ライフサイエンスコーパス: neuroepithelial

1 thinning, impaired neurogenesis and loss of neuroepithelial adherens junctions.

2 ease eIF2alpha phosphorylation levels in NT2 neuroepithelial and C2C12 myoblast cells and activate HR

3 lar surface and disordered cell migration of neuroepithelial and differentiated cells were seen at va

4 ion, Tbeta4 expression has a large impact on neuroepithelial and macroscopic brain development.

5 which ALT is prevalent, including cancers of neuroepithelial and mesenchymal origin.

6 Our work further implies that neuroepithelial and neural crest cells are particularly

7 ion, the HFD stimulated the proliferation of neuroepithelial and neuronal precursor cells of the embr

8 nd mediolateral gene expression gradients in neuroepithelial and radial glial progenitors (the 'proto

9 ges in conducting airways, and distinguishes neuroepithelial and tracheal-bronchial gland cell lineag

10 cle arrest that underpins the specificity of neuroepithelial apoptosis and neural crest cell hypoplas

11 Neuroepithelial apoptosis diminished the size of the neu

12 neural tube closure coincided with increased neuroepithelial apoptosis specifically in the hindbrain

13 is; thus suggesting that it is p53-dependent neuroepithelial apoptosis that is the primary mechanism

14 some TiPSC-OVs maintained their distinctive neuroepithelial appearance and spontaneously formed prim

15 Neuroepithelial attachments at adherens junctions are es

16 elial cells, promotes the enlargement of the neuroepithelial basal end-foot and traps Hh protein, the

17 that is required for its localization to the neuroepithelial basement membrane (BM) to effectively an

18 opore closure, accompanied by suppression of neuroepithelial bending at the median hinge point and ac

19 cretory precursors juxtaposed to presumptive neuroepithelial bodies (NEBs), distinguished by their st

20 We mapped the formation of lung neuroepithelial bodies (NEBs), innervated clusters of ne

21 esulted in considerable depletion of CGRP in neuroepithelial bodies and submucosal nerve plexuses wit

22 these mice manifested an increased number of neuroepithelial bodies but a reduced number of solitary

23 al and central modulation via stimulation of neuroepithelial bodies of the lung.

24 bers that contact pulmonary endocrine cells (neuroepithelial bodies).

25 submucosal glands/intercartilagenous rings, neuroepithelial bodies, and terminal bronchioles/broncho

26 ly proposed airway chemosensory element, the neuroepithelial body (and its immortalized cellular coun

27 )-expressing stem cells that localize to the neuroepithelial body (NEB) and contribute to renewal of

28 other cancer cell lines including breast and neuroepithelial cancer, melanoma, and leukemia cell line

29 kinase activity is essential for regulating neuroepithelial cell adhesion, migration and morphogenes

30 ic reticulum stress, caspase activation, and neuroepithelial cell apoptosis (causal events in type 1

31 (K1361R) embryos show a striking increase in neuroepithelial cell apoptosis and a dramatic loss of ph

32 predominantly leads to exencephaly, induces neuroepithelial cell apoptosis and suppresses autophagy

33 How a naive human neuroepithelial cell becomes an electrophysiologically a

34 Endothelial coculture stimulates neuroepithelial cell contact, activating Notch and Hes 1

35 gotes also manifest embryonic lethality with neuroepithelial cell death.

36 s in oxidative stress-induced DNA damage and neuroepithelial cell death.

37 ited only by the apical daughter cell when a neuroepithelial cell divides vertically.

38 Additionally, neuroepithelial cell junctions in the embryonic rat brai

39 event(s) involved in the maintenance of the neuroepithelial cell layer shortly after its formation.

40 erebral cortical precursor cells reside in a neuroepithelial cell layer that regulates their prolifer

41 All cells expressed nestin, an early neuroepithelial cell marker.

42 autonomously within the retina and brain, as neuroepithelial cell morphology and polarity in these ti

43 as key regulatory elements in the control of neuroepithelial cell proliferation and the neuroblast tr

44 Neuroepithelial cell proliferation must be carefully bal

45 helial stem cell phenotype and regulation of neuroepithelial cell proliferation, suggesting that a mu

46 3, MAD2, CDC28 protein kinase (CKS)1 and 2, neuroepithelial cell transforming gene (NET)1, activator

47 We identify neuroepithelial cell transforming gene 1 (Net1) as a dow

48 athological angiogenesis caused by defective neuroepithelial cell-endothelial cell adhesion and imbal

49 basal progenitors (BPs), differentiate from neuroepithelial cells (NCs) with stem cell properties.

50 Subsequently, neuroepithelial cells (NE) convert into neuroblasts (NB)

51 In this study, we show that neuroepithelial cells (NECs), including neural crest cel

52 racellular calcium concentration of isolated neuroepithelial cells (NECs), which are putative oxygen

53 F or EGF promotes the proliferation of mouse neuroepithelial cells (NECs).

54 ptor, CXCR4, are constitutively expressed on neuroepithelial cells and are believed to be involved in

55 at mediate the transition, we microdissected neuroepithelial cells and compared their transcriptional

56 of activated Yorkie, promotes overgrowth of neuroepithelial cells and delays or blocks their differe

57 rt to a molecular state resembling embryonic neuroepithelial cells and functionally acquire rapid pro

58 In medium that allows growth of neuroepithelial cells and glial progenitors, mutant cell

59 g of postmitotic neurons is mediated through neuroepithelial cells and is necessary for guiding neuro

60 ver, Cited2 was required for the survival of neuroepithelial cells and its absence led to massive apo

61 is driven by neuronal progenitors, including neuroepithelial cells and radial glia.

62 ment, CPECs differentiate from preneurogenic neuroepithelial cells and require bone morphogenetic pro

63 We first isolate early neuroepithelial cells and show their broad Notch-depende

64 rmalities in the cell adhesive properties of neuroepithelial cells and suggest that NMHC-B is essenti

65 migration differentially positions nuclei in neuroepithelial cells and therefore influences selection

66 iating adhesion and signaling events between neuroepithelial cells and vascular endothelial cells.

67 to be expressed along the apical surfaces of neuroepithelial cells and was coexpressed with Shh in th

68 n contrast, proliferation of MEKK4-deficient neuroepithelial cells appeared to be largely unaffected.

69 tudies on CXCR4 expression and regulation in neuroepithelial cells are fundamental for understanding

70 trations ([Ca2+]i) in proliferating cortical neuroepithelial cells are markedly dependent on Ca2+ ent

71 In the following 10 d, these neuroepithelial cells are specified to OLIG2-expressing

72 Neuroepithelial cells are transformed into asymmetricall

73 ches, we show that dystroglycan functions in neuroepithelial cells as an extracellular scaffold to ma

74 of the glass onion phenotype in a subset of neuroepithelial cells as well as its onset following the

75 It was widely localized in retinal neuroepithelial cells at 1 day postfertilization (dpf),

76 These results suggest that the remaining neuroepithelial cells at later stages of animal life are

77 Neuroepithelial cells at the medial edge of the OOA, sho

78 initially detectable when the first retinal neuroepithelial cells began to leave the cell cycle.

79 Alternatively, both neuroblasts and neuroepithelial cells could be capable of dividing asymm

80 , in dissociated cell cultures, some retinal neuroepithelial cells divide asymmetrically and distribu

81 We show that, whereas most retinal neuroepithelial cells divide with their mitotic spindles

82 is localized to apical adhesive junctions of neuroepithelial cells during neurulation and that Xena k

83 uced swollen/enlarged ER lumens in embryonic neuroepithelial cells during neurulation.

84 Following in ovo misexpression of NKL, neuroepithelial cells exit the cell cycle and differenti

85 ex, sequential transcriptional programs take neuroepithelial cells from proliferating progenitors to

86 rreversible retraction of the endfeet of the neuroepithelial cells from the vitreal surface of the re

87 gene expression between neuroblasts and the neuroepithelial cells from which they derive.

88 Neuroepithelial cells further yield successive Notch-dep

89 ion strategy involves symmetrically dividing neuroepithelial cells generating large numbers of asymme

90 Our results indicate that neuroepithelial cells have all the necessary components

91 Neuroepithelial cells in both mutants fail to apically c

92 The architecture of primary cilia on neuroepithelial cells in Pam(-/-) mouse embryos was also

93 not support the commonly held view that most neuroepithelial cells in the embryonic CNS VZ are stem c

94 Whereas neuroepithelial cells in the medial OPC directly convert

95 g stem cells, which comprise the majority of neuroepithelial cells in the ventricular zone (VZ) of th

96 Here, we report that apicobasally polarized neuroepithelial cells in Xenopus laevis have a shorter c

97 Neuroepithelial cells infected with Toxoplasma type I ex

98 opment, from a seemingly homogenous sheet of neuroepithelial cells into a complex structure that is t

99 l expansion of the Olig1/Olig2 expression in neuroepithelial cells into the Nkx2.2 domain and a conse

100 Production of astrocytes from cultured neuroepithelial cells is hedgehog independent, whereas o

101 ural tube, and forced expression of Foxj1 in neuroepithelial cells is sufficient to increase cilia le

102 ce of focal adhesions in dissociated retinal neuroepithelial cells isolated from St 25 embryos.

103 urons, like projection neurons, develop from neuroepithelial cells located near the ventricular layer

104 rsal- and ventral-most regions of the brain, neuroepithelial cells lose their integrity and begin to

105 ound in cobblestone lissencephalies in which neuroepithelial cells migrate into superficial layers of

106 port that the knockdown of Htt expression in neuroepithelial cells of neocortex results in disturbed

107 determined that NDE1 is highly expressed in neuroepithelial cells of the developing cerebral cortex,

108 t is a major challenge to understand how the neuroepithelial cells of the developing CNS choose betwe

109 e capable of dividing asymmetrically, but in neuroepithelial cells other polarity cues might prevent

110 l, human PSCs are first induced to primitive neuroepithelial cells over 10 d, and then patterned to N

111 re, with peak CPEC competency correlating to neuroepithelial cells rather than radial glia.

112 NA and protein was specifically expressed in neuroepithelial cells surrounding retinal axons at the o

113 aste buds are aggregates of 50-100 polarized neuroepithelial cells that detect nutrients and other co

114 The ability of neuroepithelial cells to generate a diverse array of neu

115 This permits the underlying neuroepithelial cells to invade the spinal canal and obs

116 ur studies establish that the progression of neuroepithelial cells to neuroblasts is regulated by Not

117 period that ends as they differentiate from neuroepithelial cells to neuronogenic radial glia.

118 that affect the transition of proliferating neuroepithelial cells to postmitotic retinal cells.

119 l stem cells (NSCs) undergo transitions from neuroepithelial cells to radial glial cells (RGCs), and

120 ion is highly reminiscent of the switch from neuroepithelial cells to radial glial cells in the devel

121 at Notch signalling augments the response of neuroepithelial cells to Shh, leading to the induction o

122 or Cx43 in which Cx43 acts through non-crest neuroepithelial cells to suppress cellular delamination

123 olecular biology techniques in both cultured neuroepithelial cells treated with a GCN5 inhibitor and

124 Neuroepithelial cells undergo a cell cycle arrest before

125 Cocultures of hES cell-derived neuroepithelial cells with exogenous astrocytes signific

126 man stem cells are first differentiated into neuroepithelial cells with or without exogenous patterni

127 ivo, yet because RGCs develop from polarized neuroepithelial cells within a polarized environment, di

128 al crest-derived, and the hypoxia-sensitive 'neuroepithelial cells' (NECs) of fish gills, whose embry

129 central nervous system (CNS) is regulated by neuroepithelial cells, although the genes and pathways t

130 romotes proliferation and differentiation of neuroepithelial cells, and identify Decorin as a novel n

131 ave transiently suppresses Notch activity in neuroepithelial cells, and that inhibition of Notch trig

132 ulation, which underlies constriction of the neuroepithelial cells, and that ultimately drive neural

133 al expression of specific metabolic genes in neuroepithelial cells, but not in neuroblasts, and highl

134 he surrounding brain parenchyma, composed of neuroepithelial cells, glia, and neuronal precursors.

135 inantly localizes to the basolateral side of neuroepithelial cells, promotes the enlargement of the n

136 niche is composed of a diverse repertoire of neuroepithelial cells, radial glia (RG), and intermediat

137 clude withdrawal from mitosis by multipotent neuroepithelial cells, specification to particular cell

138 serve nuclear movements in zebrafish retinal neuroepithelial cells, we show that, except for brief ap

139 Neuroblasts originate from neuroepithelial cells, which are polarized along the api

140 expanding cores of undifferentiated mitotic neuroepithelial cells, which can be tumorigenic.

141 eks, hESCs are induced to differentiate into neuroepithelial cells, which form neural tube-like roset

142 The Drosophila optic lobe develops from neuroepithelial cells, which function as symmetrically d

143 nd adherens-junctions within chick and mouse neuroepithelial cells.

144 P4 to generate dCPECs from human ESC-derived neuroepithelial cells.

145 ng members of the Notch pathway expressed in neuroepithelial cells.

146 rical divisions of polarized radial glial or neuroepithelial cells.

147 re significantly heterogeneous among retinal neuroepithelial cells.

148 h neutral red and appear to be the branchial neuroepithelial cells.

149 ma3d inhibits the proliferation of hindbrain neuroepithelial cells.

150 tid body Type I (glomus) cells and pulmonary neuroepithelial cells.

151 ell polarity in neoplastic transformation of neuroepithelial cells.

152 localization and cell fate determination in neuroepithelial cells.

153 e vertebrate retina develops from a sheet of neuroepithelial cells.

154 al life are derived from the Nkx6.1+ ventral neuroepithelial cells.

155 oteins on the vitreal endfeet of the retinal neuroepithelial cells.

156 ed in proliferating, undifferentiated retina neuroepithelial cells.

157 ehog protein, ptc-2, is expressed by retinal neuroepithelial cells.

158 musashi1) that are expressed in uncommitted neuroepithelial cells.

159 x can regulate survival and proliferation of neuroepithelial cells.

160 vered along with rare clusters of persistent neuroepithelial cells.

161 transcriptional program similar to embryonic neuroepithelial cells.

162 orts neurulation by stimulating autophagy in neuroepithelial cells.

163 resent in the developing neural tube (E10.5, neuroepithelial cells; NEP) were examined for the expres

164 cells acquire apicobasal polarity and adopt neuroepithelial character prior to other regions of the

165 This neuroepithelial circuit can serve as both a sensory cond

166 This neuroepithelial circuit was reconstituted in vitro by co

167 Treatment of dissociated mouse cortical neuroepithelial cluster cell cultures with the NO syntha

168 Regular dissociation of the neuroepithelial clusters in suspension, and in the prese

169 rchestrates the differential growth of optic neuroepithelial compartments during vertebrate eye devel

170 The optic neuroepithelial continuum of vertebrate eye develops int

171 Cell expulsion through neuroepithelial contraction represents a mechanism for r

172 the lumen of the nasal cavity than are their neuroepithelial counterparts, therefore having quicker a

173 These findings identify neuroepithelial cross-talk as a potential novel target i

174 of the ECM in the development of complex 3D neuroepithelial cysts that recapitulate key steps in ear

175 This suggests that the neuroepithelial default state is IPC-like, whereas OPC i

176 aring loss and vestibular dysfunction due to neuroepithelial defects in the inner ear.

177 aring loss and vestibular dysfunction due to neuroepithelial defects in the inner ear.

178 se cortical progenitors, we found that early neuroepithelial deletion of ciliary Arl13b led to a reve

179 growth factor A (VEGF) isoforms during early neuroepithelial development in the mammalian central ner

180 ch pathway, known to be involved in cochlear neuroepithelial development.

181 The process of neuroepithelial differentiation from human pluripotent s

182 pothesized that hypomethylation of Pax3 upon neuroepithelial differentiation may be inhibited by hype

183 egulators, including E2f1 and CyclinD, delay neuroepithelial differentiation, and Fat-Hippo signaling

184 ctrum of neural tube defects associated with neuroepithelial disorganization and enhanced progenitor

185 ns intrathalamica (ZLI), a transverse linear neuroepithelial domain in the alar plate of the dienceph

186 genetic manipulations, we found that one IPC neuroepithelial domain progressively transformed into mi

187 scription factor MITF plays central roles in neuroepithelial domain specification and differentiation

188 SFRP-2 is expressed in several discrete neuroepithelial domains, including the diencephalon, the

189 , the Pax proteins start being detectable in neuroepithelial domains.

190 normal by reconstituting along the retracted neuroepithelial endfeet demonstrating that these endfeet

191 sic fibroblast growth factor), activated the neuroepithelial enhancer of the nestin gene, and gave ri

192 We found neuroepithelial expression of the genes during prenatal

193 to their correct location before they adopt neuroepithelial fate.

194 xpression of Tbeta4 led to the production of neuroepithelial folds resembling gyri and sulci, which a

195 s characterization of Fat-Hippo signaling in neuroepithelial growth and differentiation also provides

196 d in the progression of stem cells along the neuroepithelial-->radial glia-->astrocyte lineage.

197 ium, suggesting a mechanism of the olfactory neuroepithelial guanylate cyclase regulation fundamental

198 permanent hearing loss through apoptosis of neuroepithelial hair cells and afferent neurons of the c

199 SC colonies at 3% O(2), where they acquire a neuroepithelial identity over a period of 2 weeks.

200 d-B) caused the transformation of OPC to IPC neuroepithelial identity.

201 Olfactory neuroepithelial injury included loss of olfactory neuron

202 However, after experimentally induced neuroepithelial injury, ckPCs are activated such that th

203 The disruption of neuroepithelial integrity may be the cause of the neuron

204 one, disrupted cell adhesion and compromised neuroepithelial integrity.

205 ed in restricted dorsoventral domains of the neuroepithelial layer at early developmental stages (E2.

206 The neuroepithelial layer is composed of ciliated sensory ne

207 The membrane fraction of the neuroepithelial layer of the rat exhibited Ca(2+)-depend

208 ated and transmitted during the folding of a neuroepithelial layer.

209 e produced during the larval period from two neuroepithelial layers called the outer and inner optic

210 mode of axogenesis and dendritogenesis from neuroepithelial-like processes may act to preserve neigh

211 mark for the production of similar long-term neuroepithelial-like stem cells (lt-NES) from pluripoten

212 bouton (B) afferents distinguished by their neuroepithelial locations into BT units near the torus a

213 m cell markers Thy-1, c-kit, and CD34 or the neuroepithelial marker neural cell adhesion molecule 1.

214 r glia partially dedifferentiate, re-express neuroepithelial markers and re-enter the cell cycle.

215 ent with ethanol induction of cell cycle and neuroepithelial maturation in the absence of apoptosis.

216 They did not stimulate the olfactory neuroepithelial membrane guanylate cyclase.

217 onal knockout of the small GTPase Cdc42 from neuroepithelial (NE) and radial glial (RG) cells in the

218 re we find that cell cycle dynamics of human neuroepithelial (NE) cells differ from radial glial (RG)

219 Pluripotent stem cells are induced to form neuroepithelial (NE) cells that form neural tube-like ro

220 igoneuronal precursors, neuronal precursors, neuroepithelial/neural crest precursors, or astrocyte pr

221 Neuroepithelial organization is first apparent at the ma

222 Here we show that proper neuroepithelial organization maintained by RhoA GTPase i

223 The early neuroepithelial organization of the eye field requires L

224 of NSCs, a property tightly linked to their neuroepithelial origin, appear to be the key determinant

225 Differences in neuroepithelial patterning and neurogenesis modes contri

226 l that alphaE-catenin is highly expressed in neuroepithelial precursor cells in the developing cortic

227 have enlarged lateral ventricles lined with neuroepithelial precursor cells, reflecting an expansion

228 n the lateral ganglionic eminence (LGE), the neuroepithelial precursor of the neostriatum.

229 By E15, expansion of the neuroepithelial precursor pool is complete and any decre

230 nic subcortical telencephalon where distinct neuroepithelial precursors generate defined interneuron

231 ll subset of OPCs arise from common olig2(+) neuroepithelial precursors in rhombomeres r5 and r6, but

232 de that (1) Fgfr3 marks astrocytes and their neuroepithelial precursors in the developing CNS and (2)

233 (mTERT) prevented telomere collapse and the neuroepithelial precursors produced continued to divide,

234 les are derived from proliferative pcdh19(+) neuroepithelial precursors.

235 ormal neuronal differentiation, with loss of neuroepithelial progenitor cell phenotype in the subvent

236 During retinal development, neuroepithelial progenitor cells divide in either a symm

237 n at early developmental stages when retinal neuroepithelial progenitor cells predominate, we sought

238 are the only glial cell type produced by the neuroepithelial progenitor cells that generate the verte

239 network and signaling processes in polarized neuroepithelial progenitor cells.

240 addition at the pallial edge from a discrete neuroepithelial progenitor pool of the posterior telence

241 e and cell-cell interactions of radial glial neuroepithelial progenitors by the Lis1-Nde1 complex is

242 nascent cell-cell adhesion clusters between neuroepithelial progenitors contribute to define orienta

243 ons differentiated, more dorsally positioned neuroepithelial progenitors descended to the pMN domain

244 Understanding how ventral neuroepithelial progenitors differentiate into MSNs is c

245 r heighten the potential for using olfactory neuroepithelial progenitors for future autologous cell r

246 The transition of dividing neuroepithelial progenitors to differentiated neurons an

247 are positioned within the apical membrane of neuroepithelial progenitors, we hypothesized that loss o

248 retina are generated from common pool(s) of neuroepithelial progenitors.

249 s as well as N-CAM-positive, nestin-positive neuroepithelial progenitors.

250 ulates the integrity and cytoarchitecture of neuroepithelial progenitors.

251 factor receptor (EGFR) activation to promote neuroepithelial proliferation and neuroblast formation.

252 n of actin-associated processes in directing neuroepithelial proliferation.

253 roglial characteristics but retain important neuroepithelial properties.

254 each olfactory lamella but scattered in the neuroepithelial region.

255 loss of Lgl1 in mice results in formation of neuroepithelial rosette-like structures, similar to the

256 ndbrain develops as a series of well-defined neuroepithelial segments or rhombomeres.

257 eodomain proteins-Vax1 and Vax2-control this neuroepithelial segregation.

258 n alleles affect the polarity of the retinal neuroepithelial sheet and, unexpectedly, both result in

259 c failure of hingepoint formation, defective neuroepithelial sheet-bending, and failure of neural tub

260 onset following the initial formation of the neuroepithelial sheets indicate the presence of genetica

261 Here, we show that hindbrain neuroepithelial stem (hbNES) cells can be derived and ma

262 Nestin, a neuroepithelial stem cell marker, was enriched in the OB

263 cal role for elf in the development of a SVZ neuroepithelial stem cell phenotype and regulation of ne

264 control of symmetric division, essential for neuroepithelial stem cell proliferation, is mediated thr

265 the orderly developmental transition from a neuroepithelial stem cell to a functional neuron.

266 conducted gene expression profiling of human neuroepithelial stem cell-derived neurons, stimulated wi

267 , suggesting a sequential differentiation of neuroepithelial stem cells (NEPs) to GRPs to ARPs and th

268 bryonic spinal cord, for example, a group of neuroepithelial stem cells (NSCs) generates motor neuron

269 ntrol of mitotic spindle orientation in both neuroepithelial stem cells and radial glial progenitor c

270 odermal domain that combines slowly dividing neuroepithelial stem cells and rapidly amplifying progen

271 lia of the mouse cerebral cortex emerge from neuroepithelial stem cells around embryonic day 11 and p

272 OPCs develop from the same neuroepithelial stem cells as neurons, can be reprogramm

273 Neuroepithelial stem cells contact CSF-filled ventricles

274 the first demonstration of highly expandable neuroepithelial stem cells derived from the human embryo

275 Mammalian neuroepithelial stem cells divide using a polarized form

276 We found that induction of cortical neuroepithelial stem cells from human ES cells and human

277 (RGPC) neurogenesis, but its role earlier in neuroepithelial stem cells is poorly understood.

278 est that, during nervous system development, neuroepithelial stem cells may not only be responsible f

279 deletion and severe apoptosis of the founder neuroepithelial stem cells, accompanied by increased hor

280 sors are sequentially generated in vivo from neuroepithelial stem cells, but do not share a common li

281 t of generation of GRP cells from totipotent neuroepithelial stem cells, of O2A/OPCs from GRP cells a

282 in a cell lineage-specific manner, mainly in neuroepithelial stem cells, radial glia, and astrocytes.

283 Controlled gene deletion of Lis1 in vivo in neuroepithelial stem cells, where cleavage is uniformly

284 a glial-restricted precursor (GRP) cell from neuroepithelial stem cells.

285 demonstrate that RhoA functions to maintain neuroepithelial structures in the developing spinal cord

286 mitotic populations that were present in the neuroepithelial subventricular zone of the developing ne

287 djacent retinal cell divisions at the apical neuroepithelial surface.

288 s in each of these structures in the tongue, neuroepithelial taste cells of the taste bud, and, possi

289 Our data suggest that epithelial cells, neuroepithelial taste cells, or olfactory sensory neuron

290 osensory machinery discovered in specialized neuroepithelial taste receptor cells of the lingual epit

291 ary identify Fgf10 as a key regulator of the neuroepithelial to radial glial transition and subsequen

292 Neuroepithelial transforming gene 1 (Net1) is a RhoA gua

293 Neuroepithelial transforming gene 1 (Net1) is a RhoA-sub

294 pithelioma is the second most common primary neuroepithelial tumor of the eye.

295 ration (CNS HGNET-MN1)," and "CNS high-grade neuroepithelial tumor with BCOR alteration (CNS HGNET-BC

296 C alteration (CNS EFT-CIC)," "CNS high-grade neuroepithelial tumor with MN1 alteration (CNS HGNET-MN1

297 of the motor cortex in patients with primary neuroepithelial tumors of the central nervous system (CN

298 euroblastic rosettes that resemble primitive neuroepithelial tumors.

299 y from three common causes, dysembryoplastic neuroepithelial tumours (DNTs; eight cases), focal corti

300 erotopia and lissencephaly, dysembryoplastic neuroepithelial tumours, and microdysgenesis.