ライフサイエンスコーパス: neuroendocrine

1 owing key words: "management" AND "PET" AND "neuroendocrine".

2 Neuroendocrine abnormalities are common in Parkinson's d

3 ased review, we describe clinically relevant neuroendocrine abnormalities in Parkinson's disease to h

4 influence numerous aspects of autonomic and neuroendocrine activity and are typically active during

5 findings identify a maladaptive sympathetic-neuroendocrine adrenal reflex mediating immunosuppressio

6 the adrenal medulla (AM) represent the main neuroendocrine adrenergic component and are believed to

7 then, we assessed whether the behavioral and neuroendocrine alterations induced by SD may be differen

8 In contrast, the neuroendocrine alterations that occur in the chronic pha

9 s to protect against hypoglycemia-associated neuroendocrine and autonomic failure in healthy humans.

10 neurons play a pivotal role in coordinating neuroendocrine and autonomic responses to maintain cardi

11 a stress-modulatory network for coordinating neuroendocrine and behavioral coping responses, wherein

12 necessary to better clarify its role in the neuroendocrine and environmental control of vertebrate r

13 s strictly regulated by a complex network of neuroendocrine and intra-ovarian signals.

14 by her oncologist for metastatic pancreatic neuroendocrine and kidney tumors.

15 nimized inpatient setting, we determined the neuroendocrine and neurobehavioral effects of repeated a

16 Deregulated neuroendocrine and neurotransmitter receptor interaction

17 childhood maltreatment including changes in neuroendocrine and neurotransmitter systems and pro-infl

18 These adipocytes express neuroendocrine and secreted factors, including the pro-p

19 highly expressed in evolutionarily conserved neuroendocrine and vocal-acoustic networks crucial for p

20 es that control cognitive-reward, autonomic, neuroendocrine, and visual modulation of eating behavior

21 HEA can acutely preserve a wide range of key neuroendocrine, ANS, and metabolic counterregulatory hom

22 Detailed neuropsychological and neuroendocrine assessments were performed at preradiothe

23 y to ataxia telangiectasia inhibition, and a neuroendocrine-associated subtype that showed increased

24 t role of these neurons in orchestrating the neuroendocrine, autonomic and behavioral response to str

25 ors with alprazolam can result in widespread neuroendocrine, autonomic nervous system, and metabolic

26 Stress elicits neuroendocrine, autonomic, and behavioral responses that

27 Stress elicits a highly coordinated set of neuroendocrine, autonomic, and behavioral responses that

28 cleus GLP-1 signaling in the coordination of neuroendocrine, autonomic, and behavioral responses to a

29 These findings suggest that neuroendocrine axes are crucial for tolerization of the

30 genetically inhibited, the activity of these neuroendocrine axes are suppressed and anxiety-like beha

31 s hallmarked by a uniform suppression of the neuroendocrine axes, predominantly of central/hypothalam

32 (31% vs 51%; P = .01) while developing a new neuroendocrine axis dysfunction in patients with preexis

33 tressful challenges, and dysfunction of this neuroendocrine axis is associated with a variety of phys

34 as the ligand-receptor pair that defines the neuroendocrine axis of serotonergic body fat loss in Cae

35 ytic picornavirus with selective tropism for neuroendocrine cancers.

36 differentiation of NEN in the direction of a neuroendocrine carcinoma (NEC - neuroednocrine carcinoma

37 l-cell lung cancer and eight with large-cell neuroendocrine carcinoma, all of whom received at least

38 all cell lung cancer (SCLC) is a devastating neuroendocrine carcinoma.

39 l carcinoma (MCC) is an aggressive cutaneous neuroendocrine carcinoma.

40 small cell lung cancer (SCLC) and high-grade neuroendocrine carcinomas.

41 ata expand our understanding of how a single neuroendocrine cell coordinates an organism-wide behavio

42 defined etiology comprising the two diseases neuroendocrine cell hyperplasia of infancy (NEHI) and pu

43 AT3 activation, a decrease in the neoplastic neuroendocrine cell population, and impaired PanIN progr

44 es augmented neural stimulation of pulmonary neuroendocrine cell secretion.

45 ressed cells and signals to activate central neuroendocrine cell(s).

46 e pixels per mm(2), P = .02), and epithelial neuroendocrine cells (4.9/mm(2) [25th-75th IQR, 0-16.4/m

47 Prostatic neuroendocrine cells (NE) are an integral part of prosta

48 Pulmonary neuroendocrine cells (PNECs) are the only innervated air

49 ptor (Robo) genes are expressed in pulmonary neuroendocrine cells (PNECs), a rare, innervated epithel

50 Neuroendocrine cells act as oxygen sensors in animals fr

51 (NEN) are rare neoplasms that originate from neuroendocrine cells and are characterized by the potent

52 d the associated regulated secretion in both neuroendocrine cells and chromogranin A-expressing COS7

53 ed nerve terminals, neuromuscular junctions, neuroendocrine cells and in hippocampal neurons.

54 cumulation of fusion-incompetent DCVs in BON neuroendocrine cells and lysosomal degradation (crinopha

55 complex formation and enhance exocytosis in neuroendocrine cells and neurons.

56 of this study demonstrate a role for ERAD in neuroendocrine cells and serve as a clinical example of

57 on of a distinctive class of Pnmt-expressing neuroendocrine cells and their descendants (i.e. Pnmt(+)

58 The dense core vesicles (DCVs) of neuroendocrine cells are a rich source of bioactive mole

59 s, likely via direct signaling to neoplastic neuroendocrine cells capable of trophic influences.

60 Nerve-responsive neuroendocrine cells exerted trophic influences and pote

61 e showed 55% double labeling of periurethral neuroendocrine cells expressing both serotonin and YFP,

62 We isolated preneoplastic neuroendocrine cells from a mouse model of SCLC and foun

63 rigin hypothesis, favoring the derivation of neuroendocrine cells from the neural crest, with the sec

64 of a major fraction of neural crest-derived neuroendocrine cells in both the human and murine prosta

65 econstruction of the distribution pattern of neuroendocrine cells in the human fetal prostate indicat

66 The histogenesis of prostatic neuroendocrine cells is controversial: a stem cell hypot

67 The uv1 neuroendocrine cells mechanically sense passage of eggs

68 pendent exocytosis of dense-core vesicles in neuroendocrine cells requires a priming step during whic

69 rkel cells, a nonproliferative population of neuroendocrine cells that arise from epidermis.

70 a heterogeneous group of tumors arising from neuroendocrine cells that includes typical carcinoid, at

71 - carotid body glomus cells, and 'pulmonary neuroendocrine cells' (PNECs) - are obscure.

72 Secretoneurin is produced in neuroendocrine cells, and the myocardium and circulating

73 Using CRISPR/Cas9, we generated HID-1 KO rat neuroendocrine cells, and we show that the absence of HI

74 secretory vesicle exocytosis in neurons and neuroendocrine cells, but the underlying mechanism remai

75 se and inhibits calcium-triggered release in neuroendocrine cells, indicating a previously unrecogniz

76 In experiments with rat neuroendocrine cells, we find that a few basic amino aci

77 pro-hormone secretory proteins released from neuroendocrine cells, with effects on control of blood p

78 regulate Ca(2+)-dependent DCV exocytosis in neuroendocrine cells.

79 rane thickness, nerve fibers, and epithelial neuroendocrine cells.

80 for secretory vesicle biogenesis in neuronal/neuroendocrine cells.

81 tic hormone (AKH) secretion from specialized neuroendocrine cells.

82 n-containing proteins by RNA interference in neuroendocrine cells.

83 culture of an extremely rare and aggressive neuroendocrine cervical cancer.

84 brane tension to shrink the Omega-profile in neuroendocrine chromaffin cells containing approximately

85 containing conventional active zones and in neuroendocrine chromaffin cells.

86 detach from the nerve and form postsynaptic neuroendocrine chromaffin cells.

87 The reproductive neuroendocrine circuit is well established, composed of

88 mmalian reproductive function depends upon a neuroendocrine circuit that evokes the pulsatile release

89 Neuroendocrine circuits encode environmental information

90 h could represent an important factor in the neuroendocrine control of flatfish reproduction.

91 Neuroendocrine control of reproduction by brain-secreted

92 d interfering with this process disrupts the neuroendocrine control of reproduction.

93 o gonadotropin-inhibitory hormone (GnIH)] in neuroendocrine control of reproductive development.

94 en assumed to have little relevance for such neuroendocrine control, we investigated whether lipid up

95 Cumulative incidence of any neuroendocrine deficit at 5 years was 55% (95% CI 41-67)

96 ining planar-supported bilayers and purified neuroendocrine dense core vesicles (DCVs) as fusion part

97 , we show that MAOA is involved in mediating neuroendocrine differentiation of PCa cells, a feature a

98 ng carcinoma and more aggressive tumors with neuroendocrine differentiation.

99 These neuroendocrine dopamine cells may contribute to the dopa

100 ese findings indicate that SSRIs may inhibit neuroendocrine dopamine release through both 5-HT-depend

101 Cumulative incidence of developing new neuroendocrine dysfunction at 5 years was significantly

102 istance, alterations of immune function, and neuroendocrine dysfunction, among others.

103 fibrotic process is initiated by cytokines, neuroendocrine effectors, and mechanical strain that pro

104 tral nervous system could include autonomic, neuroendocrine, enteric, and immune systems, with pathol

105 and bronchial reactivity, particularly ASM, neuroendocrine epithelial cells, and bronchial nerve end

106 associated with gene signatures of the known neuroendocrine/epithelial (NE) and non-neuroendocrine/me

107 a multipass transmembrane glycoprotein with neuroendocrine expression, had physical characteristics

108 Galanin is a neuroendocrine factor responsible for regulation of phys

109 Here we report that high expression of a neuroendocrine factor termed VGF induces the transcripti

110 h pathway results in a neuroendocrine to non-neuroendocrine fate switch in 10-50% of tumour cells in

111 the differentiation of precursor cells to a neuroendocrine fate.

112 Our findings suggest that the major neuroendocrine features of PWS are due to PC1 deficiency

113 e low to absent AR expression and often have neuroendocrine features.

114 g, score on the Pubertal Development Scale), neuroendocrine function (diurnal salivary cortisol, dexa

115 ConvRT achieves superior neurocognitive and neuroendocrine functional outcomes over 5 years without

116 ticotropin-releasing hormone (CRH) regulates neuroendocrine functions such as adrenal glucocorticoid

117 ), a transcriptional repressor that inhibits neuroendocrine gene expression.

118 tant prostate cancers express neither AR nor neuroendocrine genes and show AR pathway-independent gro

119 Mechanistically, MYC binding is enriched at neuroendocrine genes in mouse tumor cells and loss of MY

120 These findings provide strong evidence that neuroendocrine genomic AR signaling is an important extr

121 the majority of tumors lacked small cell or neuroendocrine histology.

122 ied, with measurements including circulating neuroendocrine hormones, affect, and, in cocaine-depende

123 forebrain behavior-regulatory nuclei and the neuroendocrine hypothalamus, integrating social and anxi

124 arly as conception, potentially via maternal neuroendocrine-immune responses to prenatal stressors, w

125 hypothalamus, is a primary neural locus for neuroendocrine integration, containing one of the riches

126 e tumor cells and loss of MYC reduces ductal-neuroendocrine lineage heterogeneity, while deregulated

127 , we demonstrate that MYC facilitates ductal-neuroendocrine lineage plasticity in pancreatic ductal a

128 methods in patients with multifocal, bilobar neuroendocrine liver metastases (NELM) after the first t

129 Importantly, MYC drives a neuroendocrine-low "variant" subset of SCLC with high NE

130 In small-cell lung cancer, an aggressive neuroendocrine lung cancer, loss-of-function mutations i

131 ing MYC levels decreases gemcitabine-induced neuroendocrine marker expression and increases chemosens

132 Neuroendocrine marker expression is associated with chem

133 a, revealing heterogeneous expression of the neuroendocrine marker Synaptophysin within ductal lesion

134 We analyzed ductal and neuroendocrine markers in pancreatic ductal adenocarcino

135 ey findings that have elucidated the central neuroendocrine mechanism of sleep during sickness.

136 c glucose homeostasis, potentially through a neuroendocrine mechanism.

137 ges affect loss aversion, yet the underlying neuroendocrine mechanisms are still poorly understood.

138 Neuroendocrine mechanisms underlying social inhibition o

139 known neuroendocrine/epithelial (NE) and non-neuroendocrine/mesenchymal-like (ML) SCLC phenotypes.

140 Neuroendocrine (NE) cancers are a diverse group of neopl

141 The mechanisms by which neuroendocrine (NE) cells arises from prostate adenocarc

142 reasing evidence suggests that enrichment of neuroendocrine (NE) cells is associated with CRPC.

143 functional requirement for AR, often through neuroendocrine (NE) transdifferentiation.

144 Lung neuroendocrine (NE) tumors are a heterogeneous group of

145 to October 2012, all gastroenteropancreatic neuroendocrine neoplasm patients undergoing restaging wi

146 oma (MCC) is a rare and aggressive cutaneous neuroendocrine neoplasm with a high risk of recurrence f

147 s are diagnosed in 1.5-5% of patients with a neuroendocrine neoplasm.

148 Neuroendocrine neoplasms (NEN) are rare neoplasms that o

149 ibes its role in the diagnostic algorithm of neuroendocrine neoplasms and its overall utility in thei

150 well as a brief summary of theranostics for neuroendocrine neoplasms and metastatic castration-refra

151 hese findings reveal new mechanisms by which neuroendocrine nerve terminal Ca(2+) can be controlled i

152 been defined as a part of these multifaceted neuroendocrine networks.

153 Many neuroendocrine neurons release neurohormones in long-dur

154 leus (MEPO), where most of the somata of the neuroendocrine neurons releasing GnRH and controlling re

155 d that the rostral PVN ERbeta-EGFP cells are neuroendocrine neurons whereas non-neuroendocrine (presu

156 Non-neuroendocrine Notch-active small-cell lung cancer cells

157 ectors of signaling via the GEF Epac2 in the neuroendocrine NS-1 cell line.

158 BMPC cancers lacked neuroendocrine or sarcomatoid differentiation, features

159 ependent gene expression in their respective neuroendocrine organs, consistent with a conserved pathw

160 differential regulation of these neurons and neuroendocrine output by estradiol.SIGNIFICANCE STATEMEN

161 ycytes) of the fetal hypothalamus, and hence neuroendocrine output.

162 erential regulation of these neurons and for neuroendocrine output.

163 ory cues with interosensory signals to drive neuroendocrine output.

164 nIN), and describe a unique subpopulation of neuroendocrine PanIN cells that express the neuropeptide

165 FLP-7 is secreted as a neuroendocrine peptide in proportion to fluctuations in

166 Remarkably, the cyclic neuroendocrine peptide somatostatin-14 (SST14) was obser

167 ators such as serotonin, the use of distinct neuroendocrine peptides for each output may be one means

168 cytes proliferate and subsequently acquire a neuroendocrine phenotype.

169 notypic levels that we investigated, shaping neuroendocrine, physiological and behavioural traits.

170 cells are neuroendocrine neurons whereas non-neuroendocrine (presumably pre-autonomic) ERbeta-EGFP ne

171 -resistant prostate adenocarcinoma (CRPC) to neuroendocrine prostate cancer (NEPC) has emerged as an

172 Neuroendocrine prostate cancer (NEPC) has increasingly b

173 rostate cancer (CRPC) or transdifferentiated neuroendocrine prostate cancer (NEPC).

174 implications for the clinical management of neuroendocrine prostate cancer as Aurora A kinase inhibi

175 By focusing on the neuroendocrine prostate cancer mutational data, we found

176 We examined neuroendocrine, PVN CRH neurons and report that social i

177 se models, the authors provide evidence that neuroendocrine PWS-associated phenotypes may be linked t

178 Neuroendocrine regulation of diverse behaviors of Caenor

179 rdiovascular regulation, pain, migraine, and neuroendocrine regulation, including reproduction.

180 pothalamic ventricular zone as a key site of neuroendocrine regulation, where the inflammatory signal

181 s to light, e.g., pupillary constriction and neuroendocrine regulation.

182 nses to stress and structural variability in neuroendocrine regulatory brain regions.

183 lved in the modulation of the behavioral and neuroendocrine response to stress.

184 Stress exposure elicits a prolonged neuroendocrine response, marked by cortisol release, whi

185 s and contributes to the regulation of acute neuroendocrine responses and their habituation to repeat

186 prospective investigation of subjective and neuroendocrine responses to alcohol measured over a 5-ye

187 Analysis of hypothalamic and neuroendocrine responses to HFS throughout the light-dar

188 the pheromones of the stud male that alters neuroendocrine responses to his chemosignals for many we

189 growth, energy metabolism, inflammation, and neuroendocrine responses to stress as key biological axe

190 posterior BNST (pBNST) to the behavioral and neuroendocrine responses to stress.

191 to affect stress-related neurobiological and neuroendocrine responses.

192 nd behavioral responses to alcohol through a neuroendocrine signal that shifts inhibitory GABA transm

193 ion, antiviral responses, and stress-related neuroendocrine signaling.

194 alamic "parvocellular" neurons release their neuroendocrine signals at the median eminence (ME) to co

195 s, a hierarchical combination of time-scaled neuroendocrine signals displays local-global integration

196 Merkel cell carcinoma (MCC) is a malignant neuroendocrine skin tumor frequently associated with the

197 re of most Merkel cell carcinomas, a primary neuroendocrine skin tumor.

198 al windows and disease processes because the neuroendocrine stress axis coordinates developmental rem

199 ch, it is unclear whether and how individual neuroendocrine stress response patterns are linked to af

200 the pBNST decreased anxiety, attenuated the neuroendocrine stress response, ameliorated stress-induc

201 Neuroendocrine stress response, depression-like behavior

202 Given that neuroendocrine stress responses are conceptualized as bi

203 rther, the role of PH GABAergic signaling in neuroendocrine stress responses was tested via inhibitio

204 ssociated with dysfunctional behavioural and neuroendocrine stress responses.

205 Neuroendocrine studies have demonstrated HPA axis overac

206 metimes coinciding with cerebral lesions and neuroendocrine symptoms, marked the acute phase of trich

207 Studies suggest alterations in the neuroendocrine system along the hypothalamus-pituitary-g

208 pheral nerves serve as a stem cell niche for neuroendocrine system development.

209 data suggest a novel role of the sympathetic neuroendocrine system for the modulation of IL-27-depend

210 ide hormones used as chemical signals by the neuroendocrine system to communicate between cells.

211 As a key component of the vertebrate neuroendocrine system, the pituitary gland relies on the

212 ese results also suggest ways in which other neuroendocrine systems can be regulated, and demonstrate

213 ch compounds on developing gut endocrine and neuroendocrine systems.

214 enin (MEN1) is a tumor-suppressor protein in neuroendocrine tissue.

215 High expression of NPSR1 was observed in neuroendocrine tissues, while no expression was observed

216 activation of the Notch pathway results in a neuroendocrine to non-neuroendocrine fate switch in 10-5

217 Secondary outcomes were neuroendocrine toxic effects and neurocognitive toxic ef

218 py-resistance in melanoma, treatment-induced neuroendocrine transdifferentiation in prostate cancer,

219 arizes and abolishes phasic discharge in rat neuroendocrine tuberoinfundibular dopamine (TIDA) neuron

220 is widely used for guiding the management of neuroendocrine tumor (NET) patients.

221 rgical treatment of patients presenting with neuroendocrine tumor (NET) with metastases.

222 TATE PET/CT interpretations in patients with neuroendocrine tumor (NET).

223 gnosis, follow-up, and treatment planning of neuroendocrine tumor (NET).

224 -hydroxycholesterol (24S-HC) in a pancreatic neuroendocrine tumor (pNET) model commonly used to study

225 tidisciplinary consensus panel consisting of neuroendocrine tumor experts, including oncologists, gas

226 Neuroendocrine tumor incidence, prevalence, and OS rates

227 sin B (CtsB) and CtsS in a murine pancreatic neuroendocrine tumor model.

228 the urinary bladder is a rare and aggressive neuroendocrine tumor of the urinary bladder.

229 highly angiogenic mouse model of pancreatic neuroendocrine tumor RIP-Tag2.

230 ignant melanoma (MM), gastroenteropancreatic neuroendocrine tumors (GEP NETs), and prostate cancer (P

231 TATE in patients with gastroenteropancreatic neuroendocrine tumors (GEP NETs).

232 Gastro-entero-pancreatic neuroendocrine tumors (GEPNETs) are increasing in incide

233 IMS: Survival times vary among patients with neuroendocrine tumors (NETs) - even among those with the

234 The incidence and prevalence of neuroendocrine tumors (NETs) are thought to be rising, b

235 Neuroendocrine tumors (NETs) are uncommon tumors with in

236 Neuroendocrine tumors (NETs) can be treated by peptide r

237 ECT have been used for functional imaging of neuroendocrine tumors (NETs) for the last 2 decades.

238 Purpose Treatment options for neuroendocrine tumors (NETs) remain limited.

239 sst2) antagonists are better tools to target neuroendocrine tumors (NETs) than sst2 agonists.

240 ate ((18)F-FET-betaAG-TOCA) in patients with neuroendocrine tumors (NETs) to evaluate biodistribution

241 ; range, 20-90 y) with histologically proven neuroendocrine tumors (NETs) underwent both (68)Ga-DOTAT

242 The molecular imaging and treatment of neuroendocrine tumors (NETs) with radiolabeled somatosta

243 iagnosis of somatostatin receptor-expressing neuroendocrine tumors (NETs), and SUV measurements are s

244 l to the diagnosis of gastroenteropancreatic neuroendocrine tumors (NETs), but detection rates, espec

245 macokinetics, and dosimetry of (18)F-MFBG in neuroendocrine tumors (NETs).

246 )In-DOTATATE) of patients with meningioma or neuroendocrine tumors (NETs).

247 use mortality after radiopeptide therapy for neuroendocrine tumors (NETs).

248 ts perform better than agonists in detecting neuroendocrine tumors (NETs).

249 y in Europe over the past decade for imaging neuroendocrine tumors (NETs).

250 Ga-DOTATATE PET/CT on managing patients with neuroendocrine tumors (NETs).

251 essed from planar images in 29 patients with neuroendocrine tumors (NETs; n = 21) or meningioma (n =

252 modulated by PDGF-DD signaling in pancreatic neuroendocrine tumors (PanNET).

253 Pancreatic neuroendocrine tumors (PanNETs) are a type of pancreatic

254 is significantly downregulated in pancreatic neuroendocrine tumors (PNETs).

255 Small intestinal neuroendocrine tumors (SI-NETs) are serotonin-secreting

256 iately differentiated to well-differentiated neuroendocrine tumors and who underwent (68)Ga-DOTATATE

257 Prognostic biomarkers for the pancreatic neuroendocrine tumors are needed.

258 are serotonin-secreting well-differentiated neuroendocrine tumors believed to originate from enteroc

259 nce from sequencing indicates that, although neuroendocrine tumors can arise de novo, they can also d

260 Paragangliomas are benign neuroendocrine tumors derived from the glomus cells of t

261 nificance of specific proteins in pancreatic neuroendocrine tumors including insulinoma.

262 MEN1 mutations frequently cause neuroendocrine tumors such as gastrinomas, characterized

263 in RIP-Tag2 transgenic mice with pancreatic neuroendocrine tumors that developed spontaneously and p

264 o had well-differentiated, metastatic midgut neuroendocrine tumors to receive either (177)Lu-Dotatate

265 of UCH-L1 and alpha-internexin in pancreatic neuroendocrine tumors was significantly associated with

266 Patients with advanced midgut neuroendocrine tumors who have had disease progression d

267 al adenocarcinoma, sarcomatoid carcinoma and neuroendocrine tumors, and all died within 65 weeks.

268 , these results have been obtained mostly in neuroendocrine tumors, and only limited data are availab

269 c radionuclide approach to the management of neuroendocrine tumors, as described by the inventor of t

270 in the clinical evaluation and management of neuroendocrine tumors, especially in neuroblastoma, para

271 rrently a mainstay in the evaluation of many neuroendocrine tumors, especially neuroblastoma.

272 In neuroendocrine tumors, NET can be targeted for imaging a

273 y neoplasms, cystic variations of pancreatic neuroendocrine tumors, pancreatic ductal adenocarcinomas

274 heranostics; review its current role in some neuroendocrine tumors, such as neuroblastoma, paragangli

275 l reported 2 female patients with pancreatic neuroendocrine tumors, WDS, and achlorhydria.

276 ty and use of DOTA analogs in the therapy of neuroendocrine tumors, we expect that (68)Ga-DOTATATE PE

277 raphy, we demonstrated that-for instance, in neuroendocrine tumors-up to 3 receptors can be coexpress

278 s an important role in staging and restaging neuroendocrine tumors.

279 tide LAR among patients with advanced midgut neuroendocrine tumors.

280 and peptide receptor radionuclide therapy of neuroendocrine tumors.

281 ependent prognostic biomarkers of pancreatic neuroendocrine tumors.

282 ssive, somatostatin-receptor-positive midgut neuroendocrine tumors.

283 tion of SQCC) shared molecular features with neuroendocrine tumors.

284 ors may be useful as a potential therapy for neuroendocrine tumors.

285 g is a widely accepted method for imaging of neuroendocrine tumors.

286 as are infrequent, genetically heterogeneous neuroendocrine tumors.

287 nesis and treatment of poorly differentiated neuroendocrine tumors.

288 hemoresistant and provide trophic support to neuroendocrine tumour cells, consistent with a pro-tumor

289 Neuroendocrine tumours (NETs) can secrete bioactive amin

290 pments and controversies in the treatment of neuroendocrine tumours (NETs) that are relevant for clin

291 well-differentiated gastrointestinal or lung neuroendocrine tumours (NETs).

292 The diagnosis of pancreatic neuroendocrine tumours (PanNETs) is increasing owing to

293 3-octreotate (DOTATATE) for the treatment of neuroendocrine tumours and (90)Y-microspheres for the tr

294 The prevalence of cerebral metastases in neuroendocrine tumours is estimated by various authors t

295 ressive, well-differentiated, non-functional neuroendocrine tumours of lung or gastrointestinal origi

296 pies for patients with advanced, progressive neuroendocrine tumours of the lung or gastrointestinal t

297 nfirmed small-cell lung cancer or large-cell neuroendocrine tumours with progressive measurable disea

298 eptable tolerability across a broad range of neuroendocrine tumours, including those arising from the

299 mouse tumors resemble human prostate cancer neuroendocrine variants; both mouse and human tumors exh

300 ivity measurements of genetically identified neuroendocrine vasopressin neurons show they can anticip