th
61 elberger State-Trait Anxiety Inventory); and neurocognitive test scores (Rey Auditory Verbal Learning
63 dings were correlated with symptom severity, neurocognitive test scores, and time to recovery with th
64 ities were correlated with symptom severity, neurocognitive test scores, and time to recovery with th
66 pe 1 diabetes is associated with deficits on neurocognitive testing that suggest central white matter
67 d with the CHR phase, measure the ability of neurocognitive tests to predict transition to psychosis,
68 oach highlights the need for development of "neurocognitive" tests to probe the function of component
69 sychological symptom reports, and results of neurocognitive testing using validated instruments were
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