ライフサイエンスコーパス: neuroendocrine cell

1 ndamental regulatory organization of diverse neuroendocrine cells.

2 nd promoted liposome fusion and secretion in neuroendocrine cells.

3 n but had no effect on ciliated or pulmonary neuroendocrine cells.

4 on PVN and peri-PVN interneurons and not on neuroendocrine cells.

5 to Golgi membranes and synaptic vesicles in neuroendocrine cells.

6 ranin family of pro-hormone cargo of DCGs in neuroendocrine cells.

7 sponsible for pulsatile hormone release from neuroendocrine cells.

8 ially by ciliated cells, with an increase in neuroendocrine cells.

9 e receptor complex on the plasma membrane of neuroendocrine cells.

10 exocytosis of large dense-core vesicles from neuroendocrine cells.

11 s to all epithelial cell lineages, including neuroendocrine cells.

12 and activated UCHL1, a marker for pulmonary neuroendocrine cells.

13 tion and altered morphology of the pulmonary neuroendocrine cells.

14 reted by islet delta-cells and by extraislet neuroendocrine cells.

15 pendent exocytosis of dense-core vesicles in neuroendocrine cells.

16 cassette in PAC1-mediated Ca2+ signaling in neuroendocrine cells.

17 secretory vesicle-plasma membrane fusion in neuroendocrine cells.

18 ing until eggs filling the uterus deform the neuroendocrine cells.

19 factor-binding protein) on SG maturation in neuroendocrine cells.

20 ations resulting from IA-2 knockout in other neuroendocrine cells.

21 lates the differentiation or function of the neuroendocrine cells.

22 h possible intracellular roles for SNAP25 in neuroendocrine cells.

23 ely unaltered by actin disruption, unlike in neuroendocrine cells.

24 cally by repressing IIS ligand expression in neuroendocrine cells.

25 ssical routes for secretion of proteins from neuroendocrine cells.

26 gulation of this process in both neurons and neuroendocrine cells.

27 tide in embryonic and adult murine pulmonary neuroendocrine cells.

28 ressed selectively in central and peripheral neuroendocrine cells.

29 of exocytosis and endocytosis in neurons and neuroendocrine cells.

30 ndent dense-core vesicle (DCV) exocytosis in neuroendocrine cells.

31 ne confers such a pro-secretory phenotype on neuroendocrine cells.

32 r gene expression to different sets of islet neuroendocrine cells.

33 e-stage tumors are composed predominantly of neuroendocrine cells.

34 n urine bombesin-like peptide and numbers of neuroendocrine cells.

35 integral component of secretory granules in neuroendocrine cells.

36 ane PAM serve as indicators of granule pH in neuroendocrine cells.

37 d to secretory granules in transfected AtT20 neuroendocrine cells.

38 dense-core vesicle exocytosis in vertebrate neuroendocrine cells.

39 e enzyme does not require features unique to neuroendocrine cells.

40 l for tumor progression in multiple types of neuroendocrine cells.

41 e-core vesicle (DCV) exocytosis in permeable neuroendocrine cells.

42 regulate Ca(2+)-dependent DCV exocytosis in neuroendocrine cells.

43 calcium-triggered exocytosis from neural and neuroendocrine cells.

44 in noradrenergic and adrenergic neurons and neuroendocrine cells.

45 rane thickness, nerve fibers, and epithelial neuroendocrine cells.

46 for secretory vesicle biogenesis in neuronal/neuroendocrine cells.

47 s neuritogenesis, upon GPCR-Gs activation in neuroendocrine cells.

48 nt with autonomic and C fibers and pulmonary neuroendocrine cells.

49 eta) were previously found to be enriched in neuroendocrine cells.

50 ia three discrete cAMP sensors identified in neuroendocrine cells.

51 tic hormone (AKH) secretion from specialized neuroendocrine cells.

52 disease, as well as the function of gastric neuroendocrine cells.

53 SCLC is thought to derive from pulmonary neuroendocrine cells.

54 rentiation and activating Uchl1, a marker of neuroendocrine cells.

55 s onto identified preautonomic, but not onto neuroendocrine cells.

56 transporter, from single vesicles in living neuroendocrine cells.

57 n-containing proteins by RNA interference in neuroendocrine cells.

58 teps during dense-core vesicle exocytosis in neuroendocrine cells.

59 in various cell types, notably in nerve and neuroendocrine cells [1, 2].

60 e pixels per mm(2), P = .02), and epithelial neuroendocrine cells (4.9/mm(2) [25th-75th IQR, 0-16.4/m

61 During differentiation, neuroendocrine cells acquire highly amplified capacities

62 Neuroendocrine cells act as oxygen sensors in animals fr

63 lower incidence of hyperplasia of pulmonary neuroendocrine cells and aggressive liver, bile duct, st

64 (NEN) are rare neoplasms that originate from neuroendocrine cells and are characterized by the potent

65 uman alpha-synuclein in mammalian kidney and neuroendocrine cells and assessing ER-to-Golgi transport

66 d the associated regulated secretion in both neuroendocrine cells and chromogranin A-expressing COS7

67 Pulmonary neuroendocrine cells and clusters of these cells termed

68 DeltaL116 mutant proteins are mistargeted in neuroendocrine cells and form SDS-resistant aggregates,

69 nins regulate secretory vesicle formation in neuroendocrine cells and granin-derived peptides are co-

70 ed nerve terminals, neuromuscular junctions, neuroendocrine cells and hippocampal neurons, but not in

71 ed nerve terminals, neuromuscular junctions, neuroendocrine cells and in hippocampal neurons.

72 ally required for the formation of pulmonary neuroendocrine cells and is a marker for human small cel

73 to sites of dense core vesicle exocytosis in neuroendocrine cells and is required for Ca(2+)-triggere

74 cumulation of fusion-incompetent DCVs in BON neuroendocrine cells and lysosomal degradation (crinopha

75 We now demonstrate hyperplasia of both neuroendocrine cells and mast cells in lungs of baboons

76 Carcinoid tumors arise from neuroendocrine cells and may develop in almost any organ

77 Calcium-regulated exocytosis in neuroendocrine cells and neurons is accompanied by the r

78 nd in specialized secretory cells, including neuroendocrine cells and neurons.

79 complex formation and enhance exocytosis in neuroendocrine cells and neurons.

80 integral membrane glycoprotein expressed in neuroendocrine cells and neurons.

81 tored in secretory granules in endocrine and neuroendocrine cells and released in response to extrace

82 of this study demonstrate a role for ERAD in neuroendocrine cells and serve as a clinical example of

83 o of the seventeen dFMRFa cell types, the Tv neuroendocrine cells and the SP2 interneurons.

84 th the secretion of catecholamines from PC12 neuroendocrine cells and the steady-state accumulation o

85 on of a distinctive class of Pnmt-expressing neuroendocrine cells and their descendants (i.e. Pnmt(+)

86 ture of lung cancer and increased numbers of neuroendocrine cells and their peptides have been descri

87 tary cells and pituitary tumors and in other neuroendocrine cells and tumors including those of the a

88 l and neoplastic human pituitaries and other neuroendocrine cells and tumors.

89 to a specialized subset of epithelial cells (neuroendocrine cells) and sensory C fibers distributed t

90 : an increase in secretory peptide levels by neuroendocrine cells, and an increase in the number of c

91 Ca(2+) sensors for exocytosis in neurons and neuroendocrine cells, and are thus likely to regulate pi

92 Hsc70, decreasing SNAP-25 levels in cultured neuroendocrine cells, and inhibited both spontaneous and

93 tained from heterologous expression systems, neuroendocrine cells, and reconstituted systems, which r

94 of the parathyroid glands, enteropancreatic neuroendocrine cells, and the anterior pituitary gland.

95 Secretoneurin is produced in neuroendocrine cells, and the myocardium and circulating

96 Using CRISPR/Cas9, we generated HID-1 KO rat neuroendocrine cells, and we show that the absence of HI

97 The dense core vesicles (DCVs) of neuroendocrine cells are a rich source of bioactive mole

98 Factors involved in the migration of these neuroendocrine cells are as yet unresolved.

99 am may explain why lung cancers arising from neuroendocrine cells are highly metastatic.

100 Secretory granules of neuroendocrine cells are inositol 1,4,5-trisphosphate (I

101 Synaptic terminals and neuroendocrine cells are packed with secretory vesicles,

102 ain PC family members expressed in mammalian neuroendocrine cells are PC2 and PC1/3.

103 n of dense core secretory granules (DCGs) of neuroendocrine cells are poorly understood.

104 Neuroendocrine cells are specialized to produce, maintai

105 more ubiquitously expressed and is found in neuroendocrine cells as well as in brain.

106 essential for the development of normal lung neuroendocrine cells as well as other endocrine and neur

107 tained from genetically modified neurons and neuroendocrine cells, as well as from reconstituted syst

108 ence of incomplete exocytosis in neurons and neuroendocrine cells, as well as recent work probing the

109 Our evidence indicates that neuroendocrine cells assemble molecularly heterogeneous

110 and coordinately regulate NE uptake in PC12 neuroendocrine cells at least in part by stimulating the

111 the hypothalamus during the period when many neuroendocrine cells become differentiated from the neur

112 er NALP, and chromogranin A) that define gut neuroendocrine cell behavior.

113 triking sensitivity to hypoxia in a range of neuroendocrine cells, being progressively inhibited from

114 of cholecystokinin and serotonin (5-HT) from neuroendocrine cells; both act peripherally, cholecystok

115 ctions in sensory neurons is also present in neuroendocrine cells but has no detectable function in t

116 CR-2 in sensory neurons, is expressed in the neuroendocrine cells but has no detectable role in egg l

117 (CHGA) in the regulated secretory pathway in neuroendocrine cells, but the mechanism by which CHGA en

118 nsmitter release both in central neurons and neuroendocrine cells, but the prevalence of this mechani

119 secretory vesicle exocytosis in neurons and neuroendocrine cells, but the underlying mechanism remai

120 ate that link metabolism and excitability in neuroendocrine cells, but their role in nonglucosensing

121 inhibited GABA neurons, including identified neuroendocrine cells, by activating GIRK conductances an

122 s, likely via direct signaling to neoplastic neuroendocrine cells capable of trophic influences.

123 iate cells (prostate stem cell antigen), and neuroendocrine cells (CD56).

124 mmonly associated with secretory granules of neuroendocrine cells, chromogranins have also been found

125 ore, a significant increase in the number of neuroendocrine cell clusters was observed in the lungs o

126 f the Kalirin and Trio isoforms expressed in neuroendocrine cells colocalize with immature granules.

127 Increased pulmonary neuroendocrine cells containing bombesin-like peptide im

128 es have been proposed to generate a class of neuroendocrine cells containing gonadotropin-releasing h

129 o storage of regulated secretory granules in neuroendocrine cells, contributes to blood pressure home

130 ata expand our understanding of how a single neuroendocrine cell coordinates an organism-wide behavio

131 tions and contained both mucin-secreting and neuroendocrine cells, demonstrating that the crypt conta

132 sis to permeable cells (platelets, mast, and neuroendocrine cells) dependent on putative Ca(2+)-bindi

133 e in hematopoietic, inner ear, and pulmonary neuroendocrine cell development and governs cell process

134 Dividing neuroendocrine cells differentiate into a neuronal-like

135 -helix protein DIMM, a critical regulator of neuroendocrine cell differentiation, controls secretory

136 nsformation in islet cells, probably late in neuroendocrine cell differentiation.

137 tes of exocytosis in hippocampal neurons and neuroendocrine cells differs markedly.

138 Nerve-responsive neuroendocrine cells exerted trophic influences and pote

139 t prostate basal cells, but not secretory or neuroendocrine cells, express p63.

140 e showed 55% double labeling of periurethral neuroendocrine cells expressing both serotonin and YFP,

141 In neuroendocrine cells expressing hASH1, such as a pulmona

142 vity, we show that activation of neurons (or neuroendocrine cells) expressing the neuropeptide allato

143 Rb is specifically required for restricting neuroendocrine cell fate despite functional compensation

144 viding evidence that pocket proteins inhibit neuroendocrine cell fate while being required for differ

145 s), the storage compartment in endocrine and neuroendocrine cells for hormones and neuropeptides.

146 We isolated preneoplastic neuroendocrine cells from a mouse model of SCLC and foun

147 rigin hypothesis, favoring the derivation of neuroendocrine cells from the neural crest, with the sec

148 of stress within four different categories (neuroendocrine, cell function, body condition and immune

149 amics of CAPS at sites of exocytosis in live neuroendocrine cells has not been determined.

150 leased from dense-core secretory granules of neuroendocrine cells, have been implicated as playing mu

151 In neuroendocrine cells, hormones and neuropeptides stored

152 GABA synapses on hypothalamic magnocellular neuroendocrine cells; however, retrograde endocannabinoi

153 Only lung neuroendocrine cell hyperplasia and a subtle neuropeptid

154 defined etiology comprising the two diseases neuroendocrine cell hyperplasia of infancy (NEHI) and pu

155 of several different forms of ILD, including neuroendocrine cell hyperplasia of infancy and ILD, due

156 Two distinctive entities, neuroendocrine cell hyperplasia of infancy and pulmonary

157 els of neprilysin increases inflammation and neuroendocrine cell hyperplasia, which may predispose to

158 yperplasia, and diffuse idiopathic pulmonary neuroendocrine cell hyperplasia.

159 phages 4 weeks after radiation and pulmonary neuroendocrine cells hyperplasia 6 weeks after radiation

160 Idiopathic diffuse hyperplasia of pulmonary neuroendocrine cells (IDHPNC) is a clinicopathological e

161 a dominant negative Cdc42 construct in human neuroendocrine cells impaired the release process by com

162 s were mimicked by ablations of serotonergic neuroendocrine cells, implicating humoral release of ser

163 of simian virus 40 T antigen to a subset of neuroendocrine cells in all lobes of the FVB/N mouse pro

164 of a major fraction of neural crest-derived neuroendocrine cells in both the human and murine prosta

165 ng peptide (GRP) is synthesized by pulmonary neuroendocrine cells in inflammatory lung diseases, such

166 antly increased numbers of type II cells and neuroendocrine cells in neuroepithelial bodies.

167 Evidence was found for basal, luminal, and neuroendocrine cells in prostatic tubules regenerated fr

168 econstruction of the distribution pattern of neuroendocrine cells in the human fetal prostate indicat

169 likely that 5-HT2A receptors are present on neuroendocrine cells in the hypothalamic paraventricular

170 s but a reduced number of solitary pulmonary neuroendocrine cells in the neonatal lung.

171 Hyperoxia increased the number of neuroendocrine cells in the peripheral lung, which was p

172 gs provide further support for a key role of neuroendocrine cells in the reparative process of airway

173 A small population of neuroendocrine cells in the rostral hypothalamus and bas

174 tal trophoblasts, renal collecting ducts and neuroendocrine cells in the stomach and colon.

175 nd an epithelial progenitor can give rise to neuroendocrine cells in vivo.

176 ion, have been identified in gastric mucosal neuroendocrine cells including parathyroid hormone-like

177 in associated with the secretory granules of neuroendocrine cells, including pancreatic beta-cells.

178 Neuroendocrine cells, including those in the gut, have a

179 the intergeniculate leaflet and hypothalamic neuroendocrine cells indicate that integration of hormon

180 se and inhibits calcium-triggered release in neuroendocrine cells, indicating a previously unrecogniz

181 I1 display abnormal development of pulmonary neuroendocrine cells, indicating that GFI1 is important

182 ibuting to the appropriate entrance of these neuroendocrine cells into the brain, and thus represent

183 ty characterized by a diffuse hyperplasia of neuroendocrine cells involving distal bronchi and bronch

184 lecular and cellular properties of all major neuroendocrine cells, irrespective of the secretory pept

185 The histogenesis of prostatic neuroendocrine cells is controversial: a stem cell hypot

186 ociated at fast synapses, the arrangement at neuroendocrine cells is less clear.

187 eature of the regulated secretory pathway in neuroendocrine cells is lumenal pH, which decreases betw

188 Ca(2+)-triggered exocytosis in neurons and neuroendocrine cells is regulated by the Ca(2+)-binding

189 Exocytosis from neurons and neuroendocrine cells is tightly regulated by intracellul

190 required for proper development of pulmonary neuroendocrine cells, is essential for the survival of a

191 sicles in neurones and secretory vesicles in neuroendocrine cells, is likely to be involved in vesicl

192 220, which is a substrate of PKD proteins in neuroendocrine cells, is localized in the ends of the pr

193 l membrane protein of dense-core granules in neuroendocrine cells, is phosphorylated in a Ca(2+)-sens

194 In neuroendocrine cells, it elicits a robust, albeit transi

195 thalamus and their similar relationship with neuroendocrine cells, it is suggested that integration o

196 However, central synapses and neuroendocrine cells lacking these synaptotagmins still

197 y pathway using the murine pituitary-derived neuroendocrine cell line AtT-20 transfected with tPA cDN

198 D3 dopamine receptor expressed in the AtT-20 neuroendocrine cell line causes robust inhibition of P/Q

199 to glycines, is expressed in AtT20 cells, a neuroendocrine cell line endogenously expressing PC1, bo

200 H is transiently expressed in GH4C1 cells, a neuroendocrine cell line lacking PC1, under pulse-chase

201 The neuroendocrine cell line PC12, derived from rat pheochro

202 PC12 cells, a model neuroendocrine cell line, express multiple isoforms of t

203 Downregulation of CGA expression in a neuroendocrine cell line, PC12, by antisense RNAs led to

204 In the neuroendocrine cell line, PC12, synaptic vesicles can be

205 This model indicates that the neuroendocrine cell lineage of the prostate is exquisite

206 inal differentiation and maturation of these neuroendocrine cell lineages.

207 mmunofluorescence microscopy in neuronal and neuroendocrine cell lines revealed that membrane-associa

208 Unlike the neuroendocrine cell lines widely used to study trafficki

209 argeting of alpha(2C)-adrenoreceptors in two neuroendocrine cell lines with the targeting in three ep

210 By using different neuroendocrine cell lines, with or without endogenous ex

211 otropin-releasing hormone (GnRH) neurons are neuroendocrine cells, located in the hypothalamus, that

212 In mammalian neuroendocrine cells, loss of AP-3 dysregulates exocytos

213 cells, together with ciliated and pulmonary neuroendocrine cells, make up the epithelium of the bron

214 tibody to calcitonin gene-related peptide, a neuroendocrine cell marker, was used to identify the loc

215 The uv1 neuroendocrine cells mechanically sense passage of eggs

216 leasing peptide (GRP), secreted by pulmonary neuroendocrine cells, mediates oxidant-induced lung inju

217 ctor for the regulation of DCG biogenesis in neuroendocrine cells, mediating the formation of functio

218 te smoking on the gene expression profile of neuroendocrine cells, microarray analysis with TaqMan co

219 spike clustering displayed by magnocellular neuroendocrine cells (MNCs) of the supraoptic (SON) and

220 bipolar extensions typical of magnocellular neuroendocrine cells (MNCs).

221 These data suggest that in neuroendocrine cells myosin XVA may have a role in secre

222 Prostatic neuroendocrine cells (NE) are an integral part of prosta

223 Both cAMP and ERK are necessary for neuroendocrine cell neuritogenesis, and pituitary adenyl

224 Secretion of hormones and peptides by neuroendocrine cells occurs through fast and slow modes

225 hydroxylating monooxygenase, is defective in neuroendocrine cells of Ctr1A mutant larvae.

226 xpression of Prox1 in three novel sites: the neuroendocrine cells of the adrenal medulla, megakaryocy

227 UCHL1 expression was evident only in neuroendocrine cells of the airway epithelium in nonsmok

228 The neuroendocrine cells of the brain receive more extensive

229 NPYR-1 is expressed in neuroendocrine cells of the central nervous system and c

230 ecently that magnocellular and parvocellular neuroendocrine cells of the hypothalamic paraventricular

231 We show that in neuroendocrine cells, PC6B is localized to a paranuclear

232 In neuroendocrine cells, PI3K-C2alpha is present on a subpo

233 is induced by hormonal therapy, suggest that neuroendocrine cells play an important role in promoting

234 n many organs, but its function in pulmonary neuroendocrine cell (PNEC) differentiation has not been

235 ted a transgenic model for primary pulmonary neuroendocrine cell (PNEC) hyperplasia/neoplasia using v

236 ls, free alveolar macrophages, and pulmonary neuroendocrine cells (PNEC).

237 ribution and abundance of solitary pulmonary neuroendocrine cells (PNECs) and neuroepithelial bodies

238 Pulmonary neuroendocrine cells (PNECs) are proposed to be the firs

239 Pulmonary neuroendocrine cells (PNECs) are the only innervated air

240 ptor (Robo) genes are expressed in pulmonary neuroendocrine cells (PNECs), a rare, innervated epithel

241 - carotid body glomus cells, and 'pulmonary neuroendocrine cells' (PNECs) - are obscure.

242 AT3 activation, a decrease in the neoplastic neuroendocrine cell population, and impaired PanIN progr

243 This region is the source of many neuroendocrine cells, primarily located in the rostral s

244 Stimulation of secretion from neuroendocrine cells produced a corresponding rapid tran

245 Pulmonary tumorlets are minute neuroendocrine cell proliferations believed to be precur

246 tegrated physiological systems requires that neuroendocrine cells remain plastic to dramatically alte

247 activated by secretory stimuli in neural and neuroendocrine cells remains unknown.

248 pendent exocytosis of dense-core vesicles in neuroendocrine cells requires a priming step during whic

249 Since CGRP-expressing neuroendocrine cells reside in bronchiolar SCPC niches,

250 ng to the plasma membrane in melanocytes and neuroendocrine cells, respectively, was altered by 4 h a

251 ressed cells and signals to activate central neuroendocrine cell(s).

252 In neuroendocrine cells, SdpII colocalizes with dynamin, co

253 In response to physiological stimuli, neuroendocrine cells secrete neurotransmitters through a

254 es augmented neural stimulation of pulmonary neuroendocrine cell secretion.

255 ecretory pathway, which suggests that EC and neuroendocrine cells share common protein targeting reco

256 Live-cell imaging of wild-type AKH neuroendocrine cells shows heightened excitability under

257 Of 11 genes considered to be neuroendocrine cell specific, only ubiquitin carboxyl-te

258 In neuroendocrine cells, splice variants of the SNARE prote

259 nger than that of exocytic vesicle fusion in neuroendocrine cells, suggesting that additional regulat

260 was found on some epithelial basal cells and neuroendocrine cells, suggesting that these cells are ta

261 In neuroendocrine cells, synaptosome-associated protein of

262 olecules mediating the exocytosis of MSGs in neuroendocrine cells, syntaxin 1, SNAP-25, and VAMP2, we

263 in CHO cells more closely resembles that of neuroendocrine cells than previously appreciated.

264 otropin-releasing hormone (GnRH) neurons are neuroendocrine cells that are born in the nasal placode

265 rkel cells, a nonproliferative population of neuroendocrine cells that arise from epidermis.

266 beta-cells are neuroendocrine cells that can take up and metabolize the

267 a heterogeneous group of tumors arising from neuroendocrine cells that includes typical carcinoid, at

268 a region of the brain that gives rise to the neuroendocrine cells that innervate the ring gland, we c

269 the corpus cardiacum (CC) cells, a group of neuroendocrine cells that produce the adipokinetic hormo

270 For the hypothalamic neuroendocrine cells that release vasopressin and oxytoc

271 Like many neuroendocrine cells, the coupling between insulin-secre

272 Ca(2+) enters pituitary and pancreatic neuroendocrine cells through dihydropyridine-sensitive c

273 ory factors, and IA-2 is widely expressed in neuroendocrine cells throughout the body.

274 They are expressed together or separately in neuroendocrine cells throughout the brain and dispersed

275 ion, can promote a differentiation defect of neuroendocrine cells thus enhancing the ability of tumor

276 cialized secretory organelles in neurons and neuroendocrine cells to make them available for regulate

277 y be part of a more general process allowing neuroendocrine cells to originate in nonneuroectodermall

278 ell as the G-protein G alpha(o), function in neuroendocrine cells to promote release of neurotransmit

279 This could be a common mechanism used by neuroendocrine cells to regulate independently the secre

280 inates expression of FMRFa in that subset of neuroendocrine cells (Tv neurons) which provide the syst

281 SLMV purification scheme on a series of non-neuroendocrine cell types including the mouse fibroblast

282 K2 is dispensable for tumorigenesis in these neuroendocrine cell types.

283 '-UTR variant system in 2 different neuronal/neuroendocrine cell types.

284 ion of GABA synaptic inputs to magnocellular neuroendocrine cells under different physiological condi

285 sing SV40 large T antigen in their prostatic neuroendocrine cells, under the control of transcription

286 lipid rafts organize exocytotic pathways in neuroendocrine cells, we examined the association of pro

287 In experiments with rat neuroendocrine cells, we find that a few basic amino aci

288 In mast cells, as in neuroendocrine cells, we show that SgIII is complexed wi

289 synaptic terminals and from several types of neuroendocrine cells, we studied its involvement in oxyt

290 re the role of AP-1 in copper homeostasis in neuroendocrine cells, we used corticotrope tumor cells i

291 aV2.3 subunits are expressed in neuronal and neuroendocrine cells where they are believed to form nat

292 subunit are expressed mainly in neurons and neuroendocrine cells, whereas those containing the Cav1.

293 pletion suppresses neurite outgrowth of PC12 neuroendocrine cells, which can be rescued by codepletio

294 mbrane protein mainly expressed in brain and neuroendocrine cells, which is a downstream target of th

295 In neuroendocrine cells, whose dense core granules are stri

296 is report shows that secretory proteins from neuroendocrine cells will activate the NF-kappaB pathway

297 distinguished in the secretory response from neuroendocrine cells with slow ATP-dependent priming ste

298 ses the same fusion machinery as neurons and neuroendocrine cells, with an additional requirement for

299 pro-hormone secretory proteins released from neuroendocrine cells, with effects on control of blood p

300 ed within the regulated secretory pathway of neuroendocrine cells yielding five TRH peptides and seve